Ortho ligaments and tendons + miscellaneous bone Flashcards

1
Q

What are ligaments and tendons made of

A

Type 1 collagen 98% in proteoglycan matrix

= made by fibroblasts

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What is the difference in blood supply to tendons and ligaments

A

Poor to both
Better to tendons since comes from multiple sites rather than the just insertion points

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

How do tendons and ligaments heal

A

Scar tissue
Very slow healing; gets back to only 60% by 6 weeks

Always susceptible to re-injury

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Ligament injury types; sprains - how we treat them

A

type I = minimeal tearing, some internal haemorrhage - just rest and support for 2 weeks

type II = partial tearing and stretching of fibres; with haemorrhage - do surgical repair = suture ligament or prosthetic

Type III = complete rupture or avulsion of attachment; surgical repair and prosthetic replacement; may need arthrodesis in some instances

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

How are collated ligaments usually injured

A

Trauma e.g RTA

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

What is a stressed radiograph useful for

A

Proving instability in a joint and checking at which level it is
By tying above and below the joint and pulling in opposite directions to stress

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

How do we treat collateral ligament injuries

A

Surgery unless no instability
Primary repair of ligament; then reinforce with prosthetic repair and immobilise joint for a while then controlled mobilisation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How should we deal with open ligament injuries

A

These are typically shear injuries from being dragged along floor by car

Need to reinforce joint and protect it
Don’t necessarily need surgery can just do external fixation and allow soft tissues to granulate

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

What is an arthrodesis

A

Permanent rigid fusion of a join
Salvage procedure

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

What are the two forms of carpal hyperextension

A

Acute rupture due to jump down injury; more paindul

Degeneratime form = insidious onset and less painful

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

How do we treat carpal hyperextension due to ligament rupture/degeneration

A

Can’t do primary repair because more than one ligament usually affected
So do carpal arthrodesis (two types)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What are the two types of carpal arthrodesis

A

Partial carpal: = from radiocarpal bone down so will have most movement of the joint BUT often poor outcomes as these is still disruption at the top level

Pancarpal arthrodesis spans the entire carpus

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are the two forms of plantar ligament rupture

A

Traumatic assocaited with jump down

Degenerative form; typically in Shetland sheedogs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What does an animal with plantar ligament rupture/degeneration look like

A

See deviation in tarsus like an extra joint

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

Treatment options for plantar ligament rupture

A

Partial tarsal arthrodesis splanning distal joints; ideally do this since retain more movement

Pantarsal arthrodesis will give no movemet

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Which animals do we tend to see congenital shoulder luxation

A

Small breed dogs
= rare

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

How can we treat congenital shoulder luxation

A

Some cope with conservative

Others need excisional arthroplasty or arthrodesis (salvage)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

Why can we only do conservative or salvage proceudres for congenital shoulder luxation

A

Because joint is very dysplastic

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

Which direction do most hip luxations go in

A

Craniodorsal
Need 2 views to tell which direction

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
20
Q

Diagnosis of hip luxation

A

Clin exam = shortened limb, inward leg torsion, see ilial crest/greater trochanter/tuber icschium in a striaght line not a trianlge

Radiographs

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
21
Q

Management of hip luxation

A

Usually closer reduction; put animal in lateral recumbency, externally rotate femur pull distally and internally rotate back in
Then must do range of motion tests for instability and and X rays

Ehmer sling applied to dogs; cats crate rested

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
22
Q

Indications for open reduction of hip luxation

A

Articular fractions
Chronic luxation
Other injuries present e.g on other leg that prevents overloading hat leg
Unsuccessful closed reduction/reluxation

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
23
Q

Open reduction techniques for hip luxation

A

Most common = toggle pin fixation

Prosthetic capsule
Iliofemoral suture
Transarticular pin not recommended since pin can snap and joint moves so is loose

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
24
Q

What are the salvage surgery options for hip luxation

A

Femoral head and neck excision
Total hip replacement

Need to consider earlier if underlying hip dysplasia or for non-reconstructbale articular frctures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
25
Q

Which tendon has a degenerative disase of chronic mineralisation

A

Common calcineam tendon

26
Q

What drugs might be involved in tendon injury

A

Corticosteroids
Fluoroquinolones

27
Q

What does it mean if we can flex the hock while stifle maintained in extension

A

There is a common calcineum tendon rupture

28
Q

How does an animal look with SDF tendon rupture

A

very flat toes

29
Q

Some tehcniques for repairing tendon ruptures

A

Locking loop (for flatter tendons)
Three loop pulley for rounder ones

30
Q

What sutures do we use to repair tendon ruptures

A

Probably permanent e.g prolene
Monofilament

PDS could be okay but doesn’t last as long

31
Q

What breed typically get biceps tendon luxation

A

Lurchers

32
Q

Treating biceps tendon luxation

A

Surgery; sutures/plates

33
Q

If we feel a popping when manipulating the shoulder what might this be

A

Biceps tendon luxation

34
Q

What signs do we get with bicipital tenosynovitis and which individuals

A

Active, large breed dogs

Insidious onset chronic front limb lameness
Pain on direct palpation (medial to greater tubercle)

