PC 615 Test 4 - Sheet1 Flashcards

1
Q

One of most common complaints presenting to ambulatory care settings

A

Chest pain

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2
Q

What to do when diagnosis is uncertain in ambulatory care settings

A

Err on the side of caution and listen carefully to the history.

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3
Q

Key to diagnosis in ambulatory care settings

A

Can be found in what the patient is telling you.

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4
Q

History-taking, physical exam, and the “working diagnosis”

A

Often they go on simultaneously and the diagnosis must be developed rapidly in an emergency situation.

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5
Q

Dyspnea in the elderly

A

Equivalent to angina and even if deny chest pain still need cardiac work up

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6
Q

Hypertensive emergency

A

Characterized by a severe elevation in BP (>180/120) complicated by evidence of pending or progressive target organ dysfunction

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7
Q

Target organ dysfunction

A

Heart - LVH, angina/prior MI, prior coronary revascularization, heart failure. Brain - stroke, dementia. Chronic kidney disease. Peripheral arterial disease. Retinopathy.

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8
Q

Examples of end target damage

A

Hypertensive encephalopathy. CVA. Acute MI. Acute L ventricular failure with pulmonary edema. Unstable angina. Dissecting aortic aneurysm.

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9
Q

Hypertensive urgency

A

Elevated BP but no evidence of end organ damage.

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10
Q

Treatment of hypertensive urgency

A

Oral agents and close follow-up be sure these patients have follow up appointments scheduled. There is no evidence to support aggressive use of IV or oral meds for rapid reduction of BP

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11
Q

Examples of hypertensive urgency without progressive organ damage

A

Upper stage 2 HTN with headache. SOA, epistaxis, anxiety

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12
Q

Treatment of hypertensive emergency

A

Requires immediate BP reduction (not to normal or goal) to prevent target organ damage. Need to be admitted to ICU for continuous monitoring and IV medication.

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13
Q

Initial goal of treatment for hypertensive emergency

A

Reduce BP by no more than 25% within minutes to one hr. If stable then to 160/100-110 within next 2-6 hrs. If stable, further gradual reduction of BP toward goal can be achieved over the next 24-48 hrs

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14
Q

Excessive falls in BP

A

Can precipitate renal, cerebral, or coronary ischemia and should be avoided.

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15
Q

Use of short acting nifedipine for hypertensive emergency or urgency

A

No longer acceptable in the initial treatment because of renal, cerebral, or coronary ischemia potential.

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16
Q

To determine target organ damage

A

Physical exam and diagnostic testing

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17
Q

Physical exam of pt presenting with hypertensive emergency or urgency

A

BP each arm sitting and standing. General appearance. Fundoscopy. Neck - palpation and auscultation of carotids, thyroid. Heart - size, rhythm, sounds. Lungs - rhonchi, rales. Abd - renal masses, bruits over aorta or renal arteries, femoral pulses. Extremities - peripheral pulses, edema. Neurologic assessment.

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18
Q

Routine lab tests for the investigation of all pts with HTN

A

Urinalysis. CBC. K+, Na+, and creatinine. Fasting glucose. Fasting total cholesterol, LDL, HDL, triglycerides. Standard 12-lead ECG

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19
Q

If target organ damage is discovered

A

Transfer to to hospital to begin care

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20
Q

Hypertensive crisis

A

Demands fast action. Can occur when BP suddenly changes in a pt with primary, chronic HTN.

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21
Q

Development of secondary hypertensive crisis

A

With conditions such as pregnancy, surgery, drug interactions or withdrawal, or cardiac problems.

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22
Q

Management of hypertensive urgency

A

Can be managed in out-pt settings with close monitoring

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23
Q

Hypertensive encephalopathy

A

May present with HTN emergency. Is the result of cerebral edema or as a thoracic aortic dissection resulting from an expanding hematoma in the wall of the aorta

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24
Q

Management o hypertensive emergency in out-pt setting

A

Under no circumstances should this occur. Stablize and refer immediately.

