OSCE Flashcards
S1 noise represents
AV vlaves close
Soft S1
AV valves close with reduced velocity
- reduced contraction (severe heart failure)
- valves don’t close properly (MR)
- valves alreaedy partially closed at end of diastole as atrial relax occurs before LV contraction (prolonged PR interval)
S4/atrial gallop
(before S1)
Pressure overload: atrial contraction into stiff hypertrophied ventricle
- LV hypertrophy
- Hypertension
-Aortic stenosis
Loud S1
AV valves close with highger velocity as they are wide open at end of diastole
- high atrial pressure (MS, AF)
- short diastole (short PR interval, tachycardia)
Split S1
= asynchronous AV valve closure
- can be normal, but wide split may suggest RBBB or ASD
What is S2
Aortic/pulmonary vallves close
Soft S2
= reduced aortic/pulmonary valve motility
- AS
- PS
Loud S2
= valves close with higher velocity due to high upstram pressure
- pulonary hypertension
- systemic hypertension
Split S2 on inspiration
= physiological. Aortic valve closes first bcause pulm valve closure slightly delayed by increased blood return to right heart, due to negative intrathoracic pressure
Wide split S2
= exaggerated split, which increases during inspiration (aortic valve closing before pulmonary)
- RBBBB
- Increased resistance to RV ejection eg pulmonary hypertension or PS
Reverse split S2
= split which icnreases during expiration (pulmonary valve closes before aortic)
- LBBB
- incr resistance to LV ejection eg systemic hypertension or AS
S3/ventricular gallop
(after S2)
= volume overload: high volume blood from atrium rapidly fills ventricle during passive filling phase
- left ventricular failure
- hyperdynamic states, eg athlete, anaemia, fever, thyrotoxicosis
Causes of aortic stenosis
Age (senile calcification)
Bicuspid aortic valve (eg in Turner’s)
Congenital
Rheumatic heart diseease
Causes of mitral regurg
papillary muscle dysfunction (post MI)
Dilated cardiomyopathy
RHeumatic heart disease
infective endocarditis
congenital
connective tissue disorders- eg Marfan’s
Causes of mitral valve prolapse
Associated with:
connective tissue disease: Marfan’s, Ehler’s Danlos, osteogenesis imperfecta
Cardiac disease: congenital heart disease, congestive cardiomyopathy, HOCM, myocarditis
Other: SLE, muscular dystrophy, ADPKD
Mitral valve prolapse features
Mid-systolic clic, and/or late syystolic murmur (so normal S1 and GAP before murmur, unlike in mitral regurg)
As in ventricular systole, mitral valve leaflet prolapses to left atriium
Causes tricuspid regurgitation
Most commonly: RV dilation in pulmonary hypertension
rheymatic heart disease
Infective endocarditis (partic IVDU)
Ebstein’s abnormality (if split S1 and S2)
Causes mitral stenosis
Rheumatic heart disease
Causes aortic regurgitation
Acute: infective endocarditis, aortic dissection
Chronic: connective tissue disorders, rhematic heart disease, syphilis, congenital/bicuspid aortic valve, long standing hypertension
Heart failure signs
Tachypnoea/tachycardia
Cool peripheries
Raised JVP
Displaced apex
S3
Bibasal fine creps
Peripheral oedema
ASD signs
Soft, ejection systolic flow murmur (pulm area). Fixed, wide split S2. RV heaveAssociations: Down’s syndrome eg low set ears, flat nasal bridge etc
Watch out for cyanosis = Eisenmenger’s
VSD signs
pansystolic murmur (loudest left sternal edge) associated thrill, RV heave
If causing R heeart failure: raised JVP, peripheral oedema
signs of cor pulmonale
Plethoric facial appearance
central cyanosis
Raised JVP (large A waves)
Giant V waves and pansystolic murmur (if secondary to tricuspid regurg)
R ventricular heave
Palpable/loud S2
Ankle oedema
Signs tetralogy of fallot repair
Sternotomy scar
Latera
Nerve root hip flexion
L2/L3
Nerve root hip extension
L4/5
Nerve root jnee extension
L3/4
Nerve root knee flexion
L5/S1
Nerve root ankle dorsiflexion
L4/5
Nerve root ankle plantar flexion
S1/S2`
Nerve root big toe extension
Pure L5
How many beats of clonus indicates UMN lesion?
> 5
Patellar reflex nerve root
L3,4 (kick the door)
Ankle reflex nerve root
S1/2 (in the shoe)