Orthoptics + Orbital disease Flashcards

1
Q

What is binocular vision and what 3 things is it based on?

A

Brains ability to perceive a single image from each eye
Based on:
Simultaneous perception (good visual acuity in both)
Depth perception
Fusion (alignment of visual axes)

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2
Q

Where are the different categories of squint?

A

Manifest (pathological) - Concomitant/Incomitant

Latent (physiological) - compensated/decompensated

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3
Q

In the cover + alternate cover tests what is seen in convergent + divergent?

A

Convergent (esotropia) - eye moves away from nose after uncovering
Divergent (exotropia) - eye moves towards nose from uncovering

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4
Q

In what cases in the cover tests may fixation not be able to occur? (2)
What sign use instead?

A
V poor visual acuity (can't fixate anyway)
Muscle paralysis (cannot fully take up fixation)

Use corneal reflections as guide

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5
Q

Who are concomitant squints mostly seen in?

What conditions are they associated with?

A

Children (eso*/exo)

Convergents: Hypermetropia (child wears glasses) + Amblyopia

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6
Q

What is amblyopia?

List 3 causes

A

Reduced acuity from failure of development of visual pathways (→ lazy eye)
Causes:
Stimulus deprivation e.g. severe ptosis, congenital cataracts (lack of sharp image on macula at critical stage of devel)
Strabismic (squint) (uncorrected myo/hypermetropia - brain suppresses deviated image)
Anisometropic (diff refractive powers + brain favours better eye)

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7
Q

When should management of a squint be started?

What is the management? (OOO)

A

As soon as noticed (>7yrs too late)
Optical: correct refractive errors
Orthoptic: patch on good eye
Operation: resection/recession of rectus muscles (cosmetic) + Botox

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8
Q

What is seen in a cover test of a manifest squint?

A

When normal eye covered → bad eye corrects itself (takes up fixation) (+good eye deviates)
When good eye uncovered → bad eye deviates again (+ good eye to normal)

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9
Q

What is seen in an alternate cover test in a latent squint?

A

If eye moves (to take up fixation) when uncovered → latent squint

NB latent squint will have in both eyes

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10
Q

What measures of visual acuity can be used in children?

A

Preferential looks (shows have some form of sight)
Cardiff cards / Kays pictures
LogMAR (reading age)

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11
Q

What is an incomitant squint? List the causes (5)

A

Due to CN3/4/6 palsy/paralysis
Microvascular (BP/DM)
Nerve compression (direct - aneurysm; indirect - space occupying lesion raising ICP)
Head injury (esp CN4)
Mechanical (thyroid; orbit inflamm; orbit trauma)
Idiopathic

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12
Q

What symptoms may be seen in orbital disease? (3)

What signs may be seen? (8)

A

Diplopia
Pain/discomfort
Reduced vision

Soft tissue involvement
Exophthalmos (proptosis)
Pseudoproptosis (high myopia/contralat endophthalmos)
Endophthalmos
Ophthalmoplegia
Visual dysfunction
Dynamic changes (pulsation)
Fundus changes
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13
Q

What are some causes of soft tissue involvement in orbital disease? (3)

A

Lid/periorbital oedema
Ptosis
Conjunctival swelling

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14
Q

What are some causes of proptosis/pseudo? (TIT)

A

Thyroid eye disease
Tumour
Inflamm/infection

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15
Q

What are some causes of endophthalmos? (3)

A

Small globe
Structural abn (e.g. blow out)
Atrophy of orbit contents (irradiation/scleroderma)

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16
Q

What are some causes of ophthalmoplegia? (5)

A
Tumour
Infection (cellulitis)
Trauma (blow out)
Restrictive myopathy
Ocular motor nn lesions
17
Q

What kinds of dynamic changes may be seen in orbital disease (2)
And give examples of conditions where these changes might arise (2/3 each)

A

Venous pressure increase: thyroid eye, vascular probs

Pulsation: AV communication, orbital floor defects, CSF pulsation

18
Q

What kinds of fundus changes may be seen in orbital disease (3)

A

Retinal/vascular changes
Disc changes
Choroidal folds

19
Q

What ocular manifestations are seen in thyroid eye disease? (2 main + 2 rare)

A

Soft tissue involvement (eyelid/periorbital oedema)
Exophthalmos (1/3rd)

Diplopia (restrictive myopathy/oedema/fibrosis)
Optic nn compression / neuropathy (open angle glaucoma)

20
Q

What can be given to patients with permanent diplopia?

