Oral Abscesses Flashcards

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1
Q

How do oral abscesses present?

Routes of prokaryotic infections of the mouth:

A
  • pain, swelling, erythema, suppuration
  • caries/trauma: periapical or dentoalveolar abscess
  • gingival: gumm tissue inflammation
  • periodontitis: periodontal abscess
  • pericoronitis: partially erupted teeth
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2
Q

List key factors of dentoalveolar abscesses:

Treatment?

A
  • remains localised
  • number/type of virulent bacteria
  • local/systemic immune response
  • anatomical damage

Treatment:

  • drainage/RCT/extraction
  • antibiotics
  • antimicrobials
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3
Q

Why do abscesses appear as swollen, inflamed areas?

What is the pus?

Whats the causative agent?

A

Swollen, inflamed areas:

  • immune response
  • recruitment of WBC
  • plasma etc

Pus:

  • dead tissue/debris/DNA
  • immune cells
  • infectious material
  • fluid

Causative agent:

  • infectious agent
  • foreign body
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4
Q

What are facultative anaerobes?

What are strict anaerobes?

Give an example of each

A

Facultative anaerobe: bacteria which can grow in the presence and absence of oxygen

e.g. S. anginosus-group, S. oralis, actinomyces, enterococcus faecalis

Strict anaerobe: microorganisms that are killed by normal atmospheric concentrations of oxygen

e.g. prevotella, fusobacterium nucleatum, tanerella forsythia

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5
Q

What is the cause and syptoms of a periodontal abscess?

What bacteria tend to be associated with periodontal abscesses?

A

Cause:

  • occlusion of opening prevents drainage
  • impaction of foreign objects

Symptoms:

  • sudden onset
  • swelling/redness/tenderness
  • may spread and destroy tissue/bone

Tend to be associated with a mixture of bacteria from gingival pockets e.g. prevotella, porphyromonas, variety of streptococci, fusobacteria nucleatum, actinomyces

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6
Q

Name bacteria associated with endodontic infections:

A

Gram positive:

  • Strep. oralis, S. mitis, S. anginosus, S. gordonii
  • enterococcus faecalis: resistant to hogh and low pH so difficult to erradicate
  • lactobacilli
  • staphylococci

Gram negative:

  • fusobacterium nucleatum
  • prevotella
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7
Q

What is ‘dry’ socket?

A
  • localised infection following extraction, the socket fails to heal
  • sparse anaerobic bacteria associated such as fusobacterium nucleatum
  • prophylaxis: chlorohexidine irrigation prior to and post extraction
  • treatment: antiseptic dressing and metronidazole
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8
Q

Explain a bit about osseointegrated implants and abscesses and the associated bacteria:

A

Periimplantitis can cause immediate or delayed abscesses:

  • staphylococcus aureus and staphylococcus epidermidis
  • aggregatibacter, prevotella, fusobacteria, anaerobic streptococci

Remove implant and antibiotic therapy to treat

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9
Q

Historically, what was associated with actinomyces?

Why is this not necessarily true?

What is the major species of actinomyces causing a mass in the head and neck?

What is it characterised by?

What is required to treat this?

A

Historically, actinomyces reported to be associated with root surface caries

Polymicrobial nature means this is now questionable and S. mutans and R. denticola are considered important

Major species: actinomyces israelii

  • characteristic ‘pus’, known as sulphur granules but not actually sulphur

Extended antibiotic treatment e.g. penicillin up to 6 weeks treatment

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10
Q

What is Treponema denticola associated with?

A

T. denticola and P. gingivalis are commonly found associated with each other in chronic perio lesions

  • grown in mixed biofilms
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11
Q

What is necrotising gingivitis?

What is it associated with?

Symptoms?

Microbiology:

Treatment?

A

An acute painful true infection of the gingivae

Associated with: immunosuppression

  • poor OH
  • plaque retentive factors
  • smoking

Symptoms: inflammation, spontaneous bleeding, intense pain, tissue destruction, bad taste

Microbiology: large amounts of fusobacterium, and spirocheates

Treatment: metronidazole therapy, OH advice

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12
Q

What is ludwig’s angina?

Symptoms?

Causes?

Microbiology:

A
  • Acute cellulitis, bilateral infection (submandibular and sublingual spaces)

Symptoms:

  • tongue and FOM swell
  • brawny oedema and swelling of neck tissues
  • airway obstruction

Cause due to extraction on lower arch

Microbiology: beta-haemolytic oral streptococci, staphylococci, enterococci, prevotella, fusobacteria

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