NREMT Flashcards

1
Q

Alpha -1 has what effect?

A

Vasoconstriction of arteries and veins

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2
Q

Alpha - 2 effect ?

A

Inhibits norepinephrine release, Inhibits insulin release, stimulates glucagon secretions, and inhibits my lipolysis

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3
Q

Beta one effect?

A

Increases heart rate – positive call real trophy, increase his cardiac contractility, increases myocardial conduction, and increases renin production to retain your

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4
Q

Beta 2 effect?

A

Smooth muscle relaxer that causes bronchodilation and stimulates glycogenolysis insulin secretion

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5
Q

Lemon pneumonic

A
L Look externally
E Evaluate 3-3-2
M MALLAMPATI
O Obstructions
N Neck mobility
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6
Q

Normal RR

Adult
Child
Infant

A

12 - 20
15-30
25-30

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7
Q

Cheyenne stokes respirations

A

Periodic breathing with cycles of increasing rate and depth of breathing followed by gradual decrease in depth and Rate breathing in between periods of apnea that can last up to 60 seconds

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8
Q

KUSSMAUL respirations

A

Continuous deep sign breast with a rapid rate usually greater than 40 when the body is responding to metabolicAcidosis

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9
Q

Normal ETC02 values range between one what and what?

A

35 and 45

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10
Q

Loud high-pitched crackling

A

Rails

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11
Q

Low pitched crackles found in the lower airway is made clear with a cough

A

RONCHI

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12
Q

Pericardium

A

Fibrous sac that protects the heart from other structures of the chest and contains lubricating fluid to reduce friction

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13
Q

Three layers of the muscular walls of the heart

A

Epicardium outer most layer
Myocardium contractile muscle
Endocardium layer that lines the inside of the heart and protect muscle tissue from the friction of the blood flowing

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14
Q

Stroke volume range

A

60 to 100

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15
Q

Frank’s darling principal

A

A property of cardiac muscle; as it gets stretched more, it contracts with greater force

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16
Q

Atherosclerosis

A

Narrowing of aorta cerebral and coronary blood vesselsBy way of fatty material deposited along the inner walls of the arteries

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17
Q

Axis deviation can be determined

A

BY looking at lead one and lead

aVF

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18
Q

Normal
Left axis
Right axis
Extreme right

A

Up and Up
Up and Down
Down and Up
Down and Down

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19
Q

Left bundle branch block and what leads

A

Leads one leaves AVF and lead V6

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20
Q

Right by the branch block

A

One, a avl , V5, and v6

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21
Q

TRhombus

A

Blood clot already formed in the near artery

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22
Q

Embolus

A

Blood clot formed elsewhere in the body can travel to Coronary artery

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23
Q

Natural effects in the respect to myocardial infarction

A

Natural dilates coronary arteries which in hopes will increase oxygen to ischemic areas… Will also significantly reduce preload because it dilates the Venus side of the vasculature

With all nitro in patient suspected of inferior myocardial infarction which includes leads two, three, and aVF

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24
Q

Vasopressor of choice during acute coronary syndrome

A

Dopamine we use dopamine because it maintains a renal bloodflow low doses while increasing myocardial contractility

