Patient Data Flashcards
Ph
(7.35-7.45)
Important to diagnose diabetes and may be a indication for sodium administration
Determine CO2 retainer (COPD) if pH is normal when PaCO2 is high = COPD
PaCO2^45 (35-45)
Not ventilating
You use to correct it
- Ippb
- mechanical ventilation
- manual resuscitation (bag)
- bipap
- mouth to mouth
- pressure support ventilation (PSV)
Below PaO2< 80. (80-100)
Means the patient is not oxygenating
Intervention is needed
Low HCO3 (22-26)
Clear indication for sodium bicarbonate administration
Hb (12-16)
Difference between low Hb and high Hb
If Hb is low the patient is hypoxic regardless of PaO2 and SaO2 so give blood
High Hb above 16 is called polycythemia found in COPD pt’s
Hemorrhaging is a indication of blood loss and indicates supplemental blood should be given!
VD/VT (20-40%)
Acceptable up to 60% if on a ventilator
The only calculation that relates to ventilation
Represents the percentage of the tidal volume that is unavailable for gas exchange
VD/VT=(PaCo2- PECo2)/ PaCo2 x100
High dead space VD/VT
Relates to pulmonary embolus
PaC02 value comes from blood gas
PECO2 comes from end-tidal CO2 monitor (infrared device or capnography) or the Douglas bag
Tube position
Tube position should always be
2cm - 1 inch above the carina or at the aortic knob/notch
Instillation of medication
Navel
Narcan.- Narcotic overdose Atropine- Bradycardia Valium/versed- Sedative Epinephrine- Asystole Lidocaine- PVC
Sinus Arrhythmia
Sinus rhythm with irregular rate
Treatment- treat any other symptoms
Sinus tachycardia
Sinus rhythm with a rate above 100
Treatment- give oxygen
Sinus bradycardia
Sinus rhythm with a rate below 60
Treatment- oxygen, atropine
PVC
Premature ventricular contractions
Treatment- oxygen lidocaine
MPVC
Multifocal premature ventricular contractions
Treatment- oxygen, lidocaine
V-tach
Ventricular tachycardia rhythm with a rate above 100
Treatment- defibrillate (if no pulse), lidocaine & (cardiovert if pulse is present)
V-fib
Ventricular fibrillation is a completely irregular ventricular rhythm
Treatment- defibrillate
Asystole
Treatment-Confirm in 2 leads first, epinephrine, atropine, CPR
1st degree heart AV block
PR interval above .20 (measured from the beginning of P wave to the beginning of the QRS
Maybe due to ischemia or digitalis
Treatment-atropine
2nd degree AV block
Irregular rhythm normal P waves but the QRS complex is missing
Treatment- atropine, electrical pacemaker
3rd degree AV block
Atrial rate above 60…. ventricular rate below 40/minute
PR interval cannot be determined; QRS complex will be widened
Treatment- electrical pacemaker
Ischemia
Reduced blood flow to tissue
Inverted T waves
Can also be caused by digitalis toxicity and hypokalemia
Injury indication
Injury is indicated by an elevated S-T segment
Infarction diagnosis
Infarction diagnosed by significant Q waves
Hyperkalemia
Will cause elevated or spike T waves
Secretions in lower lobes part of the lunges
put pt head down
Secretions in higher
Indications:
Coughing helps us get the gunk up and out
Impaired mucociliary transport Excessive pulmonary secretions *(pts who have Chronic Bronchitis & Cystic Fibrosis) Ineffective cough Absent cough
CaO2
(17-20%)
Oxygen molecules in the atrial blood
Can be reduced by low Hb anemia or CO
CaO2 has the best relationship to tissues oxygenation
CaO2=(Hb x 1.34)
CvO2
(14-16%) Oxygen retuning to the right side of the heart
Best place to obtain mixed venous blood is from the pulmonary artery
Decreasing values relate to decreasing cardiac output
Mixed venous oxygen content
CvO2=Hb
C(a-v)O2
Arterial-venous oxygen content difference from
(4-5 vol%)
C(a-v)O2= CaO2 - CvO2
Value increases as C.O decrease. (Inverse relationship)
CvO2 is dropping, cardiac output is decreasing. If the SVO2 or PVO2 is dropping while the arterial counterparts (SaO2 & PaO2) remain steady reduction of cardiac output can be assumed
Measure the oxygen consumption of the tissue
PAO2
Alveolar oxygen tension
PAO2=((PB-PH2O)FIO2)- PaCO2/0.8
The alveolar air equation is the method for calculating partial pressure of alveolar gas (PAO2). The equation is used in assessing if the lungs are properly transferring oxygen into the blood.
