Orthopaedics Flashcards
Principles of fracture management
Reduce
Hold
Rehabilitate
Pathophysiology of osteoarthritis
Degradation of cartilage and remodelling bone due to active chondrocytes
Release of enzymes break down collagen and proteoglycans destroying articular cartilage
Exposure of underlying subchondral bone -> sclerosis
Reactive remodelling -> formation of osteophytes and subchondral bone cysts
Loss of joint space
Risk factors for OA
Primary Secondary - trauma - infiltrative disease - connective tissue disease Obesity Advancing age Female gender Manual labour occupations
Clinical features of OA
Small joints of hands and feet, hip joint and knee joint
Insidious, chronic and gradually worsening
Pain and stiffness - worsened with activity
Deformity and reduced range of movement
Bouchard nodes - swelling of PIPJs
Heberden nodes - swelling of DIPJs
X-ray features of OA
Loss of joint space
Osteophytes
Subchondral cysts
Subchondral sclerosis
Management of OA
Conservative - education - weight loss - physiotherapy Medical - simple anagesics - topical NSAIDs - intra-articular steriod injections Surgical - osteotomy - arthrodesis - arthroplasty
Outcomes of open fractions
Skin
Soft tissues
Neurovascular injury
Infection
Management of open fractures
Urgent realignment and splinting of limb Broad spectrum antibiotic cover Tetanus vaccination Photograph wound Remove gross debris Dress in saline-soaked gauze Debridement of wound and fracture site Skeletal stabilisation
Define compartment syndrome
Critical pressure increase within a confined compartmental space
Pathophysiology of compartment syndrome
Fluid deposition in compartment -> intra-compartmental pressure
Increase in hydrostatic pressure
Compression of traversing nerves - paraesthesia
Arterial inflow compromised
Clinical features of compartment syndrome
Usually present within hours Severe pain - disproportionate to injury - worse by passively stretching muscle bellies of muscles - not improved by analgesia Parasthesia
Investigations for compartment syndrome
Diagnosis is clinical
Intra-compartmental pressure monitor
Creatine kinase
Management of compartment syndrome
Keep limb at neutral level
Improve oxygen delivery with high flow oxygen
Augment blood pressure with bolus of IV crystalloid fluids
Remove all dressings/splints/casts
Treat symptomatically
Surgical fasciotomy
Monitor renal function - rhabdomyolysis or reperfusion injury
Main causative organisms of septic arthritis
S.aureus
Streptococcus spp
Gonorrhoea
Salmonella
Risk factors for septic arthritis
Age > 80yrs Pre-exisiting joint disease DM Immunosuppression Chronic renal failure Hip or knee joint prosthesis IV drug use
Clinical features of septic arthritis
Single swollen joint causing severe pain
Pyrexia
Red, swollen warm joint that’s painful on active and passive movements
Investigations for septic arthritis
Routine bloods - FBC, CRP, ESR
Blood cultures
Joint aspiration
Plain radiograph
Management of septic arthritis
Empirical antibiotic treatment
Surgical irrigation and debridement
Complications of septic arthritis
Osteoarthritis
Osteomyelitis
Define osteomyelitis
Infection of bone
- caused by haematogenous spread, direct inoculation or direct spread from nearby infection
Common causative organisms of osteomyelitis
S. aureus Streptococci Enterobacteur spp H.influnzae P.aeruginosa Salmonella spp.
Risk factors for osteomyelitis
Diabetes mellitus
Immunosuppression
Alcohol excess
IV drug use
Clinical features of osteomyelitis
Severe pain
- constant
- worse at night
Low grade pyrexia
Investigations for osteomyelitis
Routine bloods Blood cultures Plain film radiographs - osteopaenia - periosteal thickening - endosteal scalloping - focal cortical bone loss Culture from bone biopsy at debridement