Npte Study Flashcards
Cerebrum is derived from what?
Prosencephlon or forebrain
What does grey matter do?
Cortex responsible got process and cognition
What does frontal lobe do?
Higher intellect, personality, mood, social conduct,and language
Parietal lobe
Language, calculation and visuals Spatial 2 pt discrimination p
Temporal lobe
Memory and language and primary auditory
This disease affected the descending motor tracts within the cerebral motor cortex,. Symptoms include: hypertonicity, hyperreflexia, and abnormal reflexes. Damaged tracts are in the lateral white column.
Upper motor neuron
Examples of UMN
CP, ALS, CVA, birth injuries, hydropchephalus, Huntington’s, MS, TBI, brain tumors, pseudobulbar palsy
These diseases occur when the nerves or axons below the level of the brain stem are affected. The ventral gray column of SC may also be affected. Flaccidity, or weakness, decreased tone, fasiculations, muscle atrophy or absent reflexes.
LMN
Examples of LMN
ALS, GB, tumors of SC, trauma, poliomyelitis, infection, Bell’s, progressive muscle atrophy, carpal tunnel, MD, spinal muscular atrophy
Damage to Anterior horn cell causes what? What diseases?
Sensory intact, motor weakness and atrophy, fasiculations, decreased reflexes
ALS, poliomyelitis (LMN)
Muscle impacted with UMN vs LMN
Sensory intact, motor weakness, no fasiculations, normal or decreased DTR
MD (LMN)
NMJ affects in LMN vs UMN
Sensory intact, motor fatigue, normal DTR
MG LMN
Affects of peripheral nerve or mononeuropathy LMN vs UMN
Sensory loss along nerve root, motor weakness and atrophy may have fasiculations
Trauma (LMN)
Peripheral polyneuropathy LMN vs UMN
Sensory impairments stocking glove, motor weakness and atrophy, dista to proximal, decreased DTR
Diabetic peripheral neuropathy
Spinal roots and nerve LMN vs UMN
Sensory will have corresponding dermatomal pattern, motor weakness via innervation, decreased DTR
Herniated disc
Athetosis
Slow, twisting writhin movements large amplitude
Seen in face tongue and trunk and extremities
Typically associated with spasticity (CP and BG pathology)
Chorea
Hyperkinesia, brief irregular contractions, rapid
Damage to caudate nucleus, fidgeting,
Ballism(form) large amp jerks-damage to subthalamic nucleus
Huntington’s
Dystonia
Sustained muscle contractions, twisting and abnormal postures, or repetitive movements
All muscles can be affected during volitional movement
Genetic, acquired, SE meds,
Present sustained of agonist/antagonist,
Parkinsons CP and encephalitis
Tics
Sudden brief repetitive coordinated movements at irregular intervals
Vocal, jerks or repetitive sounds
Tourette’s
Tremors
Involuntary rhythmic oscillatory movement 3 groups
Resting: pill rolling Parkinson’s
Postural: rapid tremor in hyperthyroidism, fatigue or anxiety and benign tumor
Intention: increase as target approaches, cerebellum efferent pathway MS
Inability to initiate a movement
Akinesia
General weakness cerebellum pathology
Asthenia