Oral diseases and RAD Flashcards

1
Q

What is a cyst. General symptoms and signs. Further investigations. What is the most common

A

-pathological cavity filled with fluid or semi-fluid contents. Lined by epithelium. Not created by pus
-asymptomatic, incidental finding, investigating unerupted tooth, slow growing. If large enough causes bony expansion, mobility, root resorption, pathological fracture or altered nerve sensation. Well-defined radiolucency
-radiograph, vitality test, aspiration, incisional biopsy
-radicular cysts most common (65%)- odontogenic inflammatory cyst

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2
Q

What is an odontome. How they look radiographically

A

-benign tumours linked to tooth development. Hard tissue growing abnormally. Limited growth potential (stop growing when mature) Made of calcified tissue (so radiopaque). Commonly cause displaced teeth rather than mobile
-tx: removal if interfering with eruption. Otherwise leave if asymtpomatic and monitor for cystic change or root resorption

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3
Q

What is an ameloblastoma. cells involved. characteristics. tx

A

-Benign odontogenic tumours- epithelial without mesenchyme
-asymptomatic, incidental finding of missing tooth, common in angle of mandible, tooth displacement,
-radiolucent, well defined and corticated, very aggressive locally and high recurrence so resected with 1cm clear margins

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4
Q

What Benign odontogenic tumour (epithelial with mesenchyme) has soap bubble appearance

A

Odontogenic myxoma

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5
Q

4 methods of surgical management of cysts. Which is most common

A

1.Enucleation: +/- peripheral ostectomy, chemical fixation or cryotherapy
2.Marsupialisation
3.Decompression
4.Resection

-enucleation most common

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6
Q

What is marsupialisation and its pros and cons

A

-Not removing cysts all in one go
-Open it up and hold open using a pack (BIPP/ gauze) so pressure is relieved which gradually decreases size of cavity and promotes bone healing
-Lining will undergo metaplasia and become normal keratinised epithelium

-Pros:
*less invasive, preserves vital structures
*Short procedure
*GA not needed. Done under LA
-Disadvantages
*Can be difficult to suture friable lining to oral mucosa
*Lining remains
*High rate of Recurrence for keratocysts (as lining not removed)
*Changing pack and having a pack = difficult for patient
*Can Close over quite easily

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7
Q

What is decompression surgical management for cysts. Pros and cons

A

-Open cyst and drain the fluid using a tube to allow shrinkage and reduce pressure
-Pros:
*Allows shrinkage
*Useful for Larger cysts where enucleation may threaten vital structures
*Relatively easily achieved vs marsupialisation / enucleation
-Cons
*Further surgery
*Drain may be lost and cavity close back up
*Doesn’t remove cyst lining, relying on it metaplasing or need surgery after to remove

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8
Q

How does enucleation work for treatment of cysts. Pros and cons

A

-Muco-periosteal flap raised, Remove ALL cyst lining, Close flap over cavity, Pack if necessary

-Advantage:
*Complete removal of cyst lining so less risk of recurrence
*Definitive treatment for most cysts. Low recurrence as removing lining
-Disadvantage
*Leaves dead space- Infection risk (Can pack / place bone graft if large)
*Damage to adjacent structures- Loss of vitality, IAN damage
*Risk of Jaw fracture (less bone support if drilling through bone to access cyst)
*Antral / nasal involvement causing communication
*Invasive
*May need GA

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9
Q

Adjuvants to enucleation to prevent recurrence of odontogenic keratocysts (high recurrence rate)

A

-carnoy’s solution (alcohol, chloroform, ferric chloride, glacial acetic acid)- removes any remaining viable lining
-cryotherapy (liquid nitrogen to freeze everything)- causes cell death
-Peripheral ostectomy -bur to remove margin around cyst
-5% 5-fluorouracil (chemotherapeutic agent)

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10
Q

What cyst has high recurrence rate. What syndrome is this cyst associated with. Tx options

A

-odontogenic keratocyst
-parakeratonised
-Found in Golin-Goltz syndrome (skeletal abnormalities and basal cell naevi -> BCCs)
-Tx: Enucleation with adjuvant of carnoy’s solution or 5% 5-fluorouracil

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11
Q

What is ATLS and what it involves

A

-advanced trauma life support
-Initial management for life-threatening trauma.
-it improves chance of survival as more in depth than BLS
1-Primary survey= ABDCE and cervical spine immobilisation (neck brace) Stabilising life-threatening injuries
2-Secondary survey: E/O and I/O assessment. Investigations
3-Soft tissue and hard tissue management