35
Q

How can we do the biceps test for pain (e.g in bicipital tenosynovitis)

A

Flex shoulder and extend elbow to do max extension of the tendon and look for pain response

36
Q

Treatment of bicipital tenosynovitis

A

Usually conservative
For non-responsive cases can do biceps tendon release via arhtroscopy

37
Q

puppy carpal laxity/hyperflexino presentation and treatment

A

3-4 months old
Non-painful

Self resolve and ensure eating correct diet

38
Q

When do we typically see quadriceps contracture

A

Associated with femoral fractures in young growing animals since muscle gets involves in the fracture and callus causing bowstring effect as femur elongates

Mild cases rehab
Severe arthrodesis

39
Q

When do we typically see infraspinatus contracture

A

Working dogs
Damage by single episode or repeat trauma
Can’t get full extension of shoulder giving tpical leg out postureT

40
Q

Treatment of infraspinatus contracture

A

Tenotomy of infraspinatus tendon

41
Q

Treatment of gracilis contracture

A

Can’t really- partial myotomy gives temporary alleviation but tends to recur

Non-painful

42
Q

What are the 2 indications for arthrodesis

A

Joints so arthritic they are painful and can’t do resection or joint replacement

Non-reconstructable articular fractures

43
Q

How to do an arthrodesis

A

Remove cartilage at end of either bone, pack with autologous bone graft and use stable fixation under compression

Want fixation at an anitomical angle

44
Q
A
45
Q

How do dogs with avascular necrosis of the femoral head present

A

Severe pain on hip flexion/extension
Unilateral (unlike many hip dysplasia cases)
Musle wastage

46
Q

What are the radiographic findings with avascular necrosis of the femoral head/neck

A

First see lucent areas of lysis (related to reperfusion injury after initial damage and avascularity)

Then get collapse and mushrooming

47
Q

What are the treatment options for avascular necrosis of the femoral head

A

Conservative; but not good if very painful
Surgery =total hip replacement or femoral head and neck excision

48
Q

What is panosteitis and how do they present

A

Focal areas of endosteal bone proliferation
Present as shifting lameness from multiple limbs, dull, anorexic/pyrexic from pain
+ painful bones on deep palpation

49
Q

Which individuals are most prone to panosteitis

A

Young dogs 5-18mo
GSDs mostly
M > F

50
Q

What does panosteitis look like on histopath

A

Very pink on H and E due to intramembranous ossification (hence ‘eosinophilic panosteitis’

See stromal cell proliferation
Medullary adipocyte degeneration

51
Q

Radiography findings with panosteitis and which sites are most typical

A

Patchy increase in density of medullary bone with whorls of new bone formation like a thumbprint

Lose cortex/medullary distinction since medulla much more dense

Classic in distal humerus, prox ulna

52
Q

Management of panosteitis

A

Conservative with pain management
Can recur until they are 18 months

53
Q

Which breeds do we see craniomandibular osteopathy in and what age

A

Terriers; mostly westies, also scotties, bostons, cairn

Young - 3-8 mnths old

54
Q

Signs of craniomandibular osteopathy

A

Enlarged mandibles, limited mouth opening, pain on attempting to open mouth
[main ddx = broken jaw]

Radiography: proliferative new bone on madibles, TMJs, bullae

55
Q

Treatment of craniomandibular osteopathy

A

Conservative: analgesia, anti-inflammatories, sterois, liquid food/feeding tube, hospitalisation

Good prognosis since self limiting but may need euthanasia due to severe pain

56
Q

What is metaphyseal osteopathy and which individuals do we see it in

A

= hypertrophic osteodystrophy at metaphyses
Seen in large and giant breeds age 2-8 months
Inclear aetiology

57
Q

Radiographic signs with metaphyseal osteopathy

A

Sclerotic line adjacent to physis with radiolucent zone adjacent to this

Periosteal new bone formation

58
Q

Treatment of metaphyseal osteopathy

A

Conservative: balanced diet, painkiller, rest, can use antibiotics if concerned about infection

Self-limiting but risk of GP closure and angular limb deformity

59
Q

What is Marie’s disease

A

= hypertrophic osteopathy; where there is new bone formation around distal bones secondary to throacic disaese
90% have pulmonary neoplasia

60
Q

What orthopaedic sign can we see in pulmonary neoplasia cases

A

Marie’s disease; hypertrophic new bone formation around distal bones

61
Q
A
62
Q
A