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25
Chest pain abdominal pain and back pain or off in the chief complaint for what aneurysm
Abdominal aortic aneurysm | Pulsating masses can also be found it
26
Drug of choice for hypertension
Labetalol because it has alpha and beta blocker effects. As an alpha blocker it relieves peripheral vasoconstriction, and it’s been a block in effect prevent the possibility of rebound tachycardia tell me a company or drop in blood pressure. The beta blockade also have negative inotropic affects
27
Beck triad
JVD hypotension and muffled heart tones
28
Biot’s respirations
Biot respirations can be described as a regular rate and depth of reading with periodic apnea
29
Shush Cheyenne stokes respirations
A gradual increase in respiration followed by a gradual decrease with apnea that the last of the 60 seconds
30
Cushing’s Triad
Ataxic respirations Low heart rate high blood pressure brainstem injury
31
Synchronize cardioversion for SVT in a flutter
50 to 100 J
32
Synchronize cardioversion for a fib
120 to 200 J
33
Synchronize cardioversion for unstable ventricular tachycardia with pulses
100 to 150 Jules
34
One cardiac cycle occurs every how many seconds
0.8
35
Stick fibrosis
Cystic fibrosis is a chronic disease primarily causing respiratory complications and deterioration. It predisposes patients to pneumonia and other components of respiratory failure as well.
36
Prior to puberty the hypothalamus restricts the production of this hormone
GRh
37
At the start of puberty the hypothalamus releases pulses of GRH which trigger the
Follicle-stimulating hormone and luteinizing hormone. These hormones trigger the production of other sex hormones that develop and maintain the reproductive system
38
The follicle stimulating hormone and luteinizing hormone secrete what hormones
Estrogen
39
The corpus luteum is secretes what
Progesterone
40
Four phases of the metro cycle
follicular phase,Ovulation, the luteal phase, Menstruation
41
Hypermenorrhea also known as menorrhagia is what
Abnormally or excessive heavy bleeding during a period
42
Polymenorrhea
Refers to a condition where a woman has a period more frequently than once every 24 days and is brought on by physical mental stress
43
Dysmenorrhea
Painful Menzies that can be so badass to interfere with their daily life
44
Metrorhagia
Spotting that occurs in between periods
45
Physiological changes in pregnant.
The heart increases in size to handle the polycythemia and increase circulating volume from 10% to 15%. Overall cardiac output increases by 40% kidneys increase in size but the 30%. Total volume and minute volume increase by as much as 50%. Blood volume increases by approximately 50%
46
A patient is suspected to be abusing recreational drugs. He currently presents with hypotension, cool and dry skin, respiratory depression, nausea, and diminishment in orientation. Vitals also are diminishing. What medication may be warranted? Dextrose Metoprolol Narcan Versed
Narcan
47
A 68-year-old male is displaying new onset, right-sided facial droop and an inability to move his left arm. When considering where to transport this patient, the most important factor to consider would be: availability of a trauma surgeon. closest location. availability of a neurosurgeon. availability of CAT scan.
CAT SCAN
48
You arrive on scene for a 70-year-old female sitting on the toilet. She has experienced pain in her upper abdomen for the past two hours. As you help her to the ambulance cot, you note dark, tarry stools in the toilet. She states her abdomen hurts worse right after eating. She was recently diagnosed as anemic by her primary care physician. What is your field impression at this time? Diverticulitis Constipation Gastric ulcer Hemorrhoids
ULCER
49
The history of abdominal pain after eating (when stomach acid is high), dark tarry stools, and recent diagnosis of anemia is a sign of
gastric ulcer
50
Trousseau sign
which is a carpopedal spasm induced by ischaemia through inflation of a sphygmomanometer cuff to a suprasystolic blood pressure
51
A patient is suspected of having cirrhosis of the liver. Which one of the following conditions would the paramedic expect to find in a patient with end-stage liver disease? A severely enlarged liver on palpation Encephalopathy Seizures Nausea and vomiting
Encephalopathy Early on, the liver becomes enlarged and signs of inflammation such as itching, pain, nausea, vomiting, and anorexia occur. As the disease progresses, bleeding tendencies and esophageal varices develop. End-stage disease is typically characterized by a buildup of toxins that leads to altered levels of consciousness, or encephalopathy.
52
Which of the following is most likely to indicate appendicitis? Umbilical pain migrating to the RLQ Pain radiating to the back Vomiting Diffuse pain in both lower abdominal quadrants
Abdominal complaints are always challenging to diagnose. Pain beginning at the umbilicus which migrates to the RLQ is most indicative of appendicitis (approximately 66% of patients report this pain pattern). The other signs and symptoms may appear in other abdominal conditions.
53
You arrive on scene for a 70-year-old female sitting on the toilet. She experienced pain in her upper abdomen for the past two hours. You note dark, tarry stools in the toilet. She states her abdomen hurts worse right after eating. What is your field impression at this time? Hemorrhoids Diverticulitis Gastric ulcer Constipation
Gastric ulcer The history of abdominal pain after eating (when stomach acid is high), dark tarry stools, and recent diagnosis of anemia are all consistent with a gastric ulcer.
54
A 78 year old male had a sudden onset of a severe headache and associated neurological dysfunction, dysphasia, and motor ataxia. He tells you the headache is progressively worsening. Based on this information, what is the likely cause? Hemorrhagic stroke Embolic stroke Thrombotic stroke Intracerebral tumor
There are usually three mechanisms behind the precipitation of a stroke. A hemorrhagic stroke usually is abrupt at onset, and rapidly progresses as blood continues to spill into the cranial vault. An embolic stroke happens suddenly, with significant findings, but usually does not progress much further, because there is a specific amount of brain tissue affected by the cessation of blood flow. Thrombotic strokes and cerebral tumors have a slower onset (possibly over days, months, and even years) as the thrombus (tumor) continues to expand in the brain. Although all these conditions may present with a headache, the progression of the syndrome is key to forming an accurate diagnosis.
55
Visceral pain
Visceral pain originates in the body's organs. Often this pain is described very generally as dull, heavy, oppressive, or as pressure or ache.
56
Pleuritic Pain
Pleuritic pain is caused by inflammation of parietal pleura of the chest that is described as sharp, stabbing, or burning. The pain often radiates.
57
Somatic Pain
Somatic pain is similar to pleuritic pain and is described as sharp and originating from the skin, muscles, or bones.
58
Referred Pain
Referred pain is when the discomfort is perceived in other parts of the body.
59
Ascites
Excessive Swelling of the abdomen with fluid | Related to liver disease
60
OPQST
Onset, Provocation, Quality, Region-Radiation, Severity, and Time
61
SAMPLE
Signs/Symptoms, Allergies, Medications, Past Medical History, Last oral Intake, and Events leading up to the occurence.
62
``` Trauma Criterion Step One Physiological Criterion Glascow Systolic Resp Rate Vent Assistance ```
GCS less than or equal to 13 Systolic less than 90mmhg Resp Rate less than 10 or more than 29 (Less than 20 if under 1 year old)
63
Trauma Criterion | Anatomic Criteria
- open or depressed skull fractures - Penetrating chest would to head, neck, torso, extremities proximal to elbow or knee. - chest wall instability or deformity - amputation to wrist or ankle - crushed, degloved, mangled, or pulseless extremity. - pelvic fractures -paralysis
64
Trauma MOI Criteria
Adult: Falls > 20ft Children: Falls > 10ft or 2/3 times the height of the child Intrusion, including roof: greater than 12 on occupants side; 18 on any other side. Ejection Death in the compartment Auto vs pedestrian, run over, thrown, or significant impact. Motorcycle crash > 20mph
65
Order of Trauma Assessment | IPAP
Inspect, Palpate, Auscultate, Percuss
66
.Glascow Coma Scale
Highest is 15 Eyes - 4 Verbal - 5 Motor - 6
67
Decorticate
elbows, wrists, and fingers, flexed, stiffened legs, and feet. OUTWARDLY
68
Decerebrate (worse)
stiffened and INTERNALLY rotated elbows, and legs, teeth clenched, legs stiff and feet extended.
69
Babinski Response
Big toe moves upward and other toes fan out when stroking bottom of foot, mean CNS Problem.
70
Term for Dialated Pupils | and Occurrences
Mydriasis Cardiac Arrest, shock, cerebral hypoxia, cocaine, epi, amphetamines.
71
Term for Constricted Pupils | and Occurrences
Miosis Narcotics, central nervous system disorder, glaucoma meds, bright light
72
Term for Unequal Pupils | and Occurrences
Brain injury, brain tumor, stroke, artificial eye, eye meds, or can be normal (called anisocoria)
73
Rapid eye movement term
Nystagmus
74
Rales/Crackles
Fluid in small airways-alveoli Found in CHF, Pulmonary edema, drowning, pneumonia, COPD
75
Rhonchi
Mucus or fluid in the larger airways - Bronchioles Found in Bronchitis, COPD, pnuemonia
76
Wheezing
Whistling sound due bronchoconstriction or marrowing of the terminal bronchioles due to edema ( swelling)
77
Vesicular
Soft, low pitch heard in lung periphery
78
Apneustic Breathing
Long, deep breathes that are stopped during inspirations then periods of apnea. found in CNS injury or stroke
79
Biots (ataxic)
Irregular periods of breathing or gasping with apnea Usually due to increased ICP
80
Cheyne-Stokes
Increasing in rate and depth, decreasingg, in rate and depth with period of apnea Neurological condition
81
Hyperressonant Percussion
Air - snare drum Hollow tension pneumothorax, emphysema, asthma
