Obstetrics Pt.3 Flashcards

1
Q

How is Rubella spread?

Why is its perinatal infection uncommon?

A
  • Togavirus spread by droplet transmission

- Very uncommon in the UK thanks to MMR

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2
Q

Describe Rubella screening

A
  • Prevalence of rubella has reached such low levels in the UK that screening is NO LONGER ROUTINELY OFFERED
  • For women who are screened and rubella antibody is NOT detected, they should be offered the MMR after pregnancy
  • NOTE: the vaccine itself is contraindicated in pregnancy because it is a live vaccine
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3
Q

What are the clinical features of Congenital Rubella Syndrome?

A
  • Sensorineural deafness
  • Congenital cataracts
  • Blindness
  • Encephalitis
  • Endocrine problems
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4
Q

What is the risk of CRS?

A
  • The risk of CRS decreases with gestation and the manifestations are less severe
  • Rubella infection before 11 weeks has 100% risk of CRS
  • Rubella infection > 20 weeks has no risk of CRS
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5
Q

What is the management of CRS?

A
  • If infection during pregnancy is confirmed, risk of CRS should be assessed
  • If it has occurred < 16 weeks, termination of pregnancy should be offered
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6
Q

What is Syphilis and its clinical features?

A
  • Caused by Treponema pallidum

Clinical Features
- Painless genital ulcer 3-6 weeks after infection is acquired (condylomata lata)

  • NOTE: this may be on the cervix and hence go unnoticed
  • Secondary manifestations occur 6 weeks to 6 months after infection with a maculopapular rash or lesions affecting the mucous membranes
  • If untreated, some will develop symptomatic cardiovascular tertiary syphilis and some will develop neurosyphilis
  • In pregnant women with early, untreated syphilis, most infants will be infected and 25% will be stillborn
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7
Q

What are the risks of syphilis in pregnancy?

A
  • FGR
  • Foetal hydrops
  • Congenital syphilis (may cause long-term disability)
  • Stillbirth
  • Preterm birth
  • Neonatal death
  • IMPORTANT: adequate treatment with benzathine penicillin markedly improved the outcome for the foetus
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8
Q

Describe Syphilis Screening in pregnancy

A
  • Routine antenatal screening is offered for ALL pregnant women
  • Treponemal antibodies are detected in serology
  • Non-treponemal tests detect non-specific treponemal antibodies
    • Venereal disease research laboratory (VDRL) test
    • Rapid plasma reagin test (RPR)
    • NOTE: they have a high false-positive rate
  • Treponemal tests detect specific treponemal
    • EIAs
      • Very sensitive and specific
    • Treponema pallidum haemagglutination assay (TPHA)
    • Fluorescent treponemal antibody-absorbed test (FTA-abs)
  • IMPORTANT: none of these tests will detect syphilis in the incubation stage
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9
Q

What is the management of syphilis in pregnancy?

A
  • Confirm the diagnosis and test for other STIs
  • GUM clinic should initiate appropriate contact tracing
  • Parenteral penicillin (benzathine penicillin) has a 98% success rate at preventing congenital syphilis
  • A Jarish-Herxheimer reaction may occur with treatment as a result of the release of proinflammatory cytokines in response to dying organisms
    • Causes worsening of symptoms and fever for 12-24 hours after starting treatment
    • May be associated with uterine contractions and foetal distress
    • So, women may be admitted during treatment for monitoring
  • If the woman is NOT treated during pregnancy, the baby should be treated after delivery immediately
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10
Q

What is Toxoplasmosis?

A
  • Caused by Toxoplasma gondii which is a protozoan found in cat faeces, soil or uncooked meat
  • 1/3 of people in the UK are probably infected with Toxoplasma at some point in their lives
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11
Q

Is toxoplasmosis screening offered?

A
  • NOT offered routinely because it is very rare for babies to be affected
  • Little evidence for the benefits of screening
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12
Q

What is some advice about toxoplasmosis prevention?

A
  • Avoiding eating rare or raw meat
  • Avoiding handling cats and cat litter
  • Wearing gloves and washing hands when gardening or handling soil
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13
Q

What are the clinical features of toxoplasmosis?

How does foetal damage and transmission rate change during pregnancy?

A
  • Initial infection is usually ASYMPTOMATIC or may cause flu-like illness
  • Parasitaemia occurs within 3 weeks
  • Infection in the first trimester is most likely to cause severe foetal damage but the transmission rate is low (10%)
  • In the third trimester, the transmission rates are much higher (85%) but the risk of foetal damage is low (10%)
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14
Q

What are the features of Severely Affected Infants of Toxoplasmosis?

A
  • Ventriculomegaly
  • Microcephaly
  • Chorioretinitis
  • Cerebral calcification
  • NOTE: most infants are asymptomatic at birth and develop symptoms later on
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15
Q

What is the management of toxoplasmosis?

A

`- Diagnosis is made by the Sabin Feldman Dye Test

  • NOTE: IgM antibody tests are also available but IgM may persist for months or years after infection
  • If an abnormal US raises suspicion of congenital toxoplasmosis, amniocentesis can be performed
  • PCR of amniotic fluid is highly accurate for identification of T. gondii
  • Spiramycin treatment can be used in pregnancy (3 week course of 2-3 g/day)
  • This reduces incidence of transplacental infection
  • If toxoplasmosis is found to be the cause of abnormalities on ultrasound, termination of pregnancy should be offered
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16
Q

What is cytomegalovirus?

A
  • CMV is a DNA herpes virus transmitted by the respiratory droplet route and excreted in the urine
  • 60% of women are seropositive for CMV when they become pregnant
  • 1-2 out of 200 infants in the UK are born with congenital CMV
  • Some will have problems at birth (e.g. hearing loss, learning difficulties) and others will be asymptomatic but go on to develop problems later on
  • Primary infection is more likely to cause congenital CMV
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17
Q

What are the clinical features of cytomegalovirus?

A
  • Primary infection usually produces no symptoms or mild flu-like symptoms in the mother
  • Diagnosis is usually made after abnormalities are seen on the ultrasound
  • Features in the foetus
    • Growth restriction
    • Microcephaly
    • Intracranial calcification
    • Ventriculomegaly
    • Ascites
    • Hydrops
  • Infants may present later with blindness, deafness or developmental delay
  • The neonate can also be anaemic and thrombocytopaenic with hepatosplenomegaly, jaundice and a purpuric rash
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18
Q

What is the management of CMV?

A
  • Serological diagnosis (CMV antibodies in an initially seronegative woman)
  • NOTE: IgM can persist for several months so IgM is insufficient to diagnose infection, it has to be a new finding in a woman who was IgM negative at the time of booking
  • The amniotic fluid can be tested by PCR
  • If abnormalities suggestive of congenital CMV are detected, termination of pregnancy should be discussed
  • CMV is a herpes virus, so it can be latent and be reactivated
  • It persists in the lymphocytes throughout life
  • Reactivation occurs via shedding in the genital, urinary or respiratory tract
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19
Q

What virus causes chickenpox?

What can chickenpox cause?

A
  • Caused by varicella zoster virus (VZV) which is transmitted by droplets and direct personal contact
  • Screening is NOT routinely recommended
  • Women identified as being seronegative can be considered for vaccination either prepregnancy or in the postnatal period
  • It can cause foetal varicella syndrome
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20
Q

What are complications of chickenpox in pregnant women?

A
  • Pneumonia
  • Hepatitis
  • Encephalitis
  • Non-immune pregnant women are more vulnerable to complications of chicken pox
  • The mortality rate is 5 times high in pregnant women compared to non-pregnant adults
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21
Q

What is the advice for pregnant women regarding chickenpox?

A
  • Asked whether they have had chickenpox before at the booking visit
  • If NOT, be advised to avoid contact during pregnancy
  • If they do come into contact with chickenpox, they should seek medical advice ASAP
  • Significant contact is defined as being in the same room as someone for 15 mins or more, or face-to-face contacts
  • Individuals with the virus are infectious 48 hours prior to the appearance of the rash until the vesicles crust over (around 5 days)
  • VZV IgG can be detected to confirm VZV immunity
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22
Q

How should non-immune women with chickenpox be managed?

A
  • Given VZIG as soon as possible
    • It is effective when given up to 10 days after contact
  • Women should be advised to inform the doctor if a rash develops
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23
Q

How is chickenpox in pregnancy managed?

A
  • Avoid contact with other pregnant women and neonates until the lesions have crusted over
  • Aciclovir 800 mg 5/day for 7 days should be prescribed if they present within 24 hours of the onset of the rash and they are > 20 weeks gestation
  • VZIG has NO therapeutic benefit once chickenpox has developed
  • Hospital admission should be considered if the following risk factors are present: smoking, chronic lung disease, corticosteroids or in latter half of pregnancy
  • Women who are hospitalised should be nursed in isolation from babies and pregnant women
  • Delivery during the viraemic period may be EXTREMELY HAZARDOUS
  • Patients should be given supportive treatment with IV aciclovir
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24
Q

What are the risks of chickenpox in pregnancy?

A
  • Thrombocytopaenia
  • DIC
  • Hepatitis
  • Varicella infection of the newborn
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25
Q

What is Congenital Varicella Syndrome?

A
  • Characterised by one or more of the following:
    • Skin scarring in a dermatomal distribution
    • Eye defects (microphthalmia, chorioretinitis, cataracts)
    • Hypoplasia of the limbs
    • Neurological abnormalities (microcephaly, cortical atrophy, mental restriction and dysfunction of bowel and bladder sphincters)
  • This occurs in a minority of infected foetuses (1%)
  • No cases have been reported if maternal infection has occurred after 28 weeks
  • If the mother has contracted chickenpox during the pregnancy, referral to foetal medicine specialist should be considered at 16-20 weeks or 5 weeks after infection for discussion and detailed ultrasound examination
    • NOTE: 5 week time lag is required as it takes time for features to manifest
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26
Q

How should maternal infection around the time of delivery be managed?

A
  • Significant risk of varicella of the newborn
  • Elective delivery should be avoided until 5-7 days after the onset of the rash to allow time for passive transfer of antibodies to the foetus
  • Neonatal ophthalmic examination done after birth
  • If birth occurs within 7 days of the onset of the rash or the mother develops chickenpox within 7 days of delivery, the neonate should be given VZIG
  • The infant should be monitored for signs of infection until 28 days after the onset of maternal infection
  • Neonatal infection should be treated with aciclovir
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27
Q

Is contact with shingles a problem?

A
  • Low risk of a non-immune pregnant woman getting chickenpox from someone with shingles
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28
Q

What is parvovirus infection and how is it transmitted?
What percentage of women are immune?
Is screening recommended?

A
  • Caused by parvovirus B19 which is transmitted through respiratory droplets
  • 50% of women of child-bearing age are immune and 50% are susceptible
  • Routine screening is NOT recommended
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29
Q

What are the clinical features of parvovirus?
When is the foetus most vulnerable?
What are complications of the virus and how is this investigated?

A
  • Asymptomatic or cause a mild flu-like illness and/or arthropathy
  • In children it causes slapped cheek syndrome (aka fifth disease, erythema infectiosum)
  • Foetus is most vulnerable in the second trimester
  • In most foetuses infected with parvovirus, there will be spontaneous resolution with no long-term sequelae
  • However, the infection can cause aplastic anaemia
  • The anaemic foetus could be come hydropic due to high output cardiac failure and liver congestion
  • Seen on ultrasound (high velocity in the foetal middle cerebral artery suggests anaemia)
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30
Q

How is parvovirus diagnosed?
When is foetal loss due to virus higher?
How is the virus managed?

