Notes Flashcards
Congenital/dysplasia atlantoaxial instability (5)
Occipitalization Odontoid agenesis Os odontoideum Down's Marfan's
Trauma atlantoaxial instability (2)
Type 2 dens fx
Transverse ligament rupture
Arthritis causing atlantoaxial instability (6)
RA AS PSA Reiter's SLE Gout
Infection causing atlantoaxial instability
Osteomyelitis
Postpharyngeal infection
Air is ___ on an xray
Black
Fat = dark gray
Epiphyseal tumors (3)
GCT
ABC
Chondroblastoma
Dipahyseal tumors (3)
Ewings
Nonhodgkins lymphoma
MM
Meatphyseal tumors
Everything else besides GCT/ABC/chondroblastoma (epiphyseal); ewings/nhl/MM (diaphyseal)
Age group of GCT
20-40
Prepubertal
ABC
Age group of ewings
10-25
Cortex
Osteoosteoma
MC arthritis of wrist
OA
RA
CPPD
Congenital anomalies rule 1
Congenital anomalies in the spine occur MC at transitional areas. Occipital cervical (occipitalization) Cervical thoracic (extra rib) Thoraco lumbar (diastometayelia) Lumbosacral
Congenital anomalies 2
Skeletal/spinal anomalies may be isolated entities but are frequentaly associated with other skeletal/spinal anomalies
Therefore look for other osseous abnormalities on the radiograph
Congenital anomalies rule 3
VATER SYNDROME = Vertebral.vascular anomalies Anal atresia Tracheo-esophageal fistual Esophageal atresia Renal and radial dysplasia
Dark CSF
T1
Only reason you’ll have a C2 megaspinous
Posterior arch at C1
Anterior arch sclerosis
Spina bifida
Os odontoideum
Posterior arch agenesis
Arcuate foramen
Posterior ponticle (15% of population)
Unfused secondary growth center after age 12
Os terminale
Limbus bones often occur with
Large schmorles nodes
Congenital blocks MC at
C2-3 and C5-6
Wasp waist
Vestigial disc
Posterior elements
Congenital block
Multiple congenital blocks
Klippel file
Signs of klippel file (5)
Low hairline Short neck Decreased aROM (d/t block vertebrae) GU anomalies (kidney malformation: horseshoe) thus do IVP Occipitalization
Types of spondy
- Congenital
- Isthumus (MC L5) - stress, elongated pars, acute trauma
- Degenerative
- Traumatic (Bi pedicle fx) C2 MC (hangman)
- Pathological
- Iatrogenic
2 MC types of spondy
2 and 3
Isthumus and degenerative
Spondylolysis
Pars interarticularis defect with or without slippage (spondylolisthesis)
90% of all spondies involve what segment
L5
13x increased incidence of what with spondy spondy
SBO
Asymmetric facets.
Only 2-3% patients with spondy over 25 show progressive dipalcement
Clinical features of spondylolisthesis
Pain maybe
Pain is usually present at onset of lysis of pars interarticularis
Hyperlordosis and gait changes may be seen
Only neuro sign = referred pain to lower extremity
Palpable “step-off” defect
Management for spondylolisthesis (for type 2-3)
Conservative care initially
Asymptomatic - no contraindications for sport etc
Spondy spondy at fresh stress fx stage may be treated with boston overlap brace (antilordotic)
Progression of spondy between
5-15 years of age
Spondy’s mc in what gender
2x mc with females
Displaced risk increased with associated what
SBO
Trapezoidal shaped L5 and dome shaped SI may lead to
Increased slippage
SI may what anteriorly to stabilize L5?
Buttress
Dysplastic/congenital features
L5 facets/posterior arch/upper sacral malformation
Often accompanying SBO
Type 2 mc at what level
L5
Young active individuals (extension activities)
3 subtypes. C = rare; b = giraffe neck
Etiology of isthmic
Repetitive microtrauma Acute fx Not recorded in infants Congenitally slender pars = predisposes Hyperlordosis. Nonunion d/t lack of immobilization (not diagnosed fast enough)
Inverted napoleon’s hat sign or bowline of brailsford
Seen with significant anterolisthesis (seen in type 3 and above)
Napoleon’s hat sign
spondy
Achondroplasia
This is recommended for patients exhibiting clinical signs of a stress fx but have negative plain films
SPECT bone scan
Imaging if pain at 6months and follwed by what
X ray
CT (spect
What imaging if trauma?
CT.
Can find out if new or old. Assessing spinal canal. Healing