P1: Psychopathology Flashcards

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1
Q

Define Statistical infrequencies in terms of abnormality

A

Abnormality: No. of times it is observed. Behaviour that is rarely seen = abnormal. E.g. Low/High IQ

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2
Q

Define Deviations from Social Norms

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Abnormality is based on social context. (collective judgement) Depends on cultural, historical context E.g.Homosexuality, Anti-social disorder = abnormal ∵ deviate from social norms

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3
Q

Evaluate Definitions of Abnormality

A

+ Practical Application: All assessments of mental disorders involves comparison to statistical norms.

  • Unusual sometimes= good:E.g.high IQ. No treatment needed ∴ cannot be used solely for diagnosis
  • No benefit from abnormal labelling:e.g. Low IQ, but happy life= no need for diagnosis. Might cause negative effect from being labelled
  • Culturally relative:E.g.Hearing voices= weird in UK, accepted in other cultures = Problem using another groups standards
  • Possible abuse of social norms: Over reliance= abuse.e.g.black slaves trying to escape,Nymphomania(hyper sexuality). Purpose to maintain control over minority ethnic groups
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4
Q

Define failure to function adequately in terms of definitions of abnormality

A

Inability to cope with everyday living.e.g. Maintaining job. Signs of failure to cope: 1)No longer conform to interpersonal rules, 2)Experience personal distress, 3) behaviour= irrational or dangerous.e.g.
-Intellectual disability disorder= failure to function adequately n inability to cope with demands of everyday life = failure to function adequately.

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5
Q

Define deviation from ideal mental health

A

Changing the emphasis. Jahoda’s ideal mental health criteria: 1)no symptoms/distress 2) Rational/accurate deception of self 3) Self-actulise 4)Copes with stress 5) Realistc view of world 6)Good self-esteem/lack guilt 7)Independent 8)successful work, love, enjoys leisure

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6
Q

Evaluate definitions of abnormality (Failure to function adequately n deviation from ideal mental health)

A
  • Requirement of patient’s perspective: Difficult to assess distress, but understands personal experiences are important
  • Deviation vs failure to function:E.g.Extreme sports, may limit freedom if behaviour treated as failures
  • Subjective judgement: Hard to tell if they are distressed.e.g. failure to diagnose
  • Culturally relative: Johoda’s classification applies to western Euro cultures .e.g.self-actulisation= self-indulgent in collectivist culture
  • Unrealistic standard for mental health:V few ppl will meet requirements ∴ abnormal, but it makes it clear how to improve
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7
Q

Define Phobia

A

Behavioural: Panic , Avoidance
Emotional: Anxiety , unreasonable emotional response
Cognitive: Selective attention, irrational beliefs

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8
Q

Define Depression

A

Behavioural: Self-harm/aggression, Low activity levels
Emotional: Low mood, anger
Cognitive: Poor concentration, dwell on negative

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9
Q

Define OCD:

A

Behavioural: Compulsions , avoidance
Emotional: Anxiety, Guilt/disgust
Cognitive: Obsessive thoughts, use coping strats

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10
Q

Explain what phobias are using the behavioural approach

A

learnt by classical conditioning, maintained by operant conditioning.UCS-UCR,NS+UCS-UCR,NS-CS,CS-CR
E.g. Little Albert Case, Fear of rats conditioning = generalised to other white furry objects

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11
Q

Evaluate the Behavioural Approach to explaining Phobias

A

+ Application: Good explanation power= use in therapy,

  • Incomplete explanation: Biological drive(Instincts)?, innate fear for certain things, evolution prep ∴ more to acquiring phobias than conditioning
  • Bad EXP ≠ phobia: Sometimes no phobia formed, maybe forms where vulnerability exists
  • Cognition Ignored: Focused on behavioural side, cognition not adequately addressed
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12
Q

Explain the Behavioural Approach to treating Phobias

A

-Systematic Desensitisation(SD):
AIM: Counter-condition.CS paired with relaxation.
Reciprocal Inhibition: Cannot be afraid n relaxed at once. Creation of anxiety hierarchy. Relaxation techniques practiced at each stage.
-Flooding:
Immediate exposure to phobic stimulus (Bombarding)
Learns through Extinction, Without avoidance learns its harmless via exhaustion.
Informed consent involved

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13
Q

Evaluate Behavioural approaches to treating Phobias

A

+ SD effective: 42 patients followed up= long-lasting n effective, reduces anxiety.
+ SD seems acceptable: When given choice, SD preferred ∵less trauma. Reflected in low refusal rates n low attribution rates(Dropping out)
- Flooding is traumatic: ppl often unwilling to finish completely ∴ ineffective, waste time n money.
- Flooding less effective for some: Less effective for complex phobias.e.g.Social phobias ∵ cognitive aspect ∴ more benefit from Cognitive therapy ∵ take irrational thinking