A
  • Diagnosed by serology in mother (development of IgM antibodies to Parvovirus B19) having previously tested negative
  • PCR of virus in maternal and foetal serum/amniotic fluid is the most accurate test
  • A hydropic foetus may recover spontaneously or may require in utero transfusion
  • Infection in the first 20 weeks can lead to hydrops fetalis and intrauterine death (intrauterine transfusion is not possible at early gestations)
  • Foetal loss rate is much higher (10%) in the first 20 weeks
  • If the anaemia is treated by in utero transfusions, the foetus can make a complete recovery
  • Parvovirus does NOT cause neurological damage so if they survive the anaemia the outcome is usually normal
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31
Q

What is Listeria?

Where does it originate?

A
  • Caused by Listeria monocytogenes which is an aerobic and facultatively anaerobic motile Gram-positive bacillus
  • It is an obligate intracellular parasite
  • People with reduced cell-mediated immunity (i.e. pregnant women) are most at risk
  • Incidence of Listeria infection in pregnant women is 18 x higher than in the non-pregnant population
  • Usually comes from contaminated food, most commonly:
    • Unpasteurised milk
    • Ripened soft cheeses
    • Paté
  • Listeria does NOT survive in cooked or frozen food but can survive in refrigerated food (e.g. milk, soft cheese, prawns)
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32
Q

What are the clinical features of Listeria?
How does transmission to the foetus occur?
What are Listeria complications in pregnancy?

A
  • Flu-like illness with fever and general malaise
  • 1/3 of women will be asymptomatic
  • Transmission to the foetus can occur via:
    • Ascending route through the cervix
    • Transplacental
  • 20% of affected pregnancies result in miscarriage or stillbirth
  • 50% result in premature delivery
  • 38% neonatal mortality (due to respiratory distress, fever, sepsis or neurological symptoms)
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33
Q

What is the management of Listeria?

A
  • Diagnosed by isolation of the organism from blood, vaginal swabs or the placenta
  • Meconium staining of amniotic fluid may raise suspicion
  • IV antibiotics (AMPICILLIN 2 g every 6 hours)
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34
Q

What is Malaria?
Which pregnant women have increased risk of malaria?
Why may the parasite be missed in blood films?

A
  • Plasmodium falciparum carries the worse prognosis for mother and foetus
  • Most common in sub-Saharan Africa
  • Pregnant women have an increased risk of infection (especially if primiparous)
  • The parasite can sequestrate in the placenta (and be missed by blood films)
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35
Q

What are the clinical features of Malaria?

A
  • Pyrexia
  • Associated with miscarriage and preterm labour
  • Hypoglycaemia is common and may be severe in pregnancy
  • Pulmonary oedema can occur (high mortality)
  • Jaundice
  • Renal failure
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36
Q

What are the effects of Malaria on the foetus?

A
  • Preterm delivery
  • FGR
  • Higher rate of HIV transmission
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37
Q

What is the management of Malaria?

A
  • Symptomatic and supportive treatment
  • Antimalarials (depends on drug resistance and local patterns of disease)
  • Pregnant women planning to travel to endemic areas should be counselled about insecticides, mosquito nets and appropriate clothing
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38
Q

Describe herpes in pregnancy

A
  • This is DANGEROUS if acquired around the time of delivery
  • Double-stranded DNA virus
  • Neonatal herpes has a high morbidity and mortality
  • It occurs due to contact with infected secretions
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39
Q

What are the clinical features of herpes?

What are the subgroups of neonatal herpes?

A
  • Genital herpes causes ulcerative lesions on the vulva, vagina and cervix
  • Primary infection is usually more severe and can cause systemic symptoms and urinary retention
  • Neonatal herpes can be caused by HSV1 and HSV2
  • Neonatal herpes has THREE subgroups:
    • Localised to the skin, eye and mouth
    • Local central nervous system disease (encephalitis alone)
    • Disseminated infection with multiple organ involvement
  • Risk is greatest when the woman acquires a new infection (primary genital herpes) particularly within 6 weeks of delivery
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40
Q

What is the general management of herpes?

A
  • Swab to diagnose HSV
  • If herpes suspected, refer to a genitourinary physician to confirm the diagnosis by viral culture and PCR
  • Aciclovir (400 mg tds) is recommended
  • For women presenting within 6 weeks of expected delivery, type-specific HSV antibody testing is advisable
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41
Q

What is the management of primary infections of herpes in pregnancy?

A
  • If the infection occurs before the 6 weeks prior to expected delivery, the pregnancy should be managed expectantly and vaginal delivery anticipated
  • C-section for ALL women developing first-episode genital herpes in the 3rd trimester (particularly if within 6 weeks of delivery)
  • If the woman chooses vaginal delivery, rupture of membranes and invasive procedures should be avoided
  • IV aciclovir should be given intrapartum to the mother
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42
Q

What is the management of recurrent episodes of herpes?

A
  • Aciclovir 400 mg tds considered from 36 weeks gestation
  • Recurrent episodes occurring during pregnancy is NOT an indication for delivery by C-section
  • Invasive procedures during labour should be avoided if there are genital lesions
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43
Q

What is Group B Streptococcus?

When does transmission occur?

A
  • Streptococcus agalactiae is a Gram-positive coccus
  • It is a vaginal commensal that can cause sepsis in the neonate
  • Transmission occurs between the time of rupture of membranes to delivery
  • It is the most common cause of severe early-onset (within 7 days of delivery) infection in newborns
  • 21% of women carry GBS as a commensal
  • Routine screening is NOT carried out in the UK
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44
Q

What are the clinical features of Group B Streptococcus?

A
  • The mother does NOT experience symptoms because GBS is a commensal
  • Sepsis in the neonate
    • Collapse
    • Tachypnoea
    • Nasal flaring
    • Poor tone
    • Jaundice
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45
Q

What is the management of Group B Streptococcus?

A
  • Antenatal
    • If detected incidentally, antenatal treatment is NOT recommended because it does not reduce the likelihood of GBS colonisation at the time of delivery
  • Intrapartum antibiotic prophylaxis
    • Infection of the neonate occurs during labour
    • Antibiotics (penicillin or clindamycin) given in labour are 60-80% effective in reducing early-onset neonatal GBS infection
    • IV penicillin 3 g should be given as soon as possible after the onset of labour and 1.5 g four-hourly until delivery
    • Clindamycin 900 mg should be given IV 8 hourly if allergic to penicillin
  • Women undergoing an elective C-section in the absence of labour or membrane rupture do NOT need antibiotic prophylaxis
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46
Q

What are risk factors requiring GBS prophylaxis?

A
  • Intrapartum fever
  • Prolonged rupture of membranes greater than 18 hours
  • Prematurity less than 37 weeks
  • Previous infant with GBS
  • Incidental detection of GBS in current pregnancy
  • GBS bacteriuria
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47
Q

How does GBS affect the Neonate?

A
  • Symptoms occur at or soon after birth
  • Neonatal sepsis can rapidly kill
  • Blood cultures should be obtained before antibiotics are commenced
  • Antibiotics should be given based on trust guidelines
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48
Q

What is Chlamydia caused by?
Is there screening?
When does transmission occur and what are complications of chlamydia in the infant?

A
  • Caused by Chlamydia trachomatis which is an obligate intracellular organism
  • NOT routinely screened
  • Women under the age of 25 booking for antenatal care should be informed about the National Screening Programme
  • Transmission occurs at the time of delivery
  • Transmission can cause conjunctivitis and pneumonia in the infant
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49
Q

What are the clinical features of chlamydia in pregnancy?

What are the pregnancy risks?

A
  • Often asymptomatic in pregnancy
  • Risks
    • Preterm rupture of membranes
    • Preterm delivery
    • LBW
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50
Q

What is the management of chlamydia?

A
  • Azithromycin or erythromycin

- NOTE: tetracyclines should be avoided in pregnancy

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51
Q

What is gonorrhoea caused by?

How does infection present?

A
  • Caused by Neisseria gonorrhoeae which is a Gram-negative diplococcus
  • Infection is often asymptomatic or may present with mucopurulent discharge or dysuria
  • Transmission occurs at the time of delivery and can cause ophthalmia neonatorum
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52
Q

What are the risks of gonorrhoea?

A
  • Preterm rupture of membranes

- Preterm birth

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53
Q

What is the management of gonorrhoea?

A
  • Bacteriological swabs should be taken
  • Cephalosporins are effective against gonococcus
  • Empirical treatment for chlamydia should also be considered
  • Contact tracing should be arranged via a GUM clinic
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54
Q

When does HIV transmission occur?

A
  • Vertical transmission mainly occurs in the late third trimester, during labour, delivery or breast feeding
  • Part of routine antenatal screening
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55
Q

What are clinical features of HIV?

A
  • Begins with an asymptomatic stage
  • Followed by gradual compromise of immune function leading to AIDS
  • The interval from HIV to AIDS can be very long
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56
Q

What is the management of HIV in the mother?

A
  • The risk of vertical transmission is affected by maternal viral load, obstetric factors and infant feeding
  • Reducing the risk of vertical transmission
    • Antiretroviral therapy (antenatally and intrapartum in the mother, for the first 4-6 weeks of life for the baby)
    • Delivery by elective C-section in the presence of high viral load
    • Avoid breastfeeding
  • Planned vaginal delivery is possible if mother’s viral load < 50 copies/mL at 36 weeks gestation
  • C-section recommended in women with hepatitis C coinfection
  • Women with a high viral load should receive IV azidothymidine (AZT) if they are undergoing a planned C-section or present with spontaneous rupture of membranes
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57
Q

What is the management of HIV in the infant?

A
  • Cord clamped ASAP and the baby bathed immediately after birth
  • Advised not to breastfeed baby
  • All infants given azidothymidine for 4-6 weeks after birth
  • Neonates test positive for HIV antibodies because of passive transfer from the mother
  • HIV diagnosis in the neonate requires PCR (normally carries out at birth, 3 weeks, 6 weeks and 6 months)
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58
Q

What Hepatitis B and how is it mainly transmitted?
Is it part of the routine screening?
How can transmission be prevented?

A
  • DNA virus that is mainly transmitted in the blood, but also saliva, semen and vaginal fluid
  • It is part of the routine screening for pregnant women
  • High rate of vertical transmission in women who are positive for HBeAg
  • Transmission is 95% preventable through administration of the vaccine and immunoglobulin at birth
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59
Q

What are the clinical features of Hepatitis B?

A
  • Many people have no symptoms

- The incubation period can be 6 weeks to 6 months

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60
Q

How and when can HepB vertical transmission be reduced?

A
  • Hepatitis B immunoglobulin and the vaccine help reduce vertical transmission
  • The immunoglobulin should be given immediately after delivery
  • The vaccine is given at birth, 1 month and 6 months
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61
Q

In what conditions is the risk of HepC transmission increased?
Is HepC included in routine screening?

A
  • Risk of transmission is higher in HIV co-infection

- NOT part of routine screening

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62
Q

What are the clinical features of Hepatitis C?

