Nurse 313R Med Surge Flashcards
Haital Hernia
- protrusion of a portion of the stomach into the esophagus through an opening, or hiatus in the diapgragm
2 Types of Hiatal Hernia
- Sliding
* Paraesophageal or rolling
Clinical Manifestations
- heartburns
- dysphagia
- severe burning pain when bending over (sit upright)
Drugs for use with Hiatal Hernias
H2 receptor blockers(dine)-Tagamet, Zantac, Pepcid
PPI’s (zole)-Prevacid, Protonix, Nexium
Cholinergics-used to increase LES pressure and increase gastric emptying-bethanechol
Prokinetic-motility enhancing-Reglan-promotes gastric emptying
Collaborative Care / Nursing Therapeutics
- reduce intraabdominal pressure
- use of antacid and antisecretory agent
- wt. loss management
Non-Conservative Therapies
Surgery
Reduction of the herniated stomach into abdomen
Herniotomy-excision of the hernia sac
Herniorraphy-closure of the hiatal defect
Gastropexy-attachment of the stomach subdiaphragmatically to prevent reherniation
Conservative
- Reduce intra-abdominal pressure
- Eat small meals, wear loose clothing, avoid heaving lifting, stop alcohol and tobacco use, keep hob up to sleep (weight loss management)
Diagnostic Studies
Barium swallow-shows protrusion of gastric mucosa through esophageal hiatus
Endoscopic visualization of lower esophagus will show mucosal abnormalities or any inflammation
Upper GI endoscopy
Motility studies
Complications
GERD, esophagitis, hemorrhage from erosion, stenosis of esophagus, ulcers in herniated part of stomach, strangulation of the hernia, regurgitation with tracheal aspiration
Gastritis
Inflammation of the gastric mucosa- a result of the breakdown of the normal gastric mucosa barrier that protects stomach from the corrosive action of HCL acid and pepsin. HCL and pepsin diffuse back into the mucosa causing edema, capillary walls lose plasma into gastric lumen, and possible hemorrhage. (mucus membrane starts to be thin)
Acute or chronic
Diffuse or localized
Chronic gastritis causes
Focus is on evaluating and then eliminating the specific cause Drugs Alcohol H. Pylori Pernicious anemia
Acute Gastritis interventions
Eliminating the cause is all that is needed to treat acute gastritis (stopping to much alcohol)
If vomiting occurs, rest, NPO status, and IVF may be prescribed.
In severe cases an NGT will be used to monitor for bleeding, for lavage of the precipitating agent from the stomach, and to keep the stomach empty
Procedure: lavage (drain out)(ng drains to keep stomach empty)
Risk Factors
Drug related gastritis
NSAIDs-risk factors for NSAID related gastritis is being female, over age 60, history of ulcer disease, concomitant use of anticoagulants, use of other NSAIDs, use of corticosteroids, having a chronic debilitating disorder
Diet –alcohol, spicy foods
H.Pylori infection
Autoimmune metaplastic atrophic gastritis an immune response against parietal cells
Clinical Manifestations
Symptoms for acute gastritis Anorexia Nausea and vomiting Epigastric tenderness Feeling of fullness Symptoms for chronic gastritis Above plus loss of parietal cells=loss of intrinsic factor=loss of absorption
Diagnostic Studies
Diagnosis based on patient’s history of drug and alcohol use
Endoscopic exams with biopsy needed for definitive diagnosis
For H. pylori there are breath, serum, stool and urine tests
Stool for occult blood
Serum for anemia or lack of intrinsic factor
Serum for antibodies to parietal cells, and IF