Paediatrics Flashcards

1
Q

What are treatment options for discoloured teeth?

A

Microabrasion, bleaching, resin infiltration, local comp rest., veneers - direct and indirect, do nothing

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2
Q

what should be recorded in the pre-op sheet for discoloured teeth

A

clinical photos, sensibility testing, shade, diagram of defect, radiographs, patient assessment

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3
Q

what is micro abrasion

A

the removal of the surface layer of opaque enamel

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4
Q

what are some advantages of micro abrasion

A

removes brown/yellow stain, effective, conservative, inexpensive, can be used before bleaching, easily performed, permanent results

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5
Q

what are some disadvantages of micro abrasion

A

removal of enamel - destructive, senstivity, may get more staining
need protective wear for patient, dentist and nurse
HCl is caustic
results are unpredictable - may appear more yellow
must be done by dentist

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6
Q

what is the clinical technique for micro abrasion

A

protect soft tissues with dental dam
place sodium bicarbonate guard behind teeth
slurry of HCl for 5 seconds on tooth - can repeat 10 times but wash off and review colour and shape after each one
once happy - place fluoride varnish - clinpro is more white in colour
polish with sandpaper - the finer/smoother prisms look less stained
polish with toothpaste

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7
Q

how much enamel is lost with micro abrasion and compare this to enamel etching

A

100 microns are lost with micro abrasion - 10 are lost with etching so losing 10x the amount

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8
Q

when and why must micro abrasion be reviewed

A

review 4-6 weeks after, advise patient not to drink or eat highly coloured foods as teeth are dehydrated and will stain. Review as can offer a second cycle but only if some improvement has been seen with first lot, if not the second wont work either. Can only do 2 rounds. Must take pre and post op photographs

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9
Q

what bleaching techniques are available for a vital tooth

A

external bleaching only. Chairside power bleaching or at home night guard bleaching

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10
Q

what bleaching techniques are available for a non-vital tooth

A

internal bleaching - inside outside technique, walking bleach technique

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11
Q

why is chairside bleaching not normally recommended

A

it uses rapidly reactive, unstable hydrogen peroxide - damage to soft tissue and eyes - causes sensitvity and more expensive

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12
Q

what instructions are given to patients for night guard at home bleaching

A

brush teeth with toothpaste
place gel in mouth guard
put in over teeth and seat - remove excess from gums
rinse gently and dont swallow
wear over night or for at least 2 hours
brush teeth and rinse with cold water
sensitive toothpaste may be required

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13
Q

what are advantages of non-vital tooth bleaching

A

conservative, good results, gingival level of adolescents is unstable for fixed restoration so this is good in mean time, simple, no irritation to gingiva

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14
Q

what are important factors when deciding if a non-vital tooth is appropriate for internal bleaching

A

good root filling - to length and condensed, no pathology
anterior teeth without large restorations
not amalgam discolouration
not fluorosis or tetracycline discolouration

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15
Q

what is the clinical technique of walking bleach - non vital internal bleaching

A

access cavity, remove GP to ECJ, place bleaching agent on cotton wool ball and place in access, place dry cotton wool ball on top and then GI over this. 2 weeks later, remove GI and replace balls - can do this up 6-10 times. Regression of 50% at 2-6 years

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16
Q

what is the clinical technique of inside out bleaching

A

access cavity, custom mouthguard made. Patient puts bleaching agent in back of tooth and mouth guard. replaces the gel every 2 hours except through the night. wear guard all the time except when eating and cleaning. 10% carbamide peroxide used

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17
Q

what are potential complications with non vital bleaching

A

over bleaching, brittleness, external cervical resorption, failure to bleach, spilling of bleaching agents

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18
Q

how can external cervical resorption be prevented

A

layer of GI cement above GP cone - but can prevent adequate bleaching
non setting calcium hydroxide placed for 2 weeks before final restoration - neutralises any acidity in PL

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19
Q

what is resin infiltration

A

infiltration of enamel lesions with low-viscosity resin and cured. surface layer is eroded with etch, lesion becomes desicated and gives access - resin placed and can infiltrate through. causes lesions to lose discolouration and appear similar to sound enamel

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20
Q

what are the parts to caries risk assessment

A

clinical evidence, diet, plaque control, fluoride exposure, medical history, social history, saliva

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21
Q

what are the parts to preventions

A

fluoride varnish, fluoride toothpaste, fluoride supplement, fissure sealants, radiographs, diet advice, change medication, toothbrushing advice

