Osteochondrosis- Bennett Flashcards
general features of osteochondroses
middle years of growthboys more than girlslower limbs15% bilateral
a group of conditions in which the primary or secondary center of ossification undergo aseptic necrosis
osteochondroses
freiberg disease ***
avascular necrosis of metatarsal head13-18 femaleunilateralsecond met 68%, third met 27%ddx: stress fx, synovitis/capsulitis, tendonitis, metatarsalgia, morton’s neuroma, JRAsmillie: secondary to traumatic processbraddock: secondary to fx modified by its proximity to epiphyseal plates/s: local pain, tenderness, swelling, limitation of mpj motion widening of jt space with effusion, initial fx, collapse of laterall margintx: below knee walking cast, metatarsal pad
*** kohler’s disease
osteochondrosis of the navicularmales 3-7ossification (18-24 mo for females and 24-30 mo in males)may occur simultaneously with legg-perthess/s: antalgic gait, local pain and tenderness over navicular, post tib tendon inflamed at insertion siteflattened navicular radiographicallytx: below knee walking cast with foot in varus and equinus 6-8 wk
buschke’s disease
osteochondrosis of the cuneifrombone5-13
thiemann’s disease
osteochondrosis of the phalanges at proximal epiphyses
sever’s disease ***
osteochondrosis of calcaneusnormal ossification 8-13contributing factors: gastroc-soleus equinus, excessive pronation, cavus foot, obesity, inflammatory conditionsddx: fracture, lytic lesion, infection, tendonitiscompare contralateral with radiographss/s pain, tenderness to palpation, antalgic gait, activity worsens, no pain morning after resttx: short leg cast, NSAIDS, control pronation
islen’s disease
osteochondrosis of the styloid processdue to traction apophysitis from the peroneus brevis, metatarsus adductus
*** leff-calve-perthes disease
ischemic necrosis of the femoral head capital ossification centermales 4-8self-limited diseasemay be due to excessive fluid pressure or inflammatory of traumatics/spain in region of the hipantalgic gaitlimited ROMdisuse atrophytx: abduction cast/brace4 stages: femoral head becomes more dense with possible fracture of supporting bone; fragmentation and reabsorption of bone; reossification when new bone has regrown; healing
*** osgood-schlatter’s disease
osteochondrosis of the tibial tuberositymales 10-15dx: pain at insertion of patellar tendon and soft tissue edema, enlargement of tibial tuberosity; may be secondary to trauma ddx: tendonitis, osteogenic sarcoma, infection, tibial tubercle fx tx: rest, ice, etc. NOT steroids type I: tibial tuberosity prominent and irregulartype II: tibial tuberosity prominent and irregular w/ sm free particl located at ant. tuberositytype III: normal tuberosity w/ bone particle
describe the early phase of necrosis (1)
AVASCULARITY- osteocytes and bone marrow cells die- ossific nucleus of the epiphysis ceases to grow- articular cartilage remains alive and grows- disuse atrophy (osteoporosis)- asymptomatic
phase of revasularization w/bone deposition and resorption (2)
stage represents a vascualr reaction of surrounding tissue to dead bone-ossificationof the thickened pre-osseous cartilage resumes and new bone is laid down on dead trabeculae- combination of irregular areas of bone deposition and resorption provides radiographic appearance of fragmentation- most vulnerable stage of osteochondrosis*
phase of bone healing (3)
bone resorption ceases, but bone depositon continues-newly formed bone still exhibits biological plasticity
phase of residual deformity (4)
- once bony healing of the epiphysis is complete its contour relatively remains unchanged- if any residual deformity is present, it will remain, as well as the associated complications
classification of freiberg disease
I: epiphyseal fissure fxII: central portionof bone reabsorpitonIII: metatarsal head begins to flattenIV: articular loose bodyV: complete flattening of metatarsal