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25
Q

Meds to use to reduce BP quickly

A

Captopril (ACEI), Nicardipine (CCB), Labetalol (BB), Clonidine (a2 adrenergic receptor agonist)

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26
Q

Components of asthma

A

A reversible airway disease that exhibits with airway obstruction, hyper-responsiveness, and inflammation.

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27
Q

Status asthmaticus

A

Asthma that lasts for weeks and requires aggressive treatment and/or hospitalization

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28
Q

Risk for all asthma patients

A

Developing a severe asthma attack that could result in status asthmaticus and respiratory failure

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29
Q

Imperative actions of a provider for a pt suffering from asthma

A

Prompt recognition, stablization, management, and referral to prevent impending respiratory failure.

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30
Q

Consideration of all abd pain complaints in ambulatory care

A

Should be considered potentially serious until proven otherwise

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31
Q

Diagnosis of abd complaints in most cases

A

Determined using the provider’s history and physical exam skills. Consider the worst possibilities first and then exclude them systematically from the differentials.

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32
Q

Female patients with abd pain

A

Pelvic-related pain can be misleading. Be absolutely certain you have ruled out pregnancy.

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33
Q

Female pain in which to caution

A

Cervical motion tenderness - rocking the uterus near an inflamed appendix can also create pain. Anytime there is unilateral, localized abd pain.

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34
Q

Most common reason pt visits ED or clinic

A

Headache

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35
Q

Narrowing down the vast differential of headaches

A

History rather than physical exam will help.

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36
Q

Type of majority of headaches

A

Primary - migraine, cluster, or tension headaches

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37
Q

Secondary headaches

A

Must not miss or misdiagnose - especially those due to subarachnoid hemorrhage, ruptured arteriovenous malformation or meningitis

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38
Q

Cataclysmic headache

A

Early recognition is imperitive for good pt outcomes

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39
Q

Descriptions of suspect headaches

A

Headache in a pt who rarely has headaches. A pt who states this is the worst headache ever.

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40
Q

Temporal arteritis

A

A do not miss diagnosis that needs immediate treatment, or loss of vision may occur. Use mnemonic SNOOP.

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41
Q

SNOOP

A

S- systemic disease (fever, weight loss, malignancy, HIV). N - neurological signs and symptoms. O - onset 50. O - onset sudden (TCH) SAH. P - pattern change (progressive headache with loss of headache free periods, or change in type of headache)

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42
Q

Thunderclap headache (TCH)

A

Severe headache with sudden onset. May or may not herald a subarachnoid hemorrhage, may be benigh, and may or may not reoccur. Migraine sufferers are more likely to have a benign TCH

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43
Q

Differential diagnosis of altered LOC

A

Must first rule out structural and metabolic causes. Use mneumonic “vowel tips”

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44
Q

Vowel tips

A

A - alcohol. E - epilepsy. I - insulin and ketoacidosis. O - opiate and overdose. U - uremia and other metabolic states. T - trauma. I - infection. P - psychiatric. S - shock or syncope

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45
Q

Consideration of altered LOC

A

Assume C-spine injury until proven otherwise. Requires a quick evaluation of ABC’s evidence from trauma and vital signs.

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46
Q

Consideration of a coma

A

Assume to be a result of drugs or toxins until proven otherwise.

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47
Q

Minor head injury

A

Result of trauma to the head that does not result in any alteration of cerebral function. Can result in severe complications (subdural or epidural hematoma) with rapid deterioration of pt’s neurologic status, disability, or death. NP only manage head injuries that do not result in altered LOC.

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48
Q

Status epilepticus

A

A continuous seizure activity which persists longer than 15-30 minutes, or when a series of seizures occur during the same time period and from which the pt does not regain consciousness between attacks.

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49
Q

Results of status epilepticus

A

An emergency situation that may result in circulatory collapse, respiratory arrest, and brain damage. 10% die.