82
Hyporessonant Percussion
Dull - Watery
83
Abdominal Quadrant Right Upper Quadrant (RUQ)
Liver Gall Bladder Stomach
84
Abdominal Quadrant Left Upper Quadrant (LUQ)
Liver Spleen Stomach Pancreas
85
Abdominal Quadrant Right Lower Quadrant (RLQ)
Appendix Right Ovary and FallTube Bladder
86
Abdominal Quadrant Left Lower Quadrant (LLQ)
Left Ovary and Tube | Bladder
87
Visceral Pn
Internal organs damaged or injured -vague not well organized, pressure-like, dull, aching, cant point to pAin, with one finger, stretches, ischemia, inflammation
88
Somatic Pain
irritation, of peritoneal lining, sharp, localized, throbbing, deep breathe increases pain
89
Referred Pain
discomfort perceived in other parts of the body such as cardiac pain referred to jaw or arm
90
Cullens Sign
Ecchymosis (bruising) around umbilicus Bleeding in the abdominal cavity
91
Grey Turner Sign
Ecchymosis (bruising) in the flank Bleeding from kidneys, hemorrhagic pancreatitis
92
Kehrs Sign
Referred pain to shoulder Ectopic or spleen injury, blood under diaphragm
93
Murphys Sign
Right Upper quadrant pain Cholecystitis (inflammation of the gall bladder gal stones)
94
Brudzinkis Sign
Hip flexes when neck is flexed Meningeal irritation, meningitis
95
Hamman's Sign
Crunching, rasping sound that correlates with heart beat. heard over mediastinum. spontaneous mediastinal emphysema tracheobronchial trauma
96
Hormans Sign
pain incalf on dorsiflexing foot while leg is straight meningeal irritation, meningitis
97
Clubbing of the fingers sign
flattening of the nail angle; Sign of chronic hypoxia - COPD
98
Measure Blood Pressure Formula
Cardiac Output X Systemic Vascular Resistance
99
Pulse PRessure
Difference between systolic and diastolic NARROWS in shock WIDENS in increased ICP
100
Orthostatic Vital Signs | positive Tilt
Decrease in Systolic BP 20mm Increase Diastolic BP 10mm Increase Heart Rate HR of 200 suggests Hypovolemia - Dehyadration
101
MAP
Mean Arterial Pressure - average pressure in arteries during one cardiac cycle, considered good indicator of perfusion normal between 70-100mmhg ADD DIASTOLIC + 1/3 of PULSE PRESSURE (Systolic minus Diastolic)
102
Heart Rates for Peds ``` Adolescent (13-18) School-Age (6-12) Preschool (3-5) Toddler (12-36 months) Infant ( Birth to 1 year) ```
Adolescent (13-18) .................... 55-105 School-Age (6-12) .....................70-110 Preschool (3-5)..........................80-120 Toddler (12-36 months)....................80-130 Infant ( Birth to 1 year)...........100-160
103
Pediatric Assessment Triangle
APPEARANCE - muscle tone, speech/cry, eye contact WORK OF BREATHING - airway sounds, positions CIRCULATION OF SKIN - pallor, mottling, cyanosis
104
Fontanel
Two Fontanels (Holes in Skull) Anterior closes 12-18 months Posterior closes by 3 months If found SUNKEN - dehydrated or shock if found BULGING - Increased ICP
105
Cerebral Palsy
group of chronic disorders, damage motor centers in brain in children; abnormal muscle spasms, hearing and vision problems, seizures
106
BGL normal measures
70 - 140mg/dl
107
Why is it difficult to judge Sp02 with sickle sell PTS
Anemia/blood disorders have inherent hemogloban issues.
108
Osmosis
Fluid movement from an area of less concentration to an area of greater concentration
109
Isotonic
stays in hlood vessels longer, field fluid replacement Normal Saline and Lactated Ringers
110
Hypertonic
more particles, fluid moves in vascular space, 10% Dextrose
111
Hypotonic
1/2 NS
112
5% Dextrose in Water
Not used for fluid replacement MI, CHF, giving drugs, KVO
113
``` Calcium chloride magnesium potassium sodium ```
``` calcemia chloremia magnesemia kalemia natremia ```
114
Hematocrit
%of red blood cells in the whole body - approx 45%
115
White blood cells - Leukocytes
fight infection
116
Platelets/Thrombocytes
promote clotting
117
ABO bloodtyping
O negative Universal Donor | AB+ Universal Recipient
118
GTTS Formula
V x SS x DD ----------------- C
119
Fluid Challenge | Infusion Rate Over Time
V x SS ---------- T
120
Blood Gases ``` pH PCO2 PO2 BiCard HCO3 ```
pH - 7.35 - 7.45 (If high, acidosis - if low, alkalosis) PCO2 - 35-45 (If high, acidosis - if low, alkalosis) PO2 - 80-100 (if under 80, hypoxic) BiCarb - 22-26 HCO3 - 22-26
121
What receptors located in this arch and this sinus sense changes in BP? once sensed what happens?
Baroreceptors located in the aortic arch and carotid sinus sense changes in BP send messages to cardio centers in medulla.
122
RE: Shock, Once changes in BP are sensed, messages sent to cardio centers in medulla. What hormone is secreted and from what gland and what happens?
Antidiuretic hormone from the pituitary - Increases Peripheral Vascular Resistance, retains water in kidneys.
123
RE: Shock... What is released by this organ to convert Angiotension 1 to angiotension 2? What does AG2 do?
Renin is secreted by the kidneys | AG2 is a potent Vasoconstrictor
124
RE: Shock...Adrenal glands secrete what?
Epinepherine and norepiniephrine to increase SVR (maintains BP) and stimulates aldosterone which retains sodium and water in kidneys
125
Hypovolemia Causes and Assessment Findings
Cause Loss of blood or fluids ``` Findings Increased HR Increased Resp Low BP Pale or Cyanotic Flat veins ```
126
Cardiogenic
``` Cause Myocardial Infarctions (paricularly Left Ventricular HF or other cardiac issue) ``` Findings Increased or Decresead HR Pulmonary edema Pale or Cyanotic
127
Anaphylactic Shock
Causes Allergic Reaction ``` Findings Increased HR Decreased BP Angioedema (fluid leaking out and appearing like swelling) wheezing, stridor, utercaria (hives) ``` ``` Treatment Airway, O2, BVM if needed Beta2 Agonist - Epi/Diphenhydramine 0.3 epi 1:1000 IM 1mg/kg (MAX 50) ```
128
Neurogenic Shock
Causes Spinal Injury - Sympathetic Nerve conduction interrupted ``` Findings Decreased BP Normal or Decreased HR Vasodilation hypothermia ```
129
Septic Shock
Overwhelming Infection Decreased BP High Temp/Fever Vasodilation Treatment Airway, O2, fluids, Dopamine - 5 - 20mcg/kg/min for hypotension or shock
130
Obstructive Shock
Causes Pulmonary Embolism Cardiac Tamponade Tension Pneumothorax
131
Controlled Substance Act
Schedule 1 no medicinal use Heroin, Weed, LSD, Mescaline, Crack Schedule 2 easily abusable Cocaine, Morphine, Fentanyl, Methadone
132
Things to remember with geriatrics per drog ingestion
Decreased liver/kidney/renal function can affect drug action/elimination. Levels can be toxic
133
Parasympathetic ANS
``` chemical control acetylcholine nervous control vagus nerve Organophosphate like effects (SLUDGE) Salivation, Lacrimation, Urination, Defacations, Gastrointestinal Distress and Emesis ``` ``` At Rest Constricted Pupils Flushed Skin (Vasodilation) Dry Skin Decreased HR, Decreased BP ``` Treatment Atropine - 0.5 mg/kg Bradycardia
134
Sympathetic ANS
Chemical control Norepinipherine then later epi Nervous Control nerve roots in thoracic and upper lumbar area of spine
135
Alpha Responses Heart Blood vESSELS Bronchioles
Heart - No effect Blood vESSELS - Constricts, Increased BP Bronchioles - No response or slight bronchoconstriction
136
Beta Responses Heart Blood vESSELS Bronchioles
``` Heart (B1) Increased HR (chronotropic) Increased Contractility (Inotropic) Increased Conductivity (Dromotropic) ``` Blood Vessels Dilate, Decreased BP Bronchioles (B2) bRONCODILATION
137
Drugs given IN (MAD)
Midazolam (Ativan), Naloxone (Narcan), Fentanyl, Lorazepam (Versed), Glucagon, max 1ml in each nostril
138
Enteral
Drugs given via GI tract(orally, SL, NG tube, rectally;
139
Parenteral
IV, IM, IN, SQ
140
Adenosine/Adenocard
Class - Antidysrhythmic MOA - slows conduction to the AV Indications - SVT
141
Albuterol
Class - Sympathomimetic, bronchodilator MOA - Beta 2 agonist Indications - Bronchospasms, COPD
142
Amioderone (Cordarone)
``` Class - Antidysrhythmic MOA - blocks sodium/potassium channels, delaying repolarization and increasing the duration of action potential Indications - VFib, PVT, Unst VTach Contraindications 2nd or 3rd degree Heart Blocks Sinus Brady ``` Dose 300mg first dose/ 150mg second dose
143
Amyl Nitrate
Class - Antidote | Indications - Cyanide Poisoning
144
Aspirin
Class - Platelete Inhibitor, anti-inflammatory Indications - Acute Chest Pn suggestive of AMI Contradictions - acute ulcer or asthma
145
Atropine
Class - Anticholinergic MOA - Inhibits the action of acetylcholine, increases HR in life threatening bradycardia. Indications - Unstable Bradycardia, Organophosphate poisoning, beta blocker/calcium channel blocker OD. Dosage and administration Adult: Unstable bradycardia: 0.5 mg IV/IO every 3–5 minutes as needed. Not to exceed total dose of 0.04 mg/kg (maximum 3 mg total). Use shorter dosing interval (3 minutes) and higher doses in severe clinical conditions. Organophosphate poisoning: Extremely large doses (2–4 mg or higher) may be needed. Pediatric: Unstable bradycardia: 0.02 mg/kg IV/IO (minimum dose: 0.1 mg). May repeat once. Maximum single dose: Child: 0.5 mg. Adolescent: 1 mg. Maximum total dose: Child: 1 mg. Adolescent: 3 mg. ET dose: 0.04–0.06 mg/kg. Rapid sequence intubation: 0.01–0.02 mg/kg IV/IO (minimum: 0.1 mg, maximum: 0.5 mg).
146
Diazepam (Valium)
Class - Benzo MOA- Induces amnesia and sedation. rasies seizure threshold Indications - Muscle relaxant, acute anxiety and agitation. Dosage and administration Adult: Seizure activity: 5–10 mg IV q 10–15 minutes PRN (5 mg over 5 minutes) (maximum dose: 30 mg). Premedication for cardioversion: 5–15 mg IV over 5–10 minutes prior to cardioversion. Pediatric: Seizure activity: 0.2 mg/kg to 0.5 mg/kg slow IV q 2–5 minutes up to 5 mg (maximum dose 10 mg/kg). Rectal diazepam: 0.5 mg/kg via 2" rectal catheter and fl ush with 2–3 mL air after administration
147
Diltiazem
Class - Calcium Channel Blocker MOA - Slows calcium channel blocker that blocks calcium ion influx during depolarization of cardiac and vascular smooth muscle. reduces preload and afterload. reduces myocardial oxygen demand. Indications - Afib, Aflutter, SVT Dosage and administration Adult: Initial dose: 0.25 mg/kg (15–20 mg for the average patient) IV over 2 minutes. If inadequate response, may re-bolus in 15 minutes. Secondary dose: 0.35 mg/kg (20–25 mg for the average patient) IV over 2 minutes