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22
Q

what guidelines are used for caries risk and prevention

A

sign 138, SDCEP

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23
Q

what toothbrushing advice should be given

A

brush twice a day, including once before bed, fluoride toothpaste, spit dont rinse, should be supervised

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24
Q

what is the indication for fissure sealants

A

for all first permanent molars of all children regardless of risk factor
in high risk children - deciduous molars caries free, and premolars

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25
Q

what are the steps for fissure sealants

A

patient and tooth selection
tooth isolation - rubber dam, cotton wool
clean tooth - pumice and slurry
etch - 35% phosphoric acid and dry
apply resin sealant
light cure
check retention, remove flash, check for air bubbles

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26
Q

what materials are required for fissure sealants

A

rubber dam or cotton wool roll, dry guard, saliva ejector
35% etch
pumice and slow speed
resin
light cure
probe, excavator, microbrush, mirror

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27
Q

how can hard tissue defects be divided and give examples

A

localised - trauma or abscess of primary
generalised - environmental or hereditary
environmental - fluorosis or MIH
hereditary - ameleogenesis imperfecta

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28
Q

what is the difference hypomineralisation and hypoplastic

A

2 phases of enamel development - secretory and mineralisation
secretory lays down jelly like shape of tooth, if there is a problem with this - the tooth will be hypoplastic - insufficient bulk/thickness of enamel
mineralisation - increasing mineral content and thus hardness - if there is a problem with this, the tooth will be hypomineralised

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29
Q

what is AI and how is it diagnosed

A

AI is an inherited condition in which there is a malfunction with enamel development - either secretory or mineralisation. diagnosis - family history, appearance (affects all teeth, tooth size, colour, shape, yellow/brown or white), radiographic - no difference between enamel and dentine

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30
Q

what medical conditions have an enamel defect - not ameleogenesis imperfecta

A

epidermolysis bullosa, prader-willi, downs

31
Q

how to tell the difference between fluorosis and MIH

A

MIH - only molars and incisors
fluoride - all teeth
ask if they lived in fluorodated area

32
Q

what questions are asked to get diagnosis of MIH

A

pre-natal - problems in last trimester of pregnancy - pre-eclampsia, gestational diabetes, infections

natal - premature birth, time special baby unit, traumatic birth

post-natal - infections in first year of life, how long they were breast fed for

33
Q

what is the calcification dates of FPMs and incisors

A

FPMs - 7-8 months after ovluation, crown formed at 1 year of life
incisors - start 3-4 months after birth, complete by 3-6 years

34
Q

when would you consider balancing an XLA

A

XLA of tooth on same arch but condralateral side - prevent midline shift
primary canines

35
Q

when would you consider compensating an XLA

A

XLA of same tooth on opposite arch - opposing tooth
prevent over eruption
if XLA of lower FPM - XLA upper FPM, over eruption would prevent drifting of 7 and space closure

36
Q

what guidelines are available for planned XLA of FPM

A

RCS Clinical Effectiveness Committee 2009

37
Q

what is the order of treatment planning

A

prevention and acclimitisation first - OHI, Diet advice, fluoride varnish - can introduce aspirator, cotton wool rolls, 3n1
fissure sealants - polish with prophy cup to introduce slow speed
small restorations with no LA - stabilisation phase
larger restorations with LA - start with maxillary
pulpotomy and extractions

38
Q

what is fluorosis

A

defect in mineralisation of enamel due to high levels of systemic fluoride during enamel development, causes white lines in mild form or brown/yellow marks in more severe form

39
Q

what age are children at risk of fluorosis until

A

until calcification is complete of all crowns - age 8 on SIGN 138 guidelines
for cosmetic purposes - until age 3 for anterior crown development

40
Q

what is the recommended fluoride levels for children

A

until age 8 - 0.1mg/kg/day
from age 8 - 10mg/day
supplement
until age 3 - 0.25mg/day
age 3-6 - 0.5mg/day
6+ 1mg/kg/day

41
Q

what are the recommendations for different levels of fluoride ingestion

A

less than 5mg/kg - give calcium (milk) orally and observe
5-15mg/kg - give calcium (milk or calcium gluconate) and take to hospital for observation
more than 15mg/kg - urgent hospital referral, intensive care and cardiac obs - IV cardiac gluconate

42
Q

what are the components fear and anxiety

A

physiological - breathlessness, perspiration, palpitations
cognitive - hypervigilance, loss of concentration, inability to remember
behavioural - avoidance, asking questions, missing appts

43
Q

how can you reduce a patients anxiety

A

acclimitisation
giving the patient control - stop signals, rest breaks, providing them information