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50
Q

Differential diagnoses of seizures

A

Convulsive syncope,, cardiac dysrhythmias, decerebrate posturing, or psychogenic seizures.

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51
Q

Causes of seizures

A

Tumors, vascular events, infection, and metabolic or toxic pathology

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52
Q

Most musculoskeletal trauma

A

Contusions, minor sprains (ligament injuries) and strains (tendon injuries)

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53
Q

Musculoskeletal injuries that require referral

A

Those of instability, obvious deformity, sudden and significant joint swelling, locking, neurovascular compromise, inability to use an extremity or obvious fracture.

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54
Q

Musculoskeletal injury with evidence of neurovascular compromise

A

Requires splinting and assistance from both an orthopedic and vascular surgeon

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55
Q

Principle of trauma care

A

Do no further injury

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56
Q

Pts with bony shoulder injuries

A

Require referral to an orthopedist

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57
Q

Soft tissue injuries

A

may be managed initially by the primary care provider

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58
Q

Ordering an ankle x-ray using Ottawa Ankle Rules

A

If pain in the malleolar zone; bony tenderness along the distal 6 cm of posterior edge of fibula or tip of lateral malleolus; bone tenderness at edge of tipic or tip of medial malleolus; inability to bear weight for 4 steps both immediately and at presentation

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59
Q

Ordering a food x-ray using Ottawa Ankel Rules

A

Pain in the midfoot zone; bone tenderness at base of 5th metatarsal; bone tenderness at the navicular bone; inability to bear weight for 4 steps both immediately and at presentation

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60
Q

Ordering a knee x-ray using Ottawa Knee Rules

A

Knee injury and age 55 or older; isolated tenderness of patella; tenderness of head of fibula; inability to flex 90 degrees; or inability to bear weight both immediately and at presentation for 4 steps.

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61
Q

Response to bioterrorism

A

Will only be as good as the availability of supplies adn medications, along with the degree of planning and training.

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62
Q

Most likely forms of biological weapons

A

Aerosol such as plague, smallpox, tularemia, or botulism. Secondary (person-to-person) such as plague and smallpox. Smaller scales such as contaminating food with botulinum, staphoylcoccus or cholera.

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63
Q

Alerts to the possibility of biological weapon use

A

Being alert to large numbers of pts seeking care for the same thing, higher morbidity or mortality for a common illness, uncommon illnesses for an area

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64
Q

Diagnosis of biological weapons

A

Based on clinical signs and symptoms, but highly specialized diagnostic test are required.

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65
Q

Definition of bioterrorism

A

Intentional release or dissemination of biological agents (bacteria, viruses, or toxins). Typically found in nature, but can be changed to increase their ability to cause disease, make them resistant to current meds, or increase their ability to spread into the environment. Can be spread through the air, through water, or in food.

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66
Q

Category A biological agents

A

High potential for adverse public health impact and that also has a serious potential for large scale dissemination. Anthroax, smallpox, botulism toxin, ebola plague, marbug

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67
Q

Category B biological agents

A

Moderately easy to disseminate adn have low mortality rates. Brucellosis, Epsilon toxin of Closteridian perfringens. Food safety threats - Salmonella, Ecoli, Shigella, Stafflococcal enterotoxin B. Water suply threats - Typhus, viral encephalitis.

