PATHOLOGY TEST 2 I MISSED THIS TEST Flashcards
BODY RESPONSE TO INJURY
Q. PROGRESSIVE TISSUE CHANGE 2. REGRESSIVE TISSUE CHANGE ,DECREASE IN SIZE
3. IRREVERSIBLE IS DEATH
CELLULAR REACTION TO INJURY . 1.DEGENERATION 2. CELLULAR SWELLING
(CLOUDY )
- ACCUMULATION OR BUILDUP OF A SUBSTANCE INTRACELLULAR . WITH IN THE PROTOPLASM . 3. ( MOST COMMON TYPE OF DEGENERATION
CELLULAR REACTION TO INJURY 1. FATTY DEGENERATION –OR FATTY METAMORPHISIS
- BUILD UP OF FAT WITH IN THE PROTOPLASM OF NORMAL CELLS
AMYLOID DISEASE
- ACCUMULATION OF WAXY STARCH LIKE SUBSTANCE IN THE PROTOPLASM –EXAMPLE–TB
CELLULAR REACTION TO INJURY (OSTEOMYLITIS )
- INFLAMMATION OF SPONGY BONE MARROW INSIDE BONES
PROGRESSIVE TISSUE CHANGE–ACCUMULATION OR BUILD UP OF A SUBSTANCE BETWEEN THE CELLS INTRACELLULAR
- INFILTRATION —
ALLERGEN
- FORIEGN PROTEIN WHICH TRIGGERS PRODUCTION OF AN INCOMPLETE ANTIBODY
- TRAUMA
ANY TYPE OF WOUND OR INJURY
PHYSICAL AGENT
RADIATION
CHEMICAL AGENT
CHEMICALS WHICH ARE TOXIC OR POISONOUS TO THE BODY
- DEFICIENCY –ALBNISM
1.DISEASE DUE TO DIETARY OR METABOLIC DEFICIENCY.
HEREDITARY
MONGOLISM, HEMOPHILIA, DOWNS SYNDROME
PROGRESSIVE TISSUE CHANGE. (INFILTRATION)
ACUMULATION OR BUILDUP OF A SUBSTANCE BETWEEN THE CELLS . ( INTERCELLULAR )
progressive tissue change ( TRUE (GANGRENE)
MOIST TRUE,SINCE SAPROPHYTES ARE PRESENT,FOUL ODOR PRESENT —- NO LINE OF DEMARCATION
DEFINITION OF TRUE GANGRENE—-ISCEMIC NECROSIS
DEAD TISSUE PRESENT IN THE LIVING BODY
DEFINITION OF TRUE GANGRENE ——PETROFASCIN
DECOMPOSITION OF PROTEIN IN THE ABSENCE OF OXYGEN
DRY (GANGRENE )
THE SAPROPHYTES HAVE NOT INVADED THE DEAD TISSUE YET AND THEIR IS NO ODOR SPREADS SLOWLY
SAPROHPYTES
LIKE TO LIVE OFF OF DEAD ORGANISMS..
3RD TYPE OF GANGRENE (INFECTOUS IN NATURE)
GAS GANGRENE—
GAS GANGRENE IS CAUSED BY (BOARDS)
CLOSTRIDIUM PERFRINGES—BACTERIA ( MO CAUSES TISSUE GAS IN DEATH )
GAS GANGRENE
ONLY OCCURS DURING LIFE
TISSUE GAS
THIS BACTERIA HAS THE ABILITY TO FERMENT AND DESTROY GLUCOSE
DRY -MOIST GANGRENE
CAUSE BY PROBLEMS IN CIRCULATION
DECAY –PUTRIFICATION
CAUSE BY DESTRUCION OF BACTERIA
DECUBITUS ULCER
BEDSORE–DEAD EPITHILIAL TISSUE
SINGEL OR POSTINE GEL
USED FOR FIXING BEDSORES OR DECUBITUS ULCER
ATROPHY
THE ABSENCE OR LACK OF GROWTH ===OR DECREASE IN SIZE OF A BODY PART OR ORGAN THAT PREVIOUSLY WAS OF NORMAL SIZE..
