Part C - Health and Unhealthy Behaviours Flashcards

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1
Q

What is coping?

A

Research recognises two major functions of coping:

  • regulating stressful emotions
  • altering the person-environment relationship causing the distress.

This makes us question: Why do some people fight whereas some people flight? Why do some people approach and some avoid?

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2
Q

How does the developmental perspective describe emotional reactions to stressors?

A

The key components are:

  • temperament
  • conditioning (behaviour)
  • emotional development
  • cognitive development
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3
Q

What is meant by ‘temperament’?

A

Temperament is generally assumed to be innate but
personality is a collection of:
- behaviours (acquired/learned),
- emotions (shaped through social relationships,
- patterns of thought (product of cognitive development).

Temperament is generally attributed to infants, e.g. easy or flexible, active or feisty, slow to warm or cautious. In adults, ‘temperament’ is normally referred to as or thought of as ‘personality. E.g. the ‘big 5’ personality (Costa & McCrae, 1996):

  • intraversion vs extraversion
  • neuroticism vs stability
  • agreeableness vs disagreeableness
  • open to experience vs resistance to change
  • consciousness vs laxity
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4
Q

Describe Goodwin and Friedman’s (2006) findings about health status and the Big Five personality traits.

A

In a National Survey, 3032 adults aged between 25 and 74. Goodwin and Friedman (2006) found that higher consciousness levels related to higher levels of education. Consciousness was lower amongst those with mental health disorders and physical illnesses (except lung problems.

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5
Q

How do children acquire healthy and unhealthy behaviours?

A

Classical conditioning, operant conditioning and social learning theory explain how children acquire healthy and unhealthy behaviours.

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6
Q

How do we learn coping habits?

A

How we learn depends on:

  • genetics
  • style of parenting
  • socioeconomic and other environmental factors.

Poor coping strategies are often attributed to unhealthy habits. They have an INDIRECT impact on health.

Socialisation patterns also have physiological implications (Monti et al., 2014). They can have a direct impact on cardiovascular and immune systems).

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7
Q

What us meant by emotional development?

A

Everyone shares the same basic emotions but we learn to regulate these emotions based on our up bringing. Children differ on their temperament. It is easier to comfort and socialise with some children than others.

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8
Q

Discuss the attachment theory (Bowlby, 1969, 1973).

A

The theory concerns the understanding of the functions of a close bond with an attachment figure. Initially the theory was concerned with the infant-caregiver bond but later extended to other attachment relationships, especially romantic partners.

Attachment styles in adults: anxious, avoidant and secure.
The attachment styles are shown to predict whether people seek support from others and their ability to provide comfort and reassurance when their partner needs it.

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9
Q

What are the implications of attachment style on patterns of health and illness according to Feeney?

A

Feeney (2000) found that secure attachment was linked with a wide range of coping strategies.

Insecure attachment was correlated with the seeking of short-term fixes which can create long-term problems.

Avoidant attachment was related with the tendency to suppress or deny problems and emotions.

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10
Q

What did Taylor et al. (2004) find regarding early environment, emotions, responses to stress and health?

A

Taylor et al., (2004) found that a harsh family environment is associated with risky heath behaviours in adulthood, e.g:

  • substance abuse
  • risky sexual behaviour
  • poor diet
  • lack of exercise
  • other risk-related behaviours

Harsh family environments refers to lack of nurturance, overt conflict and aggression, neglect, cold/unaffectionate.

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11
Q

What are some of the influences of stress that are ignored?

A

Socio-economic status. Being of a lower status can be stressful. E.g. if there are money issues causing stress, children may pick this up from their parents. Parents may mistreat children if stressed.

Genetics. Shared genetic inheritance can contribute to negative emotional states.

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12
Q

How does cognitive development describe learning?

A

Beliefs are constructed through interaction with the world including other people. Genetics may impact on propesity to learn but thought processes are constructed through experience. We learn through self-discovery and shared experiences.

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13
Q

Discuss ‘Project Ice Storm’ and what this tells us about stress.

A

‘Project Ice Storm’ by King and Laplante (2005) gives us some interesting findings on prenatal experiences of stress.

There was a freezing rain storm in Quebec, Canada that left 3 million people with no power for a few hours however some were left with no power for weeks.

