Part 3 Podiatry exam Flashcards
wound healing
Hemostasis Phase.- is the process of the wound being closed by clotting. …starts when blood leaks out of the body
Inflammatory Phase. …econd stage of wound healing and begins right after the injury when the injured blood vessels leak transudate (made of water, salt, and protein) causing localized swelling. Inflammation both controls blleding and prevents infections
Proliferative Phase. …wound healing is when the wound is rebuilt with new tissue made up of collagen and extracellular matrix. In the proliferative phase, the wound contracts as new tissues are built. In addition, a new network of blood vessels must be constructed so that the granulation tissue can be healthy and receive sufficient O2 and nutrients
Maturation Phase.the maturation phase is when collagen is remodeled from type III to type I and the wound fully closes. The cells that had been used to repair the wound but which are no longer needed are removed by apoptosis, or programmed cell death.
2 main approaches for fracture plate fixation
Interfragmentry compression and internal splinting
2 test to evaluate progression of OM
ESR
CBC
2nd layer muscle on the plantar foot
Quadratus Plantar (Lateral plantar Nerve)
1st Lumbricals Medial plantar branch nerve)
FHL and FDL run in this layer

3rd layer of the foot
Flexor Hallucis Brevis (Medial plantar nerve,proper digital N to hallux)
Adductor Hallucis(Lateral plantar nerve, deep branch)
Flexor digitii minimi brevis( Lateral plantar nerve, superficial)

4 stages of bone healing
Inflammation, soft callus, hard, callous, remodeling
4th layer
Interosseous
PAD
DAB

5 Minute reactive hyperemic test
Patient lies supine legs raided 30 degress
foot DF and PF several time to empty venous blood
Apply and inflate cuff to 100mm above ankle systolic
Place foot heart level
After 5 minutesquickly deflate cuff
Time the interval between cuff let down and color returns to foot
Normal= instant with max erythema t 1 minute
Vasospastic disease- Return of color is uniform slight delayed 5-8 seconds-max erythema 2 minutes
Organic Occlusive Disease-Return uniform requires 15 seconds to reach toes,erythema less then normal
5th ray runs at
proximal, lateral plantar, distal medial dorsal
;Conization
remove cone shape bone
A 2 agonst
reduce central and peripheral sympathetic overflow and via peripheral presynaptic receptors may reduce peripheral neurotransmitter release. Alpha 2 agonists lower blood pressure in many patients either alone or in combination with diuretics.
Drugs: Guanabenz, guanfacine, clonidine, tizanidine, medetomidine, and dexmedetomidine are all α-2 agonists that vary in their potency and affinities for the various α-2 receptor subtypes. Clonidine, tizanidine, and dexmedetomidine ha
A antagonist
Phentolamine
Phenoxybenzamine also use for pheochromocytoma
Will cause reflex tachycardia and 1st dose syncope
A farmer ran over his brother’s foot with a plow, causing
a degloving injury and comminuted fracture of the first
metatarsal. He makes it to the hospital within 1 hour
Take a culture. Start a cephalosporin,
aminoglycoside and Penicillin G. Surgical
repair.
A patient received an allograft bone graft while
undergoing surgical excision of active osteomyelitis. The
graft is rejected. Which type of hypersensitivity reaction
TYPE IV
A Type 4 Hawkins talar neck fracture represents:
displacement of the ankle joint
Displacement of the talar neck, subtalar
joint, ankle joint and talonavicular joint
Abduction test
supine, hips and knees flexed to 90 degrees
Abduct the knee to resistance
A dislocated hip will have limitation of abduction on affected side
Absorable sutures
Plain gut-natural multifilament-digested own enz, tensile strength maint 7-10 days–70 days complete
Chromic gut-natural multifilament- chromeic NacL solution resist body enz, prolong>>90 days]
Vicryl-Synthetic monofilament-75% of original tensile strength remains day 14, absorpt 56-60 days by hydrolysis
Monocryl- synthetic monofilament-tensile strength-60% at 7 day 30% 14 day, original strength lost 21 day absorbed at 91-119
PDS (polydiazone)-synthetic monofil-70% original strength at day 14, absorption minimal until 90 days and complete absorbed 6 months
Ace inhibitors
Angiotensin converting enzyme inhibitors (ACE inhibitors) are medications that slow (inhibit) the activity of the enzyme ACE, which decreases the production of angiotensin II. As a result, blood vessels enlarge or dilate, and blood pressure is reduced
Benazepril (Lotensin) Side effects
Captopril. Cough and hyperkalemia by inhib of aldosterone
Enalapril (Vasotec)
Fosinopril.
Lisinopril (Prinivil, Zestril)
Moexipril.
Perindopril.
Quinapril (Accupr
acetabular index
angle greater then 30 is dislocated

Acrocyanosis
Acrocyanosis is persistent blue or cyanotic discoloration of the extremities, most commonly occurring in the hands, although it also occurs in the feet and distal parts of face.
Can mimic Raynauds contrictive S/S
adult range in malleolar position
13-18 degrees
Alcoholic Neuropathy
dAngthrapist
similiar to beri beri
Thiamine essential for CHO catabolism
Decrease Thiamine (B1)=CVD>>Wet beri beri
N Sys Dz>>Dry Beri beri
Dry Beri beri–Wernicke-Korsakoff Syndrome which alcohol related brain damage>>>language & thinking(xs alcohol consuption
TX-100mg IV Thiamine
Alkaline Phosphatase
30-85
Mainly in liver and bone
released by osteoblast when secreted into bone
Increased-Live/bone DZ,healing fracture &bone growth,hyperparathyroidism,obstructive biliary dz, Pagets,Sarcoma
Decreased-Hypothyroidism, malnutrition,scurvy, pernicous anema, Diabetes
Alkaline Phsphatase ALP
30-85
Liver-heat stable
Bone- heat labile
Most often measure bile duct obstruction
INCREASED- Liver DZ/Bone DZ/Hyperparathyroidism/Healing bone growth/Obstruction biliary DZ
Decreased- Hypothyroidism/Malnutrition/Pernicous anemia/
A Jones fracture occurs
Metaphyseal/diaphyseal junction
allodynia
r pain out of proportion
Allograft
a tissue graft from a donor of the same species as the recipient but not genetically identical.
Allografts
osteogenesis
osteoconduction
immogenicity
individual of same species but different genertic background
Dynamization of an external fixator
The process of making the fixator more
flexibleDyn
An axis is a straight line around which an object rotates. Movement at the joint takes place in a plane about an axis. There are three axes of rotation.
Name the 3 axises
Sagittal axis - passes horizontally from posterior to anterior and is formed by the intersection of the sagittal and transverse planes.
Frontal axis - passes horizontally from left to right and is formed by the intersection of the frontal and transverse planes.
Vertical axis - passes vertically from inferior to superior and is formed by the intersection of the sagittal and frontal planes.
Anatomy associated with Lisfranc complex
- Complex composed of dorsal (weakest) Plantar (strongest) and interosseus ligaments
- 2nd TMT joint is the keystone of the arch
- No interosseus ligament between 1st and 2nd MT
Anatomy CT
High density to Low
Cortical
cancellous
muscle
nerve
tendon
ligament
fat
air
Anatomy of nerve
Perikarya
Axon
Dendrite
Oligodentrocytes
Schwann Cell
Myelin
Ganglia
Nuclei
Nissl bodies
Node of Ranvier
ANATOMY OF SCREW
Head

Land-underside of head
shank- only in cancellous screw
run out- weakest point of screw-starts at the beginning of the threads
pitch-distance between threads
rake angle- thread to axis angle
core diameter-diameter of the screw between the threads
thread diameter- describes the screw size(ie: 2.7mm has a2.7 mm thread diameter
tip angle- tip to axis
Anchor sign
baby prone, legs adducted and extended
look for asymmetry of thigh and gluteal fold
more fold on the dislocated side
Andersons disease
rare genetic disorder of glycogen metabolism. It is caused by the deficient activity of the glycogen-branching enzyme, resulting in accumulation of abnormal glycogen in the liver, muscle, and/or other tissues. The disease is inherited as an autosomal recessive trait.
Anesthesic Supplementary medication
1- Opoids-
Fentanyl- Short-term analgesia during anesthetic periods, premedication, induction and maintenance; and in the immediate postoperative periods as need arises.
Morphine- Serves to alleviate periop pain and decrease somatic and autonomic response to airway manipulation, improve hemopdynamic stability, lower req for inhales anes and decrease anxiety. Older
Demerol-(meperidine) Induces amnesia and controls post anesthetic shivering. Can casue tachycardia and caution with pts with heartt ireg. like narcotics SE: N&V, constipation (vistaril used in conjunction)
2- IV Pain management
1-Toradol NSAID reduce post op pain, however risk of bleed
2- Acetaminiophen (IV Ofirmev) deecrease risk of bleed
ANGINA PECTORIS
PRESSURE SQUEEZINF, IMPENDING DOOM
LOCATION- ARM AND JAW
TIME-15 MINUTES
CAUSE: CAD<<<
TREATMENT: NITROGLYCERIN
Anterior Motor Horn Disease
Diseases that cause selective damage that affect voluntary movement and rarely attack other path in S.C
Poliomyelitis
Symp: malaise & Myalgias- flu-like symptoms, low grade fever
muscle tightness in hamstring, thigh and back
LMN weakness or parylsis>>gradual tightening muscle spasm, muscle weakness
Amyotrophic Lateral Sclerosis (Lou Gerhigs )
Lateral colum and anterior gray matter which are close connect with SC
ALS involves voluntary motor system inv/ degeneration of corticospinal tracts and alpha motor neurons
Presents with both UMN and LMN
Anterior Spinalthalamic tract
information about pain and temperature.touch
Anti-Angina Meds
Nitrates-
CA+ channel blockers
B Blockers
Anticoagulant
Indication::Venous thrombosis, DVT/PE/A-FIB WITH EMBOLI/MI, SURGICAL PROPHYLAXIS
TYPES:HEPARIN:LMWH-ENOXAPRIN(LEVENOX =COUMADIN
Production Vit K dependent factors-II,VII,IX,X
Contraindication: Allergy//active bleed, CNS sx//eye ex//Severe HTN, Ulcer
Heparin-works faster then warfarin-injectable
Warfarin-Slows down process in liver that use Vit K to make certain Proteins (clotting factor) that causes clotting
Coumadin-oral outpatient-stop 5 days prior to Sx
Antidysrythmiacs
Class Ia NA channel blocker-Quinidine
Procainamide
Disopyramide
Class 1b Weak NA channel blockers-Lidocaine
Phenytoin
Class 2 Beta blockers vent arrhythmias
Class 3 K channel blocker Amiodarone Sotol
Btretylium
Class 4 L-type CA channel blocker Verapamil
Antiplatelet Types
ASA (-) Cox
2) PDEinhibitor
Persantine-PDE III inhibitor >>decrease cAMP>>decrease platelet aggregation
Pletal(clopidiel) PDE III inhibitor>>decreases cAMP>>decreases platelet aggregation
3)Ticlid-250Mg PO BID
4)Trental (pantoxyfilline) alters rheology RBC>>increase flexibility>>decrease blood viscosity by decrease RBC>>decrease elevated plasma level of fibrogen>>and release plasminogen activator thereby promoting fibrinolysis
Arterial and Venous testing
Duplex ultrasound-8-10 hx of U/S triphasic-faster Q wave form resemble a teepee
biphasic may be normal in patients with diabetes
monophasic-slow Q indicates signaficant abnormal

Arthrograms for ankle ligament injury
ruputure of C-F ligament is always associated with the ATFL with dye flowing into the anterior and lateral malleolus
ASA Classification Anesthesia
ASA-
PS1 Healthy non smoker
PS2-Mild dz without substantive func limits. ie: smoker, well controlled DM/HTN mild lung disease (30
PS3 Severe systemic dz with substantive functional limitation. 1 or more moderate to severe disease
ie: poor controleed dm, HTN,COPD (BMI>40), acute hepatitis, reduction of ejection fraction
PS4 Incapacitating Systemic dz-threat to life ie; < MI,CVA,TIA or CAD/stents, severe reduction ejection fraction
PS5 Moribund patient not expected to live ie; ruptured abdominal/thoracic aneuysm
PS6 Declared brain dead
PS7 If procedure is an emergency, physical status is followed by E
Assmann DZ
Osteochondritis of the 1st metatarsal
At what point during tendon healing can isometric
exercises begin?
3 weeks
Atropine Sulfate
Scopolamine
Glycopyrrolate
Reduce resp tract secretion
protect reflex bradycardia, decrease gastric secretion
Used gen+endotracheal tube insertion
SE: Dry sore mouth, poor visual, relax lower spincter bofy temp rise
if use with preanesesthetic combo with atropine and meperidine
GLLCOPYRROLLATE-MORE POP BC NO CNS EFFECT
ANTODOTE:PHYSOSTIGMINE
Autogenerous bone graph has
a. osteoconduction
b. osteoinduction
c. viable osteocytes
Autogenous bone grafts have
oeteogenesis
osteoconduction
osteoinduction
Autograft
An autograft is a bone or tissue that is transferred from one spot to another on the patient’s body
Barlow sign
thumb on lesser trochanter and middle finger of the same hand is on the greater trochanter
The dislocated hio become displaced with a palpable clunk as the head slips over the posterior aspect of the acetabulum
Basal Ganglia Disorders
Huntingtons Chorea
Sydenhams Chorea (St Vitus Disease
Parkingtons Disease (PARK) Wilson Disease
Basophilia
Bands
Platelet count
Basophilia- Polycythemia, chronic myelogenous leukemia
chicken pox- hypothyroid myxoderma Renal DZ
Bands increase-shift to the left– acute bacterial infection
Platelet Count- 140-340,000
Increased Vascular Dz, iron def, acute infection, cardiac Dz, malignancy, PCV, cancer, RA,
BEBAX SHOE
TREAT RF ABNORMALITIES (MET ADDUCTUS)
USE AFTER SERIAL CASTING FOR MET ADDUCTUS

Best meds for post op shivering
Demerol
Best treatment for osteochondral bone lesion
osteochongral autogenous graphs
Beta blockers
Beta blockers, also known as beta-adrenergic blocking agents, are medications that reduce your blood pressure. Beta blockers work by blocking the effects of the hormone epinephrine, also known as adrenaline. Beta blockers cause your heart to beat more slowly and with less force, which lowers blood pressure.
atenolol (Tenormin) and metoprolol (Toprol,Lopressor)
Side effects—-
Dizziness.
Headaches.
Flushing of your face and neck.
Upset stomach or throwing up.
Low blood pressure (hypotension)
Irregular heart rhythms (arrhythmia
Bier Block
Intravenous reghional anesthesia
Bilirubin
.1-1.3
Is breakdown of HGB
Catabolism of HGB and breakdown myoglobin,cytochromes,catalose
Unconjugated (indirect) is lipid soluable and doesn’t spill into the urine
BREAKDOWN- occurs in Kupffer cells using cytochrome P450 to breakdown heme
INCREASED- Bile duct obstruction hemolytic anemia,hepatocellar damage, Crigler-Najjar Syndrome/Gilbets disease,XS destruction of RBC or Liver unable to excrete noram amt produced
DECREASED-MEDS:PENICILLIN/SULFONAMIDES
Conjugated (direct) increase is obstructive Jaundice
Increase in Unconjugates (indirect) destruction of RBC
>>3 jaundice
Bimalleolar equivalent
rupture of the deltoid ligament instead of medial malleolar fracture. More unstable than actual fracture of the medial malleolus
bioburden testing
is the measure of microbial contamination levels on or in a product. Bioburden can be introduced from the raw materials used in the manufacturing process, or be introduced via the workforce or manufacturing environment.
biological lock plates provide
indirect healing
Biomaterials suture
Nature and synthetic
absorable and non absorable
monofilament and multifilament
Synthetic materials cause less rxn with less inflam rxn
Absorable applies to a wound that heals quickly and need minimal temp support. ITS PURPOSE TO ALLEVIATE TENSION ON WOUND EDGES
New synthetic sutures retain their strength until the absorption process starts
Non-absorable sutures offer longer mechanical support
Mono-filament less drag thru tissue but susceptible to instrumentation damage.Infection is avoided unlike braided multifilimant can cause sustain bacterial inocula
biopsy of lesion <2 cm
b. Excisional biopsy using 2 semi-elliptical incisions
c. Dimensions should be 3 to 1 width of the lesion
d. Full thickness, including fat
Bleck classification of metatarsal adductus
bisect heel and extend line distally to see where it falls on the toes
Normal-2-3rd toe
Mild-line to 3rd toe
Moderate- line thru 3-4th toe
Severe-Line thru 4-5th toe
Bleeding time
INR
2-9 Minutes Bleeding Time
3 - 4 INR
measure the primary phase of hemostasis, the ineraction of the platelets with the blood vessel wall and the formation of the hemostatic plug
Forearm scratch
Increased–Von Willibrand,Thrombocytopenia
DIC Platelet dysfunction and ASA/ NAIAD therapy
Blood Gas coefficient for anesthesia(Ostswald)
Solubility of an agent, speed of induction
The blood:gas partition coefficient is an important determinant of the speed of anesthetic induction and recovery. It describes the partition of an agent between a gaseous phase, such as alveolar air, and the blood. The greater the blood:gas partition coefficient, the greater the solubility in blood.
High BGC=more soluble=more agent needed in blood before equilibrium is met
High BGC=More potent, slower induction
MIC=amount of inhaled at 1 ATM that prevents movement and response to noxious stimulus in 5-% pts
Blood work muscle
Creatine kinase( CK or CPK) 6-30
Cardiac ezymes- CK-MB 20% Cardiac-hi during 48 hrs MI and flip LDH-DX made
CK-BB brain 90%
CK-MM-80% cardiac muscle, high in skeletal muscle injury
Myoglobin- sensitive indicator muscle injury
Troponin increase 3-12 hour after injury
Aldolase-glycolytic involved in metabolism of glucose
Increase AST and LDH w/n 1-2 days after episode chest apin=Pulmonary infact
BLOUNT DZ
OSTEOCHONDROSIS OF MEDIAL PORTION PROXIMAL EPIPHYSEAL OSSIFICATION CENTER IN TIBIA CAUSING BOWLING OF LEGS
SYMPTOMS INCLUDE LIMPING, LARERAL BOWING
RADIOGRAPH- SCLEROTIC MEDIAL CORTEX WITH SPURRING
BEFORE 6 YEARS OLD
CAUSE EARLY WALKING AND OBESITY
blount dz
true tibial varum
progressive tibial varum
disruption of the medial capital ephiphysis
BMI
BMI <25 Desirable
26-27 Mild obese
28-29 Moderately obese
>30 Morbid obese
Bone graphs
Autogenous bone grafts provide both
osteoconductive and osteoinductive properties
Bone metabolism
Alkaline Phosphatase 30-85
Calcium 8.5-11
Phosphous 3-4.5
Bone scan of reflex sympathetic dystrophy
Increased blood flow
3rd phase of affected limb reveals increased periarticular activity
Bone scan scintigraphy phases
technetium-99m (commonly Tc-99m-methylene diphosphonate (MDP)) as the active agent. The study has 4 phases which follow intravenous injection of the tracer. Sometimes a fourth (delayed/delayed) phase is performed.
The scan is positive for osteomyelitis if images show progressively increasing lesion to background activity ratios over time.
1st Phase ====Dynamic
2nd- Blood pool image and 5 minutes after injection
3rd- after 3 hours when urinary excretion has decreased the amount of the radionuclide in soft tissues.
4th- after 24 hours
Bone stimulaors good used
An oligotrophic nonunion.
Bosworth definition
avulsion fracture of the fibula from the PITFL
Brachymetatarsia
Type I=shortening of 1st metatarsal only
Type II-Shortening of 1 or 2 lesser metatarsal (usually 3rd or 4th)
Type III-Shortening of the 1st and one or more of the lesser metatarsals
Type IV-shortening of all the metatarsals
Broden view: position of foot and beam
- Ankle is dorsiflexed, with leg internally rotated 30 degrees
- X-ray beam is cephalic tilt of 10-40 degrees
Brodie Abscess
rare form of osteomyelitis. It involves a subacute or chronic infection of the bone with development of a localized abscess, usually within the metaphysis of long bones. The tibia is the most common bone involved and staphylococcus aureus is the most common organism identified.