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68
Q

Category C biological agents

A

Pathogens that might be engineered for mass dissemination because they are easy to produce and have potential for hgh morbidity or mortality. Nipah virus, hantavirus, multi-drug resistant TB

69
Q

Major difference between a hypertensive emergency and hypertensive urgency is…

A

Optimal timeframe of treatment

70
Q

Clinical assessment of chest pain due to cardiac causes can vary between men and women, primarily because of …

A

Presenting symptoms

71
Q

Elderly patients experiencing a MI may frequently only complain of

A

Weakness

72
Q

Which is true when diagnosing a patient with temporal arteritis

A

Initiation of treatment is imperative if this diagnosis is suspected, as left untreated, it could result in blindness

73
Q

Most appropriate radiologic diagnostic test to order when pt presents with suspected limb fracture

A

Plain film

74
Q

Best plan for a pt with brief new-onset seizures

A

Refer to a neurologist

75
Q

An x-ray of the ankle is indiciated for all patients with complaints of ankle pain

A

FALSE

76
Q

Pregnancy should be ruled-out for all female patients with complaints of abd pain

A

FALSE

77
Q

A cause of secondary headache

A

Subarachnoid hemorrhage

78
Q

Cardiac presentation that should be further evaluated with troponin levels adn cardiac stress testing

A

CP that is predictably exertional, ECG abnormalities, or with cardiac risk factors

79
Q

Chest pain in women

A

Usually a late sign of CAD with prodromal symptoms 1-4 months prior. Two most severe prodromal symptoms are fatigue or tiredness and sleep disturbances. Other sx are SOA, pain in shoulder / upper back, anxiety, and indigestion.

80
Q

Atypical presentation of women with unstable angina

A

Pressure pain in the chest, stomach, back, and high, higher intensity of pain, dizziness, palpitations, and irritability.

81
Q

Risk factors for CV events

A

The composition, morphology, and stability of the coronary artery plaque, not the degree of plaque stenosis.

82
Q

Pain with chronic stable angina

A

Precipitated by exertion and relieved by rest

83
Q

Diagnosis of microvascular angina (syndrome X)

A

Suspected when there is a convincing history of angina chest pain with or without documented reversible ischemic ECG changes, angiography fails to demonstrate obstruction or spasm of a major coronary artery, and other conditions have been excluded from the differentials.

84
Q

Chronic stable angina

A

Demonstrates symptoms that occur with predictable frequency, severity, duration, and provication. Symptoms occur with exertion and are relieved by rest or one nitro and generally lasts only for 1-3 minutes.

85
Q

CK lab values after MI

A

Rise - 3-12 hr. Peaks - 24 hrs. Normalizes - 48-72 hrs

86
Q

Myoglobin lab values after MI

A

Rises - 1-3 hrs. Peaks - 6 hrs. Normalizes - 24 hrs

87
Q

Management of hypertnesive emergency

A

Admission to ICU for continuous BP monitoring and parenteral administration of meds. Lower BP by no more than 25% (within minutes to 1 hr), then if stable, to 160/100-110 within next 2-6 hrs. If stable, further normalization can be implemented in next 24-48 hrs.

88
Q

Determining end-organ damage with hypertensive emergencies

A

Chest x-ray, ECG, CBC, serum electrolytes, BUN, serum creatinine. CT of head in pt with neurologic symptoms

89
Q

Symptoms of asthma

A

Recurrent episodes of wheezing, SOA, chest tightnes, cough, and sputum production.

90
Q

Triggers of asthma episodes

A

Allergens, infections, exercise, abrupt changes in weather, and exposure to airway irritants such as tobacco smoke.

91
Q

When physician consult is indicated for asthma pt

A

SpO2 <90% room air, peak flow less than 70%, and failure to improve with three nebulizer treatments or 3 epi injections

92
Q

Characteristics of an acute asthmatic episode

A

Airway obstruction, manifested by symptoms of breathlessness, anxiety, wheezing, adn sometimes coughing.

93
Q

Common allegens that precipitate asthma

A

Cat dander, house dust mite allergen, cockroach allergen, adn tree and grass pollen

94
Q

Diagnosis of asthma

A

Based on history, physical exam, and certain diagnostic tests, particularly spirometry.

