Pediatric orthopedic conditions Flashcards
What are the risk factors for developmental dysplasia of the hip (DDH) during the perinatal period?
DDH risk factors include congenital issues, poor positioning after birth, higher incidence in females, breech births, family history of DDH, oligohydramnios, being the firstborn, having a moulded baby (e.g., twins), high birth weight (>4kg), and multiple pregnancies.
Tight lower extremity swaddling
What are the clinical signs and symptoms of DDH in infants?
Signs of DDH include asymmetrical hip creases, uneven thigh or gluteal folds, limited hip movement (reduced abduction), apparent leg length differences, and positive findings on Barlow’s and Ortolani’s tests.
How does untreated DDH impact a child’s mobility and gait?
Untreated DDH can lead to severe arthritis due to reduced contact area in the hip joint, severely affecting a child’s gait and overall mobility.
What methods are used to diagnose DDH in routine infant screenings?
Diagnosis methods involve neonatal baby checks, selective ultrasound screenings for specific risk factors, general practitioner (GP) checks at 6-8 weeks, and late presentations usually when the child starts to walk (12-18 months).
What are the potential outcomes of untreated developmental dysplasia of the hip?
If untreated, DDH can result in a shallow acetabulum, formation of a false acetabulum in severe cases, leading to a shortened lower limb, increased risk of severe arthritis at a young age, and significant impairment in gait and mobility.
What imaging modalities are used in the diagnosis of developmental dysplasia of the hip (DDH), and why is each preferred or limited at certain ages?
Ultrasound (USS) is preferred, particularly in early infancy, but becomes less helpful after three months as ossification begins. X-rays are the preferred choice after ossification starts at around 4-6 months of age.
What are the primary management approaches for early and late developmental dysplasia of the hip (DDH)?
Early DDH is commonly managed with a Pavlik harness, used for up to 12 weeks until USS shows normal hip positioning, with a 95% success rate. Late DDH often requires surgical interventions such as closed reduction (CR) or open reduction (OR) spica. OR becomes more likely for persistent dislocation in children over 18 months, often needing corrective procedures on the femur or acetabulum.
How is DDH managed in its early stages, and what is the primary device used for treatment?
Early DDH is managed primarily using a Pavlik harness, worn for a specific duration with the hip abducted and flexed. Night splints might follow the harness treatment for a few weeks.
What surgical interventions are considered for late-stage DDH, and under what circumstances are these procedures typically required?
Late-stage DDH may necessitate closed reduction (CR) spica or open reduction (OR) spica surgery. OR is more probable for children over 18 months with persistent dislocation, often requiring corrective procedures on the femur or acetabulum due to shallow acetabulum development.
What are the prognostic outcomes for persistent or undiagnosed DDH in later stages, and what percentage of cases might necessitate further surgery?
Persistent or undiagnosed DDH in later stages often results in a poorer prognosis, making it challenging to reconstruct a normal hip. Approximately 30% of such cases might need further surgical intervention.
What defines Developmental Dysplasia of the Hip (DDH) and what potential complications can arise in adulthood due to this condition?
DDH is a condition resulting from a structural abnormality in the hip due to irregular fetal bone development, potentially leading to instability, subluxation, or dislocation. If untreated, it may persist into adulthood, causing weakness, recurrent subluxation or dislocation, abnormal gait, and early degenerative changes.
How is DDH usually detected, and at what stages can it become apparent in a child’s life?
DDH might be detected during newborn examinations or later when a child presents with hip asymmetry, limited hip movement, or a limp.
What are the risk factors associated with DDH as mentioned in the notes?
Risk factors for DDH include a first-degree family history, breech presentation from 36 weeks onwards, breech presentation at birth from 28 weeks onwards, and multiple pregnancies.
What elements are examined during DDH screenings, and what findings might indicate the possibility of DDH?
During DDH screenings, symmetry in hips, leg length, skin folds, and hip movements are examined. Findings indicating DDH include different leg lengths, restricted hip abduction, bilateral restriction in abduction, and variations in knee level when hips are flexed.
What are the specific tests used to check for DDH, and how are they performed?
The two tests used for DDH are the Ortolani test, checking for anterior hip dislocation, and the Barlow test, assessing posterior femoral head dislocation.
What distinguishes “clicking” from “clunking” during a physical examination in the context of DDH, and what further steps might be necessary based on these findings?
“Clicking” during an examination is generally due to soft tissue moving over bone, while “clunking” is more indicative of DDH and requires an ultrasound for confirmation.
What diagnostic methods are employed to confirm DDH in suspected cases, and when is each method particularly useful?
Ultrasound is the preferred method for diagnosing DDH in suspected cases, particularly in children with risk factors or suggestive examination findings. X-rays can be useful, especially in older infants.
What constitutes the primary treatment approach for DDH in infants below six months, and how does it function?
The primary treatment for infants under six months involves a Pavlik harness, which aims to maintain the femoral head in the correct position for normal acetabular development.
When is surgery considered in the management of DDH, and what is the subsequent post-surgery immobilization method utilized?
Surgery becomes necessary when the Pavlik harness fails or when the diagnosis is made after six months, with subsequent post-surgery immobilization using a hip spica cast for an extended period.
What are the typical ages and gender predilection for idiopathic osteochondritis of the femoral head?
Idiopathic osteochondritis typically occurs between the ages of 4 to 9 and is more common in boys, particularly very active boys of short stature (around 5:1 ratio).
What happens during the pathophysiology of idiopathic osteochondritis, leading to potential outcomes?
This condition occurs due to a transient loss of blood supply to the femoral head, resulting in necrosis and subsequent abnormal growth. It may lead to femoral head fracture and collapse, with remodelling that depends on age of onset and the amount of collapse. An incongruent joint might lead to early-onset arthritis and may necessitate hip replacement in severe cases.
What clinical symptoms characterize children affected by idiopathic osteochondritis of the femoral head?
Affected children present with pain and a limp, mostly unilateral. Bilateral cases may indicate an underlying skeletal dysplasia or thrombophilia.
What signs are observed during a clinical examination of children with this condition?
Clinical signs include initial loss of internal rotation, followed by loss of abduction, and a positive Trendellenburg test due to gluteal weakness.
What imaging modalities are used for diagnosing idiopathic osteochondritis of the femoral head, and what is their efficacy?
Imaging, primarily X-rays, may sometimes appear normal. MRI or bone scans can be used to identify pathology more clearly.
What are the general management approaches for this condition, and what proportion of cases tend to show positive outcomes?
There is no specific treatment except for regular X-ray observation and avoiding physical activity. Around 50% of cases have positive outcomes, but in severe instances, the femoral head might become aspherical, leading to muscular weakness. In rare cases, femoral head subluxation may require osteotomy of the femur or acetabulum.
What potential complications might arise in severe cases of idiopathic osteochondritis, and what interventions could be required?
Severe cases might lead to femoral head deformation, resulting in muscular weakness (positive Trendellenburg), or even femoral head subluxation necessitating surgical intervention like osteotomy of the femur or acetabulum.