Ocular Motor Disorders Flashcards

1
Q

Nerves responsible for eye movement are

A

CN III Ocular motor
CN IV Trochlear
CN VI Abducens

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2
Q

Extraocular muscle function

A

, runs on a continuum from normal to weak to paralysis
Instead of using the suffix “plegia” and “paresis” to describe the state of muscle function, the terms “phoria” and “tropia” are used

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3
Q

Phoria

A

Indicates weakness of the muscle

The eye would drift but drifting is held in check by sensory fusion

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4
Q

Tropia

A

Indicates paralysis of the muscle

The eye drifts even when the person is focusing on a target

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5
Q

Prefixes indicate the direction of the misalignment

A
Eso
Eye is deviating inward (crossed eyes)
Esophoria or esotropia
Exo
Eye is deviating outward (wall eyes)
Exophoria or exotropia
Hyper
Eye is deviating upward
Hyperphoria or hypertropia
Hypo
Eye is deviating downward
Hypophoria or hypotropia
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6
Q

Oculomotor Nerve (CN III)

A
The third cranial nerve supplies: 
levator muscle of the eyelid
medial rectus
superior rectus
inferior rectus
inferior oblique
CN III constricts the pupil through its parasympathetic fibers that supply the smooth muscle of the ciliary body and the sphincter of the iris.
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7
Q

Acquired CN III Palsy

A

Sudden onset of binocular horizontal, vertical, or oblique diplopia
Ptosis or a droopy eyelid

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8
Q

Congenital CN III Palsy

A

Found in young children
May not complain of diplopia because they suppress the second image or because they have superimposed amblyopia
Often they are brought in by their parents, who have noticed ptosis or strabismus.
Amblyopia is the major complication of 3rd nerve palsy in children

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9
Q

CN III Palsy Eye examination

A

May have a partial or complete ptosis
The pupil may be dilated and poorly reactive, or dilated and non-reactive to light
Deficits in ipsilateral adduction (medial rectus), elevation (superior rectus, inferior oblique),and/ordepression (inferior rectus) may be present.

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10
Q

Complete 3rd nerve palsies

A

Considered complete if impairment of the majority of function of all the somatic branches of the oculomotor nerve is present and ptosis is complete or almost complete.

Complete 3rd nerve palsies usually are associated with a large-angle exotropia and hypotropia (down and out

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11
Q

CN3 Palsy Differential

A
Vasculopathic
HTN
Diabetes
Tumor
Congenital
Aneurysm
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12
Q

CN III Palsy Neuroimaging

A

Neuroimaging is almost always necessary. The approach to neuroimaging varies depending upon age, and whether the 3rd nerve palsy is isolated, and the degree of involvement of the extraocular muscles and pupil.
Consultation with a neuroradiologist may be helpful in determining the optimal imaging modalities and the order of imaging, particularly when aneurysm is a consideration.

Treatment is directed at the underlying etiology. The goal is to maximize visual function, including ocular alignmen

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13
Q

Trochlear Nerve (CN IV)

A

The fourth cranial nerve has the longest intracranial course and is the only cranial nerve that has a dorsal exit from the brainstem. CN IV innervates:
superior oblique muscle

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14
Q

CN IV Palsy Clinical Presentation

A

Binocular vertical diplopiaand/or subjective tilting of objects (torsional diplopia)
Objects viewed in primary position or especially in down-gaze may appear double (when going down a flight of stairs so that the pt. does not know which step to select).
The patient may adopt an anomalous head position to avoid diplopia because torsional and vertical diplopia often improve with head tilting to the side opposite the paralyzed muscle

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15
Q

CN IV Palsy Eye Examination

A

Ipsilateral hypertropia (the involved eye is deviated upward “nasal upshoot”) is present because the action of the superior oblique muscle (to rotate downward) is weak. The deviation is greater when gaze is in the direction toward the weak muscle. Thus, a right fourth nerve palsy causes greater hypertropia in left gaze.

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16
Q

CN4 Palsy Differential

A
Vasculopathic
HTN
Diabetes
Tumor
Congenital
Trauma
17
Q

CN IV Palsy neuroimaging

A

May or may not need neuroimaging, but it is best in evaluating the etiology.
Lumbar puncture may be warranted in patients who have normal imaging studies but are suspected of having subarachnoid space lesions.

Treatment for 4th nerve palsy is directed at the underlying etiology. The goal is to maximize visual function, and ocular alignment.

18
Q

Abducens Nerve (CN VI)

A

The sixth cranial nerve enters the orbit via the superior orbital fissure to innervate:
lateral rectus muscle

19
Q

CN VI Palsy Clinical Presentation

A

Binocular horizontal diplopia that worsens with gaze toward the defective lateral rectus muscle.
Early in the course, strabismus may be present only in the gaze toward the paralyzed side, but with time, the strabismus may be present when gazing straight ahead.

20
Q

CN VI Palsy Eye Examination

A

Primary position esotropia (eye turned in “crossed eyed”), worse in gaze toward the paretic muscle (lateral incomitance), and an ipsilateral abduction deficit

21
Q

CN6 Palsy Differential

A
Vasculopathic
HTN
Diabetes
Tumor
Elevated cranial pressure
Temporal arteritis
Pseudotumor cerebri
22
Q

CN VI Palsy Neuroimaging

A

Neuroimaging is indicated to exclude occult neoplasm in patients with nonisolated palsies, bilateral, progressive, or nonresolving palsy that persists beyond 6 to 12 weeks.
Imaging should be performed emergently when focal signs or papilledema are present.
The treatment depends upon the underlying cause. The goal is to maximize visual function, including alignment.
Treatment modalities may include alternate patching, prism therapy, strabismus surgery,andbotulinum toxin

23
Q

Primary Care Take Home Points

A

“Down and out” = CN 3 Palsy
“Nasal upshoot” = CN 4 Palsy
“Cross eyed” = CN 6 Palsy