Buchingham classification
Type A-Medial STJ disloc-AKA basket-ball or acquired clubfoot,calcaneus medial to talus
Type B-Lateral STJ dislocation,calcaneus lateral to talus
Type C- Anterior & posterior STJ dislocation
Buergers Disease
Thromboangiitis Obliterans
inflam changes small and medium vessels
20:1
Raynauds phenomenon common
Decrease pulse
Inflammatory occlusions more distal
Patient usually have HLA-A9,,HLA-B5
BUN
10-20%
End produce of amino group removal in degrdation of amino acids
Measure urea produce in liver
Measure of liver function and kidney excretion
INCREASED-Renal DZ/Dehydration/ High protein diet/DM
DECREASED-Severe Liver damage,ie: poison//hepatits
BUN alone not reliable indicator of renal function because it depends on many extra renal factos
Buschke DZ
Osteochondritis of the cuneiforms
c. Muscular dystrophy with a waddling gait.
d. Cerbral palsy with a scissoring GAIT
hemiphagia with circumductive gait
Calcaneal erosions on xray
R.A,
Reiters
Ankylosing Spondylitis
Psoriatic OA
Hyperparathyroidism
Lipid dermata OA
Calcaneonavicular Coalition CN
45 % extra articular
8-10 y.o
Pain localized to the area of coalition
decrease ROM STJ and MTJ
MEDIAL OBLIQUE- calc and nav are close proximity or connet
LATERAL VIEW shows classic elongated ant process of calc-ANTEATER SIGN
TX- Resection and place the EDB belly in void (Cowell procedure)

Calcium
8.5-11
98% stored in skeletal and teeth
acts main resevoir for maintaining blood levels needed for muscle contraction
kidney important regulation of calcium
Any condition causing bone deminerization or atrophy>>>increase Ca Level
Hypo-Hypoparthyroidism,Vit D dif, renal failure secondary alkalosis
Hyper-cancer,hyperparathyroidism,Iatrognic, multiple myeloma
parathyroidism,m sarcoidosis, acidosis(CHIMPS), Lymphoma
Cause of congenital flatfoot
Compensated FF varus and valgus
RF equinus
abducted and adducted foot
neutrophic feet
muscle imbalance
PTT rupture
Ligamentous laxity (ehler-danlos, Marfans, osteogenesis imperfecta)
Calcaneal valgus
Enlarged or accessory navicular
Cavus foot description
elevated arch
primary sagittal plane deformity
less surface area touching the ground
painful callous may develop under met heads
chronic ankle sprains
CIA-norm 20-25 in cavus->30 degrees
Angle od meary 0 in cavus >6
Angle of Hibbs 135-140 cavus >150
Cavus foot cause and types
usually congenitial
due to failure of segmentation of primitive mesenchyme
TYPES
1-Syndesmosis-fibrous
2-Synchondrosis-cartilaginous
3-Synostosis-osseous
Cavus foot causes
neuromuscular ie: spina bifida, CMT, Friedreich ataxia
polio, spinal cord tumors, myelomeningocele
CP, infection, stphillis, trauma and S.C. lesions
Cavus foot treatment osseous
Cole-DF osteotomy Cuneiform-Nav Jt
Japas- V osteotomy thru entire forefoot, apex usually at the navicular
Devries-DF fusion at MTJ
Dwyer- Lateral closing wedge or open medial wedge calcaneal
McElvenny-Caldwell Procedure- DF fusion 1st metatarsal-medial cuneiform JT, if severe
nav-cuneiform jt
DFWO- DF wedge osteotomy 1sr metatarsals or all of the metatarsals
Jahss- DF wedge osteotomy across tarsometatarsal joint
Cavus foot treatments surgical soft tissue
Plantar fasciotomy
Steindler Stripping- plantar fascia with long plantar lgt, abductor hallucis,FDB, abd digiti quinti are stripped
Jones tenosuspension
Heyman procedure- transfer all 4 extensor tendons to their met heads
Hibbs procedure
Splitt T.A..T (STATT)
Peroneus longus tendon transfer
TPT transfer
Cavus foot treatments conservative
Shoe modification
extra depth shoes with metatarsal abr
Young patient passive stretching, manipulation
CBC
Measure total # of White cells in blood
HGB–Male 13.5-17
female 12.5-16
HCT- Male 40-50
Femal 37-47
RBC Male 5.4-
Female-4.8
WBC 5,000-10,000
CBC Diff
WBC Granulocytes ( 5,000-10,000)
Neutrphils (40-60%)
Eosinphiles (1-5%)
Basophils( .1%)
Bands
Lymphocytes 20-40%
Monocytes 4-8%
Cefdinir
For type 1 DM toe infections
Cerebellar disorders
Cerebellar-smooth coordination of voluntary, skilled movements.
Helps maintain normal posture, balance and unconscience propioception
Contibutes to vestibular function>>>maintains equilibrium
Cerebellar lesion >>>awkwardness and uncoordination of voluntary movements
Cerebellar Disorders Test
PATTING TEST; ASSESES THE INTEGRITY OF EPS OR SPINOCEREBELLAR TRACT
Alternative tap the heel and toe and ask to maintain heel against floor and abduct, the adduct foot
Heel-Knee Test- Asses integrity of EPS or spinocerebellar tract**MOST RELIABLE OF CEREBELLAR FUNCTION OF LOWER EXTREMITY
Romberg’s Test-test propioception. ability sense where one is-propioception 2 tracts-
conscience proprioception on the dorsal column
unconscience propioception spinocerebellar
Cerebellar and dorsal column-Eyes open and eyes closed= sway
Cerebellar if Eyes open steady and eyes closed sway
Clinical Muscle testing-0=absent- no evidence of contractility
1=Trace-evidence of slight contracture-no joint motion
2=Poor-complete ROM w/ gravity eliminated
3-Fair-Complete ROM against gravity
4-Good-complete ROM vs gravity with some resistance
5=Normal ROM vs gravity with full resistance
Cerebellar Lesion movement disorders
Asthenia- muscle tiring easy
Hyporeflexia decease DTR
Dysmetria-loss of ability to gauge distance, speed, power or movements
Speech- slurred, jerky or explosive in nature
Intention tremor- with initiation of voluntary movement often intesifies as goal is neared
Nystagmus- dancing eye
Cerebral palsey
Non-progressive neuromuscular disorder from brain damage
Spastic CP- most common 25%
Athetoid CP-20%
Ataxic CP-10%
Rigidity CP
Tremor CP
Atonic CP
SCISSOR GAIT SECONDARY TO SPASTIC ADDUCTOR MUSCLES.
Speech defect, retardation, seizures, visual defects, ankle equinus
TX-PT-OT-Splinting-Bracing
Charcot Marie Tooth discription
cavus foot type, decreased distal sensation,
decreased distal muscle power and
decreased nerve conduction velocity
Charcot-Marie Tooth
- Signs of muscle weakness in your arms, legs, hands and feet 2. Decreased muscle bulk in your lower legs, resulting in an inverted champagne bottle appearance 3. Reduced reflexes 4. Sensory loss in your feet and hands 5. Foot deformities, such as high arches or hammertoes 6. Other orthopedic problems, such as mild scoliosis or hip dysplasia provide information about the ext…
PERIPHERAL NERVE DISORDER
Chemical matrixectomies application
Phenol- 89% X 3 for 30 seconds Sodium hydrozide-1980
no neutralizer>>>flush with alcohol 10% NaOH X 2 15 seconds each
longer shelf life neutralizer-5 % acetic acid
recurrence 5-10% d/t old phenol or inadeq application LOW
or not removing enoug nail-need to remove nail
matrix is integral to prevent recurrence
Chloride
98-109
major excellular
acid/base balance
depletes with massice loss of G.I, fluid
Hypo-N&V, Ulcerative colitis,severe burns heat exhaustion, DM ketoacidosis
Hyper-Dehydration,XS IV fluid, kidney disease
CHOLESTEROL
LESS THEN 180
L=LOUSY LDL= IF HIGH…BAD
HDL=
DIRTY======TRIGLYCERIDES-fat in blood from food we eat
Choreoathetosis
A rapid (chorea) or slow (athetosis) involuntary movements of the fingers or toes (flexion–extension, adduction–abduction, writhing, sometimes piano-playing movements) which are irregular, nonrhythmic, and purposeless (Fahn, 1997).
Classification of metatarsal adductus
TYPE 1
Flexible- FF will crrect past neutral into slight over correction
TYPE II
Partial Flexible (doesn’t correct to neutral actively, but does passively)
Type III
Rigid (doesn’t correct to neutral)
Classification of Syndactyly
Type 1-(MMost common)-Zyngodactyly- partial or complete webbing 2.3rd toes
Type II- Synpolydactyly- 1 soft tissue mass covering 4th, 5th and 6th toe
Type III_ass/ with metatarsal fusion
Claudication causes
1-ASO
2-Deep thrombophlebitis of tibial, popliteal or femoral veins
3-Popliteal entrapement
4-Sciatica
5-Femoral or popliteal arterial calcification
6-Anemia (hypochromic, microcytic, sickle cell, thalassemia
Clinical presentation of Calcaneal fracture
Mondors sign
back pain between T12 and L2
Compartment syndrome
Hoffa’s sign- less taut Achilles tendon
Lateral wall blowout
Clinical presentation of compartment syndrome
6p’s
Pain out of proportion
Paresthesia
Pallor
Pulselessness
Paresis
Paralysis
Pressure
Clinical presentation sign seen with posterior process fractures talus
Nutcracker sign: pain with forced ankle PF
Clinical signs present with Lisfranc injury
Plantar ecchymosis sign
Apprehension sign: with FF DF and abduction
Stress exam of midfoot: unstable TMTJ with pronation and eversion
RULE OUT COMPARTMENT SYNDROME
clinodactyly
the medical term for an abnormally bent or curved finger. The affected finger abnormally curves to the side and may overlap other fingers. While the condition is relatively rare among the general population (3 percent), it affects one in four children born with Down syndrome (trisomy 21).
Club foot evaluation
Normal Club foot
Kite angle 20-40 0-15
Calc Inclination angle 20-25 17
Talar neck Add 10-20 80-90
Plantarflex 25-30 45-65
CMT Foot
Active plantarflexion of the medial forefoot
by the peroneus longus in conjunction with
loss of strength in the anterior tibial muscle
CNS disorders
Progrfessive Multifocal Leukoencephalopathy
CMV
Cerebral Toxoplasmosis
Multiple Sclerosis
Tabes Dorsalis
Coagulation Cascade
Extrinsic Pathway- Activated by external trauma cause blood to escape from vascular system
Intrinsic Pathway- Activated by trauma inside vessel system and activated by plaelet, exposed endothelium,or chemical
Cell injury..Vessel constrict..platelet adhesion>>stabilization and reinforcement of plug by intrinsic and extrinsic system>>>>Fibrinolysis
Phase I Generation of tissue thromboplastin (factorIII) intrinsic
Phase II-Activation of thromplastin end product of intrinsic and extrinsic
FINAL COMMON ATHWAY BEGINS WITH ACTIVATION OF FACTOR X
Phase 3- Concersion prothrombin>>>thrombin by factor Xa
Phase 4- Conversion of fibrogen into fibrin by thrombin which stabilzed by factor XIII
Coagulation Pathway part 2
Coagulation factors
Intrinsic- III + VII + VIIa>>>>activates X>>>Xa
Extrinsic XII>>>XIIa by surface contact>> XI>XIa>IX>IXa>VIIIa>>X>>Xa
Factors require Vit K
Heparin intrinsic

Codman triangle
periosteal elevation and spicules
formation represents tumor extensions into the perisoteum and calcification
Cohort studies
Cohort studies are a type of medical research used to investigate the causes of disease and to establish links between risk factors and health outcomes. The word cohort means a group of people. These types of studies look at groups of people.
Collagen diseases that affect microcirculation
1-R.A. 6-Giants Cell Arteritis
2-SLE 7-Erythema nodosum
3-Systemic Sclerosis 8-Erythema Induratum
4-Polymyositis 9-Nodular Vasculitis
5-Polyarteritis nodosum 10-Nonsuppurative panniculitis
Color Changes to nail
Yellow-Addisonsdz- Diabetes
Blue-Cyanosis
Red-Cancer
Green-Pseudomonas-Candida
Black/brown-Normal-Addison Dz-Junctional nevi, melanoma-
White-heredity-anemia-fungal infection
White lines-Arsenic poison pr drug toxicity
White spots- Injury-psoriasis
White & Pink- Anemia of chronic dz-nephrotic syndrome
Common Peroneal N Injury
L4-L5-S1
Sensory & Muscular
Vulnerable to external compression injury–NEUROPRAXIA-crossing leg c/p paresthesia,hyperesthesia,
entrapement more unusual
Diagnosis: NC studies; radiculopathy L5>>EMG IN GLUTEAL AND PARASPINAL
Clinical signs>> weakness TA& EH muscles>>>>DROP FOOT
weakness of anrterior muscle group (NC Studies)
Radiculopathy-L5 EMG
Common site for OATS procedure
non-portion weight bearing of the knee
Restores hyaline cartilage
Compartment syndrome is best characterized
A predictable, recurrent, well-localized pain
relieved by rest.
Component of open chain pronation
dorsiflexion, adbuction, evrsion
Components to look for on X-ray with ankle injury (5). The measurements and which is most reliable
-Medial clear space: >4mm abnormal
-Tib fib clear space: greater than or equal to 6mm abnormal. MOST RELIABLE
- Inversion stress view
Talocrural angle
Congeniotal Vertical talus open reduction
3 months of age
most procedure posterior release and reduction of the talonavicular joint
3-6 years-extra articular arthrodesis (Green-Grice type)
>6 wait until 10-14 when bones mature
Congenital dislocated hip signs
females, usually occurs 2 weeks after birth
children with older siblings, breech, joint laxity, first born
Classic signs in older children– limited abduction
asymmetric thigh fold, (+) trendelenburg test, externaslly rotates foot
Waddling gait.
when dislocation occurs, femeral head posterior and superior to the acetabulum
Assc/ with met adductus, calcaneal valgus
gohydramnios, torticollis
Congenital Vertical talus
congenital covex pes planovalgus assoc wi arthrogyrosis, right foot more common
reverse clubfoot, persian slipper, rockerbottom flatfoot
The talus is perpendicular so severly the navicular dislocated dorsally ontot he talus neck, locking the talus
Forefoot is abducted and DF at the MTJ and Calc is in valgus
Rgidity is the hallmark of this
gastroc soleus is contracted, spring lgt is elongated
usually are B/L/STJ facet abnormal/
Talus –Anterior absent/middle hypoplastic, posterior malformed