148
Diphenhydramine (Benadryl)
Class - Antihistamine, anticholinergic MOA - blocks histamine receptors, decreases vasodilation, reverses extrapyramidal reactions. Indications - Anaphylaxis, Extrapyramidal, Dosage and administration Adult: 25–50 mg IM, IV, PO. Pediatric: 1–2 mg/kg IV, IO slowly, or IM. If PO: 5 mg/kg/24h.
149
Dopamine
Class- Sympathomimetic, vasopressor, inotropic agent MOA - produces positive chronotropic and inotropic effects. Constricts systemic vasculature, increasing BP, and preload. increases myocardial contractility and stroke volume. Indications - Cardiogenic, distributive and septic shock, hypotension with low cardiac output states. Dosage and administration Adult: IV/IO infusion at 2–20 µg/kg/ min, slowly titrated to patient response. Pediatric: IV/IO infusion at 2–20 µg/kg/min, slowly titrated to patient response
150
Calcium Chloride
Class Electrolyte (anion). Mechanism of action Increases cardiac contractile state (positive inotropic effect). May enhance ventricular automaticity. Indications Hypocalcemia, hyperkalemia, hypermagnesemia, beta blocker and calcium channel blocker toxicity. Contraindications Hypercalcemia, ventricular fi brillation, digitalis toxicity. Adverse reactions/side effects Syncope, cardiac arrest, dysrhythmia, bradycardia, hypotension, asystole, peripheral vasodilation, nausea, vomiting, metallic taste, tissue necrosis at injection site, coronary and cerebral artery spasm. Drug interactions May worsen dysrhythmias secondary to digitalis toxicity. May antagonize the effects of calcium channel blockers. Do not mix or infuse immediately before or after sodium bicarbonate without intervening fl ush. How supplied 10% solution in 10 mL (100 mg/mL) ampules, vials, and prefi lled syringes. Dosage and administration Adult: Calcium channel blocker overdose and hyperkalemia: 500 mg to 1,000 mg (5–10 mL of 10% solution) IV push. May repeat as needed. Pediatric: Calcium channel blocker overdose and hyperkalemia: 20 mg/ kg (0.2 mL/kg) slow IV
151
Epinephrine
Class Sympathomimetic. Mechanism of action Direct-acting alpha and beta agonist. Alpha: vasoconstriction. Beta-1: positive inotropic, chronotropic, and dromotropic effects. Beta-2: bronchial smooth muscle relaxation and dilation of skeletal vasculature. Blocks histamine receptors. Indications Cardiac arrest (asystole, PEA, ventricular fi brillation and pulseless ventricular tachycardia), symptomatic bradycardia as an alternative infusion to dopamine, severe hypotension secondary to bradycardia when atropine and transcutaneous pacing are unsuccessful, allergic reaction, anaphylaxis, asthma. Contraindications Hypertension, hypothermia, pulmonary edema, myocardial ischemia, hypovolemic shock. Adverse reactions/side effects Nervousness, restlessness, headache, tremor, pulmonary edema, dysrhythmias, chest pain, hypertension, tachycardia, nausea, vomiting. Drug interactions Potentiates other sympathomimetics. Deactivated by alkaline solutions. MAOIs may potentiate effect. Beta blockers may blunt effects. How supplied 1:1,000 solution: Ampules and vials containing 1 mg/mL. 1:10,000 solution: Prefi lled syringes containing 0.1 mg/mL. Auto-injector (EpiPen): 0.5 mg/mL (1:2,000). Dosage and administration Adult: Mild allergic reactions and asthma: 0.3–0.5 mg (0.3–0.5 mL 1:1,000) SC. Anaphylaxis: 1 mg (10 mL of 1:10,000) IV, IO over 5 minutes. Cardiac arrest: IV/IO dose: 1 mg (10 mL, 1:10,000 solution) 3–5 minutes during resuscitation. Follow each dose with a 20-mL fl ush and elevate arm for 10–20 seconds after dose. Continuous infusion: Add 1 mg (1 mL of a 1:1,000 solution) to 250 mL normal saline or D 5 W (4 µg/mL). Initial infusion rate of 1 µg/ min titrated to effect (typical dose: 2–10 µg/min). Endotracheal (ET) dose: 2–2.5 mg diluted in 10 mL normal saline. Profound bradycardia or hypotension: 2–10 µg/min; titrate to patient response. Higher dose: Higher doses (up to 0.2 mg/kg) may be used for specifi c indications: (beta blocker or calcium channel blocker overdose). Pediatric: Mild allergic reactions and asthma: 0.01 mg/kg (0.01 mL/kg) of a 1:1,000 solution SC (maximum of 0.3 mL). Anaphylaxis/severe status asthmaticus: 0.01 mg/kg (0.01 mL/kg) IM of a 1:1,000 solution (maximum single dose: 0.3 mg). Cardiac arrest: IV/IO dose: 0.01 mg/kg (0.1 mL/kg) of a 1:10,000 solution every 3–5 minutes during arrest. All ET doses 0.1 mg/kg (0.1 mL/kg) of a 1:1,000 solution mixed in 3–5 mL of saline until IV/IO access is achieved. Maximum single dose 1 mg. Symptomatic bradycardia: IV/IO dose: 0.01 mg/kg (0.01 mL/kg) of a 1:10,000 solution. All ET doses 0.1 mg/kg (0.1 mL/kg) of a 1:1,000 solution. Continuous IV/IO infusion: Begin with rapid infusion, and then titrate to response. Typical initial infusion: 0.1–1 µg/min. Higher doses may be effective.
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Fentanyl
Class Opioid analgesic, schedule II narcotic. Mechanism of action Binds to opiate receptors, producing analgesia and euphoria. Indications Pain management, anesthesia adjunct. Contraindications Known hypersensitivity. Use with caution in traumatic brain injury. Adverse reactions/side effects Confusion, paradoxical excitation, delirium, drowsiness, CNS depression, sedation, respiratory depression, apnea, dyspnea, dysrhythmias, bradycardia, tachycardia, hypotension, syncope, nausea, vomiting, abdominal pain, dehydration, fatigue. Drug interactions Increased respiratory effects when given with other CNS depressants. How supplied 50 µg/mL ampules and Tubex syringes. Dosage and administration Adult: 50 to 100 µg (1µg/kg) IM or IV, IO slow push (over 1–2 minutes) to maximum of 150 µg. Pediatric: 1–2 µg/kg IM, IV, or IO slow push (over 1–2 minutes). The safety and effi cacy in children younger than 2 years has not been established.
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Flumazenil
Class Benzodiazepine antagonist, antidote. Mechanism of action Antagonizes the action of benzodiazepines on the central nervous system, reversing the sedative effects
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Furosemide (Lasix)
Class Loop diuretic. Mechanism of action Blocks the absorption of sodium and chloride at the distal and proximal tubules and the loop of Henle, causing increased urine output. Indications CHF, pulmonary edema, hypertensive crisis. Contraindications Hypovolemia, anuria, hypotension (relative contraindication), hypersensitivity, hepatic coma, suspected electrolyte imbalances.
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Glucagon
Class Hyperglycemic agent, pancreatic hormone, insulin antagonist. Mechanism of action Increases blood glucose level by stimulating glycogenesis. Unknown mechanism of stabilizing cardiac rhythm in beta blocker overdose. Minimal positive inotropic and chronotropic response. Decreases gastrointestinal motility and secretions. Indications Altered level of consciousness when hypoglycemia is suspected. May be used as a reversal agent in beta blocker and calcium channel blocker overdoses
156
Ipratropium (Atrovent)
Class Anticholinergic, bronchodilator. Mechanism of action Inhibits interaction of acetylcholine at receptor sites of bronchial smooth muscle, resulting in decreased cyclic guanosine monophosphate and bronchodilation. Indications Persistent bronchospasm, COPD exacerbation. Contraindications Hypersensitivity to ipratropium, atropine, alkaloids, peanuts.
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Lorazepam (Ativan)
Class Benzodiazepine, short/intermediate acting; sedative, anticonvulsant, schedule IV drug. Mechanism of action Anxiolytic, anticonvulsant, and sedative effect; suppresses propagation of seizure activity produced by foci in cortex, thalamus, and limbic areas. Indications Initial control of status epilepticus or severe recurrent seizures, severe anxiety, sedation. Contraindications Acute narrow-angle glaucoma, coma, shock, suspected drug abuse. Dosage and administration Note: When given IV/IO, must be diluted with equal volume of sterile water or sterile saline. When given IM, lorazepam is not diluted. Adult: 2–4 mg slow IM/IV at 2 mg/min; may be repeated in 15–20 minutes. Maximum dose of 8 mg. For sedation: 0.05 mg/kg up to 4 mg IM. Pediatric: 0.05– 0.20 mg/kg slow IV/IO/IM over 2 minutes. May be repeated once in 5–20 minutes. Maximum dose of 0.2 mg/kg.
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Magnesium Sulfate
Class Electrolyte, anti-infl ammatory. Mechanism of action Reduces striated muscle contractions and blocks peripheral neuromuscular transmission by reducing acetylcholine release at the myoneural junction. Manages seizures in toxemia of pregnancy. Induces uterine relaxation. Can cause bronchodilation after beta-agonists and anti-cholinergics have been administered. Indications Seizures of eclampsia (toxemia of pregnancy), torsades de pointes, hypomagnesaemia, ventricular fi brillation/ pulseless ventricular tachycardia that is refractory to amiodarone, life-threatening dysrhythmias due to digitalis toxicity. Contraindications Heart block, myocardial damage. Dosage and administration Adult: Seizure activity associated with pregnancy: 1–4 g of a 10% solution IV/IO over 3 minutes; maximum dose of 30–40 g/day. Cardiac arrest due to hypomagnesaemia or torsades de pointes: 1–2 g of a 10% solution IV/IO over 5–20 minutes. Torsades de pointes with a pulse: Loading dose of 1–2 g in 50–100 mL of D 5 W over 5–60 minutes IV. Follow with 0.5–1 g/h IV (titrate dose to control torsades). Pediatric: Pulseless ventricular tachycardias with torsades de pointes: 25–50 mg/kg IV/IO bolus of a 10% solution to a maximum dose of 2 grams. Torsades de pointes with pulses/hypomagnesaemia: 25–50 mg/kg IV/IO of a 10% solution over 10–20 minutes to maximum dose of 2 grams. Status asthmaticus: 25–50 mg/kg IV/IO of a 10% so