1400 pregnant women were recruited in a longitudinal study.

Their objective reaction to exposure to the ice storm and their subjective stress was measured. The mothers scored on average of 11.9 points. More than 22 points = clinical range for potential PTSD. This affected 16.6% of the mothers.

Lab assessments at 2 years old looking at cognitive development, language development and functional play.

The results showed that when mother’s had moderate-high prenatal maternal stress, the child showed significantly lower cognitive development and less word use and comprehension.

Cognitive assessments of the children at 5.5years showed the effects persisted into middle childhood.

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14
Q

What leads to coping?

A
  • appraisal of the environment
  • goal has been threatened, lost or harmed
  • appraisal is characterised by negative emotions
  • basic emotions such as anger and fear result in quick fixes to the immediate situation
  • secondary emotions such as annoyance and anxiety lead to repetitive actions and rumination, e.g. getting stressed

Positive emotions lead to more long-term, appropriate solutions to problems. But, how can you experience positive emotions when you are stressed?

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15
Q

How do we cope?

A

First, we attempt to reduce negative emotions as they can be stressful and may interfere with effective coping. Emotions are integral to the coping process as an outcome, a response and a result of appraisal.

The Fight or Flight theory talks about quick fixes, whereas Fredrickson’s Broaden and Build Theory talks about long term solutions.

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16
Q

What is emotion regulation?

A

Attempting to manage the emotional states. Two commonly used methods are:

  • cognitive reappraisal
  • expressive suppression

Reappraisal involves adapting behaviour to suit a different or other point of view.

Suppression comes late in the response process where behaviour is modified without being reduced.

John and Gross (2004) found that reappraisal was more prevalent to the anticipation of the event whereas expressive suppression was more prevalent in response to an unanticipated event. They tested this by showing people emotionally charged film and found that individuals who used reappraisal in negative situations showed greater positive emotion, lower negative and better psych health. This was the opposite for suppression in a negative situation.

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17
Q

What are the different types of coping strategies?

A
  • Emotion-focused
  • Problem-focused
  • Avoidant Strategies
  • Social support
  • Pets as social support
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18
Q

Can food really affect our behaviour?

A

There are constantly new claims and new “research” coming out saying how food can affect us. E.g. lack of vitamins = cancer, omega 3 = better brain health, earlier this week there were claims saying that eggs are extremely good for you, whereas years ago there were claims they are bad for the heart. We are always being hit with claims but there needs to be more evidence.

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19
Q

What is a macronutrient and what is a micronutrient?

A

Macronutrient - dietary fat, carbohydrates, protein.

Micronutrient - vitamins and minerals.

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20
Q

What did the Netherlands food famine in 1944-1945 tell us about our relationship with food?

A

For 6 months there was a food famine affecting a specific part of the Netherlands. On average, less than 1000 calories were consumed daily. A relationship was found between low calories and schizophrenia.

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21
Q

Give an example of evidence for a relationship between food and antisocial/criminal behaviour.

A

The Quolla Indians in Peru live at high altitudes and there was a high incidence of family feuds and murder. The violence often seemed irrational. Researchers observed a craving for sugar and prompted that their diet/glucose regulation might play a part in the violence.

Glucose tolerance tests are where people fast for a 12 hour period, their fasting blood/glucose are recorded, then they are given a small glucose drink and their blood glucose levels are recorded.

Bolton (1979) found an association between level of aggression and hypoglycaemia (falling blood/glucose levels)

Virkkunen (1982) found similar findings in violent offenders.

22
Q

Why might glucose have an effect on behaviour?

A

Glucose is the main source of energy for the brain. The main source of dietary glucose comes from simple sugars and carbs (converted into glucose during digestion).
Hence, intake of carbs increases blood glucose levels making more energy available to the brain. This then influences a variety of different systems (memory, arousal, memory).

23
Q

What is Anorexia Nervosa?

A

An intense fear of gaining weight, disturbance in the way their own shape or weight is experienced.

Has one of the highest death rates of any psychiatric condition.

24
Q

What are the possible causes of anorexia nervosa?

A

Genetic - 10 fold lifetime risk if a relative has the disorder. Concordance rates - MZ = 50%, (Bulimia, MZ = 30&)
Whereas DZ = 5-10%

Biological - serotonin and dopamine receptors both implicated. Stronger case for serotonin.

