opthalmology Flashcards

1
Q

which part of the eye produces aqueous humour

A

cilliary body

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2
Q

which cranial nerve palsy makes the eye go down and out when looking forward

A

oculomotor (cn3)

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3
Q

which cranial nerve palsy makes the eye turn upwards and inwards when looking forward

A

trochlear (cn4)

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4
Q

which cranial nerve palsy makes the eye turn inwards when looking forward

A

abducens (cn6)

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5
Q

where abouts is the angle that gets blocked in acute angle closure glaucoma

A

where the trabecular meshwork is where the iris meets the cornea in the anterior chamber

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6
Q

what is the function of the aqueous humour

A

to nourish an hydrate the eye

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7
Q

where is the anterior chamber located

A

between the cornea and the iris

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8
Q

where is the posterior chamber located

A

between the iris and the lens

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9
Q

where is the viterous chamber located

A

behind the lens

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10
Q

what is the function of the choroid

A

essentially a spongey vascular layer that helps to provide nutrients to the outer layer of the retina and contains pigment to absorb excess light

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11
Q

what is the function of cilliary body

A

pupillary constriction / dilatation

produces aqueous humour

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12
Q

what is the function of the lens

A

to focus (refract) light from the pupil onto the retina

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13
Q

what is the function of the conjunctiva

A

a moist clear membrane that covers the sclera, it protects and lubricates the eye ball

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14
Q

where are most cone cells found

A

fovea

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15
Q

what is the function of the fovea and where is it found

A

found at the centre of the macula, produces a highly focused quality image to send to the brain

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16
Q

what is the function of the iris

A

to control the amount of light that is able to enter the eye

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17
Q

function of cone cells

A

colour vision
better in high light levels
high spatial acuity

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18
Q

function of rod cells

A

night vision
better in low light levels
low spatial acuity

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19
Q

pathophysiology of primary open angle glaucoma

A

slowly increasing resistance of trabecular meshwork to outflow of humour = gradually increasing IOP over time = asymptomatic for ages / fluctuating onset of symptoms

20
Q

symptoms of primary open angle glaucoma

A
asymptomatic / incidental finding by optometist
gradual loss of peripheral vision
nasal scotomas / tunnel vision
halos around lights at night 
fluctuating pain (not always)
headache
21
Q

findings on fundoscopy in primary open angle glaucoma

A

optic disc cupping - meaning the indent in the optic disc (optic cup) is more than 0.7
pale optic disc from optic atrophy

22
Q

investigations to do in suspected primary open angle glaucoma

A
  1. fundoscopy
  2. assess visual fields with automated perimetry
  3. use slit lamp and dilate pupil to look for signs of optic nerve damage and assess baseline
  4. goldmann applanation tonometry to assess intra ocular pressure
23
Q

1st line treatment of primary open angle glaucoma

A

latanoprost drops - prostaglandin analogue to increase uveosacral outflow

24
Q

how does timolol drops work

A

beta blocker - decreases production of aqueous

25
how does latanoprost drops work
increases uveosacral outflow
26
how does pilocarpine drops work
antimuscarinic that acts on the parasympathetic fibres to induce pupillary muscle constriction = pain relief and opens the angle to allow some drainage of humour
27
how does oral / iv Acetazolamide work
reduces aqueous production by inhibiting carbonic anhydrase
28
what investigations should you do in suspected acute angle closure glaucoma
1. goinoscopy - examines the anterior chamber angle 2. slit lamp examination 3. applanation tonometry to measure IOP
29
findings on examination in acute angle closure glaucoma
hard, red eye fixed dilated non reactive pupil corneal oedema
30
what should you give someone in the primary care setting pre-hospital with suspected acute angle closure glaucoma
1. pilocarpine eye drops 2% if blue eyes 4% if brown eyes 2. acetazolamide 500mg po stat 3. antiemetic + analgesia
31
presentation of acute angle closure glaucoma
``` severely painful, red, hard eye haloes around lights worse in dark reduced visual acuity (blurred vision) semi dilated non reacting pupil corneal oedema - dull / hazy N+V ```
32
which drugs can precipitate acute angle closure
anticholinergics eg oxybutinin | pupil dilating eye drops eg atropine
33
what is seen on fundoscopy in both types of ARMD
drusen (yellow spots of lipids on the retina)
34
pathophysiology in ARMD
both: drusen formation from lipid deposits and degeneration of retinal photoreceptors wet: get neovascularisation causing new vessels to leak fluid or haemorrhage into the retina causing retinal oedema = faster degeneration of vision
35
describe the vision loss in ARMD
central scotoma / slow central loss of vision / loss of visual acuity of up close things / deterioration in night vision
36
symptoms of ARMD
central scotoma, declining night vision, distortion of straight lines, seeing things flickering / flashing lights
37
loss of peripheral vision and haloes around lights
acute glaucoma
38
loss of central vision / central scotoma and blurring / waving of lines
ARMD
39
generalised reduction in visual acuity and starbursts around lights at night time
cataracts
40
key sign on examination in cataracts
loss of red reflex - will be white or grey instead
41
symptoms of cataracts
reduced visual acuity things may go yellowy or brown in colour starbusts around lights at night asymmetrical as both lenses affected differently
42
treatment of cataracts
if not an issue then leave it alone | if reduced visual acuity severe then surgery
43
findings on fundoscopy in cataracts
normal
44
complications following cataracts surgery
1. posterior capsule opacification 2. retinal detachment 3. endopthalmitis --> inflammation of aqueous and / or viterous humour
45
name a cause of endopthalmitis
complication of cataracts surgery