Shortcut: PAO2=(O2%x7)-(PaCO2+10)
A-a Gradient
A-aDO2= PAO2- PaO2
(25-65 mm Hg)
Above 65 but less than 300 mm Hg= V/Q mismatch
Above 300 mm Hg= shunt (venous admixture)
Get value on 100% FIO2
Hypoxemia - cause interpretation
P/F Ratio
(PaO2/FIO2)
Normal is 380 or greater
300 or less signifies Acute lung Injury (ALI)
200 or less signifies Acute Respiratory Distress Syndrome (ARDS)
Oxygen Index (OI)
(Mean airway pressure x %oxygen) / PaO2
Fluffy infiltrates
Pulmonary edema
Tracheal shift from midline
Pneumothorax, hemothorax, significant atelectasis
Obliterated costophrenic angles
Pleural effusion
Flattened diaphragm
COPD, significant air trapping
Wedge shaped infiltrates
Pulmonary embolism
Butterfly or bat wing
Pulmonary edema
Plate like or patchy infiltrates
ARDS, or atelectasis
Scattered patchy infiltrates
ARDS
Ground glass or honeycomb pattern
ARDS or IRDS
Reticulogranular or granular pattern
ARDS or IRDS
Concave superior border or interface
Pleural effusion
Consolidation or haziness
Pneumonia
Sudden onset of shortness of breath
Pulmonary embolus
Steeple sign lateral neck x-ray
Croup (laryngotracheobronchitis)
Tachycardia
Hypoxemia
(Thumb sign) lateral neck x-ray
Acute epiglottitis
Flat to percussion
Atelectasis
Cold clammy skin
Myocardial infarction
Fine rales
CHF & pulmonary edema
Diurese the patient, cardiac medications, positive pressure ventilation
Venous distension
CHF
Decreased flows
FEV1/FVC%
FEV1, FEF25-75
FEF 200-1200
Obstructive pulmonary disorder
Pt
Prothrombin time
Warfarin (Coumadin)
Therapy
Heparin therapy
3 layer Sputum
Bronchiectasis
Retraction
Infant respiratory distress
PTT
Partial Thromblastin time
Heparin therapy
Sudden onset tachypnea
Pneumothorax
Hyperresonant to percussion
Pneumothorax
Medium rales
Secretions in mid-size airways
Any therapy to mobilize secretions (IPPB, SMI, PEP, CPT)
Chest movement without nasal flow
Obstructive sleep apnea
Double lumen endotracheal tube
Independent lung ventilation
After lung surgery, transplant, or with lung cancer
Hypernatremia
Dehydration
Angry, irritable combative
Electrolyte imbalance
Pitting edema
cHF
Hyperlucency
Excess pulmonary air
COPD, Asthma, Pneumothorax
Dull to percussion
Fluid filled
Pneumonia or pleural effusion
Pulses paradoxus
Severe air trapping
As seen in status asthmaticus
Consolidation
Chest x ray
Heavy collection of infiltrates
Ashen or pallor
Anemia or acute blood loss
Diaphoresis
Heart failure
Opaque
Chest x ray
Fluid filled lungs
No chest movement without nasal flow
Central sleep apnea
Pleural friction rub
Tuberculosis, lung cancer,
Pulmonary infarction
General malaise
Electrolyte imbalance
Pronounced Q waves
Previous myocardial infarction
With tissue death
Struporous confused
Drug overdose
Weakness in legs
Lower extremities
Gullain Barre
Syndrome
Bronchogram
Bronchiectasis
Minimum spontaneous tidal volume (to sustain life)
5 mL/kg
Minimum vital capacity (to sustain life)
10 mL/kg
Unilateral wheezing
Lung mass or foreign body in lungs
Elevated eosinophil count
Asthma
Reticulgranular pattern on x ray
ARDS, IRDS
Lethargic sleepy, somnolent
CO2 overdose
Marked or severe
Emergent condition
Flattened t waves
Hypokalemia
Vascular markings
Increased with CHF absent with pneumonia
Spiked t waves
Hyperkalemia
H cylinder tank factory
3.14
Drooping eyelids double vision dysphagia
Myasthenia gravis
Purulent sputum
Chronic bronchitis
Tree in winter pattern chest x ray
Bronchiectasis
Paradoxical chest movement
Fail chest
Ribs broken in multiple places
Kussmaul’s breathing
Metabolic acidosis, diabetes, renal failure
Mucoid sputum
Chronic bronchitis
E cylinder tank factor
0.28
Radiolucent
Normal lungs
Rhonchi (course rales)
Mucus in large airways
Suctioning, cough coaching and anything to mobilize secretions
Night sweets
Tuberculosis
Spag unit
Small particle aerosol generator
Deliver Ribavirin for RSV
5 obstructive diseases
Chronic bronchitis Emphysema Bronchiectasis Asthma cystic fibrosis
Minimum MIP (to sustain breathing)
20 cmH2O
Myasthenia gravis
Descending (mind to ground)
Positive tensilon test
Monitor VC/MIP (serial testing)
Drugs: neostigmine, pyridostigmine
Intubation/ mech vent- short term
Gullain barre syndrome
Ascending (ground to the brain)
Spinal tap- protein in spinal fluid
Monitor VC/MIP
Drugs: steroids, prophylactic, antibiotics
Mech vent/ trach- long term
Plasmapheresis