95
Q

Characteristics of severe asthma exacerbations

A

Labored respirations, diaphoresis, anxiety, and breathlessness (inability to finish a complete sentence), resp rate of 30 or more, HR of 120 or more

96
Q

Goals of successful asthma management

A

Achieve and maintain control of symptoms. Maintain normal activity levels, including exercise. Maintain pulmonary function as close to normal as possible. Prevent asthma exacerbations. Avoid adverse effects from asthma meds. Prevent asthma mortality.

97
Q

Corticosteroid use and asthma control

A

Most potent and effective anti-inflammatory meds available for the treatment of moderate to severe asthma.

98
Q

Managing exacerbations of asthma

A

Patient ed - written asthma action plan. Recognition of early signs. Appropriate therapy - short acting beta agonists. Remove allergens. O2 to relieve hypoxemia. Serial lung function tests. Prevent relapse.

99
Q

Symptoms of anal fissure

A

Severe intense pain with defecation. Described as knifelike, cutting, or tearing in character. Persists for hours, with a tight, throbbing quality followed by relative comfort prior to the next bowel movement.

100
Q

Mainstay of medical treatment for acute and chronic anal fissures

A

Avoidance of hard stools. Use sitz baths after painful BMs.

101
Q

Infant with colic

A

Episodes of inconsolable crying that last more than 3 hrs per day for more than 3 days/week and longer than 3 weeks

102
Q

Symptoms of inflammatory diarrhea

A

Fever, tenesmus, abd pain, and hemoccult-positive stool

103
Q

Symptoms of noninflammatory diarrhea

A

Usually watery, milder, without significant fever, with only mild abd cramping, and without blood or WBCs in the stool.

104
Q

If pt with diarrhea has taken antibiotics within past 2 months

A

Test for C. difficile

105
Q

Medications to treat pinworms

A

One oral dose of pyrantel pamoate to all family members and can be purchased OTC

106
Q

Esophageal food bolus obstruction or perforation

A

Sever pain and diaphoresis after swallowing a sharp object, such as a bone

107
Q

When a foreign body ingestion is suspected

A

Obtain PA and lat x-ray of throat adn chest to at least the midabd

108
Q

Initial management of non-threatening rectal bleeding from hemorrhoids

A

Include a high-fiber diet, stool softeners, and bulk laxatives, and the pt should be instructed to spend less time sitting and straining on the commode.

109
Q

Prolapsed or strangulated hemorrhoids

A

Warrant surgical consultaiton and possible hospital admission

110
Q

Treatment for acutely thrombosed hemorrhoid that is engorged, causing severe pain, and no anticipated bleeding problems

A

Should be incised to provide pain relief.

111
Q

Singultus

A

Hiccups - Most resolve spontaneously. If no resolve after using simple measures, try chlorpromazine (Thorazine) 25 to 50 mg PO tid or qid.

112
Q

Visceral pain

A

Usually the result of distention or spasm of a hollow organ and described as dull and cramply and is poorly localized.

113
Q

Parietal pain

A

Sharp, well-localized and is from irritation of the parietal peritoneum.

114
Q

Referred pain

A

Aching and is experienced away from the disease process and is perceived to be near the surface of the body

115
Q

Right upper quadrant pain

A

Emanates from the chest cavity, liver, gallbladder, stomach, bowel, or right kidney or ureter.

116
Q

Left upper quadrant pain

A

Associated with heart or chest cavity, spllen, stomach, pancreas

117
Q

Right lower quadrant pain

A

Associated with the appendix, bowel, right ureter, or pelvis

118
Q

Left lower quadrant pain

A

Bowel, left ureter, or pelvis

119
Q

Indication for physician consultation of abd pain

A

For suspected GI bleeding, bowel obstruction, postural vital signs changes, abnormal findings, jaundice, positive preg test, severe localized or unilateral lower abd pain, hx of trauma or any indication of peritoneal irritation

120
Q

3 signs and symptoms most predictive of acute appendicitis

A

Starts in the epigastrium or periumbilical area, migration of pain to right lower quadrant and abd rigidity

121
Q

Small bowel obstruction symptoms

A

Intermittent and crampy abd pain, vomiting, obstipation, abd distention, and fever, pain relieved by vomiting, abd decompression.