Environment - many factors including stress of mother during pregnancy

  • neglect of the individuals
  • physical/sexual abuse

Social/cultural factors - “ideal” body size, size 0 models.
BUT, how come only SOME people develop ED’s.

Some people are predisposed. Sensitivity to body size, they might seek an environment where this is important (ballet, sports)

Family attitudes to food.

Psych/cognitive theories. Links to perfectionism, low self-esteem.

25
Q

Why is obesity such a problem recently?

A

We live in an “obesogenic” environment. Food is readily available, it is cheaper to eat unhealthily and people’s lifestyles sometimes neglect healthy habits.

Supersize me culture!

Obese people normally have high leptin levels or are leptin reisistant.

26
Q

Why do we appear to need drugs?

A

To relieve boredom, to temporarily “switch off” conscious mind and to become more spiritual.

There is a difference between taking drugs and being addicted.

27
Q

What is the definition of “addiction”?

A

A psychological and physical dependency on a behaviour that can include (but not essential) ingestion of a mind altering substance.

28
Q

What are the hallmarks of addiction?

A

Dependency- potent desire, craving, inability to control behaviour.

Withdrawal- negative feelings when you don’t have the substance.

Harm- continued use despite the knowledge of harm.

Tolerance- more of the drug/behaviour is needed to obtain the same effect.

29
Q

How does gambling addiction fit in to this framework? (Despite it being behavioural rather than a substance)

A

Dependency - inability to control behaviour

Withdrawal - signs are irritability, sweating, trembling

Harm - financial implications

Tolerance - higher stake bets

30
Q

How do we become addicted?

A

The initial consumption may be driven by a number of factors:
-psychological (loneliness, isolation, low self-esteem)
-social (peer pressure)
The first attempt is generally positive, but not always.

Researching addiction helps to clarify how addiction can hijack natural reward systems. Opioids naturally activated in brain due to natural highs (food, sex, exercise). Morphine/heroin act on these same opioid systems.

‘Falsely’ signals an advantage, that something good is happening.

31
Q

What are the different explanations of addiction?

A

Cognitive - sensitisation to drug related paraphernalia, induces craving. But, why doesn’t everyone who has tried drugs become addicted? Early warning signs could include high tolerance.

Genetic (Enoch, 2011)
Heritability - alcohol: 50%
cocaine/opiate: 60-70%

Cultural differences - dry vs wet culture, alcohol ‘woven’ into culture. UK, binge! Aspect of control.

32
Q

Discuss how cannabis has been associated with mental health problems.

A
  • paranoia
  • hallucinations

Buckner (2007) found that cannabis motives was related to social anxiety (e.g. to enjoy a party).

A UoP UG project (2012) found that anxiety and cannabis were positively correlated.
Anxiety and psychosis positively correlated.
Cannabis and psychosis positively correlated.
NO relationship between cannabis USE and anxiety.

Limitation - correlation not cause.

33
Q

Describe Johann Hari’s (2015) TED talk on addiction. What research did he mention?

A

Hari questioned why doesn’t everyone become addicted to drugs. For example when people are in hospital they may be given large amounts of morphine (pure opioid).

Professor Alexander studied rats and gave them 2 water bottles, 1 = water and 1 = drug water. They always overdosed.
But! When they were in “rat park”, lots of food, friends, toys, and water bottles, they almost never drank drug water.

100% overdose when isolated, but 0% overdose when in rat park .

Vietnam war - troops and heroine. Needed no rehab or help for addiction when they came home (95% just stopped)

Alexander said, what if addiction isn’t about chemical hooks, its about BONDING. Bonds = connections, e.g. work, hobbies, relationships.

Portugal - decriminalised all drugs. The money spent on arresting people etc for drugs was spent on reconnecting them with society, 15 years later the amount of people addicted went down because they had a reason to get out of bed. Show them love.

“THE OPPOSITE OF ADDICTION IS CONNECTION” - Hari, 2015

34
Q

Why are people’s health behaviours and attitudes important?

A

What people think about their health and illness is fundamental to health psychology. People put faith in what they believe, regardless of whether it’s accurate. There are lots of models, they are all useful.

35
Q

What is the Attribution Theory (Heider, 1958)?

