Notes Flashcards
What is the first line antibiotic for dental abscesses?
According to SDCEP drug prescribing guidelines it is a 5 day regimen of either:
Phenoxymethypenicillin tablets, 250mg
Send: 40 tablets
Label: 2 tablets four times daily for five days
Amoxicillin 500mg
Send: 15 tablets
Label: 1 tablet 3 times daily for five days
If pt is allergic to penicillin - metronidazole (avoid w alcohol)
What antibiotics would you provide for a patient with spreading infection from a dental abscess and an allergy to penicillin?
SDCEP - June 21 update
Metronidazole - 400mg
Send: 15 tablets
Label: 1 tablet three times daily
What antibiotics would you provide for a patient with spreading infection from a dental abscess who has an allergy to penicillin and is on warfarin?
Clindamycin capsules, 150mg
Send: 20 capsules
Label: 1 capsule four times daily swallowed with water for five days
During an IV sedation assessment, what important things should you explain to the patient after introducing yourself.
Take hx - establish nature of fear/phobia/anxiety (cant do IV sedation for people with needle phobia)
Explain to pt:
- Need an escort
- No alcohol before
- No responsibilities or work the following day
- No driving for 12 hours
What medications would you be looking for in a MH during a sedation assessment?
Drug interactions - alcohol, opioids, erythromycin, antidepressants, antipsychotics, antihistamines, recreational drugs
What are the ASA classifications?
ASA Class I - Normal healthy pts
ASA Class II - Mild systemic disease, BP <160/95
ASA Class III - Severe systemic disease
ASA Class IV - Incapacitating disease which is a constant threat to life
ASA Class V - Moribund pt not expected to live >24h
What are the indications for IV sedation?
MH aggravated by stress - ischaemic heart disease, hypertension, asthma, IBS, epilepsy
Handicap/parkinsons/learning difficulties
Phobia/gagging/fainting
Especially long/unpleasant procedure
What are contraindications for IV sedation?
COPD
Hepatic insufficiency (midazolam is metabolised in the liver)
Pregnancy
Severe special needs
What drugs are used in IV sedation?
Midazolam - 5mg/ml
Reversal agent:
Flumazenil - 200 microgram
What are the indications for inhalation sedation?
Anxiety
Needle fear
Gagging
Traumatic tx
MH that increases stress
Unaccompanied adults needing sedation
What are contra-indications for inhalation sedation?
Common cold
Enlarged tonsils/adenoids
Severe COPD
1st Trimester pregnancy
Limited understanding
What are the pre-op instructions for IS?
Light meal pre-appt
Routine medication should be taken
Children accompanied by adult
Adults need accompanied at 1st appt only
No alcohol
Sensible clothing
Arrange childcare post appt
Plan to remain in clinic 30mins post appt
What categories are involved in a caries risk assessment in paediatric dentistry?
- MH
- SH
- Fluoride use
- Saliva
- Diet
- Clinical evidence
- Plaque control
What are some reasons for delayed eruption of a permanent tooth?
Abnormal development position
Supernumerary tooth
Displacement of permanent teeth due to trauma to primary tooth causing ankylosis
Dilaceration
Impaction
Eruption cysts
Early loss of primary teeth
What are signs of a supernumerary tooth?
Delayed eruption
Midline discrepancy
Midline diastema
Crowding of permanent teeth
Displacement of permanent teeth
Rotation of permanent teeth
What OHI advice would you give to a parent?
Standard prevention, 1x yearly
Brush as soon as 1st primary tooth erupts
x2 daily, 2 minutes, morning + last thing before bed - no food or drink (except water) afterwards
Spit out toothpaste, don’t rinse - removes action of fluoride
Assist child until 7 years old, supervise after until child is confident
Use correct amount of fluoride for childs age:
<3 - smear, >3 - pea
Age <3
LR = 1000ppm
HR = 1350-1500ppm
Age 3-9
LR = 1350-1500ppm
HR = 1350-1500ppm
Age >10
LR = 1350-1500ppm
HR - 2800ppm (>16 - 5000ppm)
If a child is at high risk of caries what actions can you take?
Enhanced fluoride toothpaste
- >3 - pea 1450ppm
- >10 - Rx duraphat 2800ppm
Standard prevention advice every recall (3 months)
3 min hands on TBI - annual
Plaque disclosing tablets
TB charts
Free TB/Toothpaste
Floss DE6’s x2 weekly
What is considered an overdose of fluoride when ingested? What is management?