A
  • Can lead to cirrhosis and hepatocellular carcinoma

- 80% are asymptomatic

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63
Q

What is the management of Hepatitis C?

A
  • Detect anti-HCV antibodies
  • Confirm with PCR for the virus
  • In non-pregnant adults, administer interferon and ribavirin (CONTRAINDICATED in pregnancy)
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64
Q

What are the three stages of labour?

A
  • 1st STAGE: begins with the onset of contractions and ends when full cervical dilatation has been reached
  • 2nd STAGE: begins with full cervical dilatation and ends with the birth of the baby
  • 3rd STAGE: begins with the birth of the baby and ends with complete delivery of the placenta and membranes
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65
Q

What are the 3Ps of Labour?

A
  • Powers
  • Passage
  • Passenger
  • If any of these three are abnormal, it is likely to result in an abnormal delivery
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66
Q

What are the boundaries of the pelvic inlet?

Read Netters Flashcards

A
  • Anteriorly - upper border of the symphysis pubis
  • Laterally - upper margin of the pubic bone, ileopectineal lien and the ala of the sacrum
  • Posteriorly - promontory of the sacrum
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67
Q

What are the normal diameters of the pelvic inlet? (Read Netters Flashcards)

A
  • Transverse = 13.5 cm
  • Anterior-posterior = 11.0 cm
  • The foetal head usually enters the pelvis orientated in the transverse position
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68
Q

What is the angle of the pelvic inlet?

A
  • The angle of the inlet is normally 60 degrees to the horizontal
  • NOTE: this can be higher in Afro-Caribbean women
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69
Q

What is the midpelvis?

A
  • Described as the area bounded anteriorly by the middle of the symphysis pubis , laterally by the pubic bones, the obturator fascia and the inner aspect of the ischial bone and spines, and posteriorly by the junction of the second and third sections of the sacrum
  • The midpelvis is almost round (similar transverse and anterior-posterior diameter of around 12 cm)
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70
Q

Why are the ischial spines of the midpelvis palpable vaginally?

A
  • To assess descent of the presenting part on vaginal examination
    • Station zero is at the level of the ischial spines
    • Instrumental delivery is only possible if the foetal head has reached the level of the ischial spines or below
  • To provide a local anaesthetic pudendal nerve block (this may be used for a vacuum or forceps delivery)
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71
Q

What are the boundaries of the pelvic outlet?

A
  • Anteriorly - lower margin of the symphysis pubis
  • Laterally - descending ramus of the pubic bone, ischial tuberosity and the sacrotuberous ligament
  • Posteriorly - last piece of the sacrum
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72
Q

What are the diameters of the pelvic outlet?

A
  • Anterior-posterior = 13.5 cm
  • Transverse = 11 cm
  • IMPORTANT: this means that at the inlet, the transverse diameter is widest, then at the outlet, the AP diameter is widest. This means that the foetal head must rotate from a transverse to AP position as it passes through the pelvis
    • This usually happens at the midpelvis where the dimensions are roughly equal
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73
Q

How does the pelvic shape change at the end of the third trimester?

A
  • Pelvic ligaments loosen towards the end of the third trimester, the pelvis becomes more flexible and the diameters may increase during labour
  • The pelvic dimensions can also be enhanced by changing the maternal position during labour
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74
Q

What are some of the different pelvic shapes?

A
  • The gynaecoid pelvis is the most favourable for labour and is also the MOST COMMON
  • The android-type pelvis predisposes to failure of rotation and deep transverse arrest
  • The anthropoid pelvis encourages an occipito-posterior position
  • A platypelloid pelvis is also associated with an increased risk of obstructed labour due to failure of the head to engage, rotate or descend
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75
Q

What is the pelvic floor and how does it affect the foetal head? (Read Netters Flashcards)

A
  • Formed by the two levator ani muscles which form a musculofascial gutter during the 2nd stage of labour
  • This encourages the foetal head to flex and rotate as it descends from the midpelvis to the pelvic outlet
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76
Q

What is the perineum?

A
  • This is the final obstacle
  • The perineal body is a mass of fibrous and muscular tissue lying between the vagina and the anus
  • This is relatively resistant in the nulliparous woman
  • Vaginal birth may result in tearing or an episiotomy
  • The perineum is more stretchy in multiparous women, resulting in faster labour
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77
Q

What is the foetal skull made out of?

A
  • Vault
  • Face
  • Base
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78
Q

How are the sutures described in a foetus?

A
  • At the time of labour the sutures of the vault are soft, unossified membranes
  • The sutures of the face and skull base are firmly united
  • The sutures allow the bones to move and overlap
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79
Q

What is the vault composed of?

A
  • The vault is composed of the parietal bones and parts of the occipital, frontal and temporal bones
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80
Q

What are between the bones of the vault?

A

4 membranous structures

  • Sagittal
  • Frontal
  • Coronal
  • Lambdoidal
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81
Q

What are fontanelles?

A
  • Fontanelles are the junctions of the sutures
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82
Q

Where is the anterior fontanelle?

A
  • The anterior fontanelle (aka bregma) is at the junction of the sagittal, frontal and coronal sutures
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83
Q

Where is the posterior fontanelle?

A
  • The posterior fontanelle lies at the junction of the sagittal suture and the lambdoidal suture (this is smaller and triangular)
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84
Q

Why are foetal bones in the skull compressible?

A
  • Allows moulding to occur as the head passes through the pelvis
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85
Q

What may severe moulding or moulding early in labour be due to?

A
  • Obstructed labour
  • Foetal malposition (failure of the head to rotate)
  • Cephalopelvic disproportion (mismatch between the size of the foetal head and maternal pelvis)
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86
Q

What is the vertex?

A
  • The vertex is the area bounded by the two parietal bones and the anterior and posterior fontanelles
  • In normal labour, this is the presenting part
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87
Q

What is the posterior fontanelle used to define?

A
  • The position of the foetal head in relation to the pubic symphysis
  • By feeling the position of the anterior and posterior fontanelles on vaginal examination, the foetal head position during labour can be ascertained
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88
Q

Which position is most favourable for spontaneous vaginal birth?

A
  • Occipito-anterior (OA) position
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89
Q

Which position is a malposition that may result in prolonged labour, instrumental delivery or C-section?

A
  • Occipito-transverse (OT) position
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90
Q

What is the shape of the foetus’ head?

A
  • Ovoid
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91
Q

What does the attitude of the foetal head mean?

A
  • The attitude of the foetal head refers to the degree of flexion and extension of the upper cervical spine
  • Different longitudinal diameters are presented to the pelvis depending on the attitude of the femoral head
  • With further extension of the head, the occipito-frontal diameter presents
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92
Q

What is the greatest longitudinal diameter which a foetus may present with?

A
  • The mento-vertical and is around 13 cm

- This is known as brow presentation and is usually too large to pass through the normal pelvis

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93
Q

What is a face presentation?

A
  • The submento-bregmatic diameter is from below the chin to the anterior fontanelle (roughly 9.5 cm)
    • This is called face presentation
    • This can deliver vaginally when the chin is anterior (mento-anterior position)
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94
Q

Describe the general physiology of labour

A
  • The trigger that initiates human labour is poorly understood
  • The cervix, which is initially long, firm and closed with a protective mucus plug must soften, shorten, thin out (effacement) and dilate for labour to progress
  • The uterus must change from a state of relaxation to an active state of regular, strong, frequent contractions to facilitate transit of the foetus through the birth canal
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95
Q

What happens to the Uterus during labour?

A
  • Prostaglandins and oxytocin increase intracellular calcium ions, thereby stimulating contraction
  • Beta-adrenergic compounds and CCBs do the opposite
  • Unique to the uterus, the actin-myosin interaction occurs along the full length of the filaments so that there is a degree of shortening with each successive interaction
  • This progressive shortening is called retraction and occurs in the upper part of the uterus
  • This results in the development of the thicker, actively contracting ‘upper segment’
  • Meanwhile, the lower segment of the uterus becomes thinner and more stretched
  • This will eventually result in the cervix being taken up (effacement) into the lower segment of the uterus thereby forming a continuum for the passage of the foetus
  • There are gap junctions between myometrial cells in the uterus which facilitates the passage of various products of metabolism and electrical current between cells
  • These gap junctions are absent during pregnancy but appear in abundance at term
  • The increase in the number of gap junctions allows greater coordination of myocyte activity
  • Prostaglandins stimulate the formation of gap junctions
  • Uterine contractions are involuntary
  • Frequency of contractions varies during labour and with parity
  • Through most of labour, they occur at intervals of 2-4 minutes
    • This is described in terms of frequency within a 10-minute period (e.g. 2 in 10)
  • The duration of contractions varies from 30-60 seconds
  • The frequency of contractions can be recorded using a CTG
  • The amplitude of intrauterine pressure generated with each contraction ranges from 30-60 mm Hg
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96
Q

What happens to the Cervix during labour?

A
  • Contains myocytes and fibroblasts separated by ECM
  • Interactions between ECM components keeps the cervix closed early in pregnancy
  • Under the influence of hormonal mediators (e.g. prostaglandins), there is an increase in proteolytic activity and a reduction in collagen and elastin
  • Interleukins cause pro-inflammatory changes with significant invasion of neutrophils
  • Dermatan sulphate is replaced by hyaluronic acid (more hydrophilic) resulting in an increase in water content of the cervix
  • This leads to cervical softening/ripening so that when contractions begin, they can bring about effacement and dilatation
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97
Q

How does progesterone maintain uterine relaxation?

A
  • Suppresses prostaglandin production
  • Inhibits communication between myometrial cells
  • Prevents oxytocin release
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98
Q

What does oestrogen do prior to labour?

A
  • Oestrogen opposes the action of progesterone
  • Prior to labour, there is a decrease in progesterone receptors and an increase in oestrogen concentration relative to progesterone
  • Prostaglandin synthesis by the chorion and decidua is enhanced, leading to an increase in calcium in the myometrial cells
  • The changes in hormones also increases gap junction formation
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99
Q

Where is CRH produced and what is its action before labour?

A
  • In the placenta

- Potentiates prostaglandins and oxytocin for myometrial contractility

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100
Q

What produces oxytocin and cortisol before labour and what are their actions?

A
  • Production of oxytocin and cortisol by the foetus also stimulates the conversion of progesterone to oestrogen
101
Q

As labour becomes established, what increases the output of oxytocin?

A
  • Ferguson reflex
102
Q

What is the ferguson reflex?

A
  • This is a phenomenon where pressure from the foetal presenting part against the cervix is relayed via a reflex arc and results in increased oxytocin release from the maternal pituitary gland
  • This stimulates uterine contractions, which, in turn, increases pressure on the cervix
103
Q

How is labour diagnosed?

A
  • Strong regular painful contractions resulting in progressive cervical change
  • In practice, a diagnosis is confirmed when a woman presents with contraction-like pains and the midwife performs a vaginal examination revealing effacement and dilatation of the cervix
  • Loss of a ‘show’ (blood-stained plug of mucus passed from the cervix) or spontaneous rupture of membranes does NOT define labour
104
Q

How long is someone’s first labour?

A
  • 8 hours
105
Q

How long is someone’s 2nd labour?

A
  • 5 hours
106
Q

What is the definition of the 1st stage of labour?