122
Q

Management of small bowel obstruction

A

Initially - NPO status, IV fluid, correction of electrolytes and acid-base, NG tube, antiemetic.

123
Q

Complications of small bowel obstruction

A

Bowel ischemia.

124
Q

Perforation of peptic ulcer

A

Life-threatening complication causing abrupt onset of severe abd pain, vomiting coffee ground emesis, hypovolemia, fever

125
Q

Peritonitis

A

Diffuse, localized, or referred abd pain with high fever, nausea, vomiting, diarrhea, or constipation, abd distension, rigidity, decreased bowel sounds, rebound tenderness and guarding

126
Q

Management of peritonitis

A

Empirical antibiotics - Cefotaxime 1-2g IV a 6-8 hrs

127
Q

AAA rupture

A

Cause of sudden death and may be asymptomatic prior. May be preceded by abd, flank, or back pain. Rupture causes severe abd pain that may radiate to flank, low back, or groin with radiation to back.

128
Q

Suspect a cerebrovascular cause if pt presents as…

A

A thunderclap headache that reaches maximal intensity within 1 min or a headache accompanied by any change in mental status, weakness, seizures, stiff neck, or persistent neurologic abnormalities. Suspect a subarachnoid hemorrhage or aneurysm

129
Q

Characteristics of polymyalgia rheumatics

A

Seen in persons older than 50 and presents as pain and stiffness in neck, shoulders, and pelvic girdle, but often associated with elevated SED and C-reactive protein levels and responds rapidly to corticosteroids

130
Q

Giant cell arteritis

A

A systemic large-vessel vasculitis commonly affecting the branches of the proximal aorta that supplies the neck and extracranial structures of the head

131
Q

When to consider giant cell arteritis

A

New onset headache or visual symptoms in an elderly pt with or without musculoskeletal symptoms. It is a medical emergency with potential for sudden and irreversible blindness.

132
Q

Management of giant cell arteritis

A

Contraversy between biospy and using prednisone or corticosteroids which can be dangerous in the elderly

133
Q

Management of polymyalgia rheumatica

A

May last 6 weeks to several years. Prednisone for 2-4 weeks while monitoring ESR and CRP closely. Prednisone should be tapered as soon as possible.

134
Q

The most significant and most common complication of polymyalgia rheumatica

A

Giant cell arteritis

135
Q

The most common complication of giant cell arteritis

A

Blindness

136
Q

Encephalitis

A

Incidence increases in late summer months and is caused by cytomegalovirus and Epstein-Barr and herpes.

137
Q

Meningitis

A

Either aseptic or septic. Aseptic caused by enteroviruses. Bacterial caused by resp infection

138
Q

Risk factors for bacterial meningitis

A

Male, malignant disease and chemo, previous basilar skull fx or neurosurgery, sickle cell, complement deficiency, asplenia, alcoholism, Navajo or Eskimo descent, HIV, and exposure to community outbreak

139
Q

Classic adult presentation of meningitis

A

Fever, headache, and stiff neck. Altered LOC, seizures, and hypotension come later.

140
Q

Treatment of bacterial meningitis

A

Immediate emperical antimicrobial therapy - vancomycin

141
Q

Complications of bacterial meningitis

A

Dehydration, septic shock, hemodynamic compromise, cerebral edema, DIC, myocarditis, hyponatremia, seizures, and death

142
Q

When to consult for headache

A

Pt with suspected temporal arteritis, change in mental status, nuchal rigidity, neurologic deficit, or new onset of headache

143
Q

Causes of secondary headaches

A

Less common - usually result of underlying disease or condition, such as aneurysm, tumor, hemorrhage, temporal arteritis, or meningitis.