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Midazolam Hydrochloride (Versed)
Class Benzodiazepine, short/intermediate acting; schedule IV drug. Mechanism of action Reversibly interacts with gamma-amino butyric acid (GABA) receptors in the central nervous system causing sedative, anxiolytic, amnesic, and hypnotic effects. Indications Sedation for medical procedures (eg, intubation, ventilated patients, cardioversion). Contraindications Acute narrow-angle glaucoma, shock, coma, alcohol intoxication, overdose, depressed vital signs. Concomitant use with barbiturates, alcohol, narcotics, or other central nervous system depressants. Adverse reactions/side effects Headache, somnolence, respiratory depression, respiratory arrest, apnea, hypotension, cardiac arrest, nausea, vomiting, pain at the injection site. Drug interactions Should not be used in patients who have taken central nervous system depressants. How supplied 1 mg/mL and 5 mg/mL vials and Tubex syringes. Dosage and administration Adult: 2–2.5 mg slow IV (over 2–3 minutes). May be repeated to total maximum: 0.1 mg/ kg. Pediatric: 0.1–0.3 mg/kg IV/IO (maximum single dose: 10 mg).
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Morphine Sulfate
Class Opioid analgesic (schedule II narcotic). Mechanism of action Alleviates pain through CNS action. Suppresses fear and anxiety centers in the brain. Depresses brainstem respiratory centers. Increases peripheral venous capacitance and decreases venous return. Decreases preload and afterload, which decreases myocardial oxygen demand. Indications Severe CHF, acute cardiogenic pulmonary edema, chest pain associated with acute myocardial infarction, analgesia for moderate to severe acute and chronic pain. Contraindications Head injury, exacerbated COPD, depressed respiratory drive, hypotension, undiagnosed abdominal pain, decreased level of consciousness, suspected hypovolemia, patients who have taken MAOIs within 14 days. Dosage and administration Adult: STEMI: Initial dose: 2–4 mg slow IV (over 1–5 minutes). Repeat dose: 2–8 mg at 5–15 minute intervals. NSTEMI/Unstable angina. 1–5 mg IV push if symptoms not relieved by nitrates, use with caution. Pediatric: 0.1–0.2 mg/kg/dose IV, IO, IM, SC. Maximum dose: 5 mg.
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Naloxone Hydrochloride (Narcan)
Class Opioid antagonist, antidote. Mechanism of action Competitive inhibition at narcotic receptor sites. Reverses respiratory depression secondary to opiate drugs. Completely inhibits the effect of morphine. Indications Opiate overdose, complete or partial reversal of central nervous system and respiratory depression induced by opioids, decreased level of consciousness, coma of unknown origin Dosage and administration Adult: 0.4–2 mg IM/IV/IO/SQ/ ET/Intranasal (diluted); minimum single dose recommended: 2 mg. Repeat at 5-minute intervals to a maximum total dose of 10 mg (medical control may request higher amounts). Pediatric: 0.1 mg/kg/dose IV/IO/IM/SQ every 2 minutes as needed. Maximum total dose of 2 mg. If no response in 10 minutes, administer an additional 0.1 mg/kg/dose.
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Nitroglycerin
Class Vasodilator. Mechanism of action Smooth muscle relaxant acting on vasculature, bronchial, uterine, intestinal smooth muscle. Dilation of arterioles and veins in the periphery. Reduces preload and afterload, decreasing workload of the heart and thereby myocardial oxygen demand. Indications Acute angina pectoris, ischemic chest pain, hypertension, CHF, pulmonary edema. Contraindications Hypotension, hypovolemia, intracranial bleeding or head injury, pericardial tamponade, severe bradycardia or tachycardia, RV infarction, previous administration in the last 24 hours: tadalafi l (Cialis) (48 hours), vardenafi l (Levitra), sildenafi l (Viagra). Dosage and administration Adult: Tablet: 0.3–0.4 mg sublingually; may repeat in 5 minutes to maximum of 3 doses. NTG spray: 1–2 sprays for 0.5–1 second at 5-minute intervals to a maximum of 3 sprays in 15 minutes. NTG IV infusion: Begin at 10 µg/min; increase by 10 µg/min every 3–5 minutes until desired effect. To a maximum of 200 µg/min. Pediatric: Norecommended. IV infusion: 0.25–0.5 µg/kg/min IV, IO titrated by 1 µg/kg/min (max dose: 5 µg/kg/min).
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Ondansetron (Zofran)
Class Serotonin receptor antagonist; antiemetic. Mechanism of action Blocks action of serotonin, which is a natural substance that causes nausea and vomiting. Indications For the prevention and control of nausea or vomiting. Used in hospital for patients undergoing chemotherapy or surgical procedures. Dosage and administration Adult: 4 mg IV/IM may repeat in 10 minutes. Pediatric: 0.1 mg/kg IV/IM.
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Promethazine Hydrochloride (Phenergan)
Class Phenothiazine, antiemetic, antihistamine. Mechanism of action H-1 receptor antagonist; blocks action of histamine; possesses sedative, anti-motion, antiemetic, and anticholinergic activity; potentiates the effects of narcotics to induce analgesia. Indications Nausea/vomiting, motion sickness, sedation for patients in labor, potentiates the analgesic effects of narcotics. Contraindications Coma, central nervous system depression from alcohol, barbiturates, or narcotics, Reye syndrome, lower respiratory symptoms (eg, asthma). Dosage and administration Adult: Dilute 1–3 mg in 10–30 mL of D 5 W. Administer slowly IV at rate of 1 mg/min. Maximum: 5 mg. Pediatric: 0.01–0.05 mg/kg/dose slow IV over 10 minutes. Maximum: 3 mg
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Hypertonic | solutions
``` - 5% Dextrose in 0.9% Sodium Chloride - 5% Dextrose in 0.9% Sodium Chloride - 10% Dextrose in Water (D 10 W) ```
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Isotonic | solutions
- Lactated Ringer’s (Hartmann’s Solution) -0.9% Sodium Chloride (normal saline) -
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Hypotonic | solutions
-0.45% Sodium Chloride (½ normal saline) - 5% Dextrose in Water (D 5 W)
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Strokes are caused by?
caused by blood clot (thrombus) or intravascular particle or clot that moves (embolus) cerebral bleed can also cause stroke
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Window for fibrolynics in stroke?
3 hrs
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Hemiparesis
numbness on one side of the body
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hemplegia
paralysis on one side of the body, dizzy, drooping of mouth
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aphasia
inability to understand or express speech
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dysarthria
difficulty speeking, speech slurred
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How do we transport stroke patient?
supine with head elevated 15-30 degrees or on side, (affected side down), IV KVO (no fluid bolus). oxygen only in indicated.
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Difference between stroke and TIA?
TIA resolves within hours
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Stages of Seizures
Aura - sensation loss of consciousness tonic phase - systemic rigidity hypertonic - arched back and rigid clonic phase - rhythmic contraction of major muscle groups, lip smacking, biting, clenching teeth post seizure - muscles relax nystagmus, eyes in back of head postictal - rest period of the brain
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absence seizures (pet mal)
little to no movement; typically children
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drugs for seizures
Lorazepam (Ativan) and Midazelam ( versed) and Mag Sulfate Ativan - 1-2 mg IV, IM, can be given IN (intranasal) Peds dose- 0.1mg/kg Versed 1-5 mg IV, IM, IO, IN (intra nasal)  Peds dose 0.1 mg/kg Mag 2-4 gm IV for eclampsia
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status elepticus
prolonged general seizure over 5 minutes or 2 or more seizures without consciousness aspiration, bone and spinal fractures, hypoxia, brain damage, hypoglycemia, dehydration, metabolic acidosis.
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sudden onset of severe headache, neuro symptoms, unconscious. "worst headache ever" usually presents in young female
subarachnoid bleed
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medical term for headache
cephalgia
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brain and spinal cord tumor causes weakness or paralysis or change in sensation of one or more limbs or side of face, dizzy, diplopia (double vision), similar to stroke
CNS neoplasm
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sudden/acute state of confusion which is reversible | elderly may experience at night
delerium
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chronic deterioration of memory, reasoning, judgement, usually progressive deterioration
dementia
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Presents with fever, head and back ache, nuchal rigidity, altered, bulging fontanel Brudzinski sign - hips and knees flex when neck flexed Kernigs Sign - unable to straighten leg when hip flexed, rash, irritability.
bacterial meningitis viral meningitis also presents with fever but not as severe. PPE droplet precautions - mask for us and PT
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Neurological Disorder Cause neurons die, buildup of plaque in brain signs memory loss, confusion, can be agressive/violent, eventually cant talk and need constant care
Alzheimers
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Neurological Disorder causes infection usually due to eating contaminated beef (fatal af) signs ataxia, jerking, visual impairment, mental disorientation
Creutzfeldt-Jakob disease
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Neurological Disorder Causes genetic disorder with loss of neurons in brain signs restless, abnormal eye movements, irritability, ataxia, difficulty standing, memory loss.