A
  • Defined as the time from the diagnosis of labour to full dilatation of the cervix (10 cm)
107
Q

What are the two phases of the 1st stage of labour?

A
  • Latent phase- ime between the onset of regular painful contractions and 3-4 cm cervical dilatation
    • The cervix becomes fully effaced
    • Effacement is a process by which the cervix shortens and becomes incorporated into the lower segment of the uterus
    • Effacement may begin in the weeks before the onset of labour but is complete by the end of the latent phase
    • Effacement and dilatation occur as consecutive events in the nulliparous woman (but may occur simultaneously in the multiparous woman)
    • Dilatation is expressed in centimetres from 0 to 10 cm
    • The latent phase usually lasts from 3-8 hours
  • Active phase- time between the end of the latent phase (3-4 cm dilatation) and full cervical dilatation (10 cm)
    • Usually lasts 2-6 hours (shorter in multiparous women)
    • Rate of cervical dilatation is considered abnormal if it is < 1 cm in 2 hours
108
Q

What is the definition of the 2nd stage of labour?

A
  • Defined as the time from full dilatation of the cervix to delivery of the foetus
109
Q

What are the two phases of the 2nd stage of labour?

A
  • Passive Phase - time between full dilatation and the onset of involuntary expulsive contractions
    • There is NO maternal urge
    • Foetal head is relatively high in the pelvis
    • Duration of 1-2 hours is recommended to allow the head to rotate and descend before active pushing
  • Active Second stage
    • There is maternal urge to push because the foetal head is low causing a reflex to bear down
    • This should last no longer than 2 hours in a nulliparous woman
110
Q

What is the third stage?

A
  • From delivery of the foetus until complete delivery of the placenta and membranes
  • The placenta is usually delivered within minutes of the baby
  • The third stage lasting > 30 mins is considered abnormal (unless the woman has opted for ‘physiological management’
111
Q

What is engagement?

A
  • The foetal head normally enters the pelvis in the transverse position
  • Engagement is said to have occurred when the widest part of the presenting part has passed successfully through the pelvic inlet
  • In most nulliparous women, this will have occurred prior to labour
  • If > 2/5 of the foetal head is palpable abdominally, the head is NOT yet engaged
112
Q

Describe descent

A
  • During the first stage and passive second stage, descent results from uterine contractions
  • In the active second stage, voluntary efforts by the mother help push the foetus along
113
Q

Describe flexion

A
  • As the head descends into the narrower midpelvis, flexion of the head occurs
  • This reduces the presenting diameter of the foetal head
114
Q

What is internal rotation?

A
  • If the head is well flexed, the occiput will be the leading point
  • As it reaches the sloping gutter of the levator ani muscles, it will be encouraged to rotate anteriorly so that the sagittal suture lies in the AP diameter of the pelvic outlet
  • If the foetus is engaged in the OP position, internal rotation can occur to an OA position (this may increase duration of labour as it is a longer internal rotation)
  • Alternatively, the OP position may persist resulting in a ‘face to pubes’ delivery
  • This may result in obstructed labour
115
Q

What is extension?

A
  • After internal rotation, the occiput is beneath the pubic symphysis and the bregma is near the lower border of the sacrum
  • The flexed head then extends
  • The occiput escapes from underneath the pubic symphysis and distends the vulva
  • This is called crowning of the head
  • The head extends further and the bregma, face and chin appear in succession over the posterior vaginal opening and the perineal body
  • Some degree of perineal tearing may occur
116
Q

What is restitution?

A
  • When the head is delivering, the occiput is directly anterior
  • After it has crossed the perineum, the head realigns itself with the shoulders which have entered the pelvis in an oblique position
  • This is a very slight movement (through 1/8 of a circle)
117
Q

What is external rotation?

A
  • To be delivered, the shoulders rotate into the direct AP plane
  • When this happens, the occiput rotates further and into the transverse position
118
Q

Describe delivery of the shoulders and foetal body

A
  • After restitution and external rotation have occurred, the shoulders will be in the AP position
  • The anterior shoulder (under the pubic symphysis) delivers first, followed by the posterior shoulder
  • Traction may be applied by pulling downwards on the foetal head to help release the anterior shoulder from beneath the pubic symphysis
  • The rest of the foetal body is usually delivered quite easily
119
Q

What should be included in the history when managing normal labour?

A
  • Previous births and size of previous babies
  • Previous C-section
  • Onset, frequency, duration and perception of strength of contractions
  • Whether membranes have ruptured and, if so, colour and amount of amniotic fluid lost
  • Presence of abnormal vaginal discharge or bleeding
  • Recent activity of the foetus (foetal movement)
  • Medical or obstetric issues of notes (e.g. diabetes, hypertension, FGR)
  • Any special requirements
  • Maternal expectations of labour and delivery
  • Birth preferences or birth plan
120
Q

What should be included in the general examination when managing normal labour?

A
  • BMI- High BMI may complicate labour
  • Temperature, pulse and blood pressure should be recorded
  • Urine tested for protein, blood, ketones, glucose and nitrates
121
Q

What should be included in the abdominal examination when managing normal labour?

A
  • Foetal lie (longitudinal, transverse or oblique)
  • Presentation (cephalic or breech)
    • If cephalic, the degree of engagement must be determined in terms of fifths of the face that are palpable abdominally
  • A head that remains high (5/5 palpable) and unengaged (>2/5 palpable) is a poor prognostic sign for vaginal delivery
  • If there is any doubt, an ultrasound scan should be performed to identify the reason for the head being high (e.g. placenta praevia, OP position)
  • Abdominal examination should include an assessment of the contractions (mainly the strength of contractions, which is not shown on CTG)
122
Q

What should be included in the vaginal examination when managing normal labour?

A
  • The cervix should be examined for position, length and effacement, dilatation and application to the presenting part
  • ‘Station’ refers to how far the foetal head has descended into the pelvis
  • The length of the cervix at 36 weeks gestation should be around 3 cm - this gradually shortens through the process of effacement
  • Dilatation is assessed digitally in centimetres
  • At around 4 cm dilated the cervix should be fully effaced
  • When no cervix can be felt, it means that the cervix is fully dilated (10 cm)
  • Vaginal examination also allows assessment of foetal head position, station, attitude and the presence of caput or moulding
  • Normally, the vertex is the presenting point
  • The occiput can be identified by feeling the triangular posterior fontanelle with the three suture lines
  • Failure to feel the posterior fontanelle may be due to the head being deflexed, the occiput is posterior or there is so much moulding that the sutures cannot be felt
  • Normally the occiput is transverse (OT position) or anterior (OA position)
  • Assessing the distance between the leading part of the head to the ischial spines will give an estimation of the station
  • If the membranes have ruptured, the amount and colour of amniotic fluid draining should be noted (a good amount of clear fluid is good)
123
Q

What care should women with no risk factors following the admission history and examination be managed under?

A
  • Midwifery care

- If there are risk factors, involvement of the on-call obstetric team may be necessary

124
Q

What observations should women in labour have done?

A
  • Pulse measured hourly
  • Blood pressure and temperature measured every 4 hours
  • Passage of urine should be noted and it should be tested for ketones and protein
  • Once the second stage of labour has been entered, blood pressure and pulse should be measured hourly and vaginal examinations should be offered every hour
125
Q

How does hypoxia occur during labour?

A
  • Contractions disrupt placental blood flow and oxygen transfer
  • The hypoxia leads to anaerobic respiration, which can result in metabolic acidosis if prolonged and severe
    • This can cause neuronal damage
  • Hypoxia and acidosis cause a characteristic change in foetal heart rate (FHR) pattern
    • This can be detected by auscultation or CTG
126
Q

What is meconium?

When is it excreted?

A
  • Meconium is often passed by a healthy foetus at or after term due to maturation of the GI tract (usually thin and dark green/brown)
    • It may also be expelled when a foetus is exposed to marked intrauterine hypoxia or acidosis (thick and brighter green)
127
Q

What are the foetal assessment options in labour?

A
  • Inspection of amniotic fluid
  • Intermittent auscultation using a Pinard stethoscope or hand-held Doppler
  • Continuous external electronic foetal monitoring (EFM) using CTG
  • Continuous internal electronic foetal monitoring (EFM) using foetal scalp electrode (FSE) and CTG
  • Foetal scalp blood sampling (FBS)
128
Q

How should FHR be auscultated?

A
  • Using a Pinard stethoscope or by using a hand-held Doppler device
  • It should be listened to for at least 1 minute after a contraction
  • Repeat every 15 mins during the first stage of labour
  • Repeat every 5 mins during the second stage of labour
  • EFM (electronic foetal monitoring using CTG) may be used if complications occur
129
Q

What are the indications for continuous EFM?

A
  • Significant meconium staining of the amniotic fluid
  • Abnormal FHR detected by intermittent auscultation
  • Maternal pyrexia
  • Fresh vaginal bleeding
  • Augmentation of contractions with oxytocin infusion
  • Maternal request
130
Q

Why may the CTG quality be poor?

A

Due to

  • foetal position
  • maternal obesity
131
Q

What can overcome poor CTG quality?

A
  • FSE may overcome this issue
  • It is fixed onto the foetal scalp and picks up the FHR directly
  • It does NOT cause any harm to the foetus but requires a certain degree of cervical dilatation and the membranes must be ruptured
132
Q

What features of a CTG can be looked at?

A
  • Baseline rate
  • Variability
  • Accelerations
  • Deccelerations
133
Q

Describe how you would use “reassuring” and “non-reassuring” to describe CTG

A
  • If all features are reassuring = normal CTG
  • If 1 feature is non-reassuring = suspicious CTG
  • If 2 or more features are non-reassuring or any one abnormal feature = pathological CTG
134
Q

Due to CTG’s high false positive rate, what can be done to prevent unnecessary intervention?

A
  • Foetal scalp blood sampling (FBS) may be performed during labour to measure foetal pH and base excess
135
Q

What is a partogram?

What are other important observations in labour?

A
  • A graphic record of labour
  • Allows instant visual assessment of the progress of labour based on the rate of cervical dilatation compared with an expected norm (according to parity)
  • Other important observations include frequency and strength of contractions, descent of the head, station, amount and colour of amniotic fluid draining and basic observations of maternal wellbeing
136
Q

Describe management during the first stage of labour?

A
  • Pain relief when needed
  • Adequate hydration and light diet to prevent ketosis (which can impair uterine contractility)
  • Women in the latent phase of labour should be encouraged to mobilise and should be managed away from the labour suite
  • Intervention should be avoided where possible
  • Vaginal examinations performed every 4 hours to determine whether the active phase has been reached (around 4 cm dilatation and full effacement)
  • Increase vaginal examinations if the midwife suspects that progress of labour is unusually slow or fast or if there are foetal concerns
  • Descent of the presenting part should also be recorded
  • During this phase, the membranes may be intact or may have ruptured
  • Standing upright may encourage progress of labour (so mobility should be encouraged)
  • Antacids should be given to women who have been given opioid analgesia
137
Q

Describe the management during the 2nd stage of labour

A
  • First sign of the second stage is likely to be an urge to push experienced by the mother
  • Full dilatation of the cervix should be confirmed by a vaginal examination if the head is not visible
  • The woman will get an expulsive reflex with each contraction and will generally take a deep breath, hold it and strain down (Valsalva)
  • Women should be discouraged from lying supine or semi-supine
  • Maternal and foetal surveillance should intensify
  • Use of regional anaesthesia (epidural or spinal) may interfere with the normal urge to push - meaning that the second stage is more often diagnosed on routine scheduled vaginal examination
  • In all cases, the baby should be delivered within 4 hours of reaching full dilatation
138
Q

How is descent and delivery of the head managed?