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12
Q

Causes of airway obstruction and its management

A

-Blood clots from perfuse bleeding
-Foreign bodies e.g. dentures, tooth
-Tongue
-Posterior displacement of maxilla
-Bilateral parasymphyseal mandibular fracture-loss of support for tongue muscles so it falls back and obstructs the airway
-swelling

-Management=head tilt chin lift or jaw thrust to open airway

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13
Q

Initial management of bleeding and blood loss. Complication

A
  • DO NOT remove foreign body
    -Control haemorrhage: use pressure, haemostat clips (arterial forceps) diathermy for small vessels, ties for large vessels, fracture reduction
    -check vital signs
    -risk of hypovolemic shock (signs = cyanosed, decreased BP, sweating & tachycardic)
    -raise legs to increase circulation to head
    -fluids, basic bloods (O rhesus –ve) then specific blood (matched)
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14
Q

Explain AVPU for assessing disability

A

-assessing neurological status and head injury
-alert? verbally responsive? Painful stimuli? Unresponsive?

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15
Q

Explain GCS for assessing disability

A

-assessing neurological status and head injury
-Eye opening, motor responses, verbal responses.
-Out of 15, lower is worse. 3 is lowest
-8 or less = coma state

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16
Q

Vital signs and their normal values

A

-Temperature: 35.5 – 37.5
-Respiratory rate: 15-20/ min
-Blood pressure: 120/80
-Pulse rate: 60-100/ min

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17
Q

Does increased intracranial pressure cause pupil dilation or constriction

A

dilation

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18
Q

Secondary survey involves extra and intra oral assessment once injuries are stabilised. List what needs to be assessed

A

-E/O:
-Inspect: lacerations, ecchymosis (brusing), oedema, facial deformity & CSF leaks (straw-like at nostril or ear)
-Assess for head injury, retro-bulbar haemorrhage & eye observations
-Palpate for tenderness, step deformities, crepitus of jaw & paraesthesia
-CN 5 & 7 tests

-I/O:
-Inspect for missing teeth, broken teeth/dentures, lacerations, ecchymosis & step deformities in the occlusal plane
-Palpate tenderness, step deformities, mobility of teeth/bone & paraesthesia
-Assess occlusion, mandibular movements

Radiographs: 2 planes at right angles

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19
Q

What is retrobulbar haemorrhage. symptoms

A

-site threatening, an emergency as a sign of increased intracranial pressure.
- bleed behind eye. Increased ocular pressure can compress on optic nerve and other nerves
-Causes pain, paralysis, proptosis (protruded), poor vision. Pressure needs to be released to treat

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20
Q

How to manage wounds once bleeding is controlled

A

-LA or GA
-decontamination and debridement (saline, iodine, scrub)
-Primary closure: Tension-free. Use sutures/ steristrips if superficial/ staples (useful in hairline)/ Dermabond glue.
-For Deep layer closure: dissolvable and undyed vicryl. Close in anatomical layers (eg. muscle to muscle, fat to fat) to remove dead spaces as a site for infection and to avoid scarring
-may need ointments or dressing during closure
-skin grafts needed if tissue loss through epidermis and full/ partial dermis
-local or regional flaps for aesthetics
-Consider antibiotic prophylaxis for dirty wounds

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21
Q

When to remove sutures etc for wounds. Explain the healing process

A
  • removed once wound strength is adequate, if left longer then it can lead to iatrogenic scarring (for face remove at 5 days)
    -wounds heal with contraction and along length due to collagen and fibroblast maturation and have a tendency to become inverted. Ensure wound edges are well apposed and slightly everted to counteract contraction
22
Q

Mandibular fractures are 2nd most common face fractures (after nasal bones) What are the sites of weakness of the mandible

A

-Socket of canine tooth
-Condylar neck (narrow)
-Mental foramen

23
Q

Describe these types of fracture descriptions: -Open, Closed, Compound, Comminuted, Linear, Greenstick, Simple, Wedge, Segmented

A

-Open-bone broken through skin/ mucosa. most mandible fractures open rather than closed
-Closed: skin/ mucosa in tact
-Compound: bone visible through skin/ mucosa
-Comminuted-multiple fragments
-Greenstick- break not the full way through
-Simple - linear fracture
-Wedge- triangular detached wedge of bone
-Segmented- 2 simple fractures causing small section

24
Q

Factors that affect favourability of fractures

A

-unfavourable if muscle pull and occlusal forces cause the fractured bones to move further apart (angle fractures affected by pull)
-favourable if fracture ends closer together to allow quicker healing, compared to displacement
-direction of the fracture is a factor

25
Q

Principle of mandibular fracture surgical management

A

ORIF: open reduction internal fixation. Use Titanium miniplates for Fixation -supports bone in position to allow healing. Plates adapted to the curve of bone

