occupational lung disease Flashcards

1
Q

what is the aim of occupational health

A

to promote and maintain the highest degree of physical, mental and social well being of workers in all occupations by preventing departures from health, controlling risks and adapting work to people

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2
Q

3 important legislations in occupational health

A

health and safety at work act (1974); control of substances hazardous to health (COSHH - 2002); report of injuries, disease and dangerous occurences at work (1995)

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3
Q

where do most occupational deaths occur (location)

A

offshore

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4
Q

10 examples of occupational lung diseases

A

asbestos related; occupation asthma; pneumoconiosis; byssinosis; berryllium disease; lung cancer; hypersensitivity pnuemonitis; infections; COPD

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5
Q

why might occupational lung diseases be under reported

A

people afraid if they report they will be told to stop working - they would be out of a job

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6
Q

what industries are highly susceptible to occupational lung diseases

A

construction; engineering; welding; foundries; mechanics; stonemasons; farmers

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7
Q

what is the largest single cause of work related fatal disease in the UK

A

asbestos

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8
Q

6 consequences of asbestos exposure (symptoms)

A

pleural plaques; diffuse pleural thickening; malignant mesothelioma; asbestosis; lung cancer; laryngeal cancer

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9
Q

what asbestos has the greater ability to reach alveoli

A

crocidolite (blue)

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10
Q

what are the types of asbestos

A

crocidolite (blue); amosite (brown); chrysotile (white)

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11
Q

what length of fibres are more resistant to phagocytosis clearance

A

> 5µm

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12
Q

when does asbestosis typically present

A

several decades following exposure

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13
Q

what fibres are usually seen in the lungs of those with asbesotsis

A

chrysotile

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14
Q

work aggravated vs irritant induced asthma

A

work aggravated - irritant provokes symptoms of pre-existing asthma;
irritant induced - asthma which is caused by inhaling a substance hazardous to heath in the workplace (reactive airways dysfunction syndrome)

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15
Q

3 levels of exposure that can result in irritant induced occupational asthma

A
  1. single exposure to high level irritant substance;
  2. multiple high-level exposures;
  3. chronic moderate-level exposure (more delayed symptom onset)
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15
Q

3 levels of exposure that can result in irritant induced occupational asthma

A
  1. single exposure to high level irritant substance;
  2. multiple high-level exposures;
  3. chronic moderate-level exposure (more delayed symptom onset)
16
Q

what is sensitiser induced occupational asthma

A

asthma caused by immunological sensitiation to agents in the workplace (T1 hypersensitivity)

17
Q

who should occupational asthma be reported to (and due to which act)

A

to Health and Safety Executive (HSE) under RIDDOR

18
Q

what is key to the history of occupational asthma

A

improves over weekends/ holidays (although this pattern may be lost over time)

19
Q

examples of causes + associated occupations for occupational asthma

A

cleaning agents - cleaners
flour - bakers
animals - lab workers
latex - healthcare
wood dust - carpentry
persulfate salts (hairdressers)
isocynates - paint spraying

20
Q

investigations for occupational asthma (5)

A

specific inhalation challenge; workplace challenge; history (improvement over holidays); serial peak flow; RAST (look for specific IgE)/skin prick testing

21
Q

treatment for occupational asthma

A

same as regular asthma (start with SABA reliever and low dose ICS preventer)

22
Q

hierachy of control measures (pyramid bottom to top, 4)

A

PPE -> administrative controls (relocation to a different job/work area) -> engineering controls (encolse work processe etc.) -> elimination or substitiution

23
Q

if your pt doesn’t want to inform their employer about occupational asthma diagnosis can you inform their employer

A

no - confidentially cannot be breached in this case

24
what is pneumoconioses
a group of diseases caused by inhalation of mineral dust - deposition of dust in alveoli
25
how are particles >10µm stopped from entering the lungs
>20µm trapped in nasal cavity 10-20µm trapped in upper branches of the RT, cleared by muco-ciliary escalator <10µm can penetrate the alveoli and are attempted to be transported to muco-cilary escalator by phagocytes these systems can be overwhelmed by large quantities
26
examples of causes + associated occupations for pneumoconioses
coal dust - coal workers asbestos - comstruction etc siliaca - miners, construction etc. slate - slate workers tin ore (stannosis) iron oxide (siderosis)
27
what harmful substances does coal dust contain (4)
carbon, kaolin, mica, silica
28
what is simple coal workers lung, symptoms and prognosis
tissue reaction to dust in the lung parenchyma, often asymptomatic and non-progressive if removed from exposure
29
coal workers lung investigations (2)
spirometry - obstructive CXR - nodular opacities
30
what is massive progressive fibrosis and symptoms
fibrotic masses in the upper/middle zones that cavitate - lung function lost (obstructuve pattern) and can caused SOB + cough w black sputum (dead lung tissue)
31
acute silicosis presentation, prog + CXR
presentation - progressive SOB and cough prognosis - often fatal over a few months CXR - pulmonary oedema
32
chronic silicosis CXR (3)
opacities (larger than CWP), more marked in upper lobes; pleural thickening; egg shell calcification of hilar lymphnodes
33
4 common occupational RTIs
viral RTIs; TB; legionnaires disease; psittacosis