Congestive Heart Failure
Inability of heart to pump blood throughout body orrequiring elevated filling pressure in oprder to pump effectively. The pooling of blood leads to congestion in body tissue
Cause-famaliar-infection, alcohol,anemia,thyrotoxicosis, arrhythmia and HTN
Plaque, stress, smoking, age. no exercise, overwork heart, obesity
S/S-Pulm edema,,peripheral edema, enlarged or pulsatile liver/ JVD(jugular vein distension)
NEVER USE PNEUMATIC COMPRESSION DEVICES
Treatment- maintaining a euvolemic state>>>diuretics>>vasodilator agents and positive inotropes
Delaying the progression>>>ACE inhibitors, beta blocker and aldosterone antagonist(diurectics)
Angiotensin converting enzyme inhibitors (ACE inhibitors) are medications that slow (inhibit) the activity of the enzyme ACE, which decreases the production of angiotensin II. As a result, blood vessels enlarge or dilate, and blood pressure is reduced.
CONSERVATIVE TREATMENT CLUBFOOT
stretch and manipulation prior to casting
apply TOB to skin
2” cast applied, ussually long cast
correction n order
1-ADDUCTION (A.V.E)
2-VARUS
3-EQUINUS
Coonradd bugg trap
interposition of PTT prevents reduction of medial malleolar fragment
Correct sequence in soft tissue reliease in bunion surgery
adductor tendon, suspensory ligament,
tenotomy of the lateral head of FHB,
excision of the fibular sesamoid
Counter rotational system Langer
correct torsional abnormalities
several hinges allow greater frredom of motion
BEST TOLERATED SPLINT, ALLOW UNENCUMBERED CRAWLING

Creatinie Clearance
Ceatinine
140-Age x weight (kg)==male 97-137
72 X Serum= female 88-128
Creatinine found in muscle
increase in age up too 20 yo
CLINICAL MARKER FOR KIDNEY
MOST SENSITIVE MARKER OF GFR>>>DOSING MEDICATION
Cross sectional studies
involves looking at data from a population at one specific point in time. The participants in this type of study are selected based on particular variables of interest.
CT views for middle subtalar facet coalition
axial and coronal because facet lies at 45 degree and is equidistant from both planes
CVD MAJOR CARDIAC
ANGINA PECTORIS-
MYOCARDIAL INFARCTION
DISSECTING ANEURYSM
ESOPHAGITIS GERD
TIETZE’S
PERICARDITIS
GALLBLADDER CHOLECYSTITIS
Cyma line: pronation vs supination
- Anterior break (pronated)- TN joint over CC
- Posterior break (supinated)-TN joint posterior to CC
Davis and German classification
Incomplete-webbing doesn’t extend to the distal toes
Complete- extends to distal toes
Complicated-phalanges involved
DDX based on Jt Fluid Analysis Chart
Group 1-Non inflammatory
DJD,trauma,osteochondritis charcot,
hyperparathyroid Pulm OA,Pigmented villous nodular
Group 2-Inflammatory condition-RA-Gout-Pseudogout
Group 3-Septa OA secondary to Bacterial Infection
Hemathrosis-results in hemorrahgic Jt fliud speciman caused by hemophilia and other bleeding diathesis, lgt with and without FX, neuropathis arthropathy, Pigmented Syn, hemangioma
DEEP PERONEAL NERVE ENTRAPEMENT
Anterior tibial nerve` SENSORY AND MOTOR
Supplies TA,EDL,EHL,PT,EDB resonsible for DF and frontal plane movement
Innervates lateral halklux and medial aspect 2nd toe
Anterior Tarsal Tunnel Sdyndrome-BLUNT TRAUMA to ant ankle///entrapped under ext ret or irriated by superior edge of Inf ext Ret (tarsal spur) compresses the DPN over ankle (max point of contact)
Clinical Finding: paresthesia dorsum of foot with numbness 1st met space//nocturnal pain relieve by movement d/t motor innervation of EDB
DX- distal motor latencies woth NCS >7ms (5 norm)
also reproduce sym PF of ankle with ext toes>>palpate the N of the ant ankle medial to dorsalis pedis
Deep Tendon Reflex
4+ associated with clonus
3+ Hyper-reflexic
2+ average
1+ low normal-hypo-reflex’
0 Absent
Patella DTR-L3 & L4 knee slight extend
Achilles S1 & S2 foot plantarflex
Define dive punch piece
- Triangular wedge of tibia that drives the rest of a pilon fracture
Defivitive treatment of atrophic non union
Decortication and stabilization of the
fragments
Delayed healing
2-6 months post
Dennis brown bar
treat met adductus
convex pes planovalgus
bar is screwed or riveted on shoes

Describe Amberry
McKeever
reverse Hohmann
long oblique distal osteotomy
Amberry-Davis+removal of base of proximal phalanx
McKeever-resection of 1/2-2/3 of 5th met
Reverse Hohmann-transverse osteotomy in neck
L. Oblique Distal -Weil osteotomy like cut at the MT neck
Describe DeVries 5th metatarsal
removal of lateral plantar condyle
Describe Hibbs tenosuspension
-EDL stlips combined and transferred to lateral cuneiform
Describe Jones tenosuspension procedure
transfer of EHL to 1st MT head
Describe Lawrence and Bott classification
1) Avulsion fracture due to lateral band of plantar fascia or PB contractures
2) Jones fracture due to ground reactive force with failure of the foot to evert
3) Diaphyseal fracture due to chronic stress, and repetitive distractive forces
Describe Steindler stripping procedure
-Sectioning of plantar fascia, 1st layer of plantar muscles, long plantar ligament
Disadvantage::::can active Charcot
Describe the Cobb procedure
-TA split proximally and rerouted through medial cuneiform. Tenodesed to PTT
Disadvantage-
sacrifices major inverter/adductor of the foot
Describe the Heyman tenosuspension procedure
transfer EDL to met necks
Describe the Stewart classification
1) Extra-articular Jones fracture
2) Intra-articular non comminuted fracture
3) Extra-articular avulsion fracture (PB)
4) Intra-articular comminuted fracture
5) Apophysis fracture
Describe the Young tenosuspension
-Reroute TA through Keyhole in the Navicular with insertion still intact
Describe Torg Classification
Type I: acute injury
Type 2: delayed union
Type 3: Nonunion
Diabetic Peripheral Neuropathy
1-Hyperglycemia and duration of disease
Small unmyelinated C fibers compose autonomic and sensory axons that transmit thermal perception and sympathetic function
-sensory changes do not correlate with N conduction deficits
2-presents prominent paresthesias and ANS with presence of orthostatic hypotension, resting tacycardia, and distal andydrosis
3-Large myelinated- (motor and sensory)- symptoms are tingling, burning, numbness, allodynia or deep lancinating pain
DTR attenuated or absent with possible distal motor wearkness
Neuopathy develps distal-proximal (Stocking & Glove) which is a cause of ulceration in 85%
Diabetic peripheral neuropathy TX
Capasaicin- to target P baciophen, muscle relax, TCA, anti-seizure meds
GAPAPENTIN-
Neurontin (900mg)initial,>>(1 tab)300mg>>(2 tbs)600mg>>900mg(3tabs)
Amytriptiline( 25mg @pm dose. if neurontin does work) +DOPA and NE receptor
OD-dizzy, drousy.. , Excrete unaltered>>no kidney stress
Gout Diagnosis
1-Light microscope-needle like crystal piercing WBC yellow needle crystals appear negatively birefrignant.Parallel to axis of lens and blue when perpendicular
2-X-Ray Rat bites, cloud sign, punched out lesions
3-Martel sign (overhanging margins)
diastematomyelia
more common in female
DIAZEPAM
LORAZEPAM
MIDAZOLAM
CNS DEPRESSES AND REDUCE ANXIETY
IV DIAZEPAM- PAIN BUT ORAL REACHES HIGHER BLD LEVEL
LORAZEPAM- 8 HR AMNESIA BC NO PRE-MED GIVEN TO OUT PT
VERSED= HIGHER % ANTEROGRADE AMNESIA-GREAT FOR ANXIOUS PT (POP)
KETAMINE DISSOCIATIVE SEDATION CAN BE COMBINE WITH VERSED OR VALIUM/ IF ADMIN ALONE-NIGHTMARES AND DELUSION
**MOA FACILITATES GABA ACTION BY INCREASE GREQ OF CL CHANNEL OPENING
ANTIDOTE: FLUMAZENZIL
DOPERIDOL+FENTANYL=NEUROLEPTIC ANESTHESIA=LIGHT GENERAL ANESTHESIA OR COGNATIVE DISS
Difference in shapes between medial and lateral OCD’s of the talus
medial: cup (PIMP CUP)
lateral: wafer
DISSECTING ANEURYSM
SHARP KNIFE LIKE SEVERE RIPPING
LOCATION-GENERALIZE
TIME->15 MIN
CAUSE-VARIES>>STROKE
TREATMENT-SURGERY AND PREVENTION
Distal Tarsal Tunnel Syndrome
Entrapment distal segments of the prox Tib N
Medial Plantar Nerve=JOGGERS FOOT SEONDARY TO COMPRESSION INTO THE PLANTAR B/C NAVICULAR TUBEROSITY AND ABDUCTOR HALLUC BELLY
Lateral plantar Nerve-1st branch to abductor digit M.Q. (BAXTERS NERVE) Enrtrap between quadratus plantar &Abductor Hallucis is accos with chronic heel spur synd and is MOST COMMON TYPE DISTAL TARSAL
TUNNEL SYNDROME
Give S/S post static dyskinesia and described as afterburn
DM Neuopathy Intrinsic muscle
Atrophy of intrinsic musculature>>> digit contracture
plantar prominence of metatarsal
abnormal distribution of the weight bearing load with ambulation
Advanced stages>>foot drop secondary gastroc complex lost its antagonist muscle group
DM neuropathy ANS
Profound vasodilation
presents::warm, erythematous and dry>>Increase blood flow
>>>>>deminerization of bone>>>>“wash away”
CHARCOT JOINT DISEASE
DM Neuropathy Sensory changes
Sensory doesn’t always correlate with Nerve conduction studies
DTR-attenuated or absent with possible distal motor weakness
Devlops in legth dependent fashion>>STOCKING GLOVE
Decrease sensory loss>>>ulceration or breakdown
LOSS OF VIBRATORY AND POSITION SENSE>>>Ulcer
CAUSE-Oxidative stress>>breakdown intraneural glucose
DM peripheral neuropathy pathology
Small ummyelinated C fibers composed autonomic & sensory axons that transmit thermal perception and sympathetic function SEEN EARLY
-Prominent paraesthesia + ANS dysfunction ie:orthostatic hypotension
resting tachycardia
distal anhidrosis
Large myelinated axons-both motor & sensory—conducts propioception, light touch & pain
Sensory does not correlate with nerve conduction deficit
Symptons:Tingling,burning, numbness, allodynia, deep lacinatying paon
Drugs for tubercolosis
Isoniazid-pyrodoxidine def is major side effect
Rifampin-cytoP50—will decrease effectiveness of co-administered drugs,::protein inhibitor, BCP, warfarin,quindine,zidovudine,itraconazole, lfluconazolw and ketakononazole
Pyrazinamide
Ethambutol or Streptomycin
Duchenne Muscular Dystrophy
DMD) is a severe type of muscular dystrophy that primarily affects boys. Muscle weakness usually begins around the age of four, and worsens quickly. Muscle loss typically occurs first in the thighs and pelvis followed by the arms.
GOWERS SIGNS-pathognomonic where child rises from sitting position by climbing on her legs
During which phase of Tendon healing do you start
cross training with lower impact exercises?
Phase 3
Ganleys closing abduccory cuboid calcaneal osteotomy
Dynamization
The process of making the fixator more
flexible
Electrolytes
Sodium (NA) 135-145
Potassium(3.5-5.5)
Chloride-(98-109)
CO2
Electromagnetic fields
low frequency magnetic fields.
Magnetic fields are created by electricity flowing through wires
Emollients
Aquaphor
Calamine
Cetaphil
Eucerin
Lac-Hydrin
Lanolin
Moisturel
Entrapement neuropathy
Definition-compression neuroapathy to gradual contriction anatomic structures about anerve
S/S- insidious & mil;d; motor and sensory changes painful
DX: EMG and NCV
TX Surgical decompression
Esses-Lopresti
Intra-articular calcaneal fracture
type 1-tongue fx d/t vertical fall primary line exist plantar secondary fx line exist posterior
Type 2-joint depression fx d/t posterior fall, primary line exist plantar and 2nd fx line posteriorsuperiorly(dorsally)
Etiology of Lisfranc fracture
- most injuries in Dorsal direction
- Forced abduction
- Twisting with an axial loading of a PF foot
- Motor vehicle accident
Etiology of posterior process fractures
-Shepard’s or cedell fractures occur with forced PF of the foot.
Steida process: intact medial tubercle
Evans procedure
procedure best treats a flat foot with a severe transverse plane abduction deformity
Exam that can be done in the OR for syndesmosis injury
Cotton test/hook test- best test for syndesmosis injury
Femur development infants
In Adults
135-155 infant
120-135 Adults
Fillauer Bar
Same as Denis-Browne Bar except the bar clamps to sole of pts shoes
Need rigid shoes

Flexible vs rigid pes planus foot
Flexible Rigid
+ Hubscher manuever (-)
+ Resupination test (-)
NOT PAINFUL PAINFUL
LA arch on weight bearing coalition, vertical talus
FRIEDMAN COUNTER SPLINT OF FLEXOSPLINT
DYNAMIC SPLINT CONSIST OF A BELT AROUND THE POSTERIOR HEEL
ALLOWING MOTION IN ALL PLANES EXCEPT INTERNAL ROTATION
INDICATED FOR INTERNAL TIBIAL TORSION
Frontal plane corection of flatfoot deformity
Chambers-raise the posterior facet of the STJ using a bone graph
Baker- Osteotomy inferior to the STJ post facet with bone graph
Selakovich- open wedge osteotomy of the sustentaculum tali with bone graph
which restricts abnormal STJ motion
Gleich- Oblique osteotomy displaced anterior- help to increase cal-inc.angle
Silver- Lateral opening wedge with graph
Koutsogiannis- Medial side calcaneal osteotomy
Triple Arthrodesis- reserved for salvage
Grice-Green Extra-articular Subtalar arthrodesis -bone graph inserted laterally in
sinus tarsi bewteen the talus and calc. good for children. Provides excellent stability
Galeazzi Sign
also known as Allis sign
hip and knees flexed, supine position
dislocated hip results in lower knee positiob on affected side
Galium 67
1/2 life 78.3 hours
diagnose if you have certain types of cancer, such as Hodgkin’s disease, lymphoma, or lung cancer.
identifies the cells that are dividing most quickly in your body. It can help detect some cancer cells. It can also help show cells that are rapidly reproducing or responding to an infection somewhere in your body. People with lymphoma (cancer of the lymph system) may need gallium scans.
principal organs that localize gallium are the liver, spleen, and bone marrow.
Ganley splint
1st splint to treat combo foot and leg disorders
Similiar to Denis-Browne
If treating internal rotation, bar placed bewteen RF plates
If treating external rotation, place bar forefoot plate