Huntington disease
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Neurological Disorder Cause autoimmune, usually viral, inflammation and destruction of myelin sheath Signs tingling in fingers and toes, progressive muscle weakness, trouble talkin and swallowing, difficulty breathing, paralysis.
Guillain-Barre syndrome
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Neurological Disorder Cause inflammation of nerve cells, loss of myelin sheathm inhibits nerve impulses, autoimmune Signs Diplopia, nystagmus, speech diff, weakness, impaired coordination, progressive
Multiple Sclerosis
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Neurological Disorder Cause unknown cause, loss of dopamine producing brain cells signs impaired voluntary movements and coordination, tremors, rigidity, chronic and progressive
Parkinsons
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Neurological Disorder Cause obsessive behavior, socially inappropriate, mental highs of depression, tremors, incontinence ``` signs genetic disease (cause unknown), damage to neurons ```
Pick Disease
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Neurological Disorder Cause Thiamine deficiency usually in chronic alcoholics signs mental confusion, abnormal eye movements, ataxia
Wernicke | ecephalopathy
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Neurological Disorder signs weakness, rapid fatigue of muscles under voluntary control, breakdown in communication between nerves and muscles, trouble talking, swallowing droopy eyelid and mouth
Myasthenia Gravis
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Organs in the RUQ LLGPSK
``` Large Liver Gallbladder Pancreas (tail) Stomach (small) Kidney (right) ```
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Organs in the LUQ SSPK
Stomach Spleen Pancreas Kidney (left)
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Organs in the RLQ AIO
Appendix, reproductive organs, (ovary fallopian tube, spermatic cord) right ureter
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Organs in the LLQ
Left ureter | reproductive organs
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Order for ABD Assessment
Inspect Auscultate Percuss Palpate
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ABD Pain in RLQ (McBurneys Point??), may have periumblical pain initially, may have rebound Ass Finding sick for several days, n/v, low grade temp (only high if ruptured), low appetite.
Appendicitis
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ABD Pain in RUQ (Murphys Sign??), pn on taking deep breathe when palpation of RUQ or epigastric pn) Ass Finding yellow or green bile emesis, usually after a fatty meal, more common in women
cholecystitis (inflammation of the gallbladder)
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pain in RUQ Ass findings jaundice, nausea, and vomiting, weight loss, fatigue, bruising easily
cirrhosis of the liver
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Pain in LLQ Ass findings fever, nausea/vomiting, diarrhea or constipation
Diverticulitis
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Absent ABD pn Ass findings bleeding in GI tract - vomiting blood (name for that?), shock
Esophageal varices
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ABD disorder; epigastric pain Ass finding black, tarry stools (name for this?), coffee ground emesis or blood (name for this), patients on anticoagulant more susceptible
GI hemorrhage-upper (ulcers)
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ABD disorder; Pn in lower abdomen, rectal, or no pain Ass findings bright red or wine-colored stool (name for this?)
GI hemorrhage- lower | hemorrhoids, anal fissures
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ABD disorder; Absent pain Assessment finding hematemesis, melena Patient been vomiting
Mallory-Weiss Tear transport in reverse trendelenburg position
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ABD issue: Pain can be anwhere, usually entire abd Ass findings rigid abdomen, fever, vomiting
Peritonoitis | inflammation of abdominal lining
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ABD issue: Pn in LUQ and may radiate to back or epigastric area Findings Diaphoresis, tachycardia, appears ill, sepsis, shock
Pancreatitis
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Tearing or shearing abdominal pain and possibly back pn findings unequal pulses in the lower extremities, urge to defecate due to blood in retroperitoneal space, many times Hypertensive history
Abdominal Aortic Aneurysm transport rapidly and carefully could rupture in route
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Tearing pain in upper chest and between shoulder blades Unequal pulses in upper extremities,
Thoracic aortic aneurysm transport rapidly and carefully could rupture in route
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Acute Kidney Renal Failure
S&S -sudden onset (days), oligaria (decrease in urine), anuria (no output), fatigue, weight loss, trauma to flank, AMS, hyperkalemia, heart dysrhythmia
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Chronic Kidney Failure
long and slow process diabetes/hypertension slower onset than AKF, may have some urine output, anemia needs dialysis for the rest of life
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What happens if PT misses dialysis?
Fluid overload, pulmonary edema, hyperkalemia, with tall peaked T waves, prominent U waves (found after T waves)
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This syndrome can occur during or right after hemodialysis treatments due to decrease in blood urea which can cause cerebral edema and increased ICP
disequilibrium syndrome
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this type of dialysis is conducted by inserting tube into abdominal cavity. uses peritoneal membrane to exchange fluids and remove toxins/waste products
peritoneal dialysis
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unilateral severe flanks pain, may have flanks to groin pain, hematuria, pale, sweating, renal colic waves of pain due to peristalsis (contraction and relaxation) of ureter.
Kidney Stone
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back or flank pain, hematuria, cloudy and foul smelling urine, high fever
pyelonephritis
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lower mid abdominal pain/tenderness, frequent urination with burning, hematuria, cloudy, foul smelling urine
Cystitis
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pain in groin, foul smelling urine
epididymitis
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Located in the lower part of the brain above the brainstem, stimulates the pituitary gland, plays a role in sleep, temperature, hunger, thirst, blood pressure and water balance
Hypothalamus
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Located in brain, called the master gland
Pituitary gland
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``` ACTH ADH FSH GH Oxytocin TSH ```
Adrenocorticotropic hormone Effects release of glucocorticoids found in adrenal glands Anti-diuretic hormone made in hypothalamus and stored inPituitary Follicle-stimulating hormone development of eggs and sperm Growth hormone growth of muscle and bones Oxytocin made and hypothalamus and stored in pituitary Thyroid stimulating hormone affects release of thyroid hormones
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Thyroid condition assessment findings: agitation and nervousness, heat intolerance, weight loss, tachycardia, hot and flushed skin, goiter (which is?), exophthalmos (which is?)
Graves’ disease Hashimoto Protrusion of eyeballs
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Thyroid condition: acute fever, sweating, diarrhea, altered mental status, hypoglycemia, seizures
Thyrotoxicosis, thyroid storm Management includes cool down, beta blocker, glucose is indicated, benzo’s for seizures
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Thyroid issue: assessment findings fatigue and lethargy, cold intolerance, bradycardia, weight gain, dry, cold skin, constipated
Myxedema: management includes common thyroid med Synthroid/levothyroxine
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Thyroid issue, assessment findings altered middle status, coma, hypothermic Slow to develop but can decline rapidly, usually triggered by sepsis, surgery, trauma
Myxedema coma Management includes active rewarming, ALS procedures in aCLS if need
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This is located in the chest between a start them in heart, produces T lymphocytes to help fight infection; not present in adults, atrophies during adolescence
Thymus
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These are located on the top of each kidney, produce epinephrine and norepinephrine, Aldosterone (which raises reabsorption of sodium/cortisol which raises energy)
Adrenal glands
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Adrenal insufficiency’s; signs and symptoms include fatigue, loss of appetite, vomiting, diarrhea, weight loss
Addison’s disease
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Adrenal insufficiency: signs and symptoms include severe vomiting and diarrhea, hypotension and even shock may be brought on by stress including trauma Presents withLow blood sugar and low blood pressure
Addisonian crisis
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This diabetic disorder has signs and symptoms of DKA, but no fruity order/breath or Kuszmaul respirations, typical and type two diabetics
Hyperosmolar non ketotic coma | Or HHNK,HONK
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Demonology of an allergic reaction
Immuno globin/IGE Mast cells stimulate each one histamine receptors, basil dilates, increases capillary permeability, Bronchoconstriction and laryngospasm Wheezing urtiCaria itching tongue and lips and Eyes swollen