26
Q

Indications for removing and leaving teeth in line of a mandibular fracture

A

Removing:
-Tooth luxated and/or interfering with reduction
-Tooth that is fractured or infected
-gross caries, advanced perio
-Mobility which would not contribute to establishment of stable occlusion.
-Existing pathology e.g. cyst, pericoronitis

Leave if:
-vital/ firm
-Does not interfere with reduction and fixation of fracture
-Requires removal of excessive bone, which may compromise plate/screw fixation
-Assists in establishing occlusion and reducing the fracture

27
Q

Aftercare for mandibular fracture surgery

A

-Maxillomandibular fixation or elastics may be used to help maintain occlusion and impacted post op swelling
-Post op radiographs and Review
-Encourage good OH
-CHX mouthwash prescribed
-Soft diet for 6 weeks
-Sometimes physiotherapy used to improve mouth opening (condylar fractures)
-Post op antibiotics and analgesics

28
Q

Healing process of mandibular fracture surgery

A

-Inflammation and bleeding from fracture ends
-Soft callus – fibrous tissue replaces haematoma. Subperiosteal bone formation begins
-Hard callus – calcification of soft callus
-Remodelling– once got a solid union get replacement by lamellar bone

29
Q

Complications after mandibular fracture surgery

A

-Malunion/non-union (due to fracture instability or inaccurate reduction)
-Fixation failure (Due to Insufficient amount of fixation, plate fracture, screw loosening, devitalisation of bone causing loosening
-Infection (Due to Non-vital tissues, fracture instability, microorganisms in fracture site
-Ankylosis (If < 3 years, or prolonged MMF, or condylar head fracture)
-Necrosis (due to Compromised Blood supply)

30
Q

Management of infection after mandibular fracture surgery

A

-Re-expose fracture site to debride wound +/- sequestrectomy
-MMF/ temporary fixation
-Recon plate that is load bearing to aid healing
-Same stage grafting
-Removal of plate if no longer functioning

31
Q

Management of nasal fractures

A

-Diagnosed clinically, as only treated if aesthetic issue
- closed reduction (manipulation under anaesthetic. No flap raised)
-May require packing & splinting or septal repositioning
-May need – septoplasty, rhinoplasty or septo-rhinoplasty

32
Q

What are facial buttresses

A

-areas of strength in mid face. designed to resist trauma and protect brain
-eg. Canine and zygomatic butresses
-they direct the forces up the face

33
Q

Common signs and symptoms of zygomatic arch and zygoma fractures

A

-Zygomatic arch: restricted opening & lateral excursions, step deformities, facial flattening
-Zygoma: black eye -Periorbital ecchymosis/oedema), subconjunctival haemorrhage, step deformities, paraesthesia, diplopia

34
Q

Explain le fort I, II, and III fractures

A

transverse fractures involving the obital, zygomatic & nasal bones

  1. Fracture of dentoalveolar complex. Runs horizontally between tooth roots and floor of the nose and & antrum. Separates the palate and alveolus from the maxillary complex
  2. Pyramidal fracture of nasomaxillary complex. Runs from frontonasal junction along the infraorbital rim then along the anterior maxillary sinus wall below the zygomatic buttress. Joins across the bridge of the nose
  3. Complete separation of maxillary complex from rest of cranium. with breaks at the frontonasal sutures, the zygomatic-frontal sutures, the zygomatic arches, and the pterygoid plates.
    Causes Facial flattening
35
Q

Signs and symptoms of le fort fractures

A

-Facial flattening/elongation
-Bilateral facial and periorbital oedema
-Epistaxis (nosebleed)
-Anterior or lateral open bite (malocclusion)
-Ecchymosis in the maxillary vestibule
-Paraesthesia of the midface
-Step deformities
-Hisotory of high velocity trauma

36
Q

Types of mobility when you manipulate the anterior maxillary teeth and alveolar ridge, whilst stabilizing the forehead. For the 3 types of le fort fractures

A

-Le Fort I- movement of the entire maxilla
-Le Fort II- maxilla and nasal complex
-Le Fort III- entire facial complex, including zygomatic buttresses

37
Q

Who does IRR and IRMER 2017 protect

A
  • keeping exposure to ionising radiations as low as reasonably practicable.
  • IR(ME)R- protects patients from unintended, excessive, incorrect medical exposure, ensures the benefits outweigh the risks
    -IRR 17: protects employees and the public working with ionising radiation
38
Q

What is involved in the IRR regulation to protect employees

A

-All employers must register “work with a radiation generator” with the Health and Safety Executive (HSE)
-Radiation protection adviser (RPA) and their roles
-ALARP- exposure are as low as reasonably practicable -eg. maximise distance, shielding, minimise time
-Have a controlled area
-Radiation protection superviser (supervises to ensure local rules and regulations are adhered to)
-Safe working procedures