General anesthesia staging based on Guedel’s
Phases on anesthesia
1- Induction
Begins with induction agent and ends with loss of consciousness. The patient can still feel pain in this stage
Stage 2-Hyperexcitable state. The time where the patient looses consciousness and when they regain autonomic stability. The patient losses the ability to maintain temperature, blood pressure and may experience irregular breathing, uncontrolled movement, GI issues (vomiting). This stage last a very short amount of time.
2-Maintenace-
Stage 3-This is desirable state or target debt of anesthesia. Patient regains autonomic stability
Plane 1-eye rolling which progresses to eyeball centrally fixed
Plane 2- Loss of corneal and laryngeal
Plane 3- Pupils dilate and loss of light reflex
Plane 4-Intercostal paralysis, Short shallow abdominal respiration (diaphragmatic respiration)
Stage 4 Overdose. Autonomic instability will begin to reemerge. Loss of BP, decrease breathing, circ failure
Emergence-Starts at stage 3 surgical anaesthesia) thru stage 2>>> stage 1 conscience awake
General induction Anesthesia
A-IV short acting Other induction agents>>Fentanyl>>>Sulfematil>>>>Alfemtanil (uses narcotic as induction reduces need for pre-medication)—-SIDE EFFECT–RESP DEPRESSION>> USE NALOXONE
B-Barbituate- 1- Thiopental-(3-5MG)-short act depress. SE long recovery>metaboloze in liver, PORPHYRIA
2- Methohexital (Brevital)short acting, faster return to consc SE: cough/singultus/ PORPHYRIA/cardiac failure/ not used with recent MI or air obstruct or severe liver damage
3 Propofol(Diprivan)-** sedative-hypnotic- metabolized fast in liver. Rapid return to clear head
SE: cause greater CV and resp depression
4- Ketamine- IV/IM great for un coop Kids ( SE dissociative anesthesia- pt appears awake eyes open but pt is unaware of surrounding)
C- Benzodiazepine 1-Midazolam-No analgesia>>Resp depression>>decrease BP-shortest act
D- Butyrophenone 1-Droperidol -if combined with other narcotic like fentanyl>>Neuroleptic Anesthesia
Giant cell tumor
This tumor usually affects patients in their 2nd and 4th
decade of life. It has a predilection for the female
population. Symptoms are often a dull ache that is
intermittent in nature and may be accompanied by a
palpable mass. The areas most commonly affected are
the distal radius, proximal tibia and the distal femur.
Radiographically one may see an expansile lesion, a
central area of radiolucency, destructive, and can extend
to the articular surface of the bone. What is
Glide and thread hole
Insertion of a fully-threaded lag screw requires drilling a glide hole in the near cortex, on the same axis as the thread hole in the far cortex.
Glide hole: The cortex under the screw head is drilled to the size of thread diameter so that the thread gets no purchase.
Thread hole: A drill hole which is the same diameter as the core of the screw is drilled, and a tap is used to cut the spiral groove that receives the thread of the screw. The result is a thread hole.
One can drill both cortices with the thread hole sized drill and then overdrill the near cortex. Alternatively, the gliding hole is drilled first, and a drill sleeve is placed within it to direct the drill bit for the thread hole in the far cortex
Goals of AO
The first screw should be placed centrally
and perpendicular to the long axis of the
bone. The secondary screw should be
placed perpendicular to the fracture line and
placed on either side of the first screw
GOUT
Affect peripheral joints, esp 1st MPJ
Monosodium crystals forms
Supersatuated hyperuricemic body fluid crytalizes
UA Crystals in joints from xs breakdown or overproduction of purines
CLASSIC BEGINS: evening or early morning
S/S=Asymmetrical monarticular OA
Sudden onset: red hot swollen with possible low fever
Joint sparing, but in chronic, joint destructive
Usually 1st attacks 1st MPJ
Crunchy tophi in ears
Ground reactve force peak in which 2 phases of gait cycle
contact, prolpulsion
GTT
Present in renal tubular epithelium and in liver
sensitive to detect chronic alcohol consumption
Guillame Barre Syndrome Landry’s Ascending Paralysis
danGthrapist
sysmetrical motor and sensory paresis
Schwann cell surface membrane targeted
frequest infection organism—Camphlobacter jejuni
Pathology Edema of nerve–degeneration of nerve and myelin sheath
S/S=distal limb first (weakness)>>decrease tednon reflex>>inv ANS>>motor and sensory weakness
TX- Plamsa phoresis with immunomodulation via infusion IgG shortens DZ
hallus abducto valgus and associated complications
prominent metatarsal head
*hypermobility of the 1st ray ( usually insufficient)
* callus
hammer toe on 2nd digit
*rearfoot valgus deformity
*gait evaluation- overpronation
arch collapse (VERY MUCH SO) {*the peroneus longus tendon is stretched with collapsed arch. }
*1st MTPJ: 60 degree ROM, 45/15 ( should be under 25 and 15)
may also see ingrown nail.
Hallux limitus and rigidus destructive procedure
Keller
Implant arthroplasty
Stone-oblique osteotomy resects 1/4 of met head leaves plantar condyle
Mayo-excise 5mm met head
Mckeever=1st MPJ arthrodesis-df 5-10 degrees
Valenti-V shape osteotomy
Lapidus-fusion of the MC joint
1st MPJ Joint preserving procedures
Cheilectomy
kessel & Bonney
Regnauld-Mexican hat prox phalanx- shorten 1st ray
Waterman
Waterman-Green-shorten and PF met head
Youngswick
Van-Ness-PF wedge base 1st met
Cotton-Open base wedge
Labrinudi-PF wedge correct met primus elevatus
Hansen’s disease (leprosy)
Leprosy is caused by infection with the bacterium Mycobacterium leprae. It mainly affects the skin, eyes, nose and peripheral nerves.
Symptoms include light colored or red skin patches with reduced sensation, numbness and weakness in hands and feet.
Leprosy can be cured with 6-12 months of multi-drug therapy. Early treatment avoids disability.
Harris Beath view :
views medial and posterior STJ facets
-great for coalitions
Hawkins Classification Type 1
20%
Non displace vertical fracture talar neck
1 of the3 bllod supplies is disrupted
artery of the sinus tarsi
15% avascular necrosis
Hawkins Classification 2
Displaced vertical fracture of the talar neck with dislocation of the STJ
2 out of 3 main blood supplies to the talar body is disrupted
AA sinus tarsi and tarsal canal
AVN 15-50%
Hawkins Class Type 3
Displaced vertical fracture talar neck with STJ and ankle joint dislocation
All 3 main blood supplied to the talar body is disrupted
AA sinus tarsi, tarsal canal and deltoid artery
AVN 91%
Hawkins Class Type 4
Displaced vertical fracture of the talar neck with dislocation of the STJ, AJ and TNJ
All main blood supply to the talar body is disrupted
AA sinus tarsi, tarsal canal and deltoid artery
100% AVN
HGB
Indices of RBC info of size and HGB content of RBC
MCHC-is the average concentration of hemoglobin in your red blood cells.
Protein in RBC help carry O2 throught the body
>>18-Polycythemia, increase Bld viscosity>>>hi risk of thrombosis
<<<<11 anema
HCT
Male 40-50
Female 37-47
Integral part of persons CBC
Is the % Volume of RBC in a sample of anticoagulated whole blood
High-Packed cells vol, PCV, CHF COMA Dehydration/ shocl
Low-Anemia and blood loss
Hematoma >25 % of nail bed, consider
nail bed laceration and distal phalanx FX
Hereditary Motor and Sensory Neuropathies
DangtHrapist
Type I CMT segental demyelination
Type II-Neuronal degeneration and onset distal limb weakness
Type III-Dejerine Sottas DZ-hypertrophic neuropathy>>onion bulb- affect ability to move
Type IV-Refsum Disease-lipid storage disorder with increase excretion of phytanic acid, connect to CMT overlap sensory and spinal path
Symptoms-repeat attacks and remisiion distal motor and sensory loss hands and feet, absent pain and Temp sense
Clinical: enlarged nerve sheath resembles Dejerine-DSottas DZ
Type V-spastic paraplegia
Type VI optic atrophy and hyperthrophic neuropathy type I
Type VII- clinical picture type I with complication retinitis pigmentosum
Hilgenreiner line (Y line)
Ombredanne (Perkins vertical line)
dislocated hip will be femoral head outer upper quadrant

Hindfoot alignment view: position of beam and what is measured
- beam at 10 degrees
- to measure RF coronal plane axis
How long do you brace with the Ponsetti method? and follow with?
recommended for 2-4 years, then dennis brown bar for 2 years
How many days sutures left in
face and neck 2-5 days
dorsum foot-7 days
plantar 10-14
retention site 3 weeks-6 weeks
How many incisions are used with Lisfranc surgery and what structures are fixated
3 incisions are used.
- medial to 1st met
- in 2nd interspace
- in 4th interspace
ORIF used to achieve arthrodesis of 1st-3rd TMTJ
Do not fuse 4 and 5 because they are essential joints.
HTN Pathology
Over time collagen fibers in aa and arteriole wall increase>>bld vessels stiffer and with reduced elasticity>> Arterial BP
Path-Inability of kidney to excrete sodium
An overactive renin/angiotension system leads to vasoconstriction and retension of sodium and water
Overactive sympathetic NS>>>increased stress response
Complication- CVA MI
Cardiomyopathy Hypertensive retinopathy
Accelerated HTN-SBP >>240 without signs of end organ damage
Hypertensive emergencies- when end organ damage is ongoing without intracaniel pp
Malignant hypertension-late phase with headaches, blurred vision, increased intracraniel pressure
Diagnostic Evaluation-**KIDNEY FUNCTION: SERUM CREATININE AND BUN ARE ELEVATED
PROTEINURIA, ELEVATED K+, EKG AND CXR
Huntington Chorea
BASAL GANGLIA DISORDER
Degenerativr CNS by involuntary movement,
progressive dementia, psychiatric and behavior distubances
Associated chromosone 4
Autosomal dominant pattern—Men=Women
Athetosis usually freq in hands and feet
Athetosis is a symptom characterized by slow, involuntary, convoluted, writhing movements of the fingers, hands, toes, and feet and in some cases, arms, legs, neck and tongue. Movements typical of athetosis are sometimes called athetoid movements.
Hemmiballism characterized by violent involuntary limb movements, on one side of the body, and can cause significant disability.
Essential tremor and choreoathetotic gait
Choreoathetotic gait- as rapid (chorea) or slow (athetosis) involuntary movements of the fingers or toes (flexion–extension, adduction–abduction, writhing, sometimes piano-playing movements) which are irregular, nonrhythmic, and purposeless
Treatment-Haloperidol and phenothiazine suppress the chorea
Hydrogenated fluourocarbons
HISMDEs
Halothane- .75 MAC///BGC= 2.3. Most potent rapid smooth induction and recovery. low irritability to resp tract
non emetic, non flammable, BRONCHODILATOR EXCELLENT PEDS> asthmatic
Disadvantage:: Decrease BP by reduce cardiac contract//sensitize myocardium ot catecholamine>>dysrhymias
myocardium depression>Halothane hepaitis> postop shiver decreases HR>>hepatic necrosis]
DON’T ADMIN W/N 3 MONTHS BECAUSE SLOW LIVER METAB>>HALOTHANE HEPATIC NECROSIS
Isoflurance-BGC 1.4 MAC=1.5 low bld solub so fastest response and recovery, bronchodilator non flam,
good muscle relax, maintains cardian rhym, compatible with epi, doesn’t sensitize heart to catecholamine
Disadv: depress CVS, shiver post op, poss acute or delayed lover injury
ENFLURANE- BGC 1.7 MAC Pleasant smell> rapid induction/recovery, non-irriate/ bronchodilator,Maintain CVS, non-emetic. comp with epi>>skeletal muscles> enhances nondepolorizing NM blocking agent
Disadv:Cardiac depresses..metabolize renal (caution) less potent then halothan
Methoxyfluranne- MOST POTENT- least volatile. great margin of safety, good relax, non inflam NEPROTOXIC
SEVOFLURANE- BGC=.65 MAC 1.7-CAN be deep sedative with local or general
Desflurance-BGC .45- MAC 6- good for same day out pt sx
Hyperlipidemia
Cholesterol turns into plaque
NORMAL FLOW-Lamina flow>>bullet shape, its is concentric layers of blood in parallel down length of blood vessels
Blood flow–hits a curve(bifurcationor trifurcation)…turbulent flow and shacks out the solid element ( lipid-plaque)>>>>lamina flow
STENTS>>>go around curve
Hypertension Charting
>>>>140/90
if DM or kidney dz>>130/80 considered a risk
Systolic Diastolic
Stage 1 140-159 90-99
Stage 2 160-179 100-109
Stage 3 180-209 110-119
Stage 4 >>210 >>129
Ilizarov divides the postoperative period after corticotomy
Latency period, Distraction period,
Consolidation period
Ill defines erosions on X-ray
Psoriatic OA
hyperparathyroidism ( hi calcium)
R.A.
Reiters
Ankylosing Spondylitis
In the Lauge Hansen classification of ankle fractures,
what would be a short oblique fracture with rupture of the deltoid
PAB III
Indium 111
An indium-111 WBC scan is used to look for a suspected infection. The scan involves the injection of radioactive white blood cells into the vein through a small needle, followed by a gamma camera scan to confirm or exclude a clinically suspected infection.
In can be used as a label for red cells, platelets, and leukocytes.
1/2 life is 67.2 hours
Infectious Neuropathy
DANGTHRAPIST
Infectious
TB Leprosy- multiple nerve palsies
Neurosyphillis T Pallidum 25% with 3rd syphilis
Poliomyelitis-RNS virus starts as flu–meningitis>>faccid paraysis Tx Vaccine
Herpes Zoster-vessicles, segmental weakness
Lyme disease-meningeal distribution with cranial neuritis, motor or sensory radiculoneuritis
Inhalational Agents
A-Gaseous
Nitrous oxide- least potent and common for sedation.
B- Volatile Liquids
Halothane (fluothane)-rarely used.SE irreg heartbeat, resp depression liver problems. Dont use if PORPHYRIA OR MALIGNANT HYPERTHERMIA
Isoflurance (forane) SE resp depression, similar to halothane. Can cause coronary aa vasodilation>>Coronary artery steal syndrome. Decrease BP
Desflurane (Suprane) same as Sevoflurane/MOST RAPID ONSET AND OFFSET. Contra for arrhythmia can trigger malignant hyerthermia
Previous MI < 6 months perioperative re-infarction 10X if older then 6 months
Intrinsic Muscle Nerve Innervation`
Deep peroneal N-–EDB & EHB
Medial plantar Nerve-Abductor hallucis & FDB &FHB
Medial plantar nerve, proper digital branch to the hallux- FHB
Medial plantar nerve, first common digital branch 1st Lumbricals
Lateral plantar nerve, trunk Quadratus Plantaw & Abductor digiti minimi
Lateral Plantar Nerve,superficial—Flexir digiti minimi brevis 3rd plantar interosseus 4th dorsal interosseous
Lateral plantar Nerve, deep- 2nd, 3rd,4th Lumbricals &Adductor halluxis (both heads) 1st & 2nd inerossei & 1st,2nd and 3rd dorsal interossei
IPOS
ANTI-ADDUCTUS ORTHOTIS TYPE 2
INDICATED METATARSAL ADDUCTUS

Internal fixation plates, examples
b. A dynamic compression plate has oblong holes with
sloped slots.
c. In a long bone, the convex side is the tension side,
and the concave side is the compression side.
d. Examples of dynamic compression implants are
tension band plate, dynamic compression plate, and
circlage wiring.
e. In tension band wiring, the band is place on the
concave, or compression side, of the fracture.
Jackson–Weiss syndrome (JWS)
is a genetic disorder characterized by foot abnormalities and the premature fusion of certain bones of the skull (craniosynostosis), which prevents further growth of the skull and affects the shape of the head and face.
Jendrassik Maneuver
The most common method of reinforcing reflexes is the Jendrassik maneuver. In 1885, Erno Jendrassik reported that having the patient “hook together the flexed fingers of his right and left hands and pull them apart as strongly as possible” while the clinician taps on the tendon enhances the reflexes of normal patients.
Joint ROM-STJ/ Ankle and Midtarsal joint
b. Subtalar joint ROM is about 16 degrees in the sagittal
and 42 degrees in the transverse plane.
c. The oblique midtarsal joint has nearly equal ROM in
the transverse and sagittal plane.
d. Ankle ROM is about 82 degrees in the sagittal and 30 degrees in the frontal plane
Joplins neuroma
benign enlargement medil plantar digital nerve located medial aspect of the 1st MPJ hallux
Cause biomechanical
JOPLINS NEUROMA
Compression or entrapement medial plantar digitial proper Nerve
Symptoms-Numbness & Pain infer-medial 1st metatarsal-phalangeal joint
Etiology: pronation>>>apropulsive gait with medial roll-off
Kirby’s sign
- posterior facet of talus abuts calcaneal floor and occludes sinus tarsi
- sign of maximum pronation
- opposite of bullet hole sinus tarsi
Kirby sign. parallel pitch line. tests for HAglunds deformity. fowler phillp angle. normal is between 44 and 69. chauveaux- liet angle. calcaneal pitch minus the angle of the most posterior point of the greater tuberosity and the apex of the postero=ior superior crest angled to a vertical line. normal is 0-12, greater than 12 is abnormal=haglunds deformity from radiopaedia online.
knee joint doing during midstance?
extending
KOHLERS DZ
BOYS<<
NAV BECOMES SCLEROTIC AND FLATTENED (COIN ON EDGE OR SILVER DOLLAR SIGN)
SELF LIMITING
Kulgerberg-Welander
mild form of spinal muscular dystrophy
Lachmans test
predislocation sysndrome
Lag Screw definition
A lag screw is used to compress fracture fragments. It is threaded into the opposite cortex, and slides through a hole in the near cortex. Tightening the screw presses the screw head against the near cortex, compressing the fracture fragments. Optimally, a lag screw should be perpendicular to the fracture plane.
Some screws are designed as lag screws. They are partially threaded so this screw threads into the far cortex, and the smooth shaft slides in the near cortex. The result is the same as if the near cortex were overdrilled.
Partially threaded lag screws are often used to compress fractures in cancellous bone. Their threads must lie completely beyond the fracture line to achieve good interfragmentary compression.
Allows compression across the osteotomy site. the head
IE: 2.7mm into cortical bone
drill thread hole (2.0mm bit) near near and far cortex
drill glide hole-2.7mm drill bit–near cortex
counter sink
measure
tap using 2.7mm
flush
insert 2.7 mm screw
Lag screw principle
Screws positioned perpendicular to the long
axis of the bone functions to provide
resistance to axial loading.
Lance DZ
Osteochondritis of the cuboid
Lange Hansen(SER)
Supination External Rotation
1-AITFL syndesmotic rupture or avulsion of it insertion
2-the talus dispaces and fractures the fibula oblique or Sprial fracture(weber b) long,
posterior spike on lateral x-ray
3-PITFL syndesmoptic rupture or avulsion of its insertion
or fracture posterior malleolus
4-tear of Deltoid ligament or transverse avulsion fracture of medial malleolus
Lange Hansen
PAB
Pronation Abduction
1-Transverse avulsion FX medial malleolus/deltoid rupture
2-AITFL syndesmotic rupture or avulsion of its insertion//or WF fracture
3-Short oblique lateral malleolus fracture (DWB Butterfly fragment)
transverse X-ray
PER
PER
1-Transverse avulsion FX medial malleolus/ deltoid rupture
2-AITFL syndesmotic rupturer avulsion of its insertion
3-Oblique or spiral fibular fracture above the talotibial joint (Weber C)
4-Involement of the PITFLor Posterior malleolus fracture
Lange Hansen Classification
Supination Adduction
1-LRupture of the AITF/PITF lgt tear,or avulsion fibular fracture(weberA)
2-Spiral fracture of the fibula DWB POSTERIOR SPIKE ON LATERAL XRAY
3- Volkmans fracture or PITF rupture
4- Rupture of the deltoid lgt/ transverse fracture of the medial malleolus