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HIV complication type of cancer with blue/red colored lesions, Pneumocystis pneumonia, TB.
Kaposis Sarcoma
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Slow onset, fever, right upper quadrant pain, nausea and vomiting, loss of appetite, jaundice, dark colored urine, light colored stools, whites of eyes are yellow
Hepatitis viral infection in the liver
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Shortness of breath, night sweats, cough, weight loss, fever, hemoptysis, Chills… Spreads by droplet must wear HEPA or N 95 mask
TB
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Drug abuse: assessment findings respiratory in central nervous system depression, decreased respirations and level of consciousness, small pupils
Possibly narcotic, opium heroin, fennel, morphine, codeine management includes protecting the airway suction as needed, BVM if respirations are too low, Narcan
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Drug abuse assessment findings: dilated pupils, increased heart rate and respiratory rate and blood pressure, agitated/restless, seizures, V fib
Stimulants Dash cocaine, methamphetamine… Management includes seizure treatment as needed, benzodiazepine, EKG, IV
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Assessment findings: salivation lacrimation, urination, diarrhea, G.I. symptoms, emesis, constricted pupils open quotation (meiosis), Bradycardia
Organophosphate poisoning treat was heavy doses of atropine
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Assessment findings metabolic acidosis, respiratory alkalosis, hyper ventilation, tachycardia, fever, sweating, ringing in the ears (tinnitus)
Salicylate - aspirin | Acetylsalicylic acid
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assessment findings dry mouth, blurred vision, hypothermia, alter mental status, seizure, EKG changes due to blockage of sodium channels – wife QRS, heart block, torsades, VTAC
Try cyclic antidepressant overdose Treat with sodium bicarb
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Treatment for a cyanide overdose/exposure
Amyl nitrate
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Treatment for extrapyramidal symptoms (dystonic) From psych drugs such as Haldol Thorazine Treat with Benadryl
Prolixin
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Drug use for a beta blocker overdose
Glucagon
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Phases of the female menstrual cycle MFOL
menstruation, follicular phase, ovulation luteal phase. Menstruation - bleeding Follicle - eggs formed; one matures Ovulation - release of a mature egg from the surface of the ovary Luteal - progesterone, along with small amounts of oestrogen. This combination of hormones maintains the thickened lining of the uterus, waiting for a fertilised egg to stick (implant) Nothing happens, hormones disappear and lining thins.. cycle repeats
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Pain in both lower quadrants, fever, foul smelling discharge, fever Most common causes gonorrhea/chlamydia
Pelvic inflammatory disease
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Early pregnancy emergencies comma:Signs and symptoms include bilateral lower of Domino pain, bleeding, passing clots a tissue, cramping
Abortion
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Early pregnancy emergencies up 20 weeks: signs and symptoms of cool unilateral lower of Domino pain, minimal bleeding, curse sign (referred pain to shoulder), syncope, shock if Russia
Ectopic pregnancy
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Late pregnancy emergencies: silence and include dark red or no visible leading, tearing up Domino pain, shock. Can be trauma induced N deceleration injury
Abruptio placenta en plus into tears away from the wall the uterus. Treat for shock fluids to keep systolic at least 100 MMHG, transport on the left side only Abrupto placenta may also occur due to trauma moi usually fall a car crash
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Late pregnancy emergencies: sign the sellers include bright red vagina bleeding, but no pain
Placenta previa - placenta implanted partially or completely over cervical os
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Late stage pregnancy emergency: in labor, painful contractions and then pain-free, labor stops, peace and
Ruptured uterus
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Stages of labor
First stage begins with contractions ends with complete Dilation Second stage begins with the delivery of the baby ends with expulsion of baby Third stage begins with the explosion of baby ends with delivery of placenta
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Indication of imminent delivery
Contractions less than two minutes apart lasting 60 to 90 seconds
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Apgar
Appearance presentation grimace activity respirations. Should be checked at the one in five-minute mark
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Pediatric Triangle W-hat? O-h? BICH DRUGS
``` Warm, Position, Dry, Clear Airway O2 BVM INTUBATE Chess Compressions Medications ```
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What do we use to combat bradycardia during signs of hypoxia in newborn
Oxygen not atropine
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Compensatory stage of shock
Perfusion: Able to maintain perfusion HEART RATE: one of the first signs is tachycardia BP: blood pressure is normal for patient S/S: anxiety/restlessness, thirst, weakness, dilated pupils, nausea vomiting, tachypnea, pale and clammy skin
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Decompensated stage of shock
Low blood pressure (late sign), decreased LOC, unable to feel peripheral pulses, modeled skin, mean arterial pressure less than 65 MMHG
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Blood loss/hemorrhage classes one through four
Class one-up to 15%/750 Class two-up to 30%/1500 Class three–30 to 40%/1500-2000 Class four greater than 40% usually irreversible
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Treatment for have a hypovolemic shock
NPO, airway add junk/control as needed, 02, NRM or BBM, control bleeding, flat position, keep warm, isotonic Crystalloid solutions, adult 20 ML/KG, rapid transport
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Myoglobin released into bloodstream due to muscle break now and burns in crush injuries, can cause acute kidney failure
Rhabdomyolsis
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Parkland formula
Four times BSA percentage times body weight This gives you the amount to be given over the 24 hours you divide this In half to determine was to be given within the first eight hours
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Why do we Reverse triage if the mask has to be involved in lightning strike
Lightning strikes often send people into lethal dysrhythmia… A patient who is in a lethal dysrhythmia can we quickly right back these patients would normally be tags black
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Head injury or trauma which presents with bleeding between the durra in the school, patient can be unconscious and conscious and then unconscious again
Suspect epidural hematoma
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What is the difference between a cerebral concussion and a cerebral contusion
Cerebral concussion occurs in the brain stem includes loss of consciousness then wakes up, no permanent damage, may have minor confusion headache nausea Cerebral contusion occurs in cerebrum, structural damage to brain tissue, patient is awake with vocal signs such a difficulty talking diplopia personality changes
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Cushing’s response
Involved in increased ICP patient will present with elevated blood pressure decreased falls and irregular respirations. Early signs include vomiting without nausea, headache and altered mental status. Low sets due to vasodilation
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During hypovolemic shock heart rate does what BP does what respirations to work
Heart rate increases BP eventually decreases respirations increase
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During neurogenic shock heart rate does what BP does work respirations do what
Neurogenic shock presents with decreased heart rate or normal, decreased blood pressure, and the respiration aren’t affected
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During moments of increased intracranial pressure heart rate does what BP does what respiration’s present how?
I see people presents with decreased heart rate, Increased blood pressure, irregular fast or slow respirations
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C1-C5 are responsible for what? | Injury to this area can present with what?
Responsible for control of diaphragm, injury to this area can cause respiratory paralysis
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C-6 and C-seven can yield what sign and symptom when injured
Quadruple easier but can breathe, diaphragm working but may have difficulty breathing
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If patient assistance injury to chest BST and will airbag deceleration complains of upper anterior chest pain, tearing pain, or pain between the shoulder blades, and has an equal radial pulses, suspect what?
Ligamentum arteriosum
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Fuck difference between simple pneumo, tension pneumo-and hemothorax?
Tension pneumo-presents with JVD tracheal deviation away from tension side, every breath patient takes gets worse decrease in blood pressure. Simple Neumos may be spontaneous usually presents and tall young males presents with chest pain and coughing no JVD Hemothorax flatJugular veins May go into hypovolemic shock
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Results as patient encounters a direct blow to chair such as the steering wheel or airbag canpresent With Bex triad open parentheses define)Also define pulses paradoxus