44
Q

how can cerebral palsy effect dental health

A

poor manual dexterity - poor OH
limb spasms - unable to get dental treatment safely
gag reflex
regurgitation - acid causing erosion
not in control of their diet or OH

45
Q

how can a patient in a wheelchair be treated

A

transfer board
wheelchair recliner
hoist

46
Q

what legislation protects children

A

children and young people act 2014

47
Q

what are types of abuse

A

sexual, physical, emotional, neglect

48
Q

what is dental neglect defined as

A

british society of paeds - the persistent failure to meet childs oral health needs likely to result in impairment of childs oral or general health or development

49
Q

what does dental disease put children at risk of

A

severe infections, teasing, multiple GAs, repeated antibiotics - resistance

50
Q

what is indicators of dental neglect

A

obvious dental disease apparent to non-dental professional, but no treatment sought after
missing multiple appoints despite support given to carer - not returned for treatment
impact on child - sleep loss, eating effected, missing school, teasing

51
Q

what guidance should be followed when managing suspected abuse/neglect

A

british dental association
child protection and dental team

52
Q

what are the stages of management of suspected dental neglect/abuse

A

stage 1 - dental team management
stage 2 - preventative multi-agency management
stage 3 - child protection referral

53
Q

what medical condition is associated with supernumerary teeth

A

cleidocranial dysplasia

54
Q

what are some anomalies related to shape of teeth

A

peg shaped laterals
dens in dentine
talon cusp
dilaceration

55
Q

what is the relevance of osteogenesis imperfecta

A

often associated with dentinogenesis imperfecta, have multiple fractured bones

56
Q

what are dentine conditions that only effect dentine

A

dentinogenesis imperfecta type 2
dentine dysplasia

57
Q

what medical conditions are associated with dentine anomalies

A

osteogenesis imperfecta
ehlers-danos syndrome
rickets

58
Q

what should be considered when deciding to extract fpm

A

AGE, skeletal pattern, future ortho needs, quality of teeth

59
Q

what are causes of delayed eruption

A

medical conditions - downs, hypothyroidism
premature, low birth weight
malnutrition
gingival hyperplasia

60
Q

what are causes of delayed exfoliation

A

trauma, infra-occlusion, ectopic successor, hypodontia

61
Q

what are causes of premature exfoliation

A

trauma
following pulpotomy
immunological deficiency

62
Q

what advice should be given to all patients post trauma

A

avoid contact sports for 2 weeks
soft diet for 2 weeks
maintain good oral hygiene - soft brush after every meal
and rinse with chlorhexidine 0.12% twice a day

63
Q

what are types of trauma complications

A

pulpal necrosis and infection
root resorption
pulp canal obliteration
break down of marginal gingiva and bone

64
Q

how does replacement resorption happen and how does it appear

A

severe damage to PDL, osteoblasts faster at healing than PDL fibroblasts, PDL becomes replaced with bone - dentine fused directly to bone
appears - tooth is infraoccluded as included in bone remodelling, no PDL space seen radiographically

65
Q

what is pulp canal obliteration

A

response of vital pulp - progressive hard tissue formation within pulp chamber - narrowing of pulp chamber and canal

66
Q

what types of trauma is pulp canal obliteration more likely to be seen

A

luxation, extrusion, intrusion, root fractures

67
Q

why do immature teeth have a better prognosis following trauma

A

wider apices - more blood vessels - better healing capacity

68
Q

what is subluxation and what are the symptoms

A

damage to surrounding tooth structures, no displacement but may have mobility
symptoms - increased mobility, TTP, bleeding at gingival crevice

69
Q

what is lateral luxation and what are the symptoms

A

displacement of tooth in a socket in any direction other than axially
symptoms - tooth immobile, ankylosis percussion note (metallic), fracture of alveolus, sensibility tests likely to be negative

70
Q

why is RCT initiated in a closed apex tooth following lateral luxation

A

high chance of pulpal necrosis, doing pulp extirpation early prevents external infection related root resorption

71
Q

how can root fractures be classified and why is this helpful

A

apical third, mid third, coronal third
coronal third fractures have the worst prognosis and must be splinted for 4 months

72
Q

if a tooth has discoloured yellow following trauma, what is this indicative of

A

pulp canal obliteration - tertiary dentine laid down in pulp chamber, reduces light transmission

73
Q

what are the properties of splints

A

must be passive and flexible
must allow for adequate oral hygiene
must allow for clinical monitoring and sensibility tests
ease of placement
aesthetics