NURS 310 2 Flashcards
bronchodilators
beta2-adrenergiuc agonists, anticholinergics, xanthine derivatives
SABA MOA
stimulate beta2 receptors to cause bronchodilation
SABA contradictions
hypertension, pregnancy
AE SABA
chest pain, palpitations, restlessness - caused by using too frequently (stimulates B1)
what receptors do non-selective bronchodilators stimulate (epinephrine)
beta1(cardiac) and beta 2(respiratory) and at high doses alpha
anticholinergic (ipratropium) MOA
block acetylcholine receptors and prevents bronchoconstriction (reduce secretions for COPD)
- prevention (long term)
Ipratropirum AE
hypotension, palpitations, urinary retention, drug toxicity if mixed with other anticholinergics
xanthine derivatives MOA
bronchodilator by increasing cAMP, dilate airways
AE with X.D.
Nausea, vommitng, anorexia
epinephrin AE
chest pain, hypertension
cation in: pregnancy, hypertension
epinephrin is high alert so:
pay attention to med concentration and administer bronchodilator first before any other
LTRA MOA
LTRA block leukotrienes from attaching and reduce inflammation and prevent SM contraction.
SE of LTRAs
liver dysfunction, nausea, headache, mood changes (depression)
order of drug administration
SABA or LABA, anticholinergic, corticosteroids
AE of corticosteroids
hoarseness, sore throat, thrush, irritability, mood changes