It depends on how much fluoride was ingested and the weight of the child
(average tube toothpaste - 90g)
5mg/kg - Give milk orally and observe for a few hours
5-15mg/kg - Give milk, calcium lactate/calcium gluconate and admit to hospital
> 15mg/kg - Give calcium gluconate, urgent referral to hospital for cardiac monitoring and IV calcium gluconate
What diet advice would you give to a parent?
Standard prevention - 1x yearly
- Sugar - reduce frequency to no more than 4x daily
- Hidden sugars - fruit juice, sweetened milk, soya milk
- Acid - limit fizzy drinks to meal times
- Sugar free snacks - cheese, breadsticks , carrots
- Water only between meals and in bedtime bottle
Enhanced:
- standard prevention at 3 month recall
- Diet diary - 2 days during week and 1 weekend, add timings of meals/snacks and accurate measurements
- Action planning
What are the elements of standard prevention in paeds?
OHI
Diet advice/diary
Fluoride varnish
Fluoride toothpaste
Fluoride supplements
Fissure sealants
Radiographs
Sugar-free medicine
How can fluoride be used in prevention for paediatrics?
Standard prevention:
>2y - NaF varnish (22600ppm) 2x yearly
2-5 - 0.25ml
>6 - 0.4ml
Enhanced - Additional 2x yearly so 4 in total unless they have received care from childsmile
0.5ml of duraphat contains 11.3mg of fluoride/0.25ml of duraphat contains 5.65mg of fluoride
· Safely tolerated dose is 1mg per kg, average 3 year old weighs 15-20kg
Fluoride Varnish Contraindications
· Allergies- colophony (Elastoplast)
· Ulcerative gingivitis
· Severe asthma (hospitalised or oral steroids)
· severe allergie
FV aftercare advice:
· Avoid eating or drinking for at least an hour
· Eat soft food all day
· Avoid brushing that night only - brush as normal from tomorrow morning (high risk as normal)
· avoid fluoride supplements for 2 days
> 7 - NaF mouthwash
When would you take radiographs for kids?
BW’s from age 4
standard - every 2 years
high risk - every 6-12 months
When would you use fissure sealants?
Place in all pits+fissures of permanent teeth if child is high risk from CRA
Standard - L6 buccal pits, U6 palatal pits
Pre-cooperative child - GI as temp sealant on PE 6/7s
Check existing fissures - may need to be topped up
What are the 5 options for caries management in primary dentition?
- Complete caries removal + rest
- Partial caries removal + rest
- No caries removal, seal
- No caries removal, prevention, make self cleansing
- XLA
What are the 9 components of the index of suspicion in paediatrics?
- Could the injury have been caused accidentally, if so - how?
- Does explanation of injury fit age and clinical findings?
- Is explanation consistent with injury, is this within normally acceptable limits of behaviour?
- If there is delay in seeking advice, are there good reasons for this?
- General demeanour
- Nature of the relationship between guardian and child
- Childs reaction to people
- Childs reaction to any medical/dental examination
- Comments by child/guardian that give concern about the upbringing or lifetstyle
What are 10 neglect markers in paediatrics?
Nutrition
Warmth, clothing, shelter
Hygiene
Stimulation and education
Affection
Failure to thrive
Cold injury/sun burn
Dental caries/head lice
Developmental delay
Withdrawn/attention seeking behaviour
Write a prescription for a conventional fixed-fixed bridge replacing tooth 25.
- Pts name, address
- Date sent, date required, disinfected
- Please pour up impressions in improved dental stone
- Please mount casts using facebow and wax bite registration provided to mount on a Denar type II semi-adjustable articulator
- Please construct conventional (fixed-fixed) bridge with teeth 24+26 as retainers
- Please provide tooth 25 pontic (modified ridge lap)
- Teeth 24+26 MCC with buccal shoulder, palatal chamfer
- Please construct in canine guidance and ensure pontic is free of excursive movements
- Shade A3
Mr R Jalif
Signature
Address of practice
Phone no.
You are conducting an intra-oral examination and you spot a lesion on the RHS buccal mucosa, how would you describe this?
