Orthopaedic hip conditions Flashcards

1
Q

what point of the femur is the attachment for the abductors and rotators?

A

greater trochanter

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2
Q

what point of the femur is the attachment of the psoas major?

A

lesser trochanter

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3
Q

what type of structure is the labrum?

A

fibrocartilage

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4
Q

what is the main blood supply of the hip joint?

A

branches of the profunda femoris to the capsule

- left and middle femoral circumflex arteries

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5
Q

a patient presents with pain and worsening stiffness of the hip joint. what is the most likely diagnosis?

A

osteoarthritis of the hip

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6
Q

where is the trochanteric bursa located?

A

between the hip abductors and the illiotibial band

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7
Q

what are the causes of trochanteric bursitis?

A

trauma

overuse; (athletes, often runners, repetitive movements)

abnormal movements;

  • distant problem i.e. scoliosis
  • local problem (muscle wasting following surgery, total hip replacement, osteoarthritis)
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8
Q

what is the presentation of trochanteric bursitis ?

A

localised pain in the lateral hip

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9
Q

what is the clinical presentation like in trochanteric bursitis?

A

look:

  • may have scars from previous usrgyer
  • may have muscle wasting of gluteals

feel:
- tenderness over greater tuberosity

move:
- worst pain in active abduction

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10
Q

what investigations would you carry out if you suspected trochanteric bursitis?

A

MRI
X-ray
USS (therapeutic as well as diagnostic i.e. guided injection)

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11
Q

what is the treatment for trochanteric bursitis?

A

non-operative:

  • NSAIDS
  • rest / activity modification
  • physiotherapy
  • steroid injection

surgical:
- bursectomy

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12
Q

what is avascular necrosis?

A

death of bone due to loss of blood supply

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13
Q

in what age and sex is avascular necrosis more common?

Is it more commonly unilateral or bilateral?

A

females > males
age: 35-50yrs
80% bilateral

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14
Q

what are the risk factors for avascular necrosis?

A
trauma i.e. intracapsular fracture 
systemic:
- idiopathic
- hypercoaguable states
- steroids
- haematological (sickle cell, lymphoma, leukaemia)
caisson's disease 
alcoholism
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15
Q

briefly describe the pathway involved in development of idiopathic avascular necrosis.

A
coagulation of interosseous microcirculation
venous congestion
retrograde arterial thrombosis
intraosseous hypertension
reduced blood flow
cell death 
chondral fracture and collapse
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16
Q

what is the presentation of avascular necrosis?

A

insidious onset of groin pain
pain with stairs, walking uphill and impact activities
limp

17
Q

what is the examination of someone with avascular necrosis like?

A

mostly normal

may present like early arthritis with stiffness and decreased range of motion (esp internal rotation)

18
Q

what action of the hip does avascular necrosis restrict?

A

internal rotation

19
Q

what are the treatments for avascular necrosis?

A

non-operative:

  • reduce weight bearing
  • NSAIDS
  • bisphosphonates
  • anti coagulants
  • physiotherapy

surgery:

  • core decompression (restore blood supply)
  • rotation osteotomy
  • total hip replacement (last resort)
20
Q

what are the 2 categories of femoral acetabular impingement ? (FAI)

A

cam lesion

pincer

21
Q

what is femoral acetabulum impingement? (FAI)

A

anatomical phenomenon which results in impingement of the femoral neck against the anterior edge of acetabulum

22
Q

what is the pathology in a cam lesion which causes FAI?

A

extra bone resulting in decreased femoral head:neck ratio

aspherical head

23
Q

in what type of patients is a cam lesion in FAI most commonly found?

A

usually in young athlete males who do a lot of hip flexion i.e. rowers

24
Q

what is the difference between cam lesion and pincer FAI?

A

cam lesion = femoral based impingement

piper = acetabulum based impingement

25
Q

in what patients does since FAI usually occur?

A

active females

26
Q

what is the pathology that causes pincer FAI?

A

abnormal acetabulum leading to;

  • anteriosuperior rim overhang
  • acetubular protrusion
27
Q

what are associated injuries with FAI?

A

label degeneration and tears
cartilage damage and flap tears
secondary hip osteoarthritis

28
Q

what is the presentation of FAI?

A

groin pain - worse with flexion
mechanical symptoms;
- block to movement
- pain with certain manoeuvres i.e. squatting, lunging, getting out of a chai

29
Q

what clinical signs are present with FAI?

A

positive FADIR test - flexion adduction and internal rotation

30
Q

what are the treatment options for FAI?

A

non-operative;

  • activity modification
  • NSAIDS
  • physiotherapy

operative:

  • arthroscopy
  • resection
  • periacetubular osteotomy
  • hip arthroplasty
31
Q

in what group of patients is labral tear most commonly found in?

A

all age groups
commonly active females
- pincer FAI
- more flexible

32
Q

what are the causes of a labral tear?

A
FAI
trauma
osteoarthritis
dysplasia 
collagen diseases i.e. Ehlers-danlos syndrome
DDH
33
Q

what is the presentation of a labral tear of the hip?

A

extreme groin or hip pain
snapping sensation
jamming or locking of the hip

34
Q

what clinical sign may be positive on examination in a patient with a labral tear?

A

positive FABER test
- flexion, abduction, external rotation (for anterior tears)

examination can be normal

35
Q

what is the treatment for a labral tear?

A

non-operative:

  • activity modification
  • NSAIDS
  • physiotherapy
  • steroid injection

operative:
- arthroscopy (repair or resection)