A

The attribution theory is an expectancy-value model which deals with how the social perceiver uses information to arrive at causal explanations. It examines what info is gathered and how it is combined to form a causal judgements.

Internal and External Attribution.

According to Kelley (1967) there are three types of causal info which influence our judgements:

  • consensus
  • distinctiveness
  • consistency

Wallston and Wallston (1982) combined the health locus of control with the attribution theory and concentrate on the internal vs. external elements of attribution theory. Three elements are:

  • internal
  • external
  • powerful others

This relates to behavioural change and required communication style.

36
Q

What does ‘risk perception’ have to do with health behaviours?

A

Risk perception is an approach concerned with ‘unrealistic optimism’, “compared to others i am LESS likely to get….”
Weinstein (1987) described 4 cognitive factors that contribute to unrealistic optimism:
1. Lack of personal experience with the problem
2. The belief that the problem is preventable by individual action
3. The belief that if the problem has not yet appeared, it will not appear in the future
4. The belief that the problem is infrequent

Risk compensation: people balance the bad health choices they make with the good.

Self-affirmation: people can be defensive when they are presented with health risk information. Self-affirmation stops defensiveness.

37
Q

What is meant by self-efficacy?

A

In Bandura’s (1977) Social Cognitive Theory, self-efficacy is described as the belief in one’s capabilities to organise and execute the sources of action required to manage one’s prospective situations. A generalised efficacy belief is “I can cope with most things life throws at me”.

Self-efficacy beliefs have been shown to be important predictors of health behaviours e.g. resisting peer pressure to use drugs, safe sex, weight loss and frequency of breast examinations.

Bennett et al., (1999) found that self-efficacy and outcome expectancies were strongly predictive of the frequency of aerobic exercise and alcohol and cigarette consumption following an MI.

Godding and Glasgow (1985) found that self-efficacy beliefs concerning the ability to resist smoking following cessation were strongly predictive of the numbers of cigarettes smoked, the amount of tobacco per cigarette and blood nicotine levels.

38
Q

Define a stage model and give some examples.

A

People are at different stages, stage models describe how people move through different stages. They have 4 basic properties:

  1. Classification system to define the different stages
  2. Ordering of stages
  3. People in the same stage face similar barriers
  4. People at different stages face different barriers

2 examples are:

  • Trans-Theoretical Stages of Change Model (Prochaska & DiClemente, 1982)
  • Health Action Process Approach (HAPA model)
39
Q

What are the stages of the Trans-Theoretical Stages of Change Model?

A

The stages of the model are:

  • pre-contemplation
  • contemplation
  • preparation
  • action
  • maintenance
40
Q

Describe some support and critique for the Trans-Theoretical Stages of Change Model.

A

DiClemente et al., (1991) categorised smokers taking part in a self-help programme into one of three groups: pre-contemplation, contemplation and prepared for action. Those in the preparation stage were most likely to attempt to quit and be abstinent for at least 6 months. However, Budd and Rollnick (1996) found that people weren’t always just in one stage at one time.

Another good point is that this model has been used to develop interventions which are tailored to each stage.

A weak point is that it is hard to know if actual stages exist and whether they are an accurate description. It also assumes a rational being. Assumes people make plans through conscious decision making.

41
Q

What is the Trans-Theoretical Stages of Change Model? (Prochaska & DiClemente, 1982)

A

The model emphasises the dynamic nature of beliefs, time, costs and benefits. There is an emphasis on decisional balance. People in the earlier stages tend to focus on the costs whereas people in the later stages tend to focus on the benefits. An example would be adopting a healthier lifestyle. E.g. the cost of a gym membership and buying fresh healthy food every week compared to focusing on the benefits such as losing more weight and looking good. It might be because results have been observed.

42
Q

Describe Health Action Process Approach (Schwarzer, 1992).

A

The HAPA emphasise the importance of self-efficacy and makes a distinction between:

  • decision making/motivational stage
  • action maintenance stage
  • intention/behavioural gap

It states that adopting health-related behaviours involves 2 stages: motivational and volition.

