Orbit, Eyes, and Lacrimal System Flashcards
Orbit floor
PaM-Z: palatine, maxillary, zygomatic
- Clinically WEAKEST
- hypoethesia due to infraoribtal nerve trauma
Medial wall of orbit
-SMEL
sphenoid (lesser wing), maxillary, ethmoid, lacrimal
-thinnest
-lacrimal sac fossa
-optic foramen
-inferior oblique muscle arising from lacrimal crest
Orbital roof:
frontal, lesser wing of sphenoid
Lateral wall
Great-Z: greater wing of sphenoid, zygomatic
-THICKEST wall
Extraocular muscles
4 rectus muscles, 2 oblique muscles
inferior oblique is the only one that doesn’t arise from back portion of the globe
Neurovasculature
cranial nerves (oculomotor does most; lateral rectus from abducents, superior oblique from trochlear) optic nerve, ophthalmic artery and vein
Sensory innervation
CN V (trigeminal) Ophthalmic division (V1): nasociliary, frontal (supraorbital, supratrochlear), lacrimal nerves
Maxially division (V2): infraorbital (cheek numbness w/ damage), zygomatic
Oculocardiac reflex
Increased pressure on eye, pulling on eye muscles–>bradycardia, asystole, arrhythmia
afferent: CN V1 (ciliary ganglion to trigeminal)
efferent: CN X
Treatment:
- release globe/muscle
- atropine
- glycopyrrolate
- preventative (or provoking): retrobulbar/peribulbar block
Orbital fractures
-floor and medial wall most commonly affected -Most often occurs medial to infraorbital canal–>leads to hypesthesia in cheek and upper lip (V2)
Repair in w/in 2-4 weeks if:
- enophthalmos (eye sunken in because of volume in orbit decreased)
- incarcerated muscle/orbital tissue
- > 50% of wall fractured
Orbital cellulitis
- presents with pain, erythematous lid swelling, TTP
- EOM restriction, proptosis
- Later: vision loss, RAPD, decreased color vision, increased IOP
- Mgmt: broad spectrum antibiotics, CT orbits with contrast, abscess I/D
Layers of eyelid below lid crease
Skin
orbicularis muscle
tarsus (Meibomian glands inside)
conjunctiva
Layers of upper eyelid above crease
skin orbicularis muscle septum fat levator aponeurosis Muller's muscle conjunctiva
cavernous sinus thrombosis
- sequelae of uncontrolled orbital cellulitis/facial cellulitis
- Orbital veins are valveless: retrograde spread of infections to cavernous sinus
- CN III, IV, V1, V2, VI palsies, proptosis, chemosis, altered mental status
- may have permanent neuropathies; death from sepsis
Layers of lower eyelid below crease
skin orbicularis septum fat LL retractors (CPF) Muller's muscle conjunctiva
Arterial supply
superior/inferior marginal arcades (2mm from margin)
Superior peripheral arcade (between levator and Muller’s)