Lateral ankle artery supply

Lateral column
lateral cortical motor pathway include spastic paresis and hyperreflexia.
LDH
Catalyses lactic acid to pyruvic acid in the citric acid cyscle(Glycolytic Cycle)
Many in tissue, liver kidney myocardium and muscle
Inceeased 12-24 hrs after MI
Increased in CVA//Hepatitis?CA/Hemolytic anemia/skeletal muscle necrosis
FALSE FLIPS-LDH1>>LDH2>>>>>MI
LDH3 Pulmonary
LDH4-Liver disease
LEGG-CALVE-PERTHES-DZ
Osteochondrosis of the femoral head
Males>>>females Limping, pain to groin with stiffnes
3-12 years of age
MOST COMON FORM OF OSTEOCHONDRITIS
SYMPTOMS-INSIDIOUS ONSET, LIMPING, GROIN PAIN REFERRED PAIN TO KNEE
childhood condition that occurs when blood supply to the ball part (femoral head) of the hip joint is temporarily interrupted and the bone begins to die. This weakened bone gradually breaks apart and can lose its round shape.
Lepow Technique
take perpendicular of a line passing thru lateral base of 5th and medial base of the 1st metatarsal and compare with the 2nd
Lewin DZ
Osteochondritis of the distal tibia
Lichtblau for clubfoot up to 4 years old
Lateral closing wedge anterior osteotmy calcaneal
LIMB LENGTHENING LENGTHENING
a. The Wagner frame
b. The Ilizarov frame
c. The Oxford frame
d. The Orthofix frame
Liver Blood Work
LDH-38-62
SGOT/AST-10-50
SGPT/ALT-10-50
GTT-2-65
Alkaline Phosphatase/ ALP (30-85)
Bilirubin (.1-1.2)
Cholesterol
Locations of bone lesions
Epiphysis
Chrondroblastoma, Giant cell tumor post growth plate
Metaphysis
Osteogenic sarcoma fibrosarcoma
unicarmeral bone cyst giant cell tumor non ossifying fibroma
Diaphysis
myeloma, ewing tumor, reticulum cell sarcoma
Loop of Henle
1) Reabsorption of 15% of filtered water and 25% of the filtered load of Na+
2) Production of a dilute (hypo-osmotic) filtrate entering the distal tubule
3) Development of a hypertonic interstitum in the medullary regions of the kidney (via Countercurrent Multiplication)
4) Recovery of H20 & NACL from urine
Loss of blood supply to the head of the femur in a 6 year
old male would likely lead
Calve-Legg Perthes Disease
ANTITHYPERTENSIVE MEDS
MANNITOL,CARBONIC INHIBITORS
LOOP DIURETIC- TYPES-FURSOSEMIDE-LASIX
ADVERSE EFFECT-CAUSES INCREASE IN K+ LOSS
THIAZIDES DIURETICS-ADVERSE EFFECTS
K SPARING DIURETICS- SPIRINOLACTONE, AMIODARONE Adverse effects
Beta Blockers-decrease HR, CO and SBP>>>>hypoglucose,hyper TG’s and ppt CHF and angina
A2 agonst- Clonidine>>>not cause reflex tachycardia
A antagonist- Phentolamine and Phenoxybenzamine
Post-ganglionic sympatholytics- Reserpine, Guanadrel
Direct vasodilators-Apresozide, hydralazioine, Minoxidil, Diozozide,Nanitroprusside, Nitroglycerin infusion
Ace inhibotors-PRILS->cough and hyperkalemia via its inhibition of aldoserone that cause hypoK+ & hyper NA
Angiotensin receptor antag-artans–blocks the angiotensin 2 receptors, has no cough
LOWER MOTOR NEURONS
LMN lesions present with muscle atrophy, fasciculations (muscle twitching), decreased reflexes, decreased tone, negative Babinsky sign, and flaccid paralysis.
ORIGNATE SPINAL CORD
ANTERIOR HORNS
WEAKNESS
amyotrophic lateral sclerosis,// progressive bulbar palsy, //primary lateral sclerosis,// progressive muscular atrophy, spinal muscular atrophy,// Kennedy’s disease, and post-polio syndrome.
Guillame Barre Syndrome
Lyme disease
Typical symptoms include fever, headache, fatigue, and a characteristic skin rash called erythema migrans.
Lyme disease causes a rash, often in a bull’s-eye pattern, and flu-like symptoms. Joint pain and weakness in the limbs also can occur.
Most people with Lyme disease recover completely with appropriate antibiotic treatment. For those who develop syndromes after their infection is treated, pain medications may provide symptomatic relief.
Magnesium
2nd greatest intracellular cation
deficiency can cause leg muscle fatigue
Maisonneuve fracture definition
Treatment
fracture of the proximal fibula corresponding with PER III
syndesmotic screw at distal fibula will reduce proximal fibular fracture
Malignant bone lesion on xray
ill defined or absent margins
cortical erosion
onion peeling
codman triange
McArdle disease
rare muscle disorder. In this disease, the muscle cells can’t break down a complex sugar called glycogen. It is part of a group of diseases called glycogen storage diseases. Another name for McArdle disease is glycogen storage disease type 5 (GSD 5 or GSD V).
Mearys angle
talo-first metatarsal angle) was measured as the angle between the line originating from the center of the body of the talus, bisecting the talar neck and head, and the line through the longitudinal axis of 1st metatarsal.
Measuring for metatarsal adductus angle
line drawn medial proximal aspect of the 1st metatarsal base and medial distal aspect of T-N articulation
2nd line-between lateral prox asp 4th metatarsal base and lateral distal aspect of the C-C joint
3rd Line- between the bisection to 2 lines
4-perpendicular line to the 3rd line down the shaft of the 2nd metatarsal
Metatarsal adductus angle a>>20 is adducted
MTA at birth 25-30 degree

Ankle blood supply

Medial Calcaneal Nerve entrapment
associated with infra-calcaneal heel spur syndrome
Medial view of ankle soft tissue

Meningitis
Bacterial- H.flu, meningitides//DX lumbar puncture
TX PCN G or Ampicillin
Coccidioidomycosis-
Cryptococcus Neoformans-pigeons—dissemination to CNS-menigitis
Brudzinski’s sign is one of the physically demonstrable symptoms of meningitis. Severe neck stiffness causes a patient’s hips and knees to flex when the neck is flexed. Indicates meningeal irritation
Kernig Sign people with meningitis weren’t able to extend their knees past a 90˚ angle without pain.
Meta-analysis
examination of data from a number of independent studies of the same subject, in order to determine overall trends:
Metatarsal adductus
adductus of the FF at the TMJ
affects 1 in 1,000 male=females
55% are B/L
CAUSE
Intrauterine position
Tight abductor hallucis muscle
Absent or hypoplastic medial cuneiform
Abnormal insertion of anterior tibial tendon
Metatarsal adductus osseous procedure
8 y.o and older
Berman and Gartland-crescentric osteotomy 1-5
Lepird-closing wedge 1-5 base
Johnson Osteochondrotomy-closing abductory-Closing abductory base wedge
1st met 2.5mm from the lesser metatarsals
Fowler-Opening wedge osteotomy of medial cuneiform with insertion bone graph
Peabody-Muro-Excise base metatarsal 2,3,4 with osteotomy 5th metatarsal
mobilize 1st met-cun joint
Steytlier and Van Der Walt-Oblique osteotomies all metatarsals
McCormick and Blount-Arthrodesis of 1st metatarsal-cuneiform joint
Metatarsal adductus soft tissue procedures
Heyman, hernon,Strong- release all soft tissue at lis franc except plantar lateral lgt
Thompson Procedure- Resection of abductor hallucis and release medial head FHB
Lange- Capsulotomy of 1sr met-cuneiform joint/ division of the abductor hallucis
Licthlau- Sectioning of hyperactive abductor hallucis
MI
N/V DIAPHORESES
LOCATION SIMILAR TO ANIGINA
TIME>>15 MINUTES
CAUSE–ANGINA MAY PRECEDE
TREATMENT: NITRATES,ASA, B-BLOCKERS,
CA+ ANTAGONIST, HEPARIN, THROMBOLYTIC TX, PCTA
Minimum Alveolar Concentration
The minimum alveolar concentration (MAC) is the minimum concentration of an inhaled anesthetic at 1 atm of pressure that prevents skeletal muscle movement in response to a surgical incision in 50% of patients.
A lower MAC value represents a more potent volatile anesthetic.
Mockenberg Medial Calcific
- is a form of arteriosclerosis or vessel hardening, where calcium deposits are found in the muscular middle layer of the walls of arteries (the tunica media).
- results in increase ankle ischemic indices
Moncheberg Disease
benign arteriosclerosis results in extensive calcium deposits in tunica media layer of medium size arteries
Monocytosis
Eosinophilia
Eosinophenia
Monocytosis Leukemia Hodgkins Bacterial Endocarditis
Collagen Vascular disease, sarcoidosis
Eosinophilia- Allergy, asthma, eczema parasitic inf, scarlet fever, Pernicious Anemia
Eosinophenia- Cushing DZ, XS ACTH, chronic steroid post opstate, shock labot
monofilament polypropylene
Least reactive
MORPHINE
MEPERIDINE (DEMEROL 1/10) MORPHINE
FENTANYL (100XPOTENT MORHINE)
ADJUNCT MINIMIZE PAIN
NOT FOR OUT PT SX\
ADR-Prolong analgesia
Hypotension, Res depression, pruritis N&V
ANTIDOTE :NALOXONE(NARCAN) OR NALMEFENE)
SULFENTANIL AND ALFENTANIL-SYNTHETIC ANALOG OF FENTANYL MORE POTENT
REMIFENTANIL-NEW AND VERY FAST ACTING OPOID FOR MINOR PROCEDURE
MRI for OM
Cortical bone destruction
bone marrow edema low signal T1
Reactive surface
well defined
Adjacent rim, well define
T2 increased signal

What does the MRI reveal in a stress fracture
Low intensity T1 linear zone poorly defined
T2 linear is dark

Mulders sign
silent palpable click that patient feels while squuzing metatarsal heads together
MUSCLE RELXANTS SURGERY
1-SUCCINYLCHOLINE (ANECTINE)-RAPID ONSET DEPOLARIZING MR WITH STRONG EFFECT, RAPID ONSET
PREVENT ASPIRATION
SE: POST MYALGIA UP TO 4 DAYS POST OP
RAPID INJECTION>>CARDIAC ARRYTHMIA
ANTIDOTE: ANTICHOLINESTERASE AGENTS-NEOSTIGMINE (PROSTIGMIN)
PYRIDOSTIGMINE(MESTINON
2-ATRACURIUM(TRACRIUM)
3-VECURONIUM (NORCURON)
BOTH non-depolaring interm. Acting MR>>slower onset then Succiny
50-75 minutes DOA Atracurium
20-30 Min DOA Vecuronium
Muscular dystrophies
progressive weaknes and degeneration of sketetal muscles
Symtoms-progressive weakness, atrophy, loss of DTR, secondary contracture and deformities
1-Duchennes-2-5 yo- most rapid progression- toe walk, diff climbing stairs, waddling gait, gower sign
2-Becker’s-5-25
3-Emery-Dreifuss-5-15
4-Facioscapulohumeral-7-20
5-Limb-Girdle-10-30
MYASTHENIA GRAVIS
AUTOIMMUNE DISORDER CAUSED BY PRODUCTION OF AUTOANTIBODIES TO ACH RECEPTOR ON MOTOR END PLATE
ASSOCIATED WITH THYMIC HYPERPLASIA OR THYMONA
neuromuscular disorder that causes weakness in the skeletal muscles, which are the muscles your body uses for movement. It occurs when communication between nerve cells and muscles becomes impaired.
Nail patholgy
Anonychia-no nail, congenital-ischemia-toxins raynauds, darier dz-lichen planus
Paronychia-infection+IGTM-staph-candida
Beau’s Line-transverse ridges .1-.5mm widex1mm deep d/t sudden arrestin Fx of nail plate-inf-typhus-syphyllis
leprosy-DM-psoriasis-vasc dz ACTH, hyperthyroidism, alopecia, exf dermatatis
Clubbed-bulbous deformity—Lovibond angle>>160 degrees-CH defects, resp, lung ca SBE
Darier White Dz-red and white longitudinal streak on nail
Mee’s lines- Horizontal striation on nail d/t arsenic & thallium poisoning
Eczema-atrophic and contact dermatitis nail colour yellow,green, gray or black
Glomus tumor-Neoplasm or AV shunts in nail beds (glomus bodies) purplish tumor & pain
Keratocantoma-subungual ulcerated lesion resembles SCC
Leukonychia- white Transverse striation, spots or total nail psoriasis, toxic metal poison, scleroderma, leprosy-anemia-cancer, hodgkins dz- darrier dz
Malignant melanoma-acral lentiginous melanoma most dreadful, MELANOTIC WHITLOW-elevation of nail
Atrophy-Lichen planus
Periungal fibroma-acq or congenital ass/ tuberous sclerosis, mental retard-seizure-adenomasebaceum
Nail procedures
Cold steel indicated for chronic reoccuring IGTN, failed chemical procedures
Frost- inverse L shape-nail, matrix and hypertrophic ungual labia: suture
Whitney-B/L frost; Mendelsohn & smith suturing
Winograd 1/4 of nail edge is removed along with matrrix and bed: snip nail fold wedge to the bone; suture
Zadik-remove nail plate & matrix c/o shortening: forced on nail matrixl; excised skin over base of nail bed prox to lunula & matrix; skin flap advanced & suture to distal nail bed
Terminal Symes-Removal entire nail plate, bed and matrix, resect 1/2 distal phalnx & close defect with plantar skin flap DISADV: shortens digit, bulbous terminal stub, slough of flap, scar, nail depth
Kaplan-stressed removal of both nails matrix and bed, H incision carried out at 2 depth
Suppan-free the eponychial fold & remove the nail, allow visual of prox nail matrix, cut lateral & ant border and remove prox attachment
Plastic lip-excise pie shape wedge of tissue from side of toe, use for hypertrophy of ungualabia
complication -recurrenc4, xs drain & bleeding, infection, exuberant granulation tissue, insuff amt removed
soft tissue migration up to dorsum of toe
Nelaton line
good for b/l dislocation
imaginary line connect the A.I. Spine to tuberosity of the ischium
if tip of the greater trochanter is palpable distal to this line>>>hip is dislocated
Neuroimaging studies
most important initial studies for children with chronic progressive ataxia
used changes in blood flow or chemistry to examine parts of the brain active while normal adults perform tasks. These studies have provided a general picture of where cognitive and emotional processing is carried out by the brain
Neuromas
Houser Neuroma 1st plantar intermetatarsal nerve
Morton Neuroma- 3rd IMS
Heuters Neurom-2nd IM nerve
Islen’s Neuroma- 4th plantar IM nerve
Neuropraxia
Radiculopathy
Neuropraxia is the mildest form of traumatic peripheral nerve injury. It is characterized by focal segmental demyelination at the site of injury without disruption of axon continuity and its surrounding connective tissues. This condition results in blockage of nerve conduction and transient weakness or paresthesia.
Radiculopathy describes a range of symptoms produced by the pinching of a nerve root in the spinal column. The pinched nerve can occur at different areas along the spine (cervical, thoracic or lumbar). Symptoms of radiculopathy vary by location but frequently include pain, weakness, numbness and tingling.
Neutrophils
Lymphocytosis
Neutrophilia-Bacterial infection, necrosis Pain
acute infection, rigious exercise post convulsion
Neutrophenia-Overwhelming infection marrow depression
autoimmunity antimetabolite
Lymphocytosis-Viral inf, measles, hepatitis, chronic TB, Lymphocytic leukemia
Non absorable sutures
Ethilon-monofil nylon-low tissue reactivity-more plaible when wet 9-0 10-0 microsx
Nerolon-braided nylon- coated to improve handling
Mersilene-multifilament polyester-minimal tissue rxn, MOST ACEPTABLE FOR VASC SYN PROCESSES
Ethibond excel-polyester braid- coated with polybutalate
prolene-relatively biolically inert, use minimal suture sx (ie:infection)
Dermabond- sterile liquid- in 3 minutes provides the same strength as healed tissue at 7 days
Steri-strips- hypoallergenic
Staples-fast method and least tissue reaction
Non Halogenated Agents
Nitrous Oxide-BGC .47
MAC 1.6
30X more soluble then NO in blood
More beneficial if used with volatile agent like halothane, enflurane ETC
MUST GIVE 100% O2 END OF SX TO PREVENT DIFFUSION HYPOXIA
CAN CAUSE CARDIA DEPRESSION
OVER LONG PERIOD>>BONE MARROW DEPRESSION
Non-invasive test to asses normal pulsation, pressure and flow for suspicion of arterial lesion
1-1Segmental pneumatic plethysmography (large vessel wall reactivwly ro pulsa)
2-Segmental pressure (indicated presence of normal to decreased heads of pressure, depending on the degree of stenosis)
3-Doppler isolation of arteries (demonstrate normal dynamic flow VS aberrant eddy current ditsal to thrombic lesions)
4-Digital photocell plethysmohraphy (demonstates pulsatility of end arteriole comprimising the glomus in the pulp of the toe)
5-Digitial pressure (asses the head of pressure in the digital arteries)
6-Thermography, skin thermometry and percutaneous oximetry (reflecs the degree of cutaneous perfusion
normal malleolar position at birth?
0 degrees
Normal metatarsal adductus at birth
22-25 degrees
Nutritional Neuropathy
daNgthrapist
Restless leh syndrome”Ekbom’s Syndrome
Decrease Zinc and folate, Vit B12 neuropathy
Clinical-persistant urge to wiggle and associated with fibromyalgia
Uncomfortable, crawling, painful grabbing sensation–Primary in L.E.
Can also have PLMS- painful limb movement sleep
TX-Dopamine agonst Pergolide (antiparkinson drug)
BZD (valium)/anticonvulsabt/opoids/dopaminergic that work on cns may help
OATS Definition
osteochondral autograft transfer system
Oral or IV anti-fungals
Itraconazole (sporanox)
Ketoconazole(Nizoral)
Amphorericin B (Abelcet, Fungizone)
Terbinafine (Lamisil)
Griseofulvin
Gentain Violet
Ortolani Sign
1-baby supine, hips and knees flexed at 90 degrees
2-grap baby thigh with middle finger over greater trochanter
and lifting and abducting the thigh while stabilizing the pelvis and opposite leg witht he other hand
+ when a palpable click is felt as the femoral head is made to enter the acetabalum
OSGOOD-SCHLATTER
OSTEOCHONDROSIS OF THE TIBIAL TUBEROSITY
CAUSES BY EXCESSIVE TRACTION OF THE PATELLAR LIGAMENT
SELF-LIMITING
Osteochondritis dissecans
s a condition that develops in joints, most often in children and adolescents. It occurs when a small segment of bone begins to separate from its surrounding region due to a lack of blood supply
**most comon femoral condyle
Other hereditary Disorders
1- Riley-Day Syndrome AKA Familail Dysautonomia- indifference to pain
Ashkenazi jews, high breech presentation//weak absent suck and poor tone
emotional, self mutilation, absent or hyporeflex DTR
2-Rousy-Levy Syndrome-Hereditary arcreflex dystasia
form of frust CMT- pes cavus, Absent DTR,POSITIVE ROMBERG SIGN-(balance with eyes closed)
3-Friedreich Ataxia AKA Spinocerebellar Ataxia- most common inherited ataxias, involving, spinocerebellar and corticospinal tract, post column
D/T protein Frataxin decreased (CNS,heart,pancrease) have abnormal high iron in cardiac tissue>> react with O2 to produce free radicals
Slowly progressive scoliosis in thoracic region
LE Finding- feet cavus, wide unsteady gait..absent DTR, +romberg sign
shin ataxia>>Cardiac failure
4-Dejerine-Sottas-Progressive hypertrophic polyneuritis
paresthersia and lightning-like pain in extremities>>>>wheelchair confined
Clincical-all sensory modalities affected, stocking glove distribution ie: light touch & pinprick>>+Romberg//like CMT slow progressive weakness
reflex diminished
TX-Accomodative
Parkinson Gait
Shuffling with short, hurried steps
Parkinson’s disease
Loss pf dopamine neurons from substantia nigra
55-60 Yo
Classic=rigidity, bradykinesia and disturbance in gait
PILL ROLLING RESTING TREMOR
AKINESIA- LACK OF POVERTY OF MOVEMENT AND COGWHEEL RIGIDITY
FESTINATING GAIT
BRADYKINSIA-SLOWNESS AND FATIGUING OF VOLUNTARY MOVEMENT
ALL SYMPTOMS PROGRESS TO MASKED FACIES
TREATMENT:LEVODOPA COMBO LEVODOPA & CARBIDOPA (Sinemet)
Dopamine agonist, AMANTADIN, Anticholinergics
COMT inhibitor which inc/ entacapone & tolcapone>>breaksdown levodopa in periphery and may increase CNS delivery of dopamine
PASA Corrections
Austin
Reverdin
Reverdin Green
Reverdin Laird
Reverdin Todd
Peabody
Hohman
Drato
Ludloff
Pathologic Reflex responses elucidates
Babinski sign- <2 yo, stropke lateral prox to distal>>>+ extension/dorsiflex of great toe with flex fanning toes
Normal>>In adults the hallux moves up
slow response plantar withdrawal response to that of tickling
Chaddocks Sign_ stroke lateral foot about lateral malleoli moving distally>>extension of hallux
Oppenheim Sign- use thumb and index finger as caliper to squeeze tibial creat>>hallux extends if deficit cause pyramidial tract
Gordon Sign- squeeze posterior calf>>hallux extends and dorsiflexs
Rossolimos Sign- tap balls or distal pulp of toes>>++ if toes Plantar flex
Pedal antiperspirants
Ostiderm roll on
Xerac
Drysol
Bromi-lotion
Dr Scholl’s antiperspirant
Pedricarditis
Sharp and generalized
Varies
Inflammation between the visceral and pericardium
Treatment-Sitting up and forwrd
Pentobarbital
Secobarbital
Sedation>>no analgesia>>RELIEVE APPREHENSION
MINIMAL RESP DEPRESISON
CAUSTION IF PT HAS PORPHYRIA (DECREASE HEME)
Performing a calcaneal osteotomy for PTTD how much translation should occur
1-1.5cm medially
peripheral neuropathies
DANGTHRAPIST
1-Diabetic peripheral neuropathy
2-Alcoholic
3-Nutrional
4-Guillame-Barre Barre
5-Toxic
6-Herditary
7-Reccurrent
8-Amyloidosis
9-Porphyria
10-Infections
11-Systemic
12-Tumors
Peroneal Muscular Atrophy Charcot Marie Tooth Disease
CMT 1- demyelination form, degeneration of post colunm of spinal cord
loss anterior horns and degeneration spinocerebellar tract
Slow nerve conduction velocities
CMT 2-neuronal form by axonal degeneration of peripheral nerve
Male>>>female
Clinical-muscle atrophy symtrical and distal: peroneals and intrinsic muscles of the feet 1st>>lose ability to evert becasue invertors mechanical advantage. Cavus type foot
Early sym varus and high arch with atrophy TA,EHL,EDL>>cavus& clawing
Finding: plumb thigh, slender legs “ostrich or stork” Inverted chanpagne bottle
Perthes Test
Detect deep vein valvular incompetence
Place tounaquet mid thighelevated leg 60mm to occlude superficial venous flow
patient walks to asses muscle pumping function of deep veins
Competent valves- blood flows thru deep veins back to heart
Valve incompetence- blood reflx thru incompetent commicators to superficial and enlarges below tournaquet
If patient feels pain on walking, could be deep venous claudication
Pes planus deformity
Transverse plane-abduction FF on RF
Frontal Plane=RF eversion with decrease 1st met declination angle
Sagittal plane-sagging midfoot-Nav-cuneiform breech//TDA increase
Phases of a 3-phase bone scan
1st immediate and seen in a few seconds
2nd is blood pool phase 2-5 minutes and represents regional blood flow
3rd delayed or static and can take 2-3 hours and represents bone uptake and urinary excretion
Phosphorus
3-4.5
controlled by parathyroid hormone
intracellular
PTH>>Increase Ca>>increae resorption Ca from bone>>suppress loss into urine
Hyper-Tissue trauma(hemolysis), burns, renal failure, healing bone
Hypo-Vomit N&D, Diuretic, starvation
Serum calcium and phosphorus>>>dependant greatly level of PTH
Planal dominence in the STJ
frontal and transverse
1st Plantar layer
Abductor hallucis( medial plantar Nerve)
Flexor digitorum brevis( Medial plantar nerve)
Abductor digital minimi(Lateral plantar nerve)