A
  • Can be judged by watching the perineum
  • Between contractions, the elastic tone of the perineal muscles will push the head back into the pelvic cavity
  • When the head no longer recedes between contractions it is called crowning
  • This indicates that delivery is imminent
  • As crowning occurs, the hands of the midwife are used to flex the foetal head and guard the perineum
  • Once the head has crowned, the woman should be discouraged from bearing down by telling her to take rapid, shallow breaths
139
Q

What is the immediate care of the neonate?

A
  • The baby will usually take its first breath within seconds
  • There is no need for immediate clamping
  • The baby’s head should be kept dependent to allow mucus in the respiratory tract to drain
  • After clamping and cutting the umbilical cord, the baby should have an Apgar score calculated at 1 minute of age and then repeated again at 5 minutes
  • Immediate skin-to-skin contact between mother and baby will help bonding and promote the further release of oxytocin which will encourage uterine contractions
  • The baby should be dried and covered with a warm blanket or towel
  • Initiation of breastfeeding should be encouraged within the first hour of life
  • Routine measurements of newborn head circumference, birthweight and temperature should be measure soon after this hour
  • The first dose of vitamin K should be given in the delivery room
140
Q

What is considered prolonged in the 3rd stage of labour?

A
  • If > 30 minutes
141
Q

What is the active management of the 3rd stage of labour?

A
  • Recommended to ALL women
  • Controlled cord traction
  • Reduces postpartum haemorrhage from 15% to 5%
  • When the signs of placental separation are recognised, controlled cord traction is used to expedite delivery of the placenta
  • When a contraction is felt, the left hand should be moved suprapubically and the fundus elevated with the palm of the hand facing towards the mother
  • Uterine inversion is a rare complication
  • In 2% of cases, the placenta will not be expelled by this method
  • If no bleeding occurs, another attempt should be made after 10 mins
  • If this fails, the placenta is ‘retained’ and requires manual removal under general or regional anaesthesia in the theatre
  • NOTE: delayed cord clamping from 1-3 minutes may be preferable
142
Q

What is the physiological management of the 3rd stage of labour?

A
  • Placenta is delivered by maternal effort with no uterotonic drugs
  • Associated with heavier bleeding
  • If haemorrhage occurs or the placenta is undelivered after 60 mins, active management should be recommended
143
Q

What should be inspected after the completion of the 3rd stage of labour?

A
  • After completion of the third stage, the placenta should be inspected for missing cotyledons or a succenturiate lobe
    • If suspected, examination under anaesthesia and manual removal of placental tissue (MROP) should be arranged
  • Finally, the vulva should be inspected for tears
144
Q

Give a summary of the features of a normal labour

A
  • Spontaneous onset at 37-42 weeks
  • Singleton pregnancy
  • Cephalic vertex presentation
  • No artificial interventions
  • Cervical dilatation of at least 1 cm every 2 hours in the active phase of the first stage
  • Active second stage no more than 2 hours in primiparous and 60 mins in multiparous women
  • Spontaneous vaginal delivery
  • Third stage lasting no more than 30 mins with active management
145
Q

What are features of abnormal labour?

A
  • Poor progress (delayed cervical dilatation or descent of presenting part)
  • Foetus shows signs of compromise
  • Foetal malpresentation
  • Multiple pregnancy
  • Uterine scar
  • Induced labour
146
Q

What are the patterns of abnormal progress in labour?

A
  • Partograms help detect poor progress
  • Prolonged Latent Phase
    • Latent phase is longer than expected
    • More common in primiparous women
    • Best managed with simple analgesics, mobilisation and reassurance
    • This is very frustrating for the woman
  • Primary Arrest
    • < 2 cm cervical dilatation per 4 hours in active 1st stage
    • More common in primiparous women
    • Most commonly caused by insufficient uterine contractions
    • Other causes: cephalopelvic disproportion, malposition and malpresentation
  • Secondary Arrest
    • Occurs when progress in the active first stage is initially good but then slows or stops altogether, usually after 7 cm dilatation
    • May be due to inefficient uterine contractions, malposition, malpresentation and cephalopelvic disproportion
  • Arrest in second stage of labour
    • Occurs when delivery is NOT imminent after the usual interval of pushing in the second stage of labour
    • May be due to inefficient uterine activity, malposition, malpresentation, CPD or resistant perineum
147
Q

How is poor progress in 1st stage of labour defined?

A
  • Defined as cervical dilatation of less than 2 cm in 4 hours
  • Usually associated with failure of descent and rotation of the foetal head
  • May be due to any of the 3 Ps: powers, passage, passenger
148
Q

What is Dysfunctional Uterine Activity? (Powers)

A
  • MOST COMMON cause of poor progress in labour
  • More common in primigravidae and in older women
  • Characterised by weak, irregular and infrequent contractions
  • Uterine contractions are usually assessed by carrying out clinical examination and by external uterine tocography
  • NOTE: intrauterine pressure catheters are available but are invasive
  • 4-5 contractions per 10 mins is ideal
  • If contractions are less frequent, more frequent examinations may be required (vaginal examination every 2 hours rather than 4 hours)
  • If delay is confirmed, the woman should be offered artificial rupture of membranes (ARM)
  • If there is still poor progress after 2 more hours, advice should be sought from an obstetrician about using an oxytocin infusion to augment the contractions
    • The infusion is started slowly initially, then increased every 30 mins according to a protocol
    • Continuous EFM is necessary because excessive contractions can cause foetal compromise
    • Women should be offered an epidural before oxytocin is started
149
Q

Are multiparous women more or less likely to experience poor progres due to dysfunctional uterine activity?

A
  • Less likely

Always exclude alternative causes e.g. malposition

150
Q

What may excessive uterine contractions in an obstructed labour result in?

A
  • Uterine rupture
151
Q

What is recommended if progress fails despite 4-6 hours of augmentation with oxytocin?

A
  • Caesarean section
152
Q

What is cephalopelvic distortion?

How may it occur? (Passages and Passenger)

A
  • CPD implies an anatomical disproportion between the foetal head and the maternal pelvis (either due to large head or small pelvis)
  • The pelvis may be abnormally small due to previous fracture or metabolic bone disease
  • Obstructive hydrocephalus can cause macrocephaly
  • Relative CPD is more common and occurs with malposition of the foetal head
  • The occipito-posterior position is associated with deflexion of the foetal head, which present a larger skull diameter to the maternal pelvis
  • Oxytocin can be given with caution in a primigravida woman with mild to moderate CPD provided that the CTG is normal
  • Oxytocin must NEVER be used in a multiparous woman where CPD is suspected
153
Q

What are findings suggestive of CPD?

A
  • Foetal head not engaged
  • Progress is slow or arrests despite efficient uterine contractions
  • Vaginal examination shows severe moulding and caput formation
  • Head is poorly applied to the cervix
  • Haematuria
154
Q

Describe malpresentation

A
  • Firm application of the foetal presenting part onto the cervix is necessary for good progress
  • A face presentation may apply poorly to the cervix resulting poor progress, but vaginal birth is still possible
  • Brow presentation (neck is slightly less extended than in face presentation) is associated with mento-vertical diameter presenting
    • This is too large to fit through the bony pelvis unless flexion or hyperextension to a face presentation occurs
    • Brow presentation often manifests as poor progress in the first stage
  • Shoulder presentations cannot be delivered vaginally so poor progress will occur
  • Malpresentation is more common in women of high parity
  • Malpresentation carries a risk of uterine rupture if labour is allowed to continue without progress
155
Q

What are some abnormalities of birth canal?

What is cervical dystocia?

A
  • CPD can delay progress
  • Unsuspected fibroids in the lower uterine segment can prevent descent of the foetal head
  • Cervical dystocia can also cause delay - this is when the cervix is non-compliant and effaces but fails to dilate because of severe scarring or rigidity (usually due to previous cervical surgery)
  • C-section may be necessary
156
Q

From the onset of the second active stage, when should birth take place within?

A
  • 3 hours = nulliparous

- 2 hours = parous

157
Q

How is delay diagnosed? (Poor progress in second stage)

A
  • if delivery is NOT imminent after:
    • 2 hours of pushing = nulliparous
    • 1 hour of pushing = parous
158
Q

What is secondary dysfunctional uterine activity?

A
  • A common cause of second stage delay
  • This may be exacerbated by epidural analgesia
  • After achieving full dilatation, the uterine contractions may become weak
  • This is sometimes associated with maternal dehydration and ketosis
  • If NO mechanical problem is anticipated and the woman is primiparous, treatment is with rehydration and IV oxytocin
  • If the woman is multiparous, a full clinical assessment should be performed by a skilled obstetrician prior to considering oxytocin
159
Q

How does android pelvis cause delay?

A
  • Android pelvis (narrow pelvis) may cause delay in the second stage because it prevent internal rotation of the foetal head
    • This may arrest descent of the foetal head at the level of the ischial spines in the transverse position (deep transverse arrest)
160
Q

What can cause second stage delay?

A
  • Android pelvis
  • Resistant perineum
  • Persistent OP position can also cause delay and would require long rotation to OA or be delivered in OP position (face to pubes)
161
Q

What do the NICE guidelines say by the time second stage delay is diagnosed?

A
  • Oxytocin should not be started

- So, inefficient uterine activity should be corrected at the beginning of the second stage

162
Q

What can be considered for prolonged second stage?

A
  • Instrumental vaginal delivery
  • Only if safety criteria are met
  • This can be done in the labour room or in the theatre (allowing C-section to be performed easily if needed)
163
Q

What may be indicated if a resistant perineum results in significant delay?

A
  • Episiotomy
164
Q

Summarise the management options of delay in second stage

A
  • Continued pushing with encouragement
  • Regular reviews of progress and foetal wellbeing
  • Oxytocin to augment contractions
  • Episiotomy for a resistant perineum
  • Instrumental vaginal birth
  • C-section
165
Q

What may foetal compromise present as?

A
  • Fresh meconium staining of the amniotic fluid
  • Abnormal CTG
  • NOTE: neither confirm foetal hypoxia or acidosis as meconium can be passed for physiological reasons (e.g. maturity) and abnormal CTG carries a high false-positive rate
166
Q

What are the risk factors for foetal compromise in labour?

A
  • Placental insufficiency (FGR and pre-eclampsia)
  • Prematurity
  • Postmaturity
  • Multiple pregnancy
  • Prolonged labour
  • Augmentation with oxytocin/hyperstimulation
  • Precipitate labour
  • Intrapartum abruption
  • Cord prolapse
  • Uterine rupture/dehiscence
  • Maternal diabetes
  • Cholestasis of pregnancy
  • Maternal pyrexia/chorioamnionitis
  • Oligohydramnios
167
Q

When is meconium staining significant?

A

If it is
- Thick or tenacious (clingy)

  • Dark green, bright green or black
168
Q

What does thin and light meconium signify?

A
  • Foetal gut maturity
169
Q

If any meconium is seen in liquor, what should be considered?