Site
Size and Shape
Surface
Surrounding tissue
Colour
Consistency
Base
Bleeding
Functional limitation
What is Lichen Planus and what are the 7 types?
Lichen planus is a chronic autoimmune disease that can affect the skin and any lining mucosa. This could be the oral, oesophageal, vaginal mucosa as well as the skin.
Reticular
Papular
Plaque-like
Atrophic
Erosive - 1-2% chance of becoming malignant
Bullous
Desquamative Gingivitis
Auto-immune, F>M, in most cases will resolve within 2y
- Causes Genetics/environment (amalgam, SLS, stress, drugs - B-blockers, NSAIDs, anti-malarials, oral hypoglycaemics)
Describe a lichen planus lesion for your notes.
Site - anywhere - commonly buccal mucosa or lateral tongue
Size - any size/shape - striae
Surface - roughness of musoca/blister/ulcer
Associated - usually bilateral, itchy skin lesion on arm, leg, trunk
Surrounding tissue
Colour - red/white/both
Consistency - homogenous/heterogenous
Base
Bleeding - no
Functional limitation
What else extraorally should you examine when looking for LP?
Most likely spotted oral lesion when doing soft tissue examination
Check flexor of wrist, ask about leg, trunk, genitals (more likely to turn malignant)
Is the lesion localised and associated with any restorations, if so likely to be lichenoid reaction to amalgam
What histopathological features are shown in LP?
Ortho or parakeratinized surface
Acanthotic or atrophic epithelium
Subepithelial band of T Lymphocytes
Liquefactive degeneration of basal cells
What special investigations are required for LP and what is the treatment?
Photograph
asymptomatic/
reticular/non-smoker - no biopsy, reassure that it is self-limiting, not to worry
If symptomatic or patient is worried -
- Avoid allergens like benzoates
- Avoid SLS containing toothpaste
- Avoid spicy food
- CHX 0.2% can be used - warn pt of risk of staining tongue and teeth - should not be used for longer than 2 weeks
- Benzydamine MW (difflam) can be used to help with soreness - 0.15% 300ml 15ml every 1.5h
In later stages medicines like corticosteroids can be used too (local - beclomethasone inhaler and systemic - betamethasone tablet)
BIOPSY IF PT SMOKER
to confirm it is LP and check for dysplasia
Explain what Lichen Planus is to a patient.
So on examination we can see you have these white patches around your mouth.
This is what we believe to be a condition called lichen planus (LP), which is a autoimmune chronic inflammation of the skin and in some cases the mouth - it is a very common condition seen in the oral medicine dept.
The whiteness arises from extra keratin in your soft tissues, keratin is a protein which is present all around your skin.
LP is kind of like an allergic reaction to something and in most cases we don’t really know what is causing it, the most common culprits are reactions to medications/amalgam fillings.
LP has a small chance to develop into something sinister like a mouth cancer but this is only in 1% of cases. LP is a spectrum disease and can be something as simple as a white patch to a more sinister erosive sore ulcerated area, depending on what area of the spectrum you’re on the risk of malignancy may be higher or lower.
This is not something we can treat other than managing the symptoms if we do not manage to remove the causative factor.
What are the cranial nerve tests?
CN1 (Olfactory) - can patient smell as normal?
CN 2, 3, 4, 6 (optical, oculomotor, trochlear, abducens) - test visual acuity and eye movement
CN 5 (Trigeminal) - Any abnormal sensations at each branch? Can pt clench jaw? Corneal reflex
CN 7 (facial) - facial muscles tests (puff out cheeks, pout, wrinkle forehead, raise eyebrows)
CN 8 (Vestibulocochlear) - can pt hear normally? Block one ear and check for differences
CN 9, 10 (glossopharyngeal and vagus) - deviation of uvula saying ahh, gag reflex
CN 11 (accessory) - can pt shrug their shoulders?
CN 12 (hypoglossal) - can patient protrude tongue? deviation on protrusion? asymmetry?
What are the tx options for a 20y/o male with a class III malocclusion?