PLEURISY
Sharp pain associated with respiration
Location-Generalized
Time>15 minutes
Causes-can be seen-pneumonia
Treatment-Control infection
Polyarteritis Nodosa
Polyarteritis nodosa (PAN), is a systemic necrotizing inflammation of blood vessels (vasculitis) affecting medium-sized muscular arteries, typically involving the arteries of the kidneys and other internal organs but generally sparing the lungs’ circulation
Polymyositis
Polymyositis is a type of muscle disease called an inflammatory myopathy. It inflames your muscles and their related tissues, like the blood vessels that supply them. It can cause muscle weakness
Eventually, people with polymyositis have trouble when rising from a sitting position, climbing stairs, lifting objects, or reaching overhead.
PONSETI SERIAL CASTING
serial casting weekly
percutaneous tendoachiles tenotomy
once corrected abduction foot orthotics 12 weeks
then at night until age 4
Complication= metatarsal adductus
heel varus
pes plano-valgus
rockerbottom foot
AVN or talar head flatening because infant connetive tissue stronger then bone
Navicular subluxation
post op complication using autologous iliac bone graph
pain
hemorrahage
hernia
Posterior column
Vibratory
sensation
proprioception
Posterior, medial and lateral release for club foot 1 year
POSTERIOR
reflic origin abductor hallacis and plantar fascia
z-plasty achilles tendon
release posterior , medial and lateral ankle joint
release posterior, medial and lateral STJ,–posterior talofib and calcfib lgts are severed
MEDIAL RELIEASE
Z-PLASTY OF PTT, SO SPRING LGY AND HENRY KNOT IS SEVERED
release entire medial STJ, inc/ superficial deltoid
Lateral release thruSTJ
release interosseous talocalc lgt
release bifurcate lgt and release lateral STJ
Posterior shin splint cause pain
3-12 cm above the medial malleolus
Potassium
3.5-5.5
Major intracellular and only 2% total body K is extracellular
Decreased--lead to arrythemia, depressed T wave
High-N&V, dehydration, Mulpiple myeloma and malignancy
Increased and decrease>>profound effect NMS>>apathy, weakness,paralysis,MI
When hypo>> exam serum bicarbonate
Pre albulmin
Distinct marker for protein synthesis
Pre empative analgesia involves preventing spinal cord windup by
using pain killers and nerve blocks pre-op to decrease pain post op
Pre op medication Class
Barbituates- Pentobarbital(nembutal)
Secobarbital (seconal)
Benzodiazpines/Sedative hynotic Diazepam (valium)
Lorazepam(ativan)
Midazolam (versed)
Narcotic/Opiods Morphine
Meperidine (demerol) 1/10morphine
Fentanyl (100x more potent then morphine
Anticholinergic(belladona derivative) Atrophine sulfate
(inhib parasympath-blocks ACY) Scopolamine
Glycopyrrolate
Pre-anesthesthic Medication
1-Sedatives- (anti-anxiety/hyponotic)
Diazepams (valium)–sedation and amnesia. Can be given PO 1-2 hr before Sx(5-10mg)
Lorazepam (ativan) 1-2 hours pre surgery 2-4mg
Midazolam (versed) pre-op +/or induction agent. Rapid onset with short duration
when IV works w/n 5 minutes. effect last between 1-6 hours
Hydroxyzine (vistaril) sedative primary, also anti-histaminic, antiemetic and bronchodilator. Good premed
2-Anticholinergic (Belladonna)-
Atropine-before anesthesia to decrease resperation secretion. Keep heart rate normal during.
prevents asystolic and bradycardia in presence of halothane and vagolytic agents
Scopolamine-Used as a preanesthetic because it produces mild respiratory stimulation and it inhibits salivary secretion. In reversing paralysis it is used to reduce parasympathetic hyperactivity.
Preanesthetic Agents principle
Purpose:
1-Reduce anxiety
2-Produce some sedation/amnesia
3- Reduce gastric PH as well as volume
4-Reduce bronchial secretion
5-Prevent N&V
Principles Lower Extremity Nerves Lower Plexus
1-Lateral fenoral cutaneous nerve L2,L3 SENSORY
1st sensory nerve of lumar plexus- supplies skin lateral/anterior thigh, Posterior branch>>lat/posterior thigh
2-Femoral Nerve L2, L3 &L4 SENSORY AND MUSCULAR-exitsvertebral canal>>inervates psoas & iliacus
Sensory branches>>ant/medial thigh medial calf
Muscular branch>>pectineus,sartarius and quad femoris
Movements:Iliopsoas>>hip flexion>>flexion of thigh:quad femoris>>>extend of leg at knee
3-Saphenous N- largest and longest sensory branch femoral nerve>>innervate skin medial thigh, leg and foot
course>>accompanies femoral AA in femoral triangle, descends and divides medially under sartorius muscle>>terminal with greater saphenous VV>>medial calf at medial malleolus to medial foot>>1st MPJ
Entrapped as it exit subsartorial canal (Hunter’s canal) proximal to knee>>no motor weakness b/c its sensory
4-Obturator Nerve (L2,L3,L4) SENSORY AND MOTOR
Anterior branch>adductor longus, brevis and greacilis
Posterior branch-obturator externus and 1/2 adductus magnus
Sensory fibers-cutaneous sensation upper medial thigh & anastomose with Saphenous N
Movement>>adductor longus,brevis and gracilis (INTERNAL ROTATES LEG>>>adduct thigh
Obturator externus>>external rotates thigh & 1/2 adductor magnus muscle>>adduct, flex&extend thigh
Proper treatment of a stage 3 PTTD would be:
Triple Arthrodesis
Protein
6-8
Albumin is a protein in liver that halps maintain normal distribution of water in body and transport blood constituents
Total blood mostly refers to Albumin 50-60%
Non-Albumins is referred to a globulin
A/G ratio diagnosis condition multiple myeloma (Bence-Jones Pr)
Increased= elevation of globulins as collagen Vascular DZ
Total Protein= 3gm globulin + 4gm Albumin====7 gm total protein
Proximal Tarsal Tunnel syndrome
Entrapment medial and lateral branch ribial N, under lacinate Lgt (flex retinaculum)
Anatomy NVS passes thru lacinate lgt
Vascular-med & post tibial N well supplied with AA. Regenerate well and susceptible ischemic injuryor aa insuffiiency>>>TARSAL TUNNEL SYNDROME
BIOMECHANICAL-compression medial & lateral plantar Nas they enter foot/seen with enlarged ABH bellies or osteophytic spurring of medial arch>>pronation syndrome>>extreme PF & adduction of talus during ambulation
enlarged navicular tuberosities D/T osseous impingement of insertion PTT
Clinical_ Buring, tireness in foot, pain totates proximally==VALLEIX PHENOMENON
REPRODUCEWITH PERCUSSION OF PTT NERVE AT LEVEL OF LACINATE LGT
DX: ELECTRO-DX distal latency (millisec) greater latency value
distal latency medial N abnormal>>6.1
lateral plantar nerve abnormal>>6.7
PTN stim just proximal to lacinate lgt
ABH muscle for Medial plantar Nerve and Abductor digit quinti to test Lateral PLantar N
PT
11-16 seconds
measure extrinsic
Increased Vit K def//Biliary obstruction//Liver disease//Coumadin tx//deficiency in extrinsic or common pathway factors
PTT
25-35 # of seconds to clot
Intrinsic pathway–M
3 stages of coagulation except factor VII
Increased- Hemophilia A factor VIII
Hemophilia B Christmas tree ( Factor IX
Von Willebrand DZ
DIC Deficiency in intrinsic pr common pathway factors
Liver DZ Heparin TX
RADICULOPATHY
PATHWAY TO SPINAL NERVE ROOTS AS EXIT SPINAL SOLUMN THRU VERTEBRAL FORAMINA
DISC HERNIATION, DEGENERATIVE JOINT DISEASE AND OTHER ARTHRITIEED AFFECTING THE SPINE
ACUTE TRAUMA
Radio Frequency
high frequency electromagnetic radiation due to the use of wireless equipment, devices and data transmission.
Radiograph congrenital vertical talus
Lateral view-witha second the foot plantarflexed showing the talo-navicular relationship doesn’t change
Navicular not evident on x-ray until age 3
line bisecting the talus is parrallet to the tibia
TC angle on A/P is increased to >>40 degrees
Talar neck hypoplastic may have hourglass shape and flat surface
Navicular articulates witht he dorsal talus
Negative hubscher maneuver

Raynauds phenomenon VS Syndrome VS Disease
Phenomenon is episodic digial palloring with paresthesias, followed by cyanosis and finally presenting with rubor and warmth with thrombing sensation
Syndrome- repeated presentation of phenomenon under coldness or anxiety and associated with collagen disorder
Disease-trilogy of colors ass/ symptoms noted for 2 years without definition of underlying collagen disease
RBC
Male 5.4-
Female 4.8
Provides info about HGB content and size of RBC
Mean Corpuscular Vol= Normocytic -82-92//Macrocytic//95-150//Miccocytic-50-80
Mean Corpuscular HGB= Normocytic-25-30//Macrocytic//30-50//Microcytic 12-25
Mean Corpuscular HGB Concentration= Normcytic 32-36/Macrocytic-32-36//Microcytic 25-30
Microcytic Anemia<<<<80 is iron deficiency, Thalasemia, blood loss, poisoning
Normocytic Anemia-(80-100) isChronic DZ, Bone marrow failure,,hemolysis
Macrocytic Anemia(>>>100 is Folate acid & Vit B12 def, Liver DZ, drugs like phenytoin
Red man syndrome
infusion-related reaction peculiar to vancomycin [3]. It typically consists of pruritus, an erythematous rash that involves the face, neck, and upper torso. Less frequently, hypotension and angioedema can occur.
Refsum’s disease:
(hereditary motor sensory neuropathy type IV, heredopathia atactica polyneuritiformis) is an autosomal recessive disorder the clinical features of which include retinitis pigmentosa, blindness, anosmia, deafness, sensory neuropathy, ataxia and accumulation of phytanic acid in plasma- and lipid-
Renandier DZ
Osteochondritis of the tibia sesamoid
Reynaulds Number
Diameter X VelocityXDensity/Viscosity
is the ratio of inertial forces to viscous forces within a fluid which is subjected to relative internal movement due to different fluid velocities. A region where these forces change behavior is known as a boundary layer, such as the bounding surface in the interior of a pipe.
Rheumatoid Disease Labs
ESR
CRP
ANA
Uric acid
ErythrocyteSedimentation Rate ESR
RBC settle out of unclotted bld
Increased>>> Infection, R.A Maignancy..A.S.Acute gout
Inflammation, Septic OA metastasis
C-Reactive Protein <
Rises before ESR and normal after NSAID,ASA Steroid
produced during acute imflam phase
Increased RA, Strep Infection
ANA
appears month after connective tissue DZ
More accurate then LE Cell test>>unaffected by steroids
Lupus.99///Scleroderma>>73 RZ>>60…Sjogrens.43
Uric acid 7-9 males
Increased in gout, malignancy, renal DZ, familial hyperuricemia
MARTINI SIGN–MONOSODIUM URATE CRYTALS NEEDLE SHAPE
Ritter DZ
osteochondritis of the fibular head
Rowe Calcaneal Type 1
1A- Fx of the medial tuberosity
1b-Fx of the sustentaculum tali
1C-Fx of the anterior process -mc of avulsion fx of the bificate ligament
Rowe Type 2
2A- Beak fx no ACH involvement
2B-Avulsion tx of Tendo achilles
NO INVOLDE THE STJ
Rowe 3
Type 3-extraarticular oblique fx of calcaneal body not involving STJ
Rowe classification 4
Type 4-Fx involving STJ w/o joint depression or comminution