39
Q

What does an RPA do

A

-Appointed by the employer to comply with IRR
-Radiation protection adviser

-assess installation plans
-Acceptance into service of engineering controls, design features, safety and warning devices in relation to new or modified radiation sources
-risk assessment, local rules and contingency plans
-Room design
-Working arrangements for pregnant employees 6
-equipment checks
-PPE
-Training programmes
-Prevention, investigation and analysis of accidents
-Quality assurance
-Periodic testing of engineering controls, design features, safety and warning devices and regular checking of systems of work

40
Q

Roles of, referrer, practitioner and operator involved in the IRMER regulation to protect patients. Who can and cannot carry out these roles

A
  1. Referrer- dentist, hygienist, therapist who can refer to a practitioner. Supply adequate info to enable justification by practitioner
  2. Practitioner -dentist, hygienist, therapist who takes responsibility and justifies the exposure
  3. Operator- patient identification, setting it up, pressing button, processing. Nurses can also do this (extra training required for setting it up)
    [4. Clinical evaluation- only dentist, hygienist or therapist can do this]
41
Q

Employer responsibilities (IRMER)

A

-legally responsible person
-identify those able to be referrer, practitioner and operator
-follows referral guidelines
-provide patients with info about benefits and risks of exposure
-setting and reviewing diagnostic reference levels
-appoints the RPA and RPS
-risk assessment
-define the controlled areas
-produce local rules
-clinical audits
-investigate incidents

42
Q

What are diagnostic reference levels. How often are they reviewed

A

-dose quantity established for each standard diagnostic examination for a standard patient cohort
-reviewed at least every 3 years or when changes are made to the equipment
-investigation needed if patient doses regularly excess the DRL

43
Q

What are the diagnostic reference levels for Intra orals, DPT and CBCT

A

-Intra-oral (mandibular molar)- 1.2mGy
-DPT- 81mGy.cm2
CBCT- 265mGy.cm2

44
Q

What are the controlled areas for CBCT, I/O and DPTs

A

-CBCT- entire room
-I/O and DPT: direction of the primary beam and 1.5m radius of the beam entry point

45
Q

Are IRMER or IRR incidents more common. When is a report needed

A

-IRR- reportable incidents are rare. eg. contingency plan triggered, an individual relieves an overexposure
-IRMER- more common. eg. wrong patient, wrong body part, wrong image

46
Q

Reasons not to justify an exposure

A

-referrer gives insufficient or unclear info
-no clinical indication
-recent images and findings available
-radiation risk not outweighed by benefits

47
Q

Diagnosis and management of cleft lip and palate

A

-from 20 week scan to 20 years old.
-2D ultrasound. Then 3D scan to know the extent. Diagnosis may not be confirmed until birth
-Primary surgery when a baby
-Alveolar bone grafting of palate at 8-10 years old, just before super 1s are about to erupt. Ortho appliance to hold in place for 6 months
-rhinoplasty, speech surgery if necessary
-restorative work, ortho
-Lifelong care monitoring speech, hearing, feeding, psychology, appearance, dental health

48
Q

Types of cleft lip and palate

A
  1. Unilateral cleft lip (+/- palate)- one side of lip. Muscle of lip attached to nose. Can be part of lip or goes all the way through lip, gum, nose and palate
  2. Bilateral cleft lip (+/- palate)- both sides of lip. Can extend up into gum, nose and palate
  3. Cleft palate – midline of palate
  4. Bilateral Cleft of palate- gap in hard and/or soft palate.
  5. Submucous cleft palate – Roof of mouth is formed but muscles in wrong place. Eg. Uvula can be doubled
49
Q

Development and causes of cleft lip and palate

A

-when don’t fuse properly during development. Happens very early in pregnancy.
-Normal palate formation-palatal shelves grow out from side of jaw. Grow down then lift up and grow towards each other to form middle of palate. If don’t join then creates a gap in the palate
Palate fuses from the front backwards. So more severe cases extend towards the incisive fossa

-Can be linked to environmental factors such as substance abuse and medications, but often no explanation. Can be genetic. -22q11 deletion associated with DiGeorge syndrome.

50
Q

Role of GDP during cleft lip and palate patient care

A

-Routine family dental care: acclimatisation, prevention
-Routine interventions- restorations, extractions
-Liaison with MDT team
-Reinforce good OH as essential for these patients- Usually increased risk of caries. But cleft does not equal caries. It is down to oral hygiene, diet, motivation. Cleft may make OH harder