A

Prisms (alleviate symptoms)

21
Q

What are some symptoms (2) and signs (4) of orbital cellulitis?

A

Severe malaise + fever
Pain

Painful ophthalmoplegia
Severe orbital oedema (+redness)
Ptosis
Optic nn dysfunction (advanced)

22
Q

What are some complications of periorbital oedema (2)

A
Optic neuropathy
Abscess formation (→ cranial if severe)
23
Q

What is idiopathic orbital inflammatory disease?
How does it usually present? (3)
Signs (4)

A

Any non-neoplastic / non-infectious orbital lesion (Dx of exclusion)

20-50yrs unilateral abrupt onset

Unilateral proptosis
Periorbital swelling
Ophthalmoplegia
Chemosis (irritation)

24
Q

What are the main vascular orbital disorders? (2)

A

Orbital varices

Carotid-canervous fistulas

25
What causes orbital varicose? What do they have a tendency to do? How do they present/signs? (2)
Congenital May bleed/thrombose Unilateral, intermittent proptosis accentuated by valsalva manouvre
26
Where is the (carotid-cavernous) fistula connection in direct + indirect?
Direct: B/wn carotid aa + cavernous sinus Indirect: B/wn meningeal branches of internal carotids + cavernous sinus
27
What are some causes of a direct carotid-cavernous fistula? (2) And causes of indirect? (2)
Direct: Head trauma / spontaneous rupture Indirect: Congenital / spontaneous rupture
28
List some clinical features (signs) of a direct carotid-cavernous fistula? (6)
``` Ptosis Chemosis / conjunctival infection Ophthalmoplegia Raised IOP Pulsatile proptosis (w. bruit + thrill) Retinal venous congestion + haemorrhage ```
29
How is direct carotid-cavernous fistula treated?
Ipsilateral carotid compression
30
List some clinical features of an indirect carotid-cavernous fistula? (4)
Dilated episcleral vessels Raised IOP Occasional ophthalmoplegia Mild proptosis
31
What are the different types of tumour that affect the orbit? (7)
Capillary haemangioma Cavernous haemangioma Pleomorphic lacrimal gland adenomas Lacrimal gland carcinoma Optic nerve glioma Optic nerve sheath meningioma Metastases
32
What is the commonest orbital tumour in children? + in adults (benign) What age do most orbital tumours present?
Children → capillary haemangioma Adult (benign) → cavernous haemangioma Most (except capillary haemangioma) middle around (40-70)
33
How may a capillary haemangioma present How is it treated?
Enlarge on coughing / straining Assoc w. systemic conditions (high output cardiac failure) Grow mainly in 1st yr Tx: systemic/IM steroids + local resection (if poss)
34
How may an optic nerve glioma present? (2) | How treated?
Young/teen girls Assoc w. NF-1 Gradual visual loss (slow growing lesion)
35
``` What is the treatment for: pleomorphic lacrimal gland adenoma lacrimal gland carcinoma cavernous haemangioma optic nerve sheath meningioma ```
Pleomorphic - surgical excision Lacrimal carcinoma - radical surgery + radiotherapy (poor prog) Cavernous - surgical excision Optic nn sheath - excision + radio
36
What are the common sites of tumour origin in orbital metastases? (6)
``` Prostate breast Bronchus GI Kidney Skin melanoma ```
37
How is orbital cellulitis managed? What are the indications for surgery? (3)
Admit to hosp + IV Abx Monitor optic nn func (complications) Abx resistance Orbital/subperiosteal abscess Optic neuropathy
38
How is idiopathic orbital inflam disease managed? (3) | What is a complication if not treated?
Steroids Radiotherapy Cytotoxics Severe prolonged activity → frozen orbit