Rowe Class Type 5
Type 5-comminuted fx of STJ w/ central or severe depression
Rowe Classification 1-II
Type Ia - tuberosity fx; beak/medial avulsion fx, vertical/horizontal body fx. (Rowe types I and II) Type Ib - calcaneo-cuboid joint involvement Type IIa - undisplaced STJ fx, secondary fx line exits posteriorly through calcaneus. Type IIb - displaced STJ fx, secondary fx line exits dorsally through calcaneus and a fragment dislocates.
Rule of Simon
Talo–1st met angle> 15 & talo-calc angle < 15= TN dislocation
Ryders test
a femoral torsion angle of 10 degree in a 6
year old female.
Sacral Plexus Nerves
Superior L4,L5, S1 (Muscular)- innervate gluteus medius, minimus and TFL
Inferior Gluteal L5,S1,S2-gluteus maximus
Superior gluteal Abducts and internal rotation thigh
Inferior N>>extension,abduction and external rotation of thigh
Sciatic Nerve-L4,L5,S1,S2> MOTOR>>peroneal component short head bicep femoris and portion add magnus, tibial branch to post thigh, hamstrings>>>>SCIATICA>>>PAIN DOWN LOWER LIMB
TIBIAL NERVE-(POST TIBIAL) sensory & motor- bifurcates w/n popliteal fossa>>medial popliteal N>>tibial
motor innerv muscle superficial/deep sural muscle (grastroc/soleus) superior 1/2 motor to deep post group :ie Tibial posterior, FHL,FDL enters foot between medial malleolus and flex retinaculum under the reticulum splits to>>>medial and lateral plantar Nerves after giving off 1st small calcenal branch
Medial Plantar N>>with medial plantar AA>>motor 4 muscles (LAFF), ANH,FDB,FHB,1st Lumbrical
Cutaneous innv medial distal 2/3 plantar foot, plantar digits 1-3 & medial 4th digit
Lateral plantar Nerve- lateral to heel and lateral plantar surface
innerv>>all other intrinsics &motor innerv sensory lateral aspect of sole & lateral 4th & entire 5th toe
Sagittal plane 1st MPJ motion occurs about which axis
transverse
Sagittal Plane correction for FF deformity
Sagittal plane correction for Pes planus
Lowman- Plantarflex T-N wedge arthrodesis with TAL
and TA tendon rerouted under the navicular
Cotton-Open dorsal wedge 1st cuneiform
transplanting TPT under the navicular
Hoke-Plantar based wedge arthrdesis of the nav-& medial cuneiform
perform TAL
Miller-Nav-1st cuneiform and 1st metarsal fusion
PTT and spring Lgt advanced using an odteoperiosteal flap
Young (keyhole technique)- reroute the TA tendon thu keyhole in the navicular
Salter Harris
S.A.L.T. ER
Salter Harris Functure
S.A.L.T.ER

Samuels Test
Elevate LE, arteriole rubor to palor pedal skin, while venogenic dependent rubor convert to normal coloration
Sanders CT classification of calcaneal fracture is bases on
number and position of the posterior facet fracture line on a coronal section
Saphenous Nerve
Branch of the femoral
Parallels the great saphaneous vein
Gives off branches to the medial crural nerve

Saphenous nerve
from the femoral and it is the only branch
branches to the medial crural nerves’

Screws
Cortical-fully threaded
fine threads with small pitch to anchor the cortical bone
cancellous-partially threaded (historically), usually lag. threads are coarser with hugh pitch inted to engage medullary bone

Rheumatoid Labs continue
Serum Compliment
Anti-Streptilysin ASO
HL A-B27
HL A-B15
Calcium Pyrophosphate
Series of enzymatic protein cobine antigen-antibody complex and effect lysis when antigen is an intact cell
ASO-Anti-streptilysin-AB against streptolysin O and group A Strep
80% acuteR>F> or other strep infection
HL-A_B27-90% A.S>//Reiters 75%//P.A &Juveline RA
HL.A B15-33%. in SLE
Calcium Pyrophosphate- crystals are rhomboid
Sever DZ
Osteochondrosis of the calcaneus
cause by excellive traction of the achilles tendon
6-12 years old
more common with equinus
radiograph are difficult
TX- Rice NSAIDS, eliminate sports, heel lift, achilles stretch exercise
SGOT/AST
10-50
When heart or liver damage, SGOT spills into blood and the amount is directly related to the damage cells
More specific for cardiac necrosis
INCREASED- MI,Liver(HEPATITIS) disease DZ, Skeltetal muscle, after strenous exercise
DECREASED-Uncontrolled DM, Beriberi
Bone disease wont cause elevation AST
SGPT/ALT
10-50mu
High in hepatitis and after strenous exercise
Found high concentration in liver>kidney.>heart
catylyzes amino group between alanine & alpha ketoglut
Bone disease wont cause elevated levels of ALT
Shenton curved line
line from femeral neck to obturator foramen
witrh hip dislocation, obturator foramen is too low
shoe anatomy

Silk
weakest
Simons Assesment Method
Simon rule of 15
talus, calcaneus and 1st metatarsal longitudinallt bisect on A/P xray in max corrected position
clubfoot talo-calc angle <<15
Talo-1st metatarsal angle >>15
Skewfoot
1-Adducted forefoot
2-normal midfoot
3-valgus hindfoot
4-Increase calc-cuboid angle (norm 0-5)
usually acquired from comp of a metatarsal varus, develops with wt. bearing or
inproper manipulation and casting
slipped capital femoral epiphysis
A 12 year old obese male who was sitting with his left
leg externally rotated rises and begins to walk. His gait
remains severely externally rotated on the left side with a
trunk-shift limp to the right side.
Snappen Fracture 1
group 1-
Transchondral or compression FX to talar dome including osteochondritis dessicans
Sneppen Fracture Group 2 Talus
Coronal, sagital or horizontal shearing fracture of entire talar body
Cause-severe DF with compression forces when talus is sandwiched b/t tibia & calcaneus
Type 1 Coronal or sagittal fx-
1a-non-displaced
1B-displace of trochlear articular surface
1C-displacement of trochlear art surface with STJ discloca
1D-total displacement of talar body with Displace STJ/AJ
TYPE 2 horizontal FX
2a-non-displaced fx dividing talar body into sup/inf halve
2b-displaced fx superior portion shifts on inf halve
Sneppen 3
fracture posterior tubercle(most common)
d/t severe plantarflex force (DDX steida process or shepaers fx, os trigonum)
pain posterior ankle causes limited rom
reproduced on FHL movement
Sneppen group 4
Fx lateral process of talus
2nd most common talar body fracture
AKA Snowboard FX eversion injury with lateral process caught b/t fibula and calcaneus

Sneppen Fracture 5
Crush Fx
Comminuted of talar body
Sodium
NA 135-145
Major cation in extracellular fluid
maintains osmotic PP in acid-base balance
Low-Dehydration, cushings Dz
High-Severe burns, N&V, excess IV, Addison DZ/ CHF
Convulsion&seizures>>Traid of low NA, Low HCT, Low BUN
ADH-Arginine vasopressin-tells kidneys how much water to conserve
Soft tissue lateral view of ankle

sphenoid bone shape
wedge shape
Spinal anesthesia
Both, requires lower volume of anesthetic agent than epidural anesthesia and difficult to control the level of anesthesia are correct.
Spinal Bifida Occulta
Incomplete closure of 1 or more vertebral arches only
L5 &S1
SPINAL CORD EVUALTION OF RADICUPATHY
NERI’S SIGN-TAKES SMALL STEPS WITH KNEE SEMI-FLEX TO PREVENT STRETCHING OF N ROOT
Minors Sign- patient rises from seated position, pt places wt on unaffected side with one hand on his back
Lumbar lordosis or thopracic kyphosis
Pseudoclaudication-relieved by flexing spine
Straight leg raise test- supine and hip passively flex with knee in full extension, should be non painful
Lasegues Test-reproduce pain when patinet leg is elevated less then 30 degree and foot DF
Bowstring-to differentiate lumbosacral ilian pain–patient supine in full extension, if nerve pain when knee is flexed pain goes away
Gaenslens Test- DDX lumbosacral from sacral ilian pain- patient supine with 1 leg in full extension and the other is lowered off the sideof the table, the twisting of the pelvis causes sacro-iliac pain
Valsalva Manuever-DDX space occupying lesion or herniated disc; b/l compression of jujular vn results in increase intra spinal PP and reproduces radicular pain
EMG Studies
STANDARD AFO
Ankle set 90 degree
various neuromuscular disorder may cause equinus
STJ AXIS ROM
Avg axis position to sagittal plane= 16 degrees
Avg axis position to the transverse plane = 42 degrees
summary sequence of ischemic necrosis
infARCTION
resorption
revascularization
remodeling
Sunderland Classification
1-Conduction eficit without axonal destruction
2- Axon is severed without reaching neural tube.(Wallerian degeneration) with regeneration
3-Degeneration of axon with destruction of fascicle with irregular regeneration
4-Destruction of axon and fascicle and no destruction of nerve truck
5- Complete loss, neuroma likely and spontaneous recovery is rare
Superficial Nerve
Common peroneal nerve within the tunnel of the P.L and neck of the fibula
upper part innervate the P.L. and PB
supplies most of anterior skin to ankle and dorsum

SUPERFICIAL PERONEAL
AKA Musclulo-cutaneous N (SENSORY. MUSCULAR
INNERVATED PL AND PB>>EVERT AND PF
DESCENDS INF BETWEEN PERONEAL MUSCLES>>INFERIOR INTO MEDIAl and INTERMEDIATE DORSAL CUTANEOUS NERVE (SENSORY BRANCHES>>ANT AND EXT RETINCULUM SUPPLY ANT-LAT ASPECT OF LOWER 1/2 LEG AND DORSUM OF FOOT & TOE MOST COMMON NERVE INJURY TO FOOT
injury and entrapement-exits the fascia w/c the calf approx 10cm about the lateral malleolus. Intermediate dorsal cutaneous N (LAMONTS N) couring just medial to the sinus tarsi; severe inversion injuries>>>>stretched Nerve & injury
DX-1- LOCAL INFILTRATE REPRODUCES TO CONFIRM WITH FOOT DORSAL FLEX AND EVERT AGAINST RESISTANT
DX 2- PASSIVE FOOT PF ABND INVERSION PLACING NERVE ON STRETCH
DX 3- DIRECT PERCUSSION FOOT HELP PF AND INVERSION
2-
Superficial Peroneal Nerve
Divides into (2)medial and intermediate (1) dorsal cutaneous nerve
medial dorsal cutaneous nerve divides into 2 dorsal digital Nerve-1-Medial proper dorsal digital nerve to the hallux(skin)
2-Common dorsal digital nerve-innervates skin 2nd & 3rd toe
3-Intermediate dorsal cutaneous nerve-splits into 2 digits nerve 3rd & 4th IDS

SURAL NERVE
sensory
From medial sural nerve (tibial) and sural commincating branch (common peroneal)
orgnates from inferior popliteal fossa b/t bellies gatroc>>inf & distally,post fibular malleolus
Cutaneous Innervation>>post-lateral distal leg/ lateral aspect of foot
Terminal branches..divides lateral and medial>medial supplies cutaneous innervation to dorsal skin of base 4th metatarsal; communicated with intermediate dorsal cutaneous nerve
Terminal branch of the sural nerve most accessible sensory nerve of the foot and NERVE OF CHOICE>BIOPSY
INJURY AND ENTRAPMENT-ANKLE IS MINIMAL SENSORY DEFICIT
Sural Nerve Anatomy
Strictly cutaneous
Forms in leg by combining branches tibial and common peroneal Nerve
Medial sural cutaneous branch is branch of tibial nerve
Sural comminicating branchof the peroneal N at fibula head
As Sural enter the foot>>lateral dorsal cuntaneous nerve0

surgical treatment club foot children 3-12 months
1- Medial hockey stick
2-Cincinnati incision
suture materials is the most
reactive
natural fibers
Sydenham’s Chorea
BASAL GANGLIA DISORDER
Major Dx R.F.
Can occur up to 6 months following Group A beta-hemolytic strep infection
Indication to intiate prophylactic antibiotic therapy to prevent subq development of other manisfestation of RF
Clinical-aimless involuntary movement, impaired coordination and muscular weakness with reduced muscle tone. Movements are worse when trying to repress
Treatment-Phenothiazine or haloperidol can control the chorea
anti-microbial tx Penicillin or erythromycin if pen allergy
Usually resolves 3-6 weeks
Syndesmotic screw: For fracture
- Material
- Size
- number of cortices
- When to remove
- Side effects
- Material: no difference between stainless steel and titanium
- Size: No difference between 3.5 and 4.5. 4.5 easier to remove but also causes greater irritation
- number of cortices: 1 screw across 4 cortices will have higher chance of fracture. 2 screws across 3 cortices have better stability, better physiological movement
- When to remove: at 3-4 months
- Side effects: limitation of ankle ROM, broken screw, pain , screw removal, syndesmosis diastasis.
talar neck fracture most stable in vitro using
6.5 mm screw & K-wire posterior to anterior
Talipes Equinus varus can be subdivided
Idiopathic, Positional, Syndromic,Terotologic
Talo-navicular coalition
2%
usally asyptomatic
if painful usually around 3-5 y.o.
CC: bump pain from shoe pressure
Lateral x-ray shows absence of Cyma line
TX- correct medial prominence
Talocalcaneal Coalition
45% and almost all the middle facet
starts around 12-14 y.o.
pain usually over the sinus tarsi
decrease ROM STJ and MTJ
X-RAY- Lateral and Harris-beath best views
TC on lateral C sign or halo sign
(this C chape is a line formed from medial outline of talar dome and inf outline of Sust tali.
thiis C shape is specific
May be absent STJ middle facet and diminished facet
Talar beaking
Medial oblique- shortening talar neck/ dysmorphic sust tali (or ovoid shape)
Talus in ankle mortise more rounded (ball and socket)