144
Q

Drugs that cause dystonic drug reactions

A

Haldol, compazine, thorazine, phenergan, reglan

145
Q

Dystonic drug reactions

A

Protruding or pulling sensation of tongue, twisted neck or facial muscle spasm, roving or deviated gaze, abd rigidity and pain, spasm of entire body

146
Q

Medications to terat dystonic drug reaction

A

1-2 mg of cogentin or 25-50 mg benadryl IV

147
Q

Typical symptoms of psychogenic seizures

A

Turning head from side to side and pelvic thrusting and no fecal or urinary incontinence

148
Q

Symptoms of acute bronchitis

A

Starts with 1-5 days of fever, malaise, and myalgias that are often resemble URIs. Followed by a second phase characterized by persistent cough, often accompanied by phegm production and wheezing that lasts 1-3 weeks.

149
Q

Delirium in older adults

A

May be first and only indicator of underlying physical illness, such as infection, MI, or drug toxicity

150
Q

Hallmark of delirium

A

Clouding of consciousness, with an inability to focus, sustain, or shift attention, as well as a change in cognition, including impairment in short-term memory, disorrientation, and perceptual disturbances

151
Q

Management of delirium

A

Definitive and pallative

152
Q

Grade 1 concussion

A

No loss of consciousness but may cause brief confusion or alteration in mental status that resolves within 15 minutes. Posttraumatic amnesia lasts less than 30 minutes

153
Q

Grade 2 concussion

A

No loss of consciousness or brief loss (less than 5 min), but confusion or mental status changes last longer than 15 min. Posttraumatic amemnia lasts longer than 30 min.

154
Q

Grade 3 concussion

A

Loss of consciousness lasting longer than 5 min or post-traumatic amnesia lasting longer than 24 hrs.

155
Q

Assessment with minimal or minor head trauma

A

Assess for signs of skull fracture, such as hemotympanum, posterior auricular or periorbital ecchymosis, bony depression.

156
Q

Most significant indicator of brain injury

A

Loss of consciousness

157
Q

Immediate ED referral for head trauma

A

Alteration of LOC, paralysis, paresthesia, rhinorrhea, raccoon’s sign, Battle’s sign, otorrhea, and hemotympanum

158
Q

Talk and deteriorate syndreom

A

Pt with this syndrome utter recognizable words after the head injury and then deteriorate to a severe, brain-injured condition within 48 hrs.

159
Q

Medication for status epilepticus

A

Lorazepam (ativan) 2-4 mg IV at 2 mg/min or valium 5-10 mg IV at 2-5 mg/min. Can give valvium PR or IM versed 5 mg if IV site can not be obtained.

160
Q

Gold standard in evaluating seizure disorders

A

MRI

161
Q

Meds for seizures and the elderly

A

Onset of epilepsy as increased related to increase in cerebrovascular disease, brain tumors, and Alzheimer’s. Monotherapy is most important for this population to reduce side effects and drug interactions.

162
Q

Treatment of isolated first- and second-degree sprains

A

RICE and no weight-bearing on swollen or acutely painful knee.

163
Q

Most common presentation of ankle sprain

A

Swollen and painful joint. Ecchymosis and decreased ROM is generally present

164
Q

Classic presentation of Achilles tendom rupture

A

There is sudden weakness in the ankle. It is impossible to rise up on the toes, and most people limp. Usually occurs in left ankle

165
Q

Clinical presentation of plantar fascitis

A

Pain with weight bearing the first thing in the morning or after periods of rest. Patients occasionally limp or avoid planting the heel when walking.

166
Q

Sprains

A

Result from a tearing of the ligaments that bind the joint as the joint is forced beyond its normal ROM

167
Q

Strains

A

Result from overstretching or overuse of muscles

168
Q

DIslocations

A

Occur when a bone is displaced at the joint so that the articulating surfaces of the bones detach.

169
Q

Indication for referral for sprains, strains, fractures

A

Indicated for compound fractures or any pt with neurovascular compromise of an extremity