Nurse 313R Med Surge Flashcards

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1
Q

Haital Hernia

A
  • protrusion of a portion of the stomach into the esophagus through an opening, or hiatus in the diapgragm
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2
Q

2 Types of Hiatal Hernia

A
  • Sliding

* Paraesophageal or rolling

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3
Q

Clinical Manifestations

A
  • heartburns
  • dysphagia
  • severe burning pain when bending over (sit upright)
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4
Q

Drugs for use with Hiatal Hernias

A

 H2 receptor blockers(dine)-Tagamet, Zantac, Pepcid
 PPI’s (zole)-Prevacid, Protonix, Nexium
 Cholinergics-used to increase LES pressure and increase gastric emptying-bethanechol
 Prokinetic-motility enhancing-Reglan-promotes gastric emptying

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5
Q

Collaborative Care / Nursing Therapeutics

A
  • reduce intraabdominal pressure
  • use of antacid and antisecretory agent
  • wt. loss management
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6
Q

Non-Conservative Therapies

A

 Surgery
 Reduction of the herniated stomach into abdomen
 Herniotomy-excision of the hernia sac
 Herniorraphy-closure of the hiatal defect
 Gastropexy-attachment of the stomach subdiaphragmatically to prevent reherniation

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7
Q

Conservative

A
  • Reduce intra-abdominal pressure
  • Eat small meals, wear loose clothing, avoid heaving lifting, stop alcohol and tobacco use, keep hob up to sleep (weight loss management)
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8
Q

Diagnostic Studies

A

 Barium swallow-shows protrusion of gastric mucosa through esophageal hiatus
 Endoscopic visualization of lower esophagus will show mucosal abnormalities or any inflammation
 Upper GI endoscopy
 Motility studies

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9
Q

Complications

A

 GERD, esophagitis, hemorrhage from erosion, stenosis of esophagus, ulcers in herniated part of stomach, strangulation of the hernia, regurgitation with tracheal aspiration

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10
Q

Gastritis

A

 Inflammation of the gastric mucosa- a result of the breakdown of the normal gastric mucosa barrier that protects stomach from the corrosive action of HCL acid and pepsin. HCL and pepsin diffuse back into the mucosa causing edema, capillary walls lose plasma into gastric lumen, and possible hemorrhage. (mucus membrane starts to be thin)
 Acute or chronic
 Diffuse or localized

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11
Q

Chronic gastritis causes

A
	Focus is on evaluating and then eliminating the specific cause
	Drugs 
	Alcohol
	H. Pylori
	Pernicious anemia
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12
Q

Acute Gastritis interventions

A

 Eliminating the cause is all that is needed to treat acute gastritis (stopping to much alcohol)
 If vomiting occurs, rest, NPO status, and IVF may be prescribed.
 In severe cases an NGT will be used to monitor for bleeding, for lavage of the precipitating agent from the stomach, and to keep the stomach empty
Procedure: lavage (drain out)(ng drains to keep stomach empty)

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13
Q

Risk Factors

A

 Drug related gastritis
NSAIDs-risk factors for NSAID related gastritis is being female, over age 60, history of ulcer disease, concomitant use of anticoagulants, use of other NSAIDs, use of corticosteroids, having a chronic debilitating disorder
 Diet –alcohol, spicy foods
 H.Pylori infection
 Autoimmune metaplastic atrophic gastritis an immune response against parietal cells

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14
Q

Clinical Manifestations

A
Symptoms for acute gastritis
	Anorexia
	Nausea and vomiting
	Epigastric tenderness
	Feeling of fullness
Symptoms for chronic gastritis
Above plus loss of parietal cells=loss of intrinsic factor=loss of absorption
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15
Q

Diagnostic Studies

A

 Diagnosis based on patient’s history of drug and alcohol use
 Endoscopic exams with biopsy needed for definitive diagnosis
 For H. pylori there are breath, serum, stool and urine tests
 Stool for occult blood
 Serum for anemia or lack of intrinsic factor
 Serum for antibodies to parietal cells, and IF

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16
Q

Peptic Ulcer Disease

A
  • condition characterized by erosion of the GI mucosa resulting from digestive action of HCl acid and pepsin.
17
Q

Type of Peptic Ulcer Disease

A
  • Acute ulcer

* Chronic ulcer

18
Q

Type of Ulcerations

A

• Gastric Ulceration

  • Duodenal Ulcers
  • Physiologic Stress Ulcer
19
Q

Clinical Manifestation

  • may have no pain
  • burning or cramp like
  • back pain
A

Complications

  • hemorrhage
  • perforation
  • gastric outlet obstruction
20
Q

Collaborative Care / Nursing Therapeutics

A
	adequate rest
	bland diet
	cessation of smoking
	aspirin and NSAIDS
	Drugs
                 - H2R Blockers
                 - Proton pump inhibitors
                 - ABT
                 - Antacid
                 - Anticholinergic drugs
                 - cytoprotective drug
	Nutritional Therapy
21
Q

Ulcerative Colitis

A
  • characterized by inflammation and ulceration of the colon and rectum
  • colon may become hyperemic and edematous may develop abscess then ulceration
22
Q

Clinical Manifestation

Collaborative Care / Nursing Therapeutics

Goals:

  • Drug therapy
  • Surgical Intervention
  • Nuritional Therapy
A

Clinical Manifestation

  • bloody diarrhea
  • abdominal pain
  • 4-5 stools BM/ day
  • Fever, malaise, anorexia
  • Wt. loss
  • Anemia, tachycardia
  • Dehydration
  • Goals:
  • rest the bowel, control inflammation, combat infections, alleviate stress, symptomatic relief

23
Q

Collaborative Care / Nursing Therapeutics

Goals:

  • Drug therapy
  • Surgical Intervention
  • Nuritional Therapy
A

Collaborative Care / Nursing Therapeutics

- rest the bowel, control inflammation, combat infections, alleviate stress, symptomatic relief

24
Q

Goals:

  • Drug therapy
  • Surgical Intervention
  • Nuritional Therapy
A

Drug therapy

   * antimicrobial
   * corticosteroid * sedative
   * antidiarrheal •	Nuritional Therapy
25
Q

Nuritional Therapy

Surgical Intervention

A
  • proctocolectomy with ileostomy

- ileostomy vs. colostomy