A
  • Do EFM and CTG

- This is MANDATORY if the meconium is THICK and DARK

170
Q

When should a CTG be started with regards to foetal compromise?

A
  • If there is
    • foetal tachycardia, bradycardia or FHR decelerations
  • CTG can be classified as normal, suspicious or pathological
171
Q

If the CTG is classified as suspicious or pathological in possible foetal compromise, what should be done?

A

If the CTG is suspicious

  • Repositioning the mother
  • IV fluids
  • Reducing or stopping oxytocin
  • Correction of epidural-associated hypotension
  • Continue CTG observation

If the CTG is pathological, consider above factors but also carry out an immediate vaginal examination to exclude malpresentation and cord prolapse and to assess the progress of labour

  • If the cervix is fully dilated, instrumental delivery may be possible
  • If the cervix is NOT fully dilated, foetal blood sampling may be considered
    • If the result is NORMAL, labour can continue but repeat samples may be needed every 30-60 mins if CTG abnormalities persist
  • An ABNORMAL result requires immediate C-section if the cervix is not fully dilated

Fresh, thick meconium with a reassuring CTG is still a cause of concern
- `Threshold for intervention should be lower and a paediatrician should be present at delivery

172
Q

Describe resuscitating the foetus in labour

A
  • Maternal dehydration and ketosis
    • Give IV fluids
  • Maternal hypotension secondary to an epidural
    • Reversed by fluid bolus (vasoconstrictor such as ephedrine may occasionally be used)
  • Uterine hyperstimulation from excess oxytocin
    • Turn off infusion temporarily and use tocolytic drugs (e.g. terbutaline)
  • Venocaval compression and reduced uterine blood flow
    • Turn woman to left lateral position
173
Q

How is foetal blood sampling done?

A
  • Woman is asked to lie in the left lateral position
  • Amnioscope is inserted into the vagina and the end is applied to the foetal head
  • The scalp is cleaned and a small cut is made and blood is collected into microtube (about 0.25 mL of blood is required)
  • Normal pH = 7.25
    • pH < 7.20 is considered foetal compromise
    • pH 7.20-7.25 is borderline
174
Q

What are non-pharmacological methods of pain relief in labour?

A
  • One-to-one care from a midwife can reduce the need for analgesia
  • Relaxation and breathing exercises
  • NOTE: prolonged hyperventilation can cause alkalosis and dizziness
  • Relaxation in warm water during first stage
  • Transcutaneous electrical nerve stimulation (TENS)
    • Blocks pain fibres in the posterior ganglia of the spinal cord
    • May be used in the latent phase
175
Q

What are pharmacological methods of pain relief in labour?

A
  • Opiates (e.g. pethidine and diamorphine) are used in most obstetric units
  • They should be available at all pregnancies but beware of side-effects
  • Opiates tend to be given as IM injections
  • Alternative route is a subcutaneous or IV infusion by a patient-controlled analgesic device (PCA)
  • If a very short-acting opiate is used, the opiate dose can be timed with the contractions
176
Q

What are the side effects of opioid analgesia?

A
  • Nausea and vomiting (should always be given with an antiemetic)
  • Maternal drowsiness and sedation
  • Delayed gastric emptying (increasing the risks of general anaesthesia)
  • Short-term respiratory depression of the baby
  • Possible interference with breastfeeding
177
Q

What is inhalational analgesia?

A
  • Nitrous oxide in the form of Entonox (NO and oxygen) is available on most labour wards
  • It has a quick onset and short duration of effect
  • May cause light-headedness and nausea
  • NOT suitable for prolonged use from early labour because hyperventilation can lead to hypocapnia, dizziness and (rarely) foetal hypoxia
  • It is most suitable later in labour or whilst awaiting epidural analgesia
178
Q

What is epidural anaesthesia?

What must woman be warned about regarding it?

A
  • MOST RELIABLE means of providing effective analgesia in labour
  • The decision to opt for epidural analgesia lies with the woman (unless there are contraindications)
  • Warn the woman that:
    • She may lose sensation and movement in her legs temporarily
    • IV access and more intensive maternal and foetal monitoring will be necessary (e.g. with continuous EFM)
  • Epidural analgesia does NOT increase C-section rates
  • It does, however, cause a longer second stage and increases risk of instrumental delivery
  • Epidural is not ideal for women in the first stage because it limits mobility
179
Q

What are the indications for epidural anaesthesia?

A
  • Prolonged labour/oxytocin augmentation
  • Maternal hypertensive disorders
  • Multiple pregnancy
  • Selected maternal medical conditions
  • A high risk of operative intervention
180
Q

What are the contraindications for epidural anaesthesia?

A
  • Coagulation disorders (e.g. low platelet count)
  • Local or systemic sepsis
  • Hypovolaemia
  • Logistical: insufficient numbers of trained staff (anaesthetic and midwifery)
181
Q

What are the complications of epidural anaesthesia?

A
  • Accidental dural puncture
  • Leakage of CSF if the subarachnoid space is reached causing a spinal headache
    • If the headache is severe or persistent, a blood patch may be necessary - involves injecting a small volume of the woman’s blood into the epidural space at the level of the accidental dural puncture
  • Bladder dysfunction (loss of awareness of needing to micturate)
  • Overdistension of the bladder can cause permanent damage and voiding problems
    • To avoid this, catheterisation should be carried out during labour if the woman does not void significant volumes of urine spontaneously
  • Hypotension
    • Treated with IV fluids and vasopressors
  • Accidental total spinal anaesthesia (injection of the analgesic into the subarachnoid space)
    • Severe hypotension
    • Respiratory failure
    • Unconsciousness
    • Death
  • Spinal haematoma
  • Drug toxicity
  • Short-term respiratory depression of the baby (because epidural solutions contain opioids)
182
Q

Describe the technique for epidural anaesthesia

A
  • Woman’s back is cleansed and local anaesthetic is used to infiltrate the skin
  • The woman may be in the left lateral position or sitting upright but leaning over
  • Aseptic technique is used
  • The epidural catheter is normally inserted at L2-L3, L3-L4 or L4-L5 interspace and should lie in the epidural space
  • The catheter is aspirated to check for position and, if no blood or CSF is obtained, a test dose is given to confirm the catheter position
  • The test dose is a small volume of dilute local anaesthetic that should not have any clinical effect if applied in the epidural space
  • If there is no change in sensation in the lower limbs, then the catheter is in the correct position
  • If there is sensory block, leg weakness and peripheral vasodilation, the catheter has been inserted into the subarachnoid space
  • If no signs have been observed within 5 mins of the test dose, the loading dose can be administered
  • The epidural solution is usually a mixture of low-concentration local anaesthetic (e.g. bupivicane) and an opioid (e.g. fentanyl)
  • After the loading dose, the mother should be kept in the right or left lateral position and her blood pressure should be measured every 5 mins for 15 mins
  • A fall in blood pressure may be caused by blocking the sympathetic tone to peripheral blood vessels
    • This can cause foetal bradycardia
    • Treat with IV fluids and vasoconstrictors (e.g. ephedrine) if necessary
  • The woman should NOT lie supine because it can cause aorto-caval compression (compression of the aorta and inferior vena cava but the gravid uterus)
  • Hourly assessment of the level of sensory block using a cold spray is critical in the detection of a block that is creeping too high and risking respiratory compromise
  • Regional anaesthesia can be maintained through labour with either intermittent boluses or continuous infusions
183
Q

Describe spinal anaesthesia

A
  • A spinal block is more effective than an epidural and has a faster onset
  • The needle is inserted through the dura into the subarachnoid space
  • They may be used for:
    • C-section
    • Trial of instrumental deliveries
    • Manual removal of retained placenta
    • Repair of difficult perineal and vaginal tears
    • Combined spinal-epidural (CSE) analgesia is a viable option for pain relief in labour
184
Q

What are pre-existing uterine scars from?

A
  • Previous C-section
185
Q

Why are 99% of C-sections performed in the lower segment of uterus?

A
  • Because risk of uterine rupture is lower
186
Q

What are the chances of uterine rupture?

A
  • Risk of uterine rupture or dehiscence (scar separation) occurs in 1 in 200 women who labour spontaneously with pre-existing lower segment uterine scar
187
Q

What are signs of uterine rupture?

A
  • Severe lower abdominal pain
  • Vaginal bleeding
  • Haematuria
  • Cessation of contractions
  • Maternal tachycardia
  • Foetal compromise (often bradycardia)
188
Q

What are the risks of uterine rupture to the mother and foetus?

A
  • Mother: shock, need for blood transfusion, operative repair, hysterectomy
  • Foetus: hypoxia, permanent neurological injury and perinatal death
189
Q

When is uterine rupture more likely to occur?

A
  • Late in the first stage of labour with induced or accelerated labour
190
Q

What percentage of women who attempt vaginal birth after caesarean will be successful?

A
  • 70-80% of women who attempt vaginal birth after caesarean (VBAC) will be successful and the remainder will require C-section
191
Q

How should a woman with a history of C-section and is admitted to labour be managed?

A
  • Close surveillance to identify early signs of uterine rupture
  • Continuous CTG monitoring is recommended with a low threshold for delivery by C-section
192
Q

What are relative contraindications for VBAC?

A
  • Two or more previous C-section scars
  • Need for induction of labour
  • Cephalopelvic disproportion in previous labour
  • Previous classical C-section is an ABSOLUTE contraindication
193
Q

What are the complications of breech presentation?

A
  • Increased risk of cord prolapse (particularly with footling breech)
  • Increased risk of CTG abnormalities as cord compression is common
  • Mechanical difficulties with delivery of the shoulders leading to damage to the visceral organs/brachial plexus
  • Delay in the delivery of the head leading to prolonged compression of the umbilical cord and asphyxia
  • Uncontrolled rapid delivery of the head may occur with a small foetus and predisposes to tentorial tears and intracranial bleeding
  • A small or preterm foetus may deliver through an incompletely dilated cervix resulting in head entrapment
194
Q

Describe face presentation and C-section

A
  • Occurs in 1 in 500 labours due to complete extension of the foetal head
  • The presenting diameter is the submento-bregmatic which is about 9.5 cm in diameter and has roughly the same dimensions as the suboccipito-bregmatic presentation
  • Engagement of the foetal head is late and progress in labour is usually slow
  • Diagnosed by palpating the nose, mouth and eyes on vaginal examination
  • If there is good progression in labour and the chin remains mento-anterior, vaginal delivery is possible with the head being delivered in flexion
  • If the chin is posterior (mento-posterior), delivery is IMPOSSIBLE
    • C-section should be performed
    • Oxytocin should NOT be used
195
Q

Describe brow presentation and C-section

A
  • Arises when there is less extreme extension of the foetal neck than with face presentation
  • It is a midway position between vertex and face presentation
  • It is the LEAST COMMON malpresentation
  • The presenting diameter is mento-vertical (13.5 cm) and is incompatible with vaginal delivery
  • It is diagnosed by palpating the anterior fontanelle, supraorbital ridges and nose on vaginal examination
  • If this position persists, C-section is necessary
196
Q

Describe shoulder presentation and C-section

A
  • Occurs in 1 in 300 pregnancies at term
  • Occurs as a result of transverse or oblique lie of the foetus
  • This can be caused by:
    • Placenta praevia
    • High parity
    • Pelvic tumour
    • Uterine anomaly
  • Delivery should be by C-section
  • Delay in diagnosing shoulder presentation could result in cord prolapse and uterine rupture
197
Q