In order to change behaviour, people need to be motivated. The motivational stage is triggered by the perception of a threat to health. Motivation (for the goal-setting stage) is made up to:

  • self-efficacy
  • outcome expectancies
  • risk perceptions

Once an intention is determined, the individual moves into the second phase. The second phase is subdivided into pre-action and action. The action phase (goal-pursuit) consist of cognitive factors: action plans and action control and situational factors: social support and absence of situational barriers.

43
Q

What are the pros and cons of the HAPA (Schwarzer, 1992)?

A

Schwarzer (1992) claimed that self-efficacy was consistently the best predictor of behavioural intentions and behaviour changes.

But, it again, assumes a rational being. It ignores the social and environmental influences and it basically just combines the Trans-Theoretical Stages of Change model, the Health Belief Model and the Theory of Reasoned Action and Planned Behaviour.

44
Q

Describe the Health Belief Model.

A

The Health Belief Model (Rosenstock, 1966) was developed to explain and predict health-related behaviours. It is well-known and widely used. Beliefs about health problems, perceived benefits of actions and barriers to action, and self-efficacy explain engagement (or lack of) in health promoting behaviour.
The model examines the predictors and precursors of health behaviour.

Perceptions -> Action.

  • threat -behaviour
  • expectations -cues to action (internal or external)

The model was revised by Rosenstock and colleagues in 1988. It incorporated the background of the individual more (demographic, psychosocial, structural).

It assumes we work to maximise our gains from the environment. States that we look at the costs and benefits before acting. We consider the:

  • perceived susceptibility to an illness
  • severity of the illness
  • health benefits of engaging in a behaviour
  • costs of carrying out a behaviour
  • cues to action

Motivation was later added by Becker.

45
Q

What are the criticisms of the Health Beliefs Model?

A
  • Assumes a rational being.
  • Emphasises the individual
  • No mention of emotional factors
46
Q

What is the Protection Motivation Theory?

A

Rogers (1983) combined elements of the health belief model and social cognitive theory and describes how fear-arousing health communications are processed.

He expanded the HBM to include:

  • threat appraisal
  • coping appraisal

Components were: severity, susceptibility, response effectiveness, self-efficacy and fear.

According to the PMT, there are 2 sources of info that inform the process and they are environmental and interpersonal.

Public Health Awareness Campaigns tap into the PMT. (susceptibility and fear) E.g. BHF anti-smoking, Teen binge drinking.

47
Q

Describe some support for the Protection Motivation Theory?

A

Boer and Seydel (1996) measured the impact of a leaflet accompanying an invitation to a breast screening.

The leaflet emphasised the high vulnerability of older women to breast cancer (susceptibility) and the efficacy of mammograms as a means of cancer control. It attempted to induce high self-efficacy by explaining how easy and non-painful it was.

Women who rated themselves as most vulnerable and who had high-self efficacy were most likely to attend.

48
Q

What are the criticisms of the Protection Motivation Theory?

A

It assumes rationality.

It doesn’t account for habitual behaviour (brushing teeth)

Doesn’t include a role for social and environmental factors.

Doesn’t tackle how attitudes might change.

49
Q

What is the Theory of Reasoned Action and when was Planned Behaviour added?

A

Fishbein (1967) said in the Theory of Reasoned Action that intention is predicted by attitude towards behaviour and subjective norm. The determinant of behaviour is a behavioural intention.

Behavioural control was added and so was “planned behaviour” by Azjen and Madden in 1986. This emphasised behavioural intentions as an outcome of several beliefs. Intentions are a result of the following beliefs:

  • attitude towards a behaviour
  • subjective norm
  • perceived behavioural control
50
Q

Discuss the support of the Theory of Reasoned Action and Planned Behaviour. (Fishbein and Azjen, 1980)

A

Povey et al., (2000) studied the intentions of people to eat 5 portions of fruit and veg per day and found the TPB was good at predicting intentions but not actual behaviour.

Rutter (2000) studied women and whether they attended 2 breast cancer screenings and found that intention and first-time attendance successfully predicted by the TPB.

51
Q

What are the criticisms of the Theory of Reasoned Action and Planned Behaviour?

A

Assumes a rational being!

They create rather than describe beliefs, which cannot be tested.

There is an overlap between models.

Shows an association not a causality.

Different variables could be added:

  • personality
  • ambivalence
  • self-identity
  • anticipated regret
  • expanded norms
  • affective beliefs

Intentional-behaviour gap