Orthopedics Flashcards

1
Q

What is a slipped capital femoral epiphysis?

A

A salter harris I type fracture where the physis of the femoral head slips
- External Rotational deformity

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2
Q

Clinical features of a slipped capital femoral epiphysis?

A
  • Acute: sudden severe pain with limp
  • Chronic: limp with medical knee or anterior thigh pain
  • Tenderness over joint capsule
  • Resistricted internal rotation, ABduction, and flexion
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3
Q

Treatment for slipped capital femoral epiphysis?

A

Mild-mod: stabalize physis with pins in current position

Severe: ORIF

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4
Q

What are the major complications of slipped capital femoral epiphysis?

A
  • Avascular necrosis
  • Chondrolysis (v articular cartilage)
  • Premature OA
  • Decreased ROM
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5
Q

What is Legg-Calve-Perthes Disease?

A
  • A degenerative disease of the hip joint where growth/loss of bone mass leads to deformity of the ball of the fumur and surface of the hip socket.
  • Characterized by Idiopathic avascular osteonecrosis of the capital femoral epiphysis
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6
Q

What are the key features of Legg-Calve-Perthes Disease?

A
  • AVN of proximal femoral epiphysis
  • Abnormal growth of physis
  • Remodeling of regenerated bone (eventually)
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7
Q

How is Legg-Calve-Perthes disease managed?

A
  • Non-weight bearing, peds ortho referral
  • ROM exercise
  • Brace in flexion and ABduction for 2-3 years
  • Pelvic or femoral osteotomy (surgical removal of bone)
  • 50% do well with conservative tx
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8
Q

Clinical features of Legg-Calve-Perthes disease?

A
  • Child with hip pain and limp
  • Tender over anterior thigh
  • Flexion contracture (decreased internal rotation and ABduction of hip)
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9
Q

What will you see on X-ray in pt with Legg-Calve-Perthes disease?

A
  • Collapse of femoral head
  • Subchondral fracture
  • Metaphyseal cyst
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10
Q

What is compartment syndrome?

A
  • Increased interstitial pressure in an anatomical compartment where muscle and tissue are bounded by faschia and bone with little room for expansion.
  • Interstitial pressure exceeds capillary perfusion pressure leading to muscle and eventually nerve necrosis
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11
Q

What is the pathogenesis of compartment syndrome?

A
  • Increased pressure from blood and intracompartment swelling
  • Decreased venous and lymphatic drainage
  • Intracompartmental pressure becomes greater than perfusion pressure
  • Muscle and nerve anoxia > necrosis
  • Leaky basement membranes cause transudarion into tissue surrounding compartment, increasing pressure
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12
Q

What are the 5 P’s of compartment syndrome?

A
  • Pain (out of proportion to injury)
    • Increased pain with passive strech is most specific sign
  • Pallor
  • Paresthesia
  • Paralysis (late)
  • Pulselessness (late)
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13
Q

What are the clinical features of compartment syndrome?

A
  • Pain with passive stretch (most specific)
  • Swollen, tense compartment
  • Suspicious history
  • 5 P’s
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14
Q

What is the treatment for compartment syndrome?

A
  • Non-operative: Remove constrictive dressing (casts, splints, etc) and elevate the limb to heart level
  • Operative: Urgent fasciotomy
  • 48-72 days post op you can close the wounds and may need to debride necrotic tissue
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15
Q

What are the compartment pressure readings associated with compartment syndrome?

A
  • Elevated compartment pressure: 30mmHg (normal is 0mmHg)

- Ischemia occurs when tissue pressures approach diastolic pressure (get concerned when it’s within 30mmHg of diastolic)

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