nursing care of normal newborn (unit 2) Flashcards
first period of reactivity
- birth-30 min
- inc. HR
- irregular resp.
- alert, inc. motor activity
- dec. body temp
- meconium/saliva possibly
sleep period (relative inactivity)
- dec. in motor activity 30-100 min after birth
- VS recovery
- temp may dip, so need warmer
second period of reactivity
- 4-8 hours after birth
- periods of tachypnea/apnea
- periods of tachycardia
- inc. muscle tone
- inc. mucous (bulb)
- meconium
mechanical stimuli initiating breathing
- compression/recoil of chest during birth
- tactile sim during/after birth
- suctioning
sensory stimuli initiating breathing
•temp (chill)
•noise
•light
*obligate nose breathehrs
signs of respiratory distress
- nasal flaring
- retractions
- grunt w/ expiration
- accessory muscle use
- seesaw resp.
- rate < 30 or > 60
- low surfactant levels
- apnea r/t analgesics or rapid cool/warm
- diaphragmatic hernia
surfactant
- lines alveoli, inc. SA
- suff. levels 34-36 wk
- deficiency leads to RDS
- measured by L/S ratio
L/S ratio
- determines fetal pulmonary maturity
- 2 or > means more mature
- 1.5 of < means inc. r/o RDS
fetal structures that close postnatally
•foramen ovale
•ductus arteriosus
•ductus venosus
*allows unoxygenated blood to circulate thru lungs
foramen ovale
- b/t RA & LA
* allows blood to bypass pulmonary circulation
ductus arteriosus
- b/t aorta & PA
* allows majority of RV output to bypass lungs
ductus venosus
- b/t umbilical vein & IVC
* allows ½ blood from umbilical vein to bypass liver/GI tract
clamping of the cord
•umbilical arteries/vein and ductus venous are functionally closed and converted to ligaments
newborn blood volume
•300 mL
newborn H&H
•14-24 g/dL
•44%-64%
*shorter lifespan of RBC
newborn leukocytes
•WBC of 18,000
•inc. after birth, then declines rapidly
*susceptible to infection
newborn clotting factors
•levels decrease b/c unable to synthesize vitamin K
nonshivering thermogenesis
- metabolism of brown fat (inter scapular, axillae, vertebrae, kidney)
- critical newborn heat production
- rapid depletion w/ cold/stress
- PT have less brown fat
heat loss via convection
- heat from body to cooler air
* keep wrapped in blanket and warm room
heat loss via radiation
- heat from body to cooler surface
* NOT direct contact
heat loss via evaporation
- insensible water loss
- can occur via respirations
- dry baby well and delay first bath if thermoregulation issues
- only expose one body part at a time
heat loss via conduction
- heat from body to cooler surface
- direct contact
- warmer
newborn hypothermia
- common b/c have inc. heat loss d/t thin skin, vessels close to surface, little fat, and high SA to body mass ratio
- s/sx: < 36.5 or 97.7 temp; cyanosis; tacypnea
newborn heat conservation
- flexed positon
- constriction of peripheral vessels
- brown fat metab.
- crying, restless, movement
cold stress
- ineffective thermoregulation
* can lead to hypoxia, acidosis, and hypoglycemia
cold stress s/sx
- drop in temp
- RR inc.
- tachy then brady
- mottled skin; acrocyanosis
- dec. activity if RD
- inc. activity if not RD
hyperthermia
- rapid BMR increase
- increase in glucose/O2 requirements
- nonfunctioning sweat glands
- vasodilation w/ increased insensible water loss
- may lead to cerebral damage, dehydration, heat stroke, death
neutral thermal environment
•where infant can maintain stable body temp w/o increase in metabolic rate
neonate renal system
- urine @ 12 wks GA
- kidney fully developed @ 35 wks
- blood flow inc. after birth -> inc. UOP
expected infant UOP
- once in 1st 24 hr
- twice 2nd 24 hr
- 3 times 3rd 24 hr
newborn sucking coordination
- after 1500 g
* 32-34 wks GA
first stools of newborn
- meconium (24-48 hr)
- transitional (day 3)
- milk (day 4)
neonate hepatic system fxn
- iron storage
- carb metab
- bilirubin conjugation
- coagulation
iron storage
- proportional to body weight
- maternal Fe transfer from 3rd trimester stored for 4-6 months
- PT have smaller stores (2-3 months)
carbohydrate metabolism
- glucose transplacentally
- glucose dec. at birth
- neonate higher glucose needs than fetus
- glycogen stored last 4-8 wks GA
hypoglycemia in infant
• < 40 mg/dl (term) •jittery •RDS •apnea •lethargy •poor suck •seizures *if no tx, possible brain damage
infant r/o hypoglycemia
- preterm
- SGA
- LGA
- stress
- maternal diabetes
coagulation of bilirubin
•short living RBC brobken into unconjugated bilirubin (fat soluble)
•liver must convert unconjugated into conjugated for metab/excretion (water soluble)
*hepatic immaturity causes accum. of unconjugated
unbound bilirubin
- conjugated
- water soluble
- yellow pigment that may leave vascular system and go to skin, sclera, oral membranes (jaundice)
why do neonates have hyperbilirubinemia
- excess production of bilirubin
- liver immaturity
- shortened life span of RBC
- delayed feeding
- trauma
r/o hyperbilirubinemia
- prematurity
- Rh or ABO incompatibility
- cephalhematoma, bruising
- delayed/poor intake
- cold stress
- sepsis
- breast feeding
physiologic jaundice
- hyperbilirubinemia
- appears after 1st 24 hrs
- benign- not normal, but common
- s/sx 2nd-3rd day of life
- usually resolves by day 4
- bili < 12 (unless PT or BF)
pathologic jaundice
- hyperbilirubinemia
- appear w/in 1st 24 hrs
- r/t abnormalities that cause excessive RBC destruction
- bili levels higher and remain high
kernicterus
- bilirubin encephalopathy
- severe jaundice
- bili > 25 mg/dl
- causes severe neurological damage
- only ½ infants survive