Orthopaedics Flashcards
Fracture Management
Resuscitate
- A-E, clinically stable
- analgesia
- Assess neurovascular status
- Obtain 2-view radiographs
Reduce
- Close e.g. MUA, Skin Traction, skeletal traction
- Open- ?where anatomic reduction is key e.g. intrarticular ‘#
Restrict (hold) prevents displacement/pain relief/facilitation of funciton and early mobilisation
-
Conservative:
- non-rigid e.g. sling
- rigid e.g. plaster of paris, bracing
-
Surgical:
- internal fixation; plates, screws, k wire
- External fixation
Rehabilitate
Early movement and physio
Fracture complications
(immediate, early, late)
Immediate <24hrs
- Neurovasc damage; arteriest, veins, nerves (neuropracia)
- Tissue damage; muscles
- Fat embolism; resp distress after #
Early <1wk
- Compartment syndrome
- Infection
- Gen; anaesthesia, prolonged bed rest (DVT)
Late <1mnth (long term >1mnth)
- Bone healing-malunion/non-union
- Avascular necrosis
- OA
- Myositis ossificans
- complex regional pain syndromes type 1 (Sudek’s atrophy)
Immediate ‘resuscitation’ Mx of open #
Resuscitation
- A-E resus
- Control bleeding; direct pressure
- Pain relief.
- Antibiotics within 3 hours. Vary with Gustilo classification (1-3: small wound/simple #, medium wound, large wound/compound #)
-
Assessment;
- Neurovascular damage?
- Soft tissue damage
- gross debris removal
- PHOTOGRAPH
- Dress with sterile saline-soaked gauze,
Splint until patient can go to theatre; Decreases pain, clot disruption and further neurovascular/soft tissue injury
‘Viva’; what is osteoarthritis (define)
OA is a degrenerative chronic joint disorder in which there is progressive loss of cartilage with new bone formation at the joint surface
Viva OA
- Presentation (history)
- Examination
- risk factors
- Joints affected (classic)
- Diagnosis
Presentation: Commonly an elderly patient complaining of joint pain. Worse on exertion and at the end of the day. Stiffness after resting.
Examination: Reduced range of movement, painful movement. Bouchards/Herbeden’s nodes.
Risk factors: Age, obesity, joint abnormality, previous trauma.
Joints classically affected: Knee, hip, DIPs and PIPs.
Diagnosis: History and examination. Imaging findings confirm.
Dx (and findings)
OA radiographic changes:
- Joint space narrowing
- Sclerosis
- Osteophytes
- subchondral cysts
OA management
(conservative/medical/surgical)
Conservative:
- Losing weight
- Physiotherapy
- Walking aids etc. (occupational therapy)
Medical:
- analgesia; paracetamol WHO pain ladder
- Steroid injections
Surgical:
- Arthroscopy +/- cartilage excision/remove loose bodies
- Arthroplasty
- Arthrodesis (palliative)
- Realignment osteotomy (biomechanics, younger)
Special Tests!
●Spine
●Shoulder
●Elbow
●Hand and wrist
●Hip
●Knee
●Foot and ankle
●Spine – Schober’s, Straight leg raise
●Shoulder – rotator cuff (Supra, Infra, Teres m, Subscap)
●Elbow – medial and lateral epicondylitis
●Hand and wrist – Tinel’s (c. tunnel) Finkelstein’s (tenosynovitis)
●Hip – Thomas’s test, Trendelenberg’s
●Knee – Cruciate, Collateral and Cartilage (Mcmurray’s)
●Foot and ankle – Simmond’s (Achilles’ rupture)
What is Trendelenburg’s test
Trendelenburg’s test of hip abductor strength. Weak muscle on the contralateral side that the pelvis drops as it fails to abduct and keep pelvis stable when foot is lifted off the floor.
‘Sound side sags’
Hip related Gaits (define and likely cause)
Antalgic
Trendelenburg
Circumduction
Antalgic;
- chronic hip pain rsults in avoidance of use and stiffness. classic ‘limp’
- likely causes= OA (other’ bursitis, musculoligamentous strain, femoroacetabular impingement)
Trendelenburg
- weakening of hip abductors causes pelvic instability. The patient corrects this by lurching their body towards the side of the weakness.
- Likely cause = Gluteus medius strain. Superior gluteal nerve lesion (compression, inflammation, trauma).
Circumduction
- Gait seen in hemiplegia/hemiparesis. With a lack of knee and hip flexion the affected foot is dragged in front of the body in a semicircle during the swing phase
- likely cause: stroke most common. Other neuro conditions.
What is Thomas test?
Thomas’s test
- Place hand under patient’s spine.
- Passively flex both legs (hips/knees) as far as you are able to.
- Your hand should detect the lumbar lordosis is obliterated.
- Ask patient to fully extend the hip you are assessing.
- Failure to flatten leg shows a fixed flexion deformity.
Causes of a fixed flexion deformity:
- Osteoarthritis of knee or hip.
- Other causes depend on the structure involved:
- Skin – burns and scar tissue cause contractures
- Muscles – hamstring contracture
- Joint – intra-articular fractures, septic arthritis.
Define: True and Apparent hip lengths
Causes of discrepancy (>2cm) in true leg length (and therefore apparent leg length):
x3
Causes of discrepancy (>2cm) in apparent leg length with equal true leg lengths:
x2
True = ASIS to medial malleoulus
Apparent = Midline point (xiphisternum/umbilicus) to medial malleolus
Significant difference = 2cm
Causes of discrepancy in true leg length (and therefore apparent leg length):
- Congenital
- Fracture (NOF acutely, chronically after growth plate fracture)
- Post total hip replacement
Causes of discrepancy in apparent leg length with equal true leg lengths:
- Spinal pathology e.g. scoliosis
- Pelvic pathology e.g. hip abduction/adduction contracture
Finding on Ex of hip OA
Gait/look/feel/move/special
GAIT – Antalgic if severe. Walking aids.
Look – ?arthroplasty/arthroscopy scars
Feel – May have pain on palpation of greater trochanter.
Move – Reduced range of active and passive movement.
Special test’s – Thomas’ positive. Fixed flexion deformity.
Hip arthroplasty scars
and potential injuries!
Hip arthroplasty scars, incisional approach:
Anterior/lateral/posterior
Anterolateral approach: Femoral nerve damage. Abductor weakness. Increased risk of infection
Posterior approach: Sciatic nerve damage. Increased risk of dislocation
Also consider femoral diaphyseal #
Reasons for hip arthroplasty
Reasons for arthroplasty:
- Fracture repair in an individual unlikely to heal with fixation alone
- Erosion of the hip joint such that a replacement will improve quality of life e.g. osteoarthritis.
Hip # types
- Intracapsular; subcapital, transcervical, basicervical
- Extracapsular
- intertrochanteric
- subtrochanteric
Why does it matter?
Intracapsular fractures are at risk of avascular necrosis of the femoral head.
What is Garden classification
Garden classification of intracapsular fractures.
Higher grade = higher risk of AVN.
- incomplete #
- Complete # (-displacement)
- Complete # + Partial Displacement
- Complete # + Full Discplacement