Tarsal Coalition
abnormal bridge bewteen tarsal bones
1% AND 50% ARE BILATERAL
most common cause of peroneal spastic flatfoot, secondary to spasm of STJ
Males>>>females
TC=CN and account for over 90%
CT scan is gold standard
TX- conservative by decrease motion of inv/ joints with shoe modification or braces,
casting , splints, RICE & NSAIDS
Tarsal tunnel
Flexor retinaculum medial/posterior
calcaneus and posterior aspect of the talus
Distal tibia and medial malleolus-anteriorly
Tc-99m scans HMPAO
used to detect a wide range of conditions including injuries, infections, tumours, heart disease, thyroid abnormalities, kidney conditions and also to guide some cancer procedures.
HMPAO-hexylmethyl propylene amids Oxide
1/2 life is 6 hours
Tendon healing
Week one is characterized by edema, erythema, and
callus formation.
b. During week two, vascularity and fibroblastic
proliferation continue.
c. Gentle range of motion exercises starting in week 3
will lead to increased strength of the tendon.
d. Passive range of motion exercises during week 4
helps realign the collagen fibers to allow optimum strength
Test determine flexible Flatfoot
Hubcheur Manuever-PF 1st ray will cause STJ to supinate and re-create arch if flex
Trunk Twist- STJ flexibility for supination and pronation
Jack test-Pt on heels, heels will invert, check the PT tendon
Tetanus
caused by toxin Clostridium tetani–affect CNS
Spore>>release bacteria>>multiply and produce>>neurotin>> Tetanospasmin
The achilles tendon consist of
paratenon, epitenon
The first ray is
Uniaxial, triplanar not pronatory/supinatory
The research design
is a broad framework that describes how the entire research project is carried out. Basically, there can be three types of research designs – exploratory research design, descriptive research design, and experimental (or causal) research design.
The windlass effect refers to
Plantarflexion of the 1st metatarsal during 1st MTP dorsiflexion
durometric Thickness of plastozoate
35
Thiemann DZ
Osteochondritis epiphyseal ossification center of the phalanges
Thompson Test
Rupture of the Achilles tendon
Tietze
Sharp, Costochondritis, tender at pressure points
Location-Chest wall
Time>days to weeks
Causes>Inflam of costoconfral joints
Treatment-Benign, assoc with aniety
Tillaux-Chaput definition
avulsion fracture of the tibia from the AITFL
Toe pressure diabetic healing
>55mm Hg Healing
45-55 Hg Range of uncertainty
<45 No wound healing
NEED AT LEAST 30MM HG REQUIRED FOR HEALINH OF A WOUND ON THE DIGIT
Topical Fungals for nails
Efinaconazle (jublia)
tavaborale (Kerydin)
Ciclopirox (penlac)
Tolfanate (Formula 3)
Underclenic (Tineacide)
Tolcylen
Torus fracture
Common pediatric fracture at the transitional zone between the metaphysis and diaphysis
TOTAL SERUM PROTEIN
6-8
Formed in liver to maintain normal distribution of water ( colloidal osmotic pp) and transport bld constiuent
Usually referred to Albumin because bld protein 60% albumin
Non-albulmin referred to a globulin A?G ratio
A/G ratio diagnosis multiple myeloma/(Bence-Jones protein is a globulin)
Increased-Hyperproteinmia,N&V,Mutiple myeloma/Malignancy
Decreased-Hypoproteinemia//chronic liver DZ//Severe burns
Total protein=3 gram%globulin+4gm%albumin===7gm total
Toxic neuropathy (drugs)
dangThrapist
Heart and HBP-Amiodarone Hydralazine perhexilline
Fight cancer: Vincristine, Cisplatin
Infetions:Metronidazole (flagyl) Nitrifurantoin, Thalidomide (leprosy)
IND (isoniazid),(TB)
Skin conditions-Dapsone
Antoconvulsants -Phenytoin
Anti drugs and alcohol-Disulfram
Fight HIV-Zisovudine(retrovir,AZT),Didanosine (Videx) Stavudine (Zerit)
Zalcitabine (Hivid) Ritonavir (norvir), Amprenavir (agenerase)
Toxic neuropathy Environment
1-Arsenic-cause whitr striae on nail>>Mees band
TX-BAL chelating agent
2-Barium-can lower lserum K>>clinical paraylsis
3-Lead-vasculopathy-edematous fluid surrounds abnorm permeable capillary
S/S convulsice seizures
4-Mercury-3 forms 1- Inorganic salts and 2 metallic>> S/S=neurological comp Mad Hatter Syndrome include tremors, personality tremor of the limb
3-Organomercurial exposure>>paresthesia limbs, visual, dysarthria tremor, incordination large expsure could be permanent
TX:BAL and EDTA for chelation
5-Lithium
6-Manganese - rigidity and hypokinsia can manage with L-dopa
7-Urate
Transverse plane correction of flatfoot
Evans-open osteotomy of calc 1.5cm to CCjoint with insertion of bone graph
Kidner-remove prominent nav tuberosity and transplant TPT underside of navicular
Transverse plane correction of pes planus
Evan’s-calcaneal osteotomy 1.5cm proximal to the C-C jt with insert bone graph to lengthen lateral column and put PT on stretch to increease supinatory motion
Kidner-removal prominant tuberosity(acc navicular) transpose PT plantar
C-C Joint distraction arthrodesis
transyndesmotic screw is:
Inserted posterior lateral to anterior medial,
inserted 2-3 cm above the syndesmosis,
and inserted with the foot neutral to slightly
dorsiflexed
Treatment of choice for type II-IV talar neck FX
Immediate ORIF and Closed reduction followed by
ORIF when patient is stable
Treatment of dislocated 5th digit
Increase the deformity, distract the toe, then
reduce the deformity.
Treatment of DM Neuropathy
Rigid glucose control
Superfical Nerve pain-Capaicin
Deep Nerve pain-tricyclic anti depressants (amytriptyllene) or anti seizure (gabapentin)
Tegretol (anti-seizure)
Gabapentin-(post-herpectic neuroligia req titration)
OD- dizzyness and Drousy
Neurontin-900mg///300mg//600>>>900mg
Amytrptiline if neurontin doesn’t work
Treatment of pilon fracture
a. Restoration of fibula length
b. Anatomic reduction of articular defect
c. Bone grafting of metaphyseal defect
d. Medial tibial buttress plating
e. All of the answers are correct
Trendelenburg Maneuver
DDX deep and superficial venous incompetence
Elevate leg to empty venous
Tounaquet upper thigh 30-60mm Hg to occlude superficial venous flow
Then stand-if varicosities fill within 20-30 seconds>>deep and perforation disease is present
If varicositie promp return>>>superficial system
Treve DZ
Osteochondritis of the fibular sesamoid
tumor of the bone marrow
myeloma
Twister cables
cables around waist
inside pant leg
CONTROLS DEGREE OF ABDUCTION AT HEEL CONTACT
TREAT SCISSOR GAIT OF CP PATIENTS

Type II navicular body fracture
Axial compression fracture with an oblique
pattern from dorsal-lateral to plantar-medial
Types of Anestheisa Inhalation
Inhalation agents
Chloroform-rapaid induction/recovery, nonflammable, good muscle relaxation
Disadvant>not in use, myocardial depresion, hepatoxic
Diethyl ether- reliable anesthesia depth-resp stimulated, bronchodilator, circ not depressed, goof muscle relax
Disadv>not in use-prolonged induction, flammable and explosive
N.O.- little effect of HR myocardial contractility,resp,BP, liver or kidney metabolism
VERY HIGH MAC
DISADV> LEAST POTENT 100%> MUST BE GIVEN AT ERMINATION OF SX TO PREVENT DIFFUSION HYPOXIA> no muscle relaxation, ?? bone marrow depression, fatal agranulocytosis
Unibar
Same as denis brown bar except has ball and socket joint beneath each foot
tighten for varus position (preventing STJ and MTJ subluxation) elimninating the need to bend the bar

UPPER MOTOR NEURONS
BRAIN
MOTOR COMPONANT OF CNS
PYRAMIDAL AND EXTRAPYRAMDIAL
SYMPTOMS USUALLY RIGID
muscle hypotonia and flaccid
Lou Gehrig’s disease (amyotrophic lateral sclerosis, or ALS)
Primary lateral sclerosis (PLS)
Traumatic brain injury.
Spinal cord injury.
Multiple sclerosis.
Stroke.
Huntington’s disease.
URIC ACID
1.5-7mg
End product of purine-not useful indicator of renal failure
U.A levels most common used to evaluate renal fsilure, gout and leukemia
ELEVATED-Renal failure
Gout
Leukemia
Alcoholism
Lead Poisoning
Decreased-uricosuric agents, Fanconi sign/ Wlson DZ
Uric Acid Gout
Mostly in males
During a gouty attack levels are normal
90% acute gout>>>>>>1st MPJ (Podagra)
2) Soft tissue
3) Ankle joint
Tophi in ears>>B/C lowerTemp>>>UA less soluable
Solubility of UA= 37C -6.8>>30% 4.5>>Ankle Jt 29C Crystalization
Virchow Triad
1-Stasis (arrythmias, MI,CHF,heart failure, immobiliation, besity , varicose veins, dehydration
2-Blood vessel injury (trauma, fractures)
3-Hypercoagulability (neoplasm, oral contraceptives,pregnancy,surgery,polycythemia
these are the 3 stages of formation of thombi
Volkmann definition
avulsion fracture of the tibia from the PITFL
Von Gierke disease
condition in which the body cannot break down glycogen. Glycogen is a form of sugar (glucose) that is stored in the liver and muscles. It is normally broken down into glucose to give you more energy when you need it. Von Gierke disease is also called Type I glycogen storage disease (GSD I).
Von Rosen View (frog leg view)
dislocated hip, the line bisects the ASIS
Wagstaffe definition
avulsion fracture of the fibula from the AITFL
Watson & Jones type 2
Dorsal avulsion lip fracture (most common) 40%
Plantarflexion & forced inversions causes avulsion
Fx via dorsal TN lgt
plantarflexion-eversion causes avulsion via dorsal TN LGT
TX short leg cast immboile 6-8 weeks
Watson & Jones 3
Transverse tracture of body (29%) with dorsal fragment dislocation vertical or horizontal
from fall from height
longitudinal force along the ray when ankle PF
Horizontal plane FX-long dorsal deformity
Watson & Jones 4
Stress FX of middle 1/3 in young atheletes
Differentiate from overuse syndrome
Watson & Jones Type 1
Tuberosity fracture (24%) forceful eversion
avulsion fracture from the PT tendon
R/O tibiale externumor MTJ subluxation
TX short leg cast NWB 4-6 weeks
WBC
5,000-10,000
Leukocytosis in acute infection, metabolic acidosis, gout, heavy metal toxin, tissue necrosis, burns gangrene and inflammation
Leukopenia- decrease reduction of W.C in blood>>>various DZ,, adverse RX to druga and certain severe infection
What antibiotic is contraindicated in pts with CHF
Timentin
What antibiotic results in increased CPK levels
Daptomycin
What antibiotics should be used to treat MRSA
- Clindamycin
- Bactrim
- Linezolid
- Tetracycline
What antibiotics should be used to treat pseudomonas
- Fortaz
- Aminoglycosides
- Timentin
- Ciprofloxacin
- Imipenem
- Atreonam
- Zosyn
What are hammertoe soft tissue procedures (4)
- Flexor or extensor percutaneous tenotomy
- Extensor tendon lengthening, Z-lengthening
- Capsulotomy
- Flexor tendon transfer
What are some distal metatarsal procedures that can be done for metatarsalgia
Weil-cut distal dorsal to proximal plantar
Complications of Weil-
- Transfer lesions
- –recurrence
- –floating toe/flail toe
- –Dorsal contracture
- *Weil+plantar plate repair**
- Jacoby-V cut to PF or DF
- Duvries- Plantar condylectomy on both sides of the joint
- Chevron-removal of bone to shorten the met
- DFWO-same as the waterman
What are some of the soft tissue procedures that can be done for overlapping 5th toe
- Incision from Distal Medial to Proximal Laterl
- Z-plasty or V-Y pasty
- tendon lengthening
What are the 3 axis of motion
Just as there are three planes of motion, there are three axes of rotation: the anterior-posterior axis, the mediolateral axis, and the longitudinal axis. Joints rotate in these axes, allowing movement to occur in the planes.
What are the 3 phases of soft tissue healing?
When do they occur?
Lasts until when?
- Inflammatory – first 48-72 hours (up to 10 days)
- Proliferative – from ~day 3 (lasts 3 - 6 wks)
- Remodeling – from ~day 9 (lasts 6 wks to 12 mths)
What are the 3 ways that a wound vac works
1) To get rid of stagnant bacteria
2) Promote new blood flow to the area with growth factors and neutrophils
3) Allows for even growth of the wound bed, and keeps callouses from forming
What are the 4 different responses in the inflammatory phase?
It begins with disruption of normal tissue physiology
It ends with complete removal of the wound debris
Initial Vasoconstriction – with the role of limiting blood loss
Followed by vasodilation
–Hemostatic response
–Vascular response
–Cellular response
–Immune response
What are the arteries that provide intraosseous circulation 5th metatarsal
1) periosteal plexus
2) nutrient artery
3) metaphyseal/epiphyseal arteries
What are the classification systems for pilon fractures
Ruedi and Allgower. AO classification
What are the classification systems to describe 5th metatarsal fractures
- Stewart classification
- Torg classification
- Lawrence and Bott classification
What are the classification systems used to describe Lisfranc injuries
Harcastle
- Quenu and Kuss
- Nunley
What are the compartment pressures during a compartment syndrome
Intra-compartmental : >30mmhg
Extra-compartmental: within 10-30mmHg of diastolic BP
What are the different names for proliferative phase?
Physiological: Proliferative, granulation, fibroblastic or repair phase
Clinical: Subacute phase
Management: Controlled motion phase
•It ends when
–Fibroblasts activity / collagen production returns to baseline levels
–Fibroblast-rich granulation tissue is replaced by scar tissue
What are the different names for remodeling phase?
Physiological: Remodeling phase
Clinical: Chronic phase
Management: Return-to-Function phase
–The ends point for this phase is unclear as the rate of tissue remodeling may return to baseline before biomechanical properties are recovered.
What are the side effects of steroid use
- Leukocytosis
- increased wound healing time
- hyperglycemia
- hypopigmentation
What are the three Flexor tendon transfers
- *-Girdlestone Taylor**- bisect the tendon
- flexor reattached dorsally and sutured together to make a sling over proximal phalan
-Kuwada/Dockery-reroute the tendon to the distal drill hole
-Schuberth-reroute the tendon thru the proximal drill hole
What bacteria is incorporated into Santyl
Chlostridium histolytica
What classification system is used for open fractures
Gustilo and Anderson-
1- Opening in the skin <1cm
2- Opening in the skin between 1-5cm
3A: Greater than 5 cm with great soft tissue coverage
3B: Greater than 5cm with periosteal stripping
3C: Greater than 5 cm with arterial damage3
What is a locking plate and when is the best time to use it
A locking plate provides no compression of any sort
-great for osteoporosis, bone deficits and comminution
What is a strain
What’s a sprain?
Strain-used to describe physical damage to “active” soft tissues (muscle, tendon, myofascia). Usually tear of muscle or tendon
Sprain: Term used to describe physical damage to “passive” soft tissues (esp. ligament and joint capsule).
What is extensor substitution and during what phase of gait
- Weak anterior muscles: EDL/EHL overpowers intrinsics during swing phase of gait
- Cavus foot- EDL in position to overpower lumbricals just by passive stretch
- Equinus: extensors overworked to prevent tripping on forefoot
During the Swing phase of gait
What is flexor stabilization and during what phase of gait does it occur
- Failure of PTT to resupinate the foot results in overpronation.
- Extrinsics (FHL/FDL) fire earlier/longer/stronger to grasp ground during midstance
Midstance phase of gait
What is flexor substitution and during what phase of gait?
Supinated foot with weak posterior muscle (achilles tendon) unable to PF foot during propulsion.
PT, FHL, FDL overpower to lumbricals
Propulsive phase of gait
What is the angle formed by the metatarsal parabola
142 degrees
What is the blair procedure and what should it be used for
When the body of the talus is removed, the foot is fused in 15 degrees dorsiflexion. This procedure is used for AVN of the talus
What is the definition of osteoarthritis?
progressive loss of articular cartilage
accompanied by
- attempted repair of articular cartilage,
- remodelling, and
- sclerosis of subchondral bone, and
in many instances the formation of
- subchondral bone cysts and
- marginal osteophytes.
What is the depth of 1st met-cuneiform joint
3.23 cm
What is the etiology of talar neck fracture and what xray view is used
Etiology: axial load+ hyper dorsiflexion. Aviators Astralagus
-Canale X-ray view-AP view with foot PF, pronated 15 degrees to view angular deformity of talar neck
What is the function of plantar plate and what is its healing potential
Plantar plate serves as a stabilizer during WB and has very poor healing potential
What is the medical term for rolled edges along wound bed
epipilee
What is the most senitive and specific signs on a peripheral blood smear for megaloblastic anemia
Hypersegmented neutrophils
what is the relationship of motion to axis at a hinge joint?
perpendicular
What is the sequential release of MTPJ for hammertoe repair
1) Arthroplasty
2) Extensor tenotomy
3) Extensor hood release
4) Extensor Tendon lengthening
5) Flexor tendon transfer
What is Volkman’s contracture
ischemic necrosis causes muscular contracture
What layer of the nerve does a Morton’s neuroma usually develop in
in the epineurium
What normally occurs 2-5 cm superior to the insertion of
the Achilles tendon?
Tendon twist 90 degrees lateral
What oral meds are okay to give during Charcot Neuropathy
bisphosphonates
What sign presents to the medial calcaneus in flatfeet
Pizogenic sign which is fat papule herniations
What test differentiates between FF and RF cavus foot
Coleman block test
Wheaton Brace
used for metatatsal adductus
alternative to serial casting

Wheaton Brace System
Additional AK piece is design to lock into BK component
Knee flexed at 90 degrees prevent twisting of femur or hip and allowing isolated unilateral tx of tibial torsion

when casting infant for clubfoot
forefoot adductus
rearfoot varus
equinus
Where in the LE do atheromatous plaques tend to develop
Sites of bifurcation
1-Distal abdominal aorta
2-Common iliac
3-Common fermerol
4-Popliteal
5-Trifurcation into tibial and peroneal trunks
Which anesthesthetic should be evaluate for malgnant hyperthermia
Elevation of creatine Kinase
Which of the following are the two types of limb length
discrepancies?
Structural
Functional
Which of the following is a lower motor neuron disorder?
achilles tendon over taking the degenerative PTT
If the DTML is severed
develops hammertoes
While performing the vertical stress test for 2nd MTPJ
dislocation, which stage would you find the phalangeal
base can be subluxed but not dislocated?
Stage 1
Why are 5th met fractures hard to heal
- Watershed area of inraosseous blood supply to the metaphyseal region
- Mechanical pull of PB
Why is FDL used as a tendon transfer with PTTD
because it is in direct opposition to PB, expendable, and proximity to PTT
Wiberg CE Angle
if femoral head is inadequately, wil develop DJD
this test reveals how much DJD will develop
treatment: aligning the femoral head in the actabulum and holding it there

Wilson Disease
BASAL CELL DISORDER
Juvenile patient, symp to parkinsons
cooper-binding ceriloplasmin leading to accumulation of cooper in tissue
KAYSER-FLEISCHER RINGS-PATHOGNOMONIC OF APPEAR COOPER
CHILDREN EXHIBIT SCHIZOPHRENIC BEHAVIOR, MULTIPLE MOVEMENT DISORDER
TREATMENT-D PENICILLSMINE
X-Ray difference Normal to Flatfoot
Normal Flatfoot
Meary’s angle 0 degree 1-15 and >>15 severe
CIA 20-25 degrees <15 degrees
A/P T/C angle <25 >25 degrees
T-N angle <50 60-70 degrees
X-ray findings associated with Lisfranc injury
Space between 1st and 2nd MT should be equal inspace with medial and intermediate cuneiform
- Drop in arch
- Fleck sign
X-ray findings to confer fibular length (2).
Shenton’s lines: line continues with spur of lateral malleolus with tibial plafond
Dime sign: assesses fibular length and talocrural angle
Diathermy
use of electric current to coaguate vessels
hinge-axis
concept?
To lengthen the metatarsal, the cut should
be proximal-medial to distal-lateral.
Sniffin Position
extend head and flex the neck for anesthesia