Pain Theories Flashcards

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1
Q

Briefly describe the intensity theory

A
  • in existence since 400BC
  • 1859: experiments to back the theory up

Theory:

  • non-noxious or sub-threshold noxious stimuli summate in the spinal cord
  • if enough stimuli and signals build up, pain will be experienced
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2
Q

Briefly describe the pattern theory

A
  • proposed in the 1920s

Theory:

  • there are not separate neurons or pathways for noxious signals
  • rather noxious and non-noxious signals have different time patterns, resulting in the creation of different sensations
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3
Q

Briefly describe the specificity theory

A
  • in existence for milennia
  • popularized by Descarte in 1600s (bell image)

Theory:

  • each modality has a separate neuron, separate signal and separate pathway
  • existence of ‘pain fibres’
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4
Q

Briefly describe the gate control theory

A
  • proposed by Melzack and Wall in 1965

Theory:

  • there are ‘gates’ (interneurons) in the dorsal horn that can either inhibit or facilitate the transmission of nociceptive signals
  • input from nociceptive neurons (A delta and C fibres) ‘open’ the gate by inhibiting inhibitory interneurons
  • input from non-nociceptive neurons (A alpha and beta) ‘closes’ the gate by activating inhibitory interneurons
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5
Q

Describe the neuromatrix theory of pain

A
  • proposed by Melzack in 1991

Theory:

  • pain is a multidimensional experience with multiple inputs and outputs
  • 3 inputs: cognitive evaluative, sensory discriminative and motivational affective
  • 3 outputs: pain perception, actin programs, stress regulation programs
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6
Q

What are the 3 outputs in the neuromatrix theory of pain?

A

Pain perception

  • sensory
  • affective
  • cognitive

Action programs
- involuntary and voluntary action patterns

Stress regulation programs

  • cortisol
  • norepinephrine
  • endorphin levels
  • immune activity
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7
Q

What are the 3 inputs in the neuromatrix model of pain?

A

Sensory discriminative

  • felt sensation of pain (quality, sensation, spatial location)
  • thalamus and primary somatosensory cortex

Motivational affective

  • emotional component
  • limbic system

Cognitive evaluative

  • interpretation of pain
  • pain beliefs
  • higher processing centres
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8
Q

What is the classical conditioning theory of motor control?

A
  • people can learn to associate pain with movement via classical conditioning
  • this can be protective in the short term when there is actual or potential tissue damage; but unhelpful in the long term when there is no threat of tissue damage
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9
Q

What is the protective response theory of motor control?

A
  • movement and motor control is altered as a response to pain and/or injury
  • changes can include stiffness, altered biomechanics, altered loading
  • changes can be protective in the short term but harmful in the long term (sub-optimal tissue loading can increase risk of future injury)
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10
Q

What is the impaired movement theory of motor control?

A
  • sub optimal loading can lead to pain and injury; and pain and injury can lead to sub optimal loading
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11
Q

What is tissue creep?

A

The insidious onset lengthening or deformation of tissue under prolonged and inappropriate load (can be caused by sub-optimal loading and altered motor control in response to pain)

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