PHYSIOLOGICAL ATROPHY
DECREASE IN SIZE OF A BODY PART OR ORGAN DUE TO DECREASE IN FUNCTIONAL DEMAND—THYMUS GLAND
THYMUS GLAND
WAS LARGEST IN INFANCY SO THAT IT COULD PRODUCE T-CELLS THAT WOULD LAST YOU FOR THE REST OF YOUR LIFE
SENILE ATROPHY
PROGRESSIVE SHRINKING OF ALL ORGANS AS PART OF A NATURAL AGINNG PROCESS
BREAST ATROPHY
BREAST TISSUE OF THE MOTHER AFTER LACTATION
PATHOLOGICAL ATROPHY
DECREASE IN SIZE DUE TO DESEASE—ANOREXIA NERVOSA–CANCER
BALEMIA
THEY BELIEVE THAT THEY WEIGH TO MUCH
DISUSE —— PARAPLEGIC–QUADRAPLEGIC
DETERIORATION OF MUSCLES B/C OF DISUSE
INFLAMMATORY LESIONS
ABSCESS LOCALLIZED COLLECTION OF PUS
CARBUNCLE
COMMUNICATING BOILS OCCUR TWO OR MORE
PHLEGMON
CELLULITIS
VESSICLE
BLISTER
FERUNCLE
NON COMMUNICATING BOILS
FISTULA
ABNORMAL PASSAGEWAY
ULCER
LOCAL AREA OF NECROTIC EPITHELEAL
ULCERS ARE TREATED WITH
SIN GEL OR POSTING GEL
ILFLAMMATORY LESION—PUSTULE
ELEVATION ON SKIN WHICH CONTAINS PUS
SEROUS EXUDAT DOES NOT
CONTAIN PUS
OUTCOME
1.REGENERATION 2. PHYSIOLOGICAL 3. REPAIR
RESOLUTION
SIGNALS AN END OF THE EXUDATION PHASE OF INFFLAMATION
OUTCOME-REPAIR OF -CONNECTIVE TISSUE CELLS
REGENERATION 2. PHYSIOLOGICAL 3. PATHOLOGICAL 4.
HOW YOUR BODY RESPONDS TO INFLAMMATION
REGENERATION 2. PHYSIOLOGICAL 3. PATHOLOGICAL
REGENERATION
REPLACEMENT OF DAMAGED OR DESEASED TISSUE WITH THE EXACT SAME TYPE OF TISSUE DO TO INCREASED FUNCTIONAL DEMAND
PHYSIOLOGICAL REGENERATION
REPLACEMENT OF DAMAGED OR DISEASED TISSUE WITH THE EXACT SAME TYPE OF TISSUE DUE TO INCREASED FUNCTIONAL DEMAND
PATHOLOGICAL REGENERATION
REPLACEMENT OF DAMAGED OR DISEASED TISSUE WITH HE EXACT SAME TYPE OF TISSUE DUE TO DISEASE—EXAMPLE— 2ND DEGREE BURNS
REPAIR REGENERATION
REPLACEMENT OF DAMAGED OR DISEASED TISSUE WITH HEALTHY NEW CELLS OR REPLACEMENT BY SURGERY–ESPECIALLY CONNECTIVE TISSUE CELLS
poliomyelitis
INFlAMMATION OF THE GREY MATTER PORTION OF THE SPINAL CORD
POLIOMYELITIS
CAN GET IT 3 DIFFERENT TIMES
MENINGITIS
INFECTION OF THE MININGIS
ANOTHER NAME FOR POLIO
INFANTILE POLIO
POLIOMYELITIS
YOU MAY NOT BECOME TOTALY PARILYSED,IF IT IS NOT A VIRULANT STRAIN
NEURITIS
INFLAMATION OF THE STRUCTURAL AND FUNCTIONAL UNIT OF THE NERVOUS SYSTEM
