Ped Derm Flashcards
Congenital melanocytic nevi CMN presentation
macules, paules, or plaques at birth
+/- hair
appearance may change with time
lesions grow in proportion to individuals size
congenital melanocytic nevus risk of malignancy
small/medium- less than 1%
large/giant 0-7.6% with most estimated risk of 2%
mongolian spots presentation
-patch of bluish-grey pigmentation with IRREGULAR BOARDER and normal skin texture
commonly buttocks and lower back
asian>black>hispanic
evident at birth
mongolian spot clinical diagnosis
none additional required
affected areas fade by age 2 and often disappear by age 10
nevus sebaceous clinical presentation
mostly on scalp or face
waxy, smooth, yellow-orange hairless patch, often oval or linear in shape
becomes MORE PRONOUNCED in adolescence
may become bumpy, warty, or scaly
nevus sebaceous treatment
intermittent interval follow up is recommended
refer to dermatologist if concerning changes are observed
aplasia cutis congenita ACC presentation
most commonly found midline posterior scalp
tuft of hair may surround defect which may indicate neural tube defect
may be associated with bulla
possible other developmental abnormalities
lesions well demarcated
aplasia cutis congenita ACC treatment
depends on size, depth and location
gentle cleansing and ointment
hypertrophic scar
refer to neurosurgery
cafe au lait macules CALM presentation
discrete uniformly pigmented macules or patches
present at birth and early childhood
conditions such as mccune albright syndrom
or neurofibromatosis type 1 NF1 (lisch nodules eye, bone defects, scoliosis, neurofibromas, optic nerve glioma) (autosomal dominant, 50% new mutations) get yearly opthalmology exams at year 1
vascular tumor classification
hemangiomas -infantile hemangioma -congenital hemangioma tufted angioma pyogenic granuloma
Vascular malformations
capillary malformations
port-wine stain
nevus simplex
port-wine stain clinical presentation
presents at birth and DOES NOT REGRESS
pink or dark red patches
soft tissue or bony overgrowth
sturge weber syndrome in the V1 distribution
congenital glaucoma concerns if patch affects eyelid
infantile hemangiomas
common benign vascular tumor
risk factors: low birth wt, famle, twin gestation, and fair skin
not present at birth but shortly after
superficial, deep, or mixed
superficial: bright red and minimally elevated
deep: larger with a bluish color
ulceration is a common complication
Proliferative:
early RAPID GROWTH during first 3 months of age
max growth 5-7w of age
Late: less rapid but still ongoing completed typically by 9 m
Involution phase:
color darkens and tumors soften
ultimate residual skin changes
nevus simplex (salmon patch) clinical presentation
faint, transient capillary malformation
-flat, pink/red patch
-typically midline of forehead, scalp, upper eyelids, posterior neck and back
commonly called a “STORK BITE”at the nape of the neck
and “ANGEL KISS” on the eyelid
LESIONS TYPICALLY FADE WITHIN 1-2y
pyogenic granuloma
common ACQUIRED lobular vascular tumor occurs at any age *affects skin particularly prone to trauma (hands finger face and mucous membranes) *extremely friable can recur despite treatment