Cardiac Tamponade Compression of the heart, can’t refill properly, decreased cardiac output. presents with my full heart sounds JVD due to increased venous pressure and decreased blood pressure due to decreased in arterial pressure Pulses paradoxes presents with a decrease in systolic BP during inspiration, narrow path pressure
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Disruption of rhythm of heart due to direct blow to the chest and children and teenagers because Chest is still pliable. CPR and defibrillation may be necessary
Commotion cordis
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Cullen Signs
Presents and patience with abdominal trauma signs and symptoms include. Umbilical bruising and pain, gardening, distention, rigid abdomen most likely due to blood pooling from Solid organs
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Gray Turner
Kidney injury hematoma in the flank may also present with hematuria, could be pancreas too
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Pregnant female may suffer up to how much blood loss before hypotension sets in
30 to 35%
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What will you notice in a hypo dermic patient on their EKG and where were you notice it
Osborne Wave, V2 through V5
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Dangers of fresh water drowning
Hemodilution, electrolyte imbalance, washed away surfactant which leads to atelectasis -collapse of alveoli
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Dangers of saltwater drowning
Pulmonary edema, salt draws water into alveolar
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Shark fin on capno | Reverse shark fin on capno
Shark fin - asthma | Reverse - emphysema
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What marking on ET tube does intubated male and female PT at Lip read?
23cm for male | 22cm for female
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Thyroid, CTM, cricoid cartilage
CTM location for thyrotomy
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Adequate respiration rates Adult Child Infant
Adult 12-20 Child 15-30 Infant 25-50
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Equation for propose Et tube
Age plus 16 then divide by 4
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Sudden onset, pleuristic chest pn, more on Inspiration, JVD, may be cyanotic from nipple line down, shock, resp distress Most commonly caused by DVT
Pulmonary Embolism
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``` Slow onset (several nights in a row) Viral, not high fever, “seal” like barking cough ```
Croup
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Lower airway wheezes | Whooping cough
Pertussis
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Genetic ailment, thick mucus which can cause life threatening infection
Cystic fibrosis
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Layers of the heart
Endometrium - lines inside of the heart Myocardium muscle layer-does the world Epicardium-outer most layer protects muscle from friction during beats Pericardium - fibrous sac that protects heart from structures in chest
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LAD sever which walls of heart? Circumflex Then RAD
LAD SEPTAL, ANTERIOR Circ LATERAL RAD INFERIOR
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cardiac issue | SOB, crackles, orthopnea or paroxysmal nocturnal Dyspnea
Left Ventricular Heart failure Management 02, lasix, nitro, morphine, CPAP, suction if needed, advanced airway if needed
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Cardiac issue No trauma JVD, pedal or sacral edema Hx of high blood pressure
Right side heart failure | Or cor pulmonale
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Excessive fluid inside pericardial sac (due to infection), diffuse st elevation, down sloping PR, sharp substernal chest pn, may radiate to neck, arms or jaw, more intense WHEN LAYING Down Low grade fever, swelling of legs and feet.
Non traumatic cardiac tamponade
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Types of Responses ``` Facilitation Reflection Clarification Silence Empathy Confrontation Interpretation Summary ```
...
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Landing zone by day and night
By day, Varies | By night 100x100
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Cheyenne stokes Biots Apneustic
Shane Stokes presents with periodic breathing with cycles of increasing rate and depth of breathing followed by gradual decrease in depth and rate periods of apnea that can last up to 60 seconds Biot’s presents with in the regular rate rhythm and depth with intermittent apnea ataxic respirations Apneustic respirations are an ominous sign of a brainstem injury, prolong inhalation fall of a short and ineffective exhalation
299
disease is a common inflammatory bowel disorder characterized by occult bleeding and frequent intestinal obstructions.
Chrons disease
300
This period is the time between infection and the ability of the infection to be transmitted to another.
Latent period
301
This period is the length of time the pathogen can be spread from one person to another.
communicable period
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clinical presentation (anxious, narrow pulse pressure, tachycardia, etc), this patient displays findings consistent with what percentage of blood loss. How much blood is in the body typical adult
15-30 percent 4500-5500 mL
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Heart Sounds S1 S2 S3 gallop S-gallop before S-1
S1 (closure of cuspid valves) S2 (closure of semilunar valves) S3 deceleration of blood flow from LA to LV, CHF S-gallop: LV failure; aortic stenosis
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U waves present when? | Delta waves present when?
Hypokalemia | Wolf Parkinson’s
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Pediatric Joule recommendation for cardioversion
2joules/kg then 4joules/kg Max is 10ug/kg
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Cardioversion Vfib/Vtach without pulse Afib-cardioversion A flutter and SVT VTACH WITH PULSE
120-200 biphasic 120-200 50-100 100
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Which heart failure categorized as SOB, crackles, S-3 gallop, blood-tinged white foam from mouth/nose, orthopnea Pulmonary edema
LV failure
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Right sided heart failure
JVD, pedal edema Enlarged liver Loss of appetite
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Sharp substernal or left precordial chest pain, more intense when lying flat, may radiate to neck arms or jaw
Non traumatic cardiac tamponade/pericarditis Excessive fluid inside pericardial sac
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Heart defect where blood passes directly from LA to RA | May be asymptomatic
Foramen Ovale | Atrial Septal Defect
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Heart defect | Narrow Aorta, reduces flow of blood to body; hypertension
Coarctation of Aorta (COA)
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Heart defect where blood flows continuously from sorts through ductus into pulmonary artery, shunts blood away from lungs
Patent ductus arteriosus | Ductus fails to close at birth
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Heart defect Narrowing of pulmonary valve, increased right ventricular pressure, right vent hyper trophy decreased blood flow to Lungs May present with JVD, cyanosis especially when feeding
Pulmonary stenosis
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Pulmonary stenosis, right ventricular hypertrophy, BSD, overriding aorta – connected to left and right ventricle instead of just left, a order will receive some blood with low oxygen from right ventricle Patient is Cyanotic all the time but worse room crying, feeding, or active
Tetralogy of Fallot
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Pulmonary artery connected to the left ventricle and a order of connected to right ventricle, blood from body goes back to body without being oxygenated, blood from the lungs goes back to Long’s without going to body Patient presents with shortness of breath, clubbing of fingers, cyanosis
Transposition of the great vessels TGA
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Heart defect where there is no tricuspid valve, small or no right ventricle
Tricuspid atresia
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Heart defect with blood passes directly from left ventricle into right ventricle Patient may be in asymptomatic, may called pulmonary hypertension or heart failure, low blood pressure
Ventricular septal defect VSD
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SIDS happens between these months
4-6months
319
Drug known to cause vasoconstriction and may help in shock states. It has been approved for massive bleeding when few choices exist for management peptide hormone formed in the hypothalamus
Vasopressin
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Calcium channel blockers are those that end in? Beta blockers end in? Nitrates end in?
Pine Olol Trate
321
hypoglycemic pediatric patient should be treated with what? | And what dose?
25% dextrose | 2-4ml/kg
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refers to a small margin between an effective dose and a harmful dose.
Narrow therapeutic index
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Drug most effective in acute congestive heart failure (CHF) because it helps to excrete the excess fluid
Furosemide
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Size needle most appropriate for IM injections
For most IM injections on nonobese patients, a | 21-gauge 1 to 1 1/2-inch needle is appropriate.
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DOPE Mnemonic for intubated peds
Displacement Obstruction Pneumo Equipment