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14
Q

Explain the cognitive approach to explain Depression

A

-Beck’s Cognitive theory of Depression:
Faulty info processing= some ppl more prone to depression.e.g.Black n white thinking
Depressed ppl have Negative schemas= interpret world negatively.
Negative triad: Negative views of SELF,WORLD,FUTURE
-Ellis’s ABC Model:
A-Activating Event: Arises from irrational thoughts, occurs from experiencing negative event
B-Beliefs: Negative events tigger irrational beliefs.
e.g. Musterbation belief we must always succeed
C-consequences: Emotional n behavioural consequences
e.g. Musterbation-> fail-> consequence= depression

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15
Q

Evaluate the cognitive approach to explaining Depression

A

+ Supporting Evidence: 65 preggo women tested for cognitive vulnerability/depression b4 n after birth, vulnerable= more likely to suffer post-natal depression ∴ cognition cane seen b4 depression develops
+ Practical evidence:Beck’s explanation forms basis of CBT. Negative triad, identified n challenged ∴ translates to successful therapy
- Explanation power: cannot explain all aspects of depression.e.g.extreme anger ∴ limited
-Partial Explanation: Ellis explains reactive depression. Cannot explain depression that arises without an obvious cause

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16
Q

Explain the Cognitive approach to treating depression

A

-CBT: work together, identify irrational/negative thoughts n challenge negative triad by patient taking active role. patient encouraged to test reality of beliefs, maybe also set homework= evidence of patients incorrect beliefs.
-Rational Emotional Behaviour Therapy(REBT): Extension of ABC model.
D-Dispute: challenge beliefs
E-Effect: effect of challenge
Empirical Argument: Evidence to support irrational belief?
Logical Argument: Negative thoughts actually follow facts?
Goal of treatment: Gradual decrease in avoidance n isolation, increase engagement in actives that can improve mood(exercise)- Behavioural Activation

17
Q

Evaluate the Cognitive approach to treating depression

A

+ CBT effective: Lots of evidence= support CBT. As effective as medicine, helpful alongside medication ∴ case to make it first choice of treatment

  • incompatibility with some cases of depression: severe= cannot motivate self ∴cannot do cognitive work. Must take meds until ready to start CBT.
  • Success due to therapist-patient relation: Variation difference= small, Key factor= relation between therapist n patient determines success not a technique
  • Overemphasis on cognition: Minimises importance of circumstance.e.g.poverty. CBT shifts focus to Cognition ∴ can demotivate ppl to change situation
18
Q

Explain the Biological Approach to explaining OCD

A

GENETIC EXPLANATION
-Candidate Genes: Genetic Vulnerability. Serotonin n dopamine genes. Neurotransmitters have role in regulating mood. OCD is polygenetic, up to 230 different genes involved.
-Aetiologically heterogeneous:Different groups of genes cause OCD in ppl, not always the same group
NEURAL EXPLANATION
-Low serotonin= low mood (Neurotransmitter relies info from 1 Neuron to another) ∴ not normal.
-Impaired Frontal Lobes(responsible:logic n decisions):(some cases) linked to abnormal functioning.e.g.hoarding.
Some Evidence to suggest LEFT Parahippocampal gyrus association: Processing unpleasant emotions functioning abnormally

19
Q

Evaluate the Biological Approach to explaining OCD

A

+ Research Support: Studies 68% identical twins(MZ) share OCD, 31%(DZ) twins do ∴ supports genetic influence on OCD
- Too many candidate genes identified: Each genetic variation increases risk of OCD by a fraction ∴ very little predictive value
+Neural support: Antidepressants work solely on serotonin= effective ∴ serotonin system involved in OCD, forms part of bio conditions.e.g. Parkinsons
- Co-Morbidity link: OCD n depression often occur together ∴ depression may cause disruption to serotonin system—–

20
Q

Explain the Biological approach to treating OCD

A

DRUG THERAPY
AIM: to change levels of neurotransmitters.e.g.increase serotonin
SSRI: Prevents reabsorption n breaks down serotonin ∴ compensates Serotonin system. Requires 3-4months of daily use for impact.
Often used alongside CBT, Drugs deal with Emotional symptoms= so can better engage with CBT
ALT to SSRI: Tricyclics n SNRI
Tricyclics= same effect, more side effects (old version)
Used as a 2nd line of defence

21
Q

Evaluate the Biological Approach to treating OCD

A

+ Effective: SSRI vs placebo= yield better results reduced symptoms for 70%ppl. Rest helped with other drugs or drugs+CBT
+ Cost-effective: Cheaper than CBT ∴ good for NHS. SSRI= non-disruptive to lives n require little effort ∵ no need to partake in psychological therapy
- Side effects: Possible severe side-effects.e.g.indigestion, irregular blood pressure ∴ reduced effectiveness n may deter usage
- Sometimes Unreliable: Controversy, Drug companies sponsor= biased results n facts ∴ suppress/report some findings to maximise profit