Paeds Flashcards

1
Q

What is suggestive of fabricated or induced illness?

A
  • Multiple unexplained symptoms.
  • multiple negative investigations
  • perpretrator may induce the child to toxins, or alter laboratory test results, fabricate PHM
  • Mother nearly always responsible and often has extensive contact with health services, very compliant
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2
Q

How can you investigate fabricated or induced illness?

A

Look for exogenous toxins, foreign substances such as blood

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3
Q

How do you manage fabricated or induced illness?

A

Refer to social care, who have a statutory order to undertake thorough assessment according to strict guidelines. Check the child protection register and call the health visitor

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4
Q

What is the most common cause of chest pain in a child with sickle cell?

A

Acute sickle chest syndrome. Thrombosis, infection and fat embolism produce pleuritic pain, shortness of breath and fever. There is often consolidation at the bottom. Needs to be managed aggressively.

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5
Q

How do you treat acute sickle chest syndrome?

A

Acidosis, hypoxia and dehydration need to be corrected> hyper hydration and supplemental oxygen. Analgesia (often IV morphine) and physiotherapy help the child to breath deeply. Start empirical antibiotics such as co-amoxiclav and clarithromycin. Discuss with senior paedatrician and haematologist as may deteriorate and require continuous ventilation, CPAP or intubation. May also require blood transfusion

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6
Q

What should all children with sickle cell receive?

A

As they develop hyposplenism, they are vulnerable to infection with encapsulated bacteria–> vaccinate and regular penicillin prophylaxis

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7
Q

7 year old girl from Zimbabwe. Fever, worsening headaches, neck stiffness and recurrent otitis media

A

Bacterial meningitis, most likely due to streptococcus pneumonia that may be secondary to the recurrent otitis media

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8
Q

How should bacterial meningitis be treated?

A

Commonly iv ceftriaxone and dexamethasone

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9
Q

What primary factors predispose to recurrent infection?

A

Immune deficiency (antibody, complement, neutropaenia) Chronic granulomatous disease, ataxia telangiectasia

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10
Q

What secondary factors predispose to recurrent infection?

A

HIV, immunosuppressive drugs, malnutrition, hyposplenism, cystic fibrosis and anatomical difficulties.

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11
Q

What would you see on the blood test of someone with rickets?

A

High ALP and borderline calcium. Frontal bossing, prominent costochondral junctions (rickety rosary), muscle weakness, tetany and hypocalcaemic fits.

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12
Q

What would point to IDA?

A

Poor nutritional intake, pallor and microcytic hypochromic anaemia

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13
Q

What is rickets? What are risk factors?

A

A failure of mineralisation of bones. Dark skin, poor nutritional intake and prolonged breast feeding (milk is a poor source of vitamin D). Diseases that affect the kidney or liver as well as malabsorption.

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14
Q

What is seen on the Xray of rickets?

A

Swolllen ankles and wrists. Widening of the growth plate, and cupping, splaying and fraying of the metaphysis

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15
Q

What should the ferritin be like in IDA?

A

Low

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16
Q

How should rickets be managed?

A

A 3 month course of oral high dose vitamin D. Bone chemistry should be measured 2 weeks after starting treatment and regularly to prevent hypercalcaemia. Then there should be a long term maintenance dose.

17
Q

How should anaemia be managed?

A

A 3 month dose of oral iron, dietary advice (dietitian).