Accept and monitor
Intercept with a URA - procline uppers (notice pts age in scenario - this might not be possible)
Growth modification - with functional appliance (reverse twin block) or (RME + protraction headgear) - notice pts age in scenario - this might not be possible
Camouflage with fixed appliances - accept underlying skeletal A/P discrepancy, move teeth with fixed ortho to hide it
Procline uppers and retrocline lowers
Risks of ortho - decal, root resorption, relapse, gingival recession
This sometimes requires XLA of U5’s and L4’s
Orthognathic surgery with combined orthodontics - to be done once the patients jaws have stopped growing -
surgical manipulation of the mandible and/or maxilla to produce optimal aesthetics/function
total time = 36 months
How would you explain denture induced stomatitis to a patient?
Redness and swelling in denture bearing area caused by fungal microorganisms called candida. Common in poor oral hygiene, ill-fitting dentures, older dentures, poor diets, immunocompromised, repeated antibiotics
Some symptoms you may have experienced could be redness and swelling of the areas covered by the denture, burning sensation, inflammation of your mucosa, discomfort, bad taste and bad breath
What is newton’s classification?
Classification system for denture induced stomatitis
Type I - localised inflammation with hyperaemic foci
Type II - diffuse inflammation and erythema confined to mucosa contacting denture without hyperplasia
Type III - granular inflammation with erythema and papillary hyperplasia
what is the management of denture induced stomatitis?
- ask pt about care of denture - cleaning and removal
- Prevention - remove at night, steep denture in milton, brushing palate
- Advice on cleaning denture - brushing after meals w soft toothbrush and non-abrasive denture cream, soaking in CHX m/w or NaOCL (if acrylic) for 15mins 2x daily
- Leave dentures out as much as possible during tx
- Consider tissue conditioner for current denture
- Consider making new denture (ill-fitting)
- Consider underlying deficiency/disease - diabetes/haematinic deficiency
- More common in diabetic patients - encourage diabetic control
- If local measures fail - Fluconazole 50mg 1 x 7 days (send 7 capsules)
- Nystatin 100,000 units/ml - 1ml after food 4x daily for 7 days (send: 30ml)
NO AZOLES if WARFARIN/STATIN
Why and how would you carry out a reline for a complete denture?
Reasoning
- Relines - when fitting surface is inadequate but denture otherwise is okay - i.e. occlusal planes, OVD, profile are acceptable but fitting surface underextended, not supportive or retentive
- Rebase - when you want to keep the occlusal surface but change fitting and polished surface
Method:
1. check all occlusal relationships are acceptable and appropriate
2. remove undercuts from dentures fitting surface using acrylic bur
3. adjust border for under/over extension with green stick
4. apply adhesive to fitting surface of the denture to be refined
5. insert impression material (light body PVS) into the fitting surface and seat the denture
6. functional impression - ask the patient to bite together so the impression is taken in OVD
7. Take a lower impression with denture in-situ
8. Take a bite registration if ICP not obvious
9. When set remove the impression and send the denture to lab for reline
“Pls pour impressions in 100% dental stone using impressions provided. Please mount to cast and create a self cure PMMA reline to change the impression surface”
What hx would you take from a patient who brings her child to the clinic and they aren’t feeling well - pics provided of small vesicles in labial mucosa and tongue.
Introduce yourself
Take hx:
- No. of days symptoms
- Does the child have a fever?
- Child less active than normal?
- Analgesia used?
- Did it work?
- PHG Signs - lymphadenopathy, malaise, pyrexia, erythematous gingivae, ulceration
- PHG symptoms - sore mouth + throat, fever, enlarged lymph nodes, loss of appetite, dehydration
You have diagnosed a child with primary herpetic gingivostomatitis, explain this in laymans terms to the parent.
From my clinical findings and from what I can see, I believe that your child has a condition which we call primary herpetic gingivostomatitis.
It is a contagious infection which is caused by the herpes simplex virus, the same virus which causes cold sores.
It is self-limiting and will disappear within 10-14 days.
The blisters do not leave scars.
The virus may reactivate in the future and can cause cold sores.
Most infections are not clinical.
How do you treat PHG?
· Encourage rest
· Encourage fluid intake
· Pain relief- ibuprofen and paracetamol (helps control fever too)- ask if patient has tried this
· Clean teeth with damp cotton wool roll and CHX swab for gums
· No AB/AV as they are ineffective- unless severe or immunocompromised
- Acyclovir tablets 200mg/oral suspension 200mg per 5 ml, 5 times daily for 5 days (send 25 tablets)- if aged 2 to 17
- Half dose for 6 months to 1 year
What is the prescription for acyