Orthopedic surgery Flashcards

1
Q

Define meta-, epi-, diaphysis.

A

epi is at the end (covered with physeal
cartilage)

meta is the “neck” of the joint

dia is mid long bone (usually has a big blood vessel going along it suppling the bone with blood)

Bleeding from bone is classified as parenchymal bleeding.

Externally, the bone is covered with periosteum, absent only at bone ends. The periosteum contains nociceptors.

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2
Q

Location of epiphyseal plates.

Note the anatomy.

A

See image.

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3
Q

What is pictured?

A

The bone cortex is one of the first things you should evaluate on a bone x-ray. Assess the density. The cortical density should be higher than medullar.

The cortex is correctly thicker along the diaphysis and thinner along the epiphysis.

Is an indicator of bone health.

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4
Q

Osseous tissue is a connective tissue and like all connective tissues contains

A

relatively few cells (2% of mass) and large amounts of extracellular matrix.

By mass, osseous tissue matrix consists of 1/3rd collagen fibers and 2/3rds calcium phosphate salt.

The collagen provides a surface for inorganic salt
crystals to adhere.

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5
Q

Osteogenic cells are undifferentiated and develop into

A

osteoblasts.

The osteoblast is the bone cell responsible for forming new bone and is found in the growing portions of bone. Osteoblasts deposit bone matrix.

Osteoclasts develop from a different cell lineage and act to resorb bone.

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6
Q

When osteoblasts get trapped within the calcified matrix, they become

A

osteocytes.

Osteocytes maintain bone tissue.

Osteoclasts develop from a different cell lineage and act to resorb bone.

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7
Q

Most common orthopaedic conditions in dogs. (8)

A

¤ Hip Dysplasia
¤ Elbow Dysplasia

¤ Cranial Cruciate Ligament Rupture
¤ Patellar Luxation

¤ Secondary osteoarthritis
¤ Panosteitis

¤ Legg-Calve-Perthes disease
¤ Bone neoplasia

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8
Q

Canine hip dysplasia (CHD) is

A

a genetic and multifactorial developmental disorder
characterized by hip joint laxity, degeneration, and
osteoarthritis (OA).

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9
Q
A
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10
Q
A
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11
Q

Canine elbow dysplasia (CED) is

A

a disease of the elbows of dogs.

Elbow dysplasia includes any mismatch of growth and development between the radius and ulna.

This in turn can cause damage to the joint cartilage and even fracture the tip of the coronoid process.

First look for a gap in the elbow joint. It should be a nice, round space so the bigger the “step”, the worse the incongruence and thus elbow dysplasia.

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12
Q
A

ununited anconeal process (UAP) a form of elbow dysplasia

Is a developmental abnormality in which the anconeal process does not fuse with the ulna.

The loose piece of ulna should be removed.

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13
Q

Panosteitis is

A

a spontaneously occurring, self-limiting (should resolve in 3ish weeks) disease of young, large breed dogs involving both the diaphyseal and metaphyseal areas of the tubular long bones.

Panosteitis begins with adipocyte degeneration,
intramembranous ossification, and bony remodeling that results in medullary fibrosis and periosteal/endosteal new bone formation.

Tx with NSAIDs for a few weeks.

palpation of the bones is painful but joint flexion is not.

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14
Q
A

left image shows some increased medullary density.

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15
Q
A

panosteitic changes in the medulla (increased density)

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16
Q

Legg-Calve-Perthes disease

A

Legg-Calve-Perthes disease is also known as avascular or aseptic necrosis of the femoral head.

This is a condition in which the head of the femur
spontaneously begins to degenerate.

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17
Q
A

osteosarcoma

Usually seen at:
* distal femur
* proximal tibia
* distal radius
* porximal humerus

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18
Q
A
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19
Q
A

osteosarcoma

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20
Q

Most common orthopaedic
conditions in cats. (3)

A

¤ Primary osteoarthritis (pictured)

¤ Hip dysplasia (Siamese cats, Maine Coon)

¤ Slipped capital femoral epiphysis which is essentially just feline legg-perthes’ (Maine Coon)

21
Q
A

which is essentially just feline legg-perthes’

22
Q

The orthopedic examination can begin prior to assessment by

A

considering the patient’s signalment. The astute clinician will consider differential diagnoses based
on:
¤ Age
¤ Breed
¤ Presenting complaint

23
Q

Breed predispositions in:
¤ Elbow dysplasia:
¤ Medial patellar luxation:
¤ Cranial cruciate ligament disease:

A

Elbow dysplasia:
¤ Labrador retriever
¤ Rottweiler
¤ German shepherd dog
¤ Staffordshire bull terrier

Medial patellar luxation:
¤ Chihuahua

Cranial cruciate ligament disease:
¤ Rottweiler
¤ Labrador retriever

24
Q

An orthopedic examination begins by

A

observing the animal for signs of lameness. The correct limb may not have been identified by owner.

The animal should be observed for reducing the weight placed on the affected limb when standing or sitting.

Unilateral or bilateral muscle atrophy and abnormal
muscle development, instability, or crepitation (e.g., fracture).

It is preferable to begin examining a healthy limb to
identify the individual’s normal response to manipulation and pressure.

Asymmetry may indicate tumor, abscess, atrophy, joint swelling, or greenstick fracture.

Greenstick fractures are a type of broken bone that happens when a bone bends enough to crack on one side without snapping into multiple pieces.

25
Animals with forelimb lameness will
lift their heads after the lame limb strikes the ground in an attempt to remove weight from the affected limb. A short stride occurs when the animal has a decreased range of motion in a diseased joint. ## Footnote "down on sound"
26
After the lame limb has been identified, the animal should be
palpated, and a screening neurologic examination should be performed. The examiner should develop a consistent evaluation procedure. You can start at the front of the animal and work toward the rear, or do opposite. Starting at the toes of each limb and progressing proximally. Joints should be isolated and moved through a complete range of motion to detect crepitation, pain, or abnormalities in range of motion.
27
Cruciate ligaments test.
Test the integrity of the cruciate ligaments by trying to elicit a cranial or caudal drawer motion or by performing a tibial compression test to elicit cranial tibial thrust. Drawer movement is caused by the tibia sliding cranially or caudally in relationship to the femur. This motion is not possible when the cruciate ligaments are intact in adult animals
28
What is the tibial compression test?
Another test for cranial cruciate ligament rupture in addition to the drawer test. an be performed with the patient standing or in lateral recumbency (affected leg up), awake or sedated. The stifle is held in slight flexion. The index finger of one hand is placed over the tibial crest. The other hand flexes & extends the hock. If the ACL is torn, the tibial tuberosity will move cranially, ever so slightly, as the hock is in the flexed position. The reason for this displacement is that hock flexion causes tension of the gastrocnemius muscle, which in turn displaces the tibia cranially. This is called tibial compression or cranial tibial thrust.
29
What are extracellular vesicles?
Extracellular vesicles (EVs) are cell-derived membrane-surrounded vesicles that carry bioactive molecules and deliver them to recipient cells. Classical EVs are exosomes, microvesicles, and apoptotic bodies. These look different in health vs with cancer. In the future, the aim is that these can be analyzed to test for neoplastic disease such as osteosarcoma.
30
Palpating for hip luxation:
Use the position of the greater trochanter in relationship to the tuber ischium as a landmark. In the standing animal, compare the distance from the greater trochanter with the tuber ischium bilaterally. With hip luxation, the trochanter rolls over the thumb.
31
To detect hip laxity associated with hip dysplasia perform...?
perform the Ortolani maneuver to detect hip laxity associated with hip dysplasia With the animal in lateral recumbency, Place one hand over the dorsal pelvis. Grasp the stifle with the other hand, and orient the femur parallel with the table surface. Simultaneously adduct and push the stifle toward the pelvis. Maintain the pressure and abduct the stifle. As the femoral head returns to the acetabulum, use the hand stabilizing the pelvis to detect a click.
32
pathologic or spontaneous fractures may be caused by internal pathologies such as: (4)
¤ neoplasia (osteoma, osteosarcoma) ¤ metabolic disorder ¤ hormonal diseases ¤ age–related changes
33
Bone fractures are classified according to several criteria: (5)
¤ etiology, ¤ presence of external injuries, ¤ localisation/course of fracture line, ¤ bone damage, ¤ fracture stability Closed fractures – are those, where the ends of the fractured bone do not contact external environment. Open fractures – are those, where the skin and surrounding soft tissues are penetrated or damaged, and bone fragments contact external environment. These fractures are commonly infected and their recovery may be complicated.
34
Bone fracture Classification according to localization:
Tubular bone fractures may be classified as: ¤ Metaphyseal fractures ¤ Epiphyseal fractures ¤ Diaphyseal fractures
35
Bone fracture Classification according of fracture line. (8)
¤ Transverse fracture – fracture line is perpendicular in relation to the longitudinal axis of the bone. ¤ Oblique fracture – there is an acute angle between the fracture line and longitudinal axis of the bone. ¤ Longitudinal fracture – fracture line goes along the longitudinal axis of the bone. ¤ Spiral fracture – fracture line goes spirally along diaphysis of the tubular bone. Simple fracture – the bone is fractured in only one place and is split into two parts. Either: ¤ Incomplete simple: Fracture through only one cortex ¤ Complete simple: Fracture through both cortices ¤ Comminuted fracture – the type of fracture that results in numerous small splinters. ¤ Articular fracture – fracture line penetrates articular cavity; this type of fracture is often termed – intraarticular fracture. ¤ Avulsion fractures occur when the insertion point of a tendon or ligament is fractured and distracted from the rest of the bone.
36
Name the fracture types.
37
A greenstick fracture is
occurring in immature animals, is an incomplete fracture in which a portion of the cortex is intact, thus stabilizing the bone to some extent.
38
Avulsion fractures occur when the insertion point of a tendon or ligament is fractured and distracted from the rest of the bone.
39
Describe Bone fracture healing.
Osseous tissue heals after the fracture via bone callus formation. Periosteum, endosteum (the layer covering the cortex from inside), bone marrow, and blood vessel endothelium participate in the regeneration process. Four phases may be distinguished in the process of callus formation. * Phase 1, inflammation * Phase 2, soft callus granulation * Phase 3, hard callus (ossification) * Phase 4, remodeling
40
Four phases may be distinguished in the process of callus formation:
* Phase 1, inflammation * Phase 2, soft callus granulation * Phase 3, hard callus (ossification) * Phase 4, remodeling
41
Describe bone fracture healing. Phase 1 – Inflammation: (4)
Occurs over the first 48–72 hours after the fracture, a serous aseptic inflammation, exudation, and migration of leukocytes and inflammatory mediators occur in the fracture area. Haematoma! Demineralisation and resorption occurs at bone ends due osteoclasts and their ferment – acidic phosphatase. Fibrin is the first supportive tissue that holds bone fragments together.
42
Describe bone fracture healing. Phase 2 – soft callus formation (granulation): (4)
Occurs over the 3–4 days after the trauma, connective or fibrous matrix starts to form. At first, osteoid tissue is formed in periosteum, endosteum, and bone marrow and, then, develops further to the fracture line. New blood vessels start growing into the clot from the vessels coming from the medullary cavity, periosteum, and other surrounding soft tissues. Granulation tissue develops above the clot and forms connective tissue callus.
43
Describe bone fracture healing. Phase 3 – hard callus formation (ossification):
Ossification starts on day 10–12. Callus size is directly related to the stability of the fracture. If the fracture is not stable, callus may be relatively large to redistribute and decrease pressure in the area. If fragment fixation is good, irregularly located osseous tissue elements appear in the primarily cartilaginous callus.
44
Describe bone fracture healing. Phase 4 – bone remodelling:
¤ The newly formed osseous tissue has not yet acquired its final structure and its function is incomplete. ¤ When the limb starts performing support and motion functions, the callus is staticodynamically restructured according to functional needs. ¤ Eventually, the fracture site becomes architecturally similar to the normal bone. ¤ This is a relatively long process that may take years, depending on the age and general state of the animal.
45
Stabilization of the bone fracture patient.
Administer proper analgesia as soon as possible. Ideal analgesics are pure mu opioids, such as: * Methadone: Dogs, 0.2 to 0.3 mg/kg; cats, 0.1 up to 0.3 mg/kg; IV or IM q6-8H * Fentanyl: Dogs/cats, 2 mcg/kg loading dose followed by 2 to 10 mcg/kg/h CRI (caution with cats). Check and stabilize vitals (temperature, pulse quality and heart rate, respiration rate, blood pressure, pulse oximetry), if needed. Perform thorough physical, orthopedic, and neurologic examinations. Pursue diagnostics such as blood analysis, thoracic and abdominal radiographs, and an AFAST ultrasound (if needed). Resolve any life-threatening issues, which means that surgery may need to be delayed for several days due to conditions, such as pulmonary contusions or hypovolemia.
46
Bone fracture clinical signs includes: (6)
¤ Pain ¤ Oedema ¤ Deformation ¤ Abnormal position of the limb ¤ Limb dysfunction ¤ Crepitation
47
Several options exist for fracture stabilization, and numerous factors must be considered when deciding on an appropriate fixation method. Such as: (4)
¤ Patient factors: Size, age, weight, breed, activity level ¤ Client factors: Finances, compliance, husbandry ¤ Fracture factors: Configuration, location, forces, contamination ¤ Veterinarian factors: Understanding of biomechanics and healing, knowledge of particular implants, implant availability
48
Describe conservative fracture tx.
The main aim of conservative treatment method is to restore anatomic position of the the injured limb and ensure the maximal immobility of the fractured area. When conservative treatment method is used, the ends of the fractured bone are only immobilised. No immobilising construction can inflexibly support the bone. There is always soft tissue between the bone and the immobilizer that cannot be pressed too strongly because it may cause oedema, blood supply problems, osteoporosis, and even necrosis.
49
Describe surgical fracture tx.
¤ Assemblage of fractured bone fragments during a surgery is termed osteosynthesis. ¤ Fractured bone ends are assembled via osteosynthesis using various equipment. ¤ Metal constructions enable assembly of bone fragments efficiently and provide them with necessary strength reserve.