NRS 110 Comfort/ Pain Management Flashcards
Exam 3
Pain
Whatever the pt says it is.
Psychological effects of pain:
- hyperglycemia
- increased cardiac workload
- immune system dysfunction
- GI ileum
- urinary retention
- decreased lung volume and fatigue
Nociceptive Pain
Ability to feel painful stimuli.
Hurtful/ injurious.
Transduction
Transmission
Perception
Modulation
Neuropathic Pain
Abnormal functioning- diabetics
Pain caused by a lesion/ Disease of the peripheral/ central nerves. Cause is unknown.
Short but frequently chronic.
Described as burning/ electric/ tingling/ stabbing
Psychogenic Pain
Physical cause can’t be identified. Mental event
Referred Pain
Perceived from point distant from point of origin.
Cutaneous Pain
Involves skin and subs tissue- superficial.
ex: paper cut
Breakthrough Pain*
RAPID ONSET of pain. Needs a rapid acting medication.
Anticipate ordering medication for breakthrough pain if pain persists after administering original pain med. OPIOIDS
Sources of Pain
Somatic
Visceral
Somatic Pain
Diffuse and scattered and originates in:
- Tendons
- Ligaments
- Bones
- Nerves
- Blood Vessels
caused by mechanical/ chemical/ thermal/ electrical disorders/ injury affecting bones, joints, muscles, skin, or other structures composed of CONNECTIVE TISSUE.
Visceral Pain
Poorly localized. Originates in Body organs:
- Cranium
- Thorax
- Abdomen
Produced by disease. Occurs as organs become distended, ischemic, or inflamed.
Reface contraction of abdomen wall may occur to protect additional trauma.- GUARDING
Organs: HEART, KIDNEYS, INTESTINES that are diseased/ injured.
Acute Pain
Rapid onset. Varies in intensity from MILD to SEVERE.
Tissue damage/ disease and triggers autonomic responses as increased HR, fight or flight, increased BP.
Ends once healing occurs. Not longer than 6 months.
ex: pricked finger, sore throat, surgery
Chronic Pain
Intermittent/ consistent.
- May lead to withdrawal, depression, anger, frustration and dependency.
- Exacerbation.
Lasts after normal healing pain.
Time varies: 6 months or longer.
Transduction
Nociceptive
Activation on pain receptors. Injured chemicals release chemicals that excite nerve endings.
- Bradykinin triggers release of histamine- produces redness, swelling, pain when inflammation happens
- Prostaglandins: hormone like substance that sends additional pain stimuli to CNS
- Substance P: sensitives receptors on nerve to feel pain and increases firing of nerves.
Transmission
Pain sensations from site of injury are sent to the spinal cord and then higher centers
Protective Pain Reflex: responsible for withdrawal of endangered tissue from a damaging stimulus.
Perception
Sensory process that occurs when a stimulus for pain is present.
Person’s interpretation of the pain.
hot shower- increasing heat- pain threshold
Modulation
Process that the sensation of pain is inhibited.
Regulated by: Neuromodulators: morphine like chemical regulators in spinal cord and brain.
Endorphine: most potent
Enkephalins: least potent. Reduce pain sensation by inhibiting the release of substance P from the terminals of afferent neurons
opioid neuromodulators.
Gate Control Theory of Pain
Relation between pain and the projection of pain info to the brain.
Factors that have an impact on opening and closing:
- past experiences
- cultural/ social environment
- personal expectations
- beliefs about pain
Interventions to close gate
- Massages, warm compress to a painful lower back area will stimulate large nerve fibers to close the gate.
Blocks impulses from that area.
Teach: self management techniques that activate closing the gate also minimize the experience of pain.
Common Responses to Pain
- Physiological
- Behavioral
- Affective
Physiological
Responses are involuntary body responses.
Sympathetic stimulation: MODERATE/ SUPERFICIAL PAIN
Parasympathetic stimulation: PROLONGED/ SEVERE/ VISCERAL PAIN
Sympathetic Stimulation
Physiological
MODERATE/ SUPERFICIAL PAIN:
- Increased vital signs (BP, P, RR)
- Pupil dilation
- Muscle tension
- Rigidity
- Pallor
- Increased adrenaline
- Increased blood glucose