Describe multiple pregnancy and C-section

A
  • 1 in 80 pregnancies are multi-foetal
  • Incidence is rising due to increasing maternal age and assisted contraception
  • High-order multiples tend to be delivered by C-section
  • For twin pregnancies, the second twin is at greater risk of intrapartum compromise than the presenting twin
  • C-section is not necessarily indicated in twin pregnancies, however they end up being performed in almost half of all twin pregnancies
  • In 70-80% of pregnancies the presenting twin is cephalic (the remainder are mainly breech)
  • Vaginal delivery is usually achievable if the presenting twin is in a cephalic vertex presentation
  • Planned C-section will usually be performed if the first twin is in breech, and almost certainly if it is transverse
198
Q

Describe induction of labour

A
  • This is the planned initiation of labour prior to its spontaneous onset
  • 20-25% or deliveries in the UK occur following IOL
  • Generally, IOL is performed when the risks to the foetus and/or the mother of the pregnancy continuing outweigh those of bringing the pregnancy to an end
  • MOST COMMON reason is prolonged pregnancy (used to be known as post-term or postdates)
  • Pregnancies extending beyond 42 weeks are associated with increased risk of stillbirth, foetal compromise in labour, meconium aspiration and mechanical problems at delivery
  • So, women are usually offered IOL between 41-42 weeks
  • Prelabour rupture of membranes (PROM) is another common indication for IOL
  • If there is a long delay between membrane rupture and delivery of the baby, there is a greater risk of ascending infection (chorioamnionitis)
  • If beyond 37 weeks, evidence supports use of IOL around 24 hours following membrane rupture
  • If < 34 weeks, another additional indication is needed to justify IOL if the membranes rupture (e.g. foetal compromise, growth restriction)
  • If 34-37 weeks, in an otherwise straight forward pregnancy, the benefits of IOL need to be assessed on an individual basis
  • Pre-eclampsia and other maternal hypertensive disorders often indicate earlier delivery
    • If it occurs at term, it is normally managed with IOL
    • If very preterm (< 34 weeks) or if there is rapid foetal or maternal compromise, C-section may be a better option
199
Q

When can IOL happen?

A
  • Prolonged pregnancy (> 41 weeks)
  • PROM
  • Pre-eclampsia and other maternal hypertensive disorders
  • FGR
  • Diabetes mellitus
  • Deteriorating maternal illness
  • Unexplained antepartum haemorrhage
  • Twin pregnancy continuing beyond 38 weeks
  • Intrahepatic cholestasis of pregnancy
  • Maternal isoimmunisation against red cell antigens
  • Social reasons
200
Q

What are absolute contraindications to IOL?

A
  • Placenta praevia

- Severe foetal compromise

201
Q

What conditions favour C-section?

A
  • Deteriorating maternal condition
  • Major antepartum haemorrhage
  • Pre-eclampsia
  • Maternal cardiac disease
  • Breech (relative contraindication)
  • Preterm
    • Not an absolute contraindication but induction < 34 weeks is associated with higher risk of failure and the need for C-section
202
Q

What is the Bishops score?

A
  • If labour is induced before cervical changes take place, the labour will take longer
  • The Bishop score is used to quantify how far the process of cervical changes has progressed before IOL
  • High scores (favourable cervix) are associated with an easier, shorter induction process
  • Low scores (unfavourable cervix) is more likely to take longer and more likely to fail and result in C-section
203
Q

What is the method for inducing labour?

A
  • Prostaglandin E2 (PGE2) is the most commonly used formulation
  • It is inserted vaginally into the posterior fornix as a tablet or gel
  • Two doses are often required, given at least 6 hours apart
  • A controlled-released pessary is also available and this is left in place for up to 24 hours
  • Labour may ensure following administration of prostaglandins but ARM and oxytocin are often also necessary (particularly in primiparous women)
  • Oxytocin is given IV as a dilute solution (as it has a short half-life)
    • NOTE: the response to oxytocin is highly variable so strict protocol must be followed
    • The starting infusion rate is low and defined increments will follow every 30 mins until 3-5 contractions are achieved every 10 mins
  • Mifepristone (antiprogesterone) and misoprostol (another prostaglandin) can be used to induce labour
  • Membrane sweeping describes insertion of a gloved finger through the cervix and its rotation around the inner rim of the cervix
    • This strips off the chorionic membrane from the underlying decidua and releases natural prostaglandins
    • It can be uncomfortable and is only possible if the cervix is beginning to dilate and efface
    • It reduces the need for induction
    • Usually only performed at term
    • Placenta praevia must be excluded before it is performed
204
Q

What are complications of inducing labour?

A
  • More pain experienced with induced labour
  • Epidural analgesia is more commonly used
  • Rates or instrumental delivery are higher when epidural analgesia is used
  • NO higher rate of C-section
  • Increased risk of PPH secondary to uterine atony
  • Uterine hyperstimulation may cause foetal compromise as a side-effect of using prostaglandins and oxytocin
  • Contraction frequency of >5 per 10 mins should be treated by stopping oxytocin and, if necessary, administering a tocolytic drug (usually SC injection of terbutaline (beta-2 agonist))
  • Uterine hyperstimulation may cause foetal bradycardia and the need for an emergency C-section if FHR fails to resolve promptly
  • If ARM is performed when the foetal head is high, cord prolapse can occur which may need a C-section
  • The risk of uterine rupture in women with a previous C-section scar who are induced is higher than if they went through spontaneous labour
  • IOL failure is said to have occurred if ARM is still impossible after the maximum number of doses of prostaglandin have been given or if the cervix remains uneffaced and < 3 cm dilated after an ARM has been performed and oxytocin has been running for 6-8 hours with regular contractions
205
Q

What are the options if IOL fails?

A
  • Rest then attempt induction again

- C-section

206
Q

What percentage of women who deliver vaginally will have some degree of perineal trauma?

A
  • 85%
207
Q

What percentage of women will need suturing after vaginal birth?

A
  • 60-70%
208
Q

What are risk factors for perineal tears?

A
  • Prolonged labour
  • Big babies
  • Instrumental delivery
  • During active second stage
209
Q

What does external sphincter incompetence lead to?

A
  • Faecal urgency
210
Q

What does internal sphincter incompetence lead to?

A
  • Faecal incontinence
211
Q

What are obstetric anal sphincter injuries? (OASIs)

A
  • Third and fourth degree tears
212
Q

Describe the surgical technique of perineal tear repair

A
  • Adequate analgesia should be provided by topping up an epidural or by infiltration with local anaesthetic
  • A pad or tampon to prevent blood loss from the uterus obscuring the view
  • Vaginal mucosa is repaired first using rapidly absorbable suture material
  • A continuous stitch should be used to close the vaginal mucosa
  • Interrupted sutures are placed to close the muscle layer
  • Closure of perineal skin is done with either interrupted sutures or a continuous subcuticular stitch
  • A gentle vaginal examination should be performed to check for any missed tears and to ensure that good opposition is achieved
  • A rectal examination should be performed to confirm that the sphincter feels intact and to ensure that no sutures have been inadvertently placed through the rectal mucosa
213
Q

Describe OASI repair

A
  • Regional or general anaesthetic (to achieve relaxation of the sphincter)
  • Repair rectal mucosa
  • Repair torn external sphincter
  • Some surgeons do an end-to-end repair whereas others prefer an overlap method
  • The rest of the perineal repair is as for second-degree trauma
214
Q

Describe OASI aftercare

A
  • Lactulose and a bulking agent (e.g. Fybogel) are recommended for 5-10 days
  • Remain in hospital until first bowel motion happens
  • An oral broad-spectrum antibiotic should be prescribed for 5-7 days to reduce the risk of infection
  • Regular oral analgesia
  • At 6-12 weeks, a full evaluation of the degree of symptoms should take place
  • Asymptomatic women should be told that the risk in future pregnancy is 6-8% and vaginal delivery is safely achievable
215
Q

What is an episiotomy?

A
  • An episiotomy is a surgical incision of the perineum performed during the second stage of labour to enlarge the vulval outlet and assist vaginal birth
  • Episiotomies are used in about 10% of spontaneous vaginal deliveries in the UK (rates vary in other countries)
216
Q

Describe the surgical technique of an episiotomy

A
  • Episiotomy is performed in the second stage when the perineum is being stretched
  • If there is NOT a good epidural, the perineum should be infiltrated with local anaesthetic
  • If an effective epidural is in place, it should be topped up for delivery with the patient upright to get best coverage of the perineal area
  • The incision can be midline or at an angle from the posterior end of the vulva
  • A mediolateral episiotomy at a 60 degree angle to midline is usually recommended
  • The midline episiotomy results in less bleeding, quicker healing and less pain but there is an increased risk of extension to involve the anal sphincter (OASI)
  • The episiotomy should be repaired in the same way as a second-degree tear unless involvement of the anal sphincter requires OASI repair
217
Q

What are the complications of an episiotomy?

A
  • Pain
  • Infection
  • Haemorrhage
  • Dyspareunia
  • Incontinence of urine
  • Incontinence of flatus or faeces
  • Risks are highest with OASI
218
Q

What is an operative vaginal delivery?

A
  • Refers to vaginal birth with the use of any type of forceps or vacuum extractor (ventouse)
  • The goal is to speed up delivery with a minimum of maternal and neonatal morbidity
  • 10-15% of deliveries in the UK are assisted with instruments
  • The rate is higher in nulliparous women (around 30%)
219
Q

What is the most common foetal factor for an OVD?

A
  • Suspected foetal compromise is the MOST COMMON foetal factor
  • It is based on an pathological CTG
220
Q

What is the most common maternal factor for an OVD?

A
  • Prolonged active second stage of labour
221
Q

Describe some contraindications of OVD?

A
  • If the safety criteria is NOT met, OVD is contraindicated
  • NOT to be used if < 34 weeks because of high risk of cephalhaematoma and intracranial haemorrhage
  • NOT used for face or breech presentation
  • Minimal risk of haemorrhage if a vacuum extractor is used following foetal blood sampling or foetal scalp electrode
  • Forceps or vacuum extractor delivery is contraindicated before full dilatation of the cervix
222
Q

What are the pros of using a ventouse for OVD?

A
  • Lower risk of pelvic floor trauma with ventouse
  • Lower risk of anal sphincter injury with ventouse
  • Higher risk of lacerations and facial palsy with forceps
  • Less likely to need maternal regional or general anaesthesia
  • Less likely to cause significant perineal pain at 24 hours
  • No difference in pelvic floor symptoms 5 years after instrumental delivery between forceps and ventouse
  • Both are associated with low 5 minute Apgar scores
223
Q

What are the pros of using forceps for OVD?

A
  • Lower failure rate than Ventouse for vaginal delivery
  • Lower risk of cephalohaematoma and cerebral haemorrhage than ventouse
  • Lower risk of retinal haemorrhage than ventouse
  • No difference in pelvic floor symptoms 5 years after instrumental delivery between forceps and ventouse
  • Both are associated with low 5 minute Apgar scores
224
Q

How does C-section compare to instrumental delivery?

