part 1 EXTRA Flashcards
بسم الله الرحمن الرحيم توكلنا علي الحي الذي لا يموت
ECG ?
هو تسجيل النشاط الكهربي للقلب
وهو مجموع جهود الحركة اللي بتوصل لسطح الجسم
U Wave ?
Repolarization of papillary mmuscle and purkinje fibers
P wave
a
Atrial deplarzition
0,1 sec
<0,2 mv
QRS
Intraventriular conduction
0,04 TO 0,08 SECODS
1mv
Porlonged in cell to cell conduction
Ventricular ectopic focus
Ventricular hypertrophy
T wave
Ventricular replariztion
is gradual process compared to deplarization so that?
longer time low amplitude
0,16-0,25 seconds amplitude 0.2 mv
pr interval ?
atrial and avn conductin 0,12 to 0,21 seconds
prolonged in
Vagal stimulation and AVN abnormalities
QT Interval
Ventriclar electric systole ( deplarization and repolarization )
0,34to0,43 seconds
prolonged in tachycardia and shortened in bradycardia
st and tp semetns
ST sements isoelectric and fully depolarized ventricles
TPsegments isoelectric fully repolarized ventricle
Amplitude in ECG is dierct proportional to mass
So QRS higher than P wave
ECG IMP
Anatomical orietation of Heart Artial and ventricualr sizes Heart rate and regularity Paacemaker and the path of action potential Muscles status
ECG IMP IN DIAGNOSIS ?
Dysrhythmia
Myocardial ischemia and infarction
Electrolytes level as potassium
Digitalis effect
why epicardium reolarize firest?
due to compression of contraction on subendocardial blood vessels delaying its reppolariztion
Augmented limb lead
=نفسه + نص مجموع الاتنين التانيين
=واحد ونصف مثله
المحور؟ AXIS
متوسط اتجاه انتشار موجة اللااستقطاب في البطين
Normal axis degrees ?
-30 to +110
Rigth axis deviation ?
Tall person
Right ventricular hypertrophy
Right bundle branch block
Left ventricular extrasystole
Left axis deviation ?
Left ventiruclar hypertrophy
Left bundle branch block
Right ventricular extrasystole\
short
Bipolar leads?
measue potential differnce betwwen 2 points by 2 active elctrodE
Lead 1 , 2 ,3
Einthovein Law ?
Lead II =Lead I + Lead III
arrythmia ?
مشاكل تكوين النبض
مشاكل توصيل النبض
مشاكل تكوين النبض ؟
Altered impulse formation اختلاف RMP Thresold Slope
Trigger activity
Afterdepolariaton oscillitations ! due to ischemia
مشاكل التوصيل؟
1- bloclk premature beats
2-Brady cardia deceleration dependent block
1-Reduced excitability and AP Amplitude فيونكات
2-degenerative process after ischemia
Sinus rhtym normal in
positive p wave in lead 2 and aVR
Heart rate 60 -100 bpm