past paper set 2 Flashcards
p gingivalis virulence factors
biofilm formation
LPS endotoxin
fimbriae - host tissue adhesion and invasion
proteases
capsule polysaccharide and outer membrane vesicles
c albicans virulence factors
hyphae (morphological change and host invasion)
proteases
s mutans virulence factors
binding proteins - glucosyl and frucosyltransferases glucans (communication and adhesion) sugar modifying enzymes polysaccharides acid tolerance and adaption (ATPase)
LA mechanism
prevents action potentials through the voltage gated Na+ channels
sugar substitutes
mannitol xylitol sorbitol aspartame saccharin sucralose cyclamates
salivary proteins
amylase cystatin gustin histatin Igs lactoferrin lactoperoxidase lipase lysozyme mucoproteins 'plasma proteins' PRPs statherins
enzymes in saliva
amylase
lactoperoxidase
lysozyme
fcts of tongue
speech mastication mechanical cleansing taste protective reflex kissing
Bell’s palsy
acute unilateral facial palsy with unknown cause
- most resolve spontaneously within a few wks
- neurological opinion if severe
- CS (+/- antiviral)
macrocytic anaemia
reduced B12, folate
reticulocytes
normocytic anaemia
bleed
chronic disease
short-term adrenal suppression effects
mood disturbance
insomnia
faint
LOC due to hypotension causing hypoperfusion of the brain due to reduced O2
heart attack
MI due to atheroma causing arterial lumen occlusion
reduced O2 causing tissue necrosis of area
detecting a conventional bridge debond
place probe under pontic and apply pressure coronally
- abutment may move
- saliva bubbles may be seen at margin
can you pass floss underneath?
if debonded for some time, abutment may be visibly grossly carious
may see caries radiographically underneath
can section into smaller pieces and check for adhesiveness
features that may lead to conventional bridge failure
poor crown prep, insufficient reduction or crown margins finished subgingivally
retainers not prepped to common PofI
lack of sufficient ferrule for crown
inadequate root surface area for support, Ante’s law
poor moisture control during cementation
implant considerations
quantity of bone quality of bone position of existing teeth - rotations, angulations OH proximity to anatomical features smoking MH
extrinsic staining
diet
smoking
CHX
Fe supplement
intrinsic staining
F
non-vital
tetracycline
physiological age changes
Glickmans theory
inflammation spreads from gingivae into supporting PDL to produce a vertical bony defect, which local trauma exacerbates
Waerhang’s theory
plaque causing bone destruction of a 2mm radius, not big enough to destroy entire width so angular defect
common causes of vertical defect
trauma calculus subgingival plaque occlusion overhanging Rxs
features in a parotid swelling that would make you suspect malignancy
fixed to underlying structures
firm
growing
unilateral
fluorosis appearance
diffuse, mottled
pitting
yellow/brown discolouration