A
  • Women who deliver by C-section more likely to have a major haemorrhage and to need a hospital stay for more than 5 days
  • Babies delivered by C-section were less likely to have trauma than babies delivered by forceps but were more likely to require intensive care
  • Women who had a C-section are much more likely to require a C-section in future pregnancies
225
Q

Describe the evaluation of OVD

A
  • Thorough vaginal and abdominal examination should take place to confirm the foetal lie, presentation, engagement, station, position, attitude and degree of caput or moulding
  • This will confirm whether the basic safety criteria for OVD have been met
  • Anthropoid (narrow), android (male/funnel-shaped) or platypelloid (elliptical) pelvises make instrumental delivereis MORE difficult
  • A trial push is helpful to determine whether descent is possible and allows attempted correction of malposition
226
Q

Describe analgesia use in OVD

A
  • More analgesia is required for forceps compared to ventouse
  • Forceps - regional anaesthesia is preferred
  • Rigid cup ventouse - pudendal block with perineal infiltration
  • Soft cup ventouse - perineal infiltration with local anaesthetic
227
Q

What is the positioning for OVD?

A
  • Classically performed with patients in the lithotomy position
  • The bladder should be emptied
228
Q

What is the contingency plans for OVD?

A
  • A failed vacuum delivery may be completed with low-pelvic forceps
  • But a failed or abandoned forceps delivery will almost always result in C-section
  • The risk of shoulder dystocia and postpartum haemorrhage should always be considered
229
Q

Describe the technique of Ventouse delivery

A
  • Soft cups are more likely to fail than rigid cups
  • However, soft cups cause less scalp injury
  • Soft cups are suitable for uncomplicated deliveries with an OA position
  • Metal cups are better for OP, transverse and difficult OA position
  • For successful use of the ventouse, determination of the flexion point is vital
  • This is at the vertex, which is on the saggital suture 3 cm anterior to the posterior fontanelle and 6 cm posterior to the anterior fontanelle
  • The centre of the cup should be positioned directly over this
  • Operating suction pressure is 0.6-0.8 kg/cm2
  • Check that no maternal tissue is caught under the edge
  • Traction must along in the plane of least resistance along the axis of the pelvis (usually at exactly 90 degrees to the cup)
  • Operator should keep a thumb and forefinger on the cup and foetal scalp to ensure traction direction is correct and to feel for slippage
  • Safe and gentle traction is applied and coordinated with uterine contractions and maternal expulsive efforts
  • The descent phase brings the head onto the perineum
  • The crowning phase occurs shortly afterwards
  • Rotation is achieved through the natural passage of the head through the pelvis
  • The operator should not allow any more than 2 episodes of the suction popping off during delivery
  • The maximum time from application to delivery should be < 15 mins
230
Q

What are the different types of forceps?

A
  • Blades may be fenestrated, pseudofenestrated or solid
  • Length of shanks and design of the lock vary between models
  • Non-rotational forceps are used when the head is OA with no more than 45 degrees deviation to the left or right
  • Examples of non-rotational forceps
    • Neville Barnes
    • Simpson
  • If the head is positioned > 45 degrees from the vertical, rotation must be accomplished before tractions
  • Examples of rotational forceps
    • Kielland
231
Q

Describe the technique of forceps delivery

A
  • The left blade is usually inserted before the right with the operators hand protecting the vaginal wall from the blades
  • The forceps blades will end up lying parallel to the axis of the foetal head
  • The operator will then lock the blades
  • Traction should be applied intermittently coordinated with uterine contractions and maternal expulsive efforts
  • The axis of traction changes during delivery and is guided along the J-shaped curve of the pelvis
  • As the head crowns, the blades are directed to the vertical and the head is delivered
  • Most forceps deliveries will be completed with no more than three pulls
  • In practice, most obstetricians cut an episiotomy routinely for forceps delivery
  • This is more likely in nulliparous women
232
Q

What are complications of OVD?

A
  • Maternal deaths have been reported with vacuum deliveries as a result of cervical tearing in women delivered before full dilatation
  • Faecal incontinence
  • Postpartum haemorrhage
  • Underestimation of blood loss
  • Cephalhaematoma (ventouse)
  • Risk of trauma to the baby correlates with duration of operative vaginal delivery and difficulty of the delivery
233
Q

What is a C-section?

A
  • A C-section is a surgical procedure where incisions are made through a woman’s abdomen (laparotomy) and uterus (hysterotomy) to deliver one or more babies
  • 25-30% of all babies in the UK are delivered by C-section
234
Q

What are four major indications for a C-section?

A
  • Previous C-section
  • Malpresentation (mainly breech)
  • Failure to progress in labour
  • Suspected foetal compromise
235
Q

What are other indications for a C-section?

A
  • Multiple pregnancy
  • Placental abruption
  • Placenta praevia
  • Foetal disease
  • Maternal disease
236
Q

What is tocophobia?

A
  • Irrational fear of childbirth
  • It is important to distinguish mothers who request C-section because they want to avoid labour from those that want to avoid it because of a previous traumatic birth experience
237
Q

What is the procedure for a C-section?

A
  • Informed consent
  • Preparation
  • Abdominal incision
  • Uterine incision
  • Closure
238
Q

Describe informed consent in a C-section

A
  • Must be obtained before surgery

- Potential indications should be discussed in the antenatal period

239
Q

Describe the preparation for a C-section

A
  • Most scheduled C-sections are performed under spinal anaesthesia with the mother awake
  • If an epidural has been placed during labour there is usually enough time to top-up the anaesthesia before emergency C-section
  • General anaesthesia is occasionally required
  • Bladder should be emptied before the procedure and a urinary catheter is usually kept in situ
  • A left lateral tilt minimises aorto-caval compression and reduces the incidence of hypotension and reduced placental perfusion
  • The anaesthetic block is confirmed and the woman’s abdomen is cleaned and draped
  • Prophylactic antibiotics should be administered IV before the surgical incision
240
Q

Describe the abdominal incision for a C-section

A
  • The skin and subcutaneous tissues are incised using either a transverse curvilinear incision 2 finger breadths above the symphysis pubis extending from and to points lateral of the lateral margins of the abdominal rectus muscles (Pfannenstiel incision)
  • An alternative incision is a transverse suprapubic incision with no curve
  • Subcutaneous tissues are separated by blunt dissection and the rectus sheath is incised transversely along the middle 2 cm
  • The incision is extended with scissors before the fascial sheath is separated form the underlying muscle by blunt dissection
  • The recti are separated, the peritoneum incised and the abdominal cavity entered
  • A vertical incision is used in cases of extreme maternal obesity, suspicion of other intra-abdominal pathology or where access to the uterine fundus is required
    • The vertical incision allows easy access to the pelvic and intra-abdominal organs but there is an increased incidence of wound dehiscence (splitting open)
241
Q

Describe the uterine incision for a C-section

A
  • A lower uterine segment transverse incision is used in >95% of C-section deliveries due to ease of repair, reduced blood loss and low incidence of dehiscence or rupture in subsequent pregnancies
  • The classical C-section incision incorporates the upper uterine segment with a vertical incision - this has few absolute indications but include:
    • Lower uterine segment obscured by fibroids
    • Lower segment covered with dense adhesions
    • Placenta praevia
    • Transverse lie with the back down
    • Foetal abnormality (e.g. conjoined twins)
    • C-section in the presence of carcinoma of the cervix
  • Once the uterus is incised, the membranes are ruptured if still intact and the operator’s hand is placed below the presenting part
  • If cephalic, the head is flexed and delivered by elevation through the uterine incision either manually or with forceps
  • Fundal pressure is applied by the assistant to aid delivery
  • Once the foetus is delivered, an oxytocic agent (5 IU syntocinon IV) is administered to aid uterine contraction and placental separation
  • The placenta is delivered by controlled cord traction
242
Q

Describe the closure of a C-section

A
  • Closure should be performed in either single or double layers with continuous or interrupted sutures
  • A running stitch is usually employed
  • A second layer is commonly used to improve haemostasis and the integrity of the scar
  • Peritoneal closure is NOT routine and depends on the operator’s preference
  • Abdominal closure is performed in the anatomical planes
  • The skin can be closed by absorbable or non-absorbable suture material or with clips
243
Q

What are general complications of a C-section?

A
  • Haemorrhage
  • Infections of the wound
  • Urinary tract infection
  • Infection of the endometrium
  • Transient tachypnoea of the newborn
  • Fatality from C-section is 5 x that of vaginal delivery (but this is mainly because of emergency C-section)
244
Q

What are intraoperative complications of a C-section?

A
  • Haemorrhage
    • May be due to damage to uterine vessels
    • May occur as a consequence of uterine atony or placenta praevia
    • In patients with an anticipated high risk of haemorrhage (e.g. placenta praevia), blood should be routinely cross-matched
  • Caesarean Hysterectomy
    • The most common indication is uncontrollable maternal haemorrhage
    • Life-threatening haemorrhage occurs in about 1 in 1000 deliveries
    • The most important risk factor for emergency postpartum hysterectomy is a previous C-section
      • This is especially bad if the placenta overlies the old scar –> increased risk of placenta accreta
    • Other indications for hysterectomy:
      • Atony
      • Uterine rupture
      • Extension of a transverse uterine incision
      • Fibroids preventing uterine closure and haemostasis
  • Placenta praevia
    • Risk of placenta praevia increases with each previous C-section
    • So, future reproductive intensions are important when considering C-section
  • Organ damage
    • Bowel damage may occur during a repeat procedure if adhesions are present from a previous surgery
  • Bladder injury
  • Damage to ureters
245
Q

What are postoperative complications of a C-section?

A
  • Infection
    • C-section has a 5-20 fold increased risk of infectious complications compared to vaginal delivery
    • Fever
    • Wound infection
    • Endometritis
    • Bacteraemia
    • UTI
    • Other causes of post-operative fever: haematoma, atelectasis, DVT
    • The duration of labour and the presence of ruptured membranes seem to be the biggest risk factors
    • Infections are often polymicrobial and include E. coli, aerobic Gram-negative rods and group B streptococcus
    • Prophylactic antibiotics reduce the risk of infection
  • Venous Thromboembolism
    • C-section is a major risk factor
    • Incidence can be reduced by:
      • Adequate hydration
      • Early mobilisation
      • Prophylactic heparin
  • Psychological
    • Delayed contact with the baby can contribute to psychological distress
246
Q

Describe subsequent delivery following C-section

A
  • Up to 70% of women with a previous C-section who labour achieve vaginal delivery
  • Two options for women with previous C-section:
    • Elective repeat caesarean section (ERCS)
    • Attempted vaginal birth after caesarean section (VBAC)
  • If the first operation was carried out for a non-recurrent indication, and providing the obstetric situation close to term in the subsequent pregnancy is favourable, then it is appropriate to offer a trial of labour after caesarean (TOLAC) to any woman with a previous uncomplicated lower segment C-section
247
Q

What are the benefits of ERCS to the mother?

A
  • Avoid pelvic floor trauma (urinary and faecal problems)
  • Avoid the need to undergo emergency C-section
  • Avoid scar dehiscence or rupture
248
Q

What are the risks of ERCS?

A
  • Increased bleeding
  • Febrile morbidity
  • Prolonged recovery
  • Thromboembolism
  • Long-term bladder dysfunction
  • Increased risk of placenta praevia in subsequent pregnancies