RHINITIS
INFLAMMATION OF THE NASAL MUCOSA–MUCOUS MEMBRANE WHICH LINES THE NASAL CAVITY
RHINITIS
HUMANS –CAN DEVELOP ACUTELY–CAN DEVELOP– THIS CAN BECOME CHRONIC ALSO
PHARYNGITIS
CAN GET A SORE THROAT–RESP. AND DIGESTIVE SYSTEMS ARE AFFECTED–NASAL PHARYNIX
SINUSITIS
INFLAMATION OF ONE OR MORE OF THE PARANASAL SINUSES–(AIR SPACES IN BONES LOCATED NEAR OR ALONGSIDE THE NASAL CAVITY..FRONTAL 2. ETHMOIDAL 3. MAXILLARY
MYELITIS
INFLAMATION OF THE SPINAL CORD
LARYNGITIS
INFLAMATION OF THE VOICE BOX
PLEURITIS
INFLAMMATION OF PLUERA –THIN MEMBRANE WHICH LINES THE LUNGS
PLEURA
OUTER LAYER IS PLEURA -PERIETAL INNER LAYER IS
TRACHEITIS
INFLAMMATION OF THE PASSAGEWAY FOR AIR BETWEEN THE VOICE BOX AND THE RHINI ..INFLAMMATION OF THE VOICEBOX
BRONCHITIS
INFLAMATION OF ONE OR BOTH OF THE BRONCHI–INFLAMMATION OF ONE OR BOTH OF THE PASSAGEWAY FOR AIR BETWEEN THE WINDPIPE AND THE LUNGS
COPD
CHRONIC BRONCHITITS AND EMPHASEMA AT THE SAME TIME
BRONCHITIS
INFECTION OF THE LUNG TISSUE ITSSELF
PNUEMONIA
INFECTION OF THE LUNG TISSUE
RIGHT LUNG IS LARGER
HAS 3 LOBES–LEFT LOBE HAS 2 LOBES
BRONCHAL PNUEMONIA
INFECTION OF THE LUNG TISSUE –MORE GENERAL
LOBAR PNUEMONIA
INFECTION OF ONE OF THE SECTIONS OF THE LUNG LOBES
STOMATITIS
MEANS OPENING—(INFLAMMATION OF THE MOUTH)
GLOSSITIS
INFLAMMATION OF THE TONGUE
GINGIVITIS
INFLAMMATION OF THE GUMS
ESOPHAGITIS
INFLAMMATION OF THE MUSCULAR TUBE THAT CONVEYS FOOD FROM THE THROAT TO THE STOMACH..( inflammation of the esophagus )
GASTRITIS
INFLAMATION OF THE STOMACHIC–COULD HAVE STOMACH CANCER–
ENTERITIS
INFLAMMATION OF THE SMALL INTESTINE ( CHROHNS DISEASE) ..
DIVERTICULITIS
INFLAMMATION OF A DIVERTICULI–( also INFLAMMATION OF a INTESTINAL HERNIA
COLITIS
INFLAMMATION OF THE LONGEST DIVISION OF THE LARGE INTESTINE
AMEBIC COLITIS
BACTERIA
ULCERATIVE COLITIS
RESOLVED WITH A COLOSTOMY–BY REMOVING YOUR COLON
PROCTITIS
INFLAMMATION OF THE (RECTUM)–WHICH IS PART OF THE LARGE INTESTINE
PERITONITIS
INFLAMMATION OF THE SEROUS MEMBRANE WHICH LINES THE ABDOMINAL VISCERA—OR THE PERITONIUM
PANCREATITIS
INFLAMMATION OF THE PANCREAS– REGULATES BLOOD SUGAR— SECRETES INSULIN
HEPATITIS
INFLAMMATION OF THE — LIVER