Ortho unit 3 Flashcards
knock knees and bow legs
and management
bow legs = genu varum (knees out, space between feet deminishes)
knock knees = genu valgum (knees in, space between feet increases)
normal space between feet is 4cm.
by the age of 7 most will have developed normal alignment
milestones for sitting independently, standing and walking
9 months
12 months
20 months
in toeing (pigeon toed) - what is it and what are the causes
often exaggerated when running
often referred because of clumsiness
causes :
- fem neck angle variation (during late development of foetus the leg rotates on the pelvis so the acetabulum points backwards and the fem head on the neck is forward. sometimes this rotary process inst complete by birth so the neck is more anteriorly rotated (anteverted) than normal. so kids born like this can internally rotate their femur lots and not externally rotate much. this is reflected in their posture and gait - in toeing)
- tibial torsion (bone is distorted/warped along its vertical axis). this is a normal variation and should be ignored
- abnormal forefeet - partic the hooked (adducted) forefoot is common. majority correct and residual hooking rarely causes functional difficulties
flat foot - types and management
normal variation, rarely causes functional abnormalities except uneven shoe wear
can be rigid or mobile
most are mobile and innocuous
all kids feet are flat at birth and arch may not form until 7.
rigid flat foot is rare and implies bony abnormality - occasionally it is a sign of a more serious disease eg RA
curly toes - management
minor overlapping especially 5th toe is common.
most correct
occasionally the crossed 5th toe causes discomfort in shoes - surgery to fix but it is discouraged
knee pain in adolescence causes
common cause of referral
usually 10-12yrs
more common in girls when they develop secondary sex characteristics
osgood schlatters
adolescence knee pain
congenital dislocation of hip
simple walking problems and difficulties in posture in childhood causes
knock knees and bow legs
in toeing
flat foot
curly toes
osgoods schlatters - describe, cause, who gets it, what are the symptoms and management
inflammation of the attachment of the patella tendon to the growing tibial epiphysis caused by excess traction by the quads.
cause unknown
more common in active kids
causes tenderness and discomfort. worse after exercise. may be swelling episodic and can be treated with rest
symptoms cease in middle adolescence when epiphysis fuses
adolescent knee pain - who, what is it, management
more common in girls
rarely on arthroscopy an area of patella cartilage is seen to be eroded - chondromalacia patellae.
most grown out of it but if symptoms persist - arthroscopy
congenital dislocation of hip
incidence screening test signs management
1-2/1000 births
better name is congenital hip dysplasia - it reflects the abnormality of the fem head, acetabulum or both.
more common in girls, with familial and racial tendency
can be bilateral
all kids are screened at birth, 3,6 and 12 months.
test - try to dislocate and relocate. may produce a slight click (suspicious) or definite clunk. if undetected it will be obvious in later life.
clinical signs = shortening of limb, asymmetrical skin creases, limited abduction and a limp
if a click - reexamine at 3 months when a radiograph can be justified
clunks - treat from birth. fem head is relocated and maintained in acetabulum using splintage
if discovered late but before wt bearing - treat with gentle traction then open/closed manipulation. then splint in plaster for 3 months.
if late and walking has commenced - major surgery is needed to deepen the underdeveloped acetabulum and reangulate the fem neck to stabilise the hip/ not great results and 2nd arthritis is common.
club foot
diff forms and causes of these
treatment for diff forms
follow up
talipes equino varus
if treat early - fully correct mild cases and major cases result can be much improved and functional.
common
mild postural form and fixed form
mild form = after breech birth (related to position in womb).
fixed form = ass. wth developmental abnormalities of nerves and muscles in leg.
can be bilateral
mild form is corrected by manipulation at birth
severer forms need surgery
both cases begin with gentle stretching, firstly correcting the hindfoot equinus and secondly correcting the mid and forefoot varus
mild cases - 6 weeks stretching and strapping in a corrected/overcorrected position is enough
in severe cases - reassess after 6 weeks if correction is incomplete - surgery.
follow up all kids until feet stop growing (14yrs) as late relapse needs surgery. the affected foot is usually smaller
spina bifida
diff types
incidence
symptoms and consequences
management
abnormal development in the first 3 months of developing foetus
spina bifida acculta
- minor bony abnormality
- 2 % population
- usually no significance but some develop mechanical back pain and some may get tethering of the spinal cord to the higher lumbar vertebrae during growth - diastamatomyelia
spina bifida cystica
- neural plate tissue open with little/no skin or bony cover
- nerve tissue may be covered with a cyst (meningomyelocele)
- may have hydrocephalus leading to mental retardation and increased size of head
-many die soon after brith
- some survive surgery to close the lesion but will have many problems eg paralysis, growth deformaties through muscle imbalance and incontinence
- many need early surgery to feet to maintain functional shape
- others develop joint contraction causing fixed flexed knees and dislocation of hips
- keep kids mobile so they grow to reasonable size
- many walk with splints and aids
at adolescence they may go into a wheelchair as its easier
what is diastamatomyelia
in spina bifida acculta - may get tethering of the spinal cord to the higher lumbar vertebrae during growth
cerebral palsy
what is it and cause
diff types
symptoms/signs
management
caused by abnormality in brain, often damaged at birth and results in delayed or arrested development of mervous and MSK system
spinal tissue develops normally so children have uninhibited reflexes but lack co-ordination and purpose of movement. normally controlled b brain - spastic paralysis. some muscles contract strongly in an uncoordinated way (spastic) whilst others are weak and flaccid. this imbalance = abnormal muscle and bone growth with secondary deformities of joints
one arm and one leg on same side = hemiparesis
2 legs = paraparesis
all 4 limbs = quadraparesis
most are retarded and some are blind/deaf. some will only have one muscle group affected so v minor spasticity - common sign is toe walking in adolescence - when examined it shows calf spasticity and some need tendo-achilles lengthening before growth ceases
deformaties are minimised by physio. splintage used with caution as it can cause increased spasm and deformity. careful use of surgery to lengthen/tighten tight muscles or denervate them or occasionally to move them
scoliosis
what is it and who gets it
cause
complaints and symptoms
management
curvature of the spine with rotatory abnormality of vertebrae. 3D deformity based on abnormal lordosis of spine which leads to bucking and twisting of vertebra column as a result of the actions of muscles and gravity
most idiopathic cases occur in teens and more common in girls
rarely causes physiological disturbances
complains of twisting ribs - causes hump on one side of shoulder. girls complain skirt hangs crooked. may be painful but this is usually secondary to anxiety
not all curves progress. if it is progressive offer treatment early. braces have NO BENEFIT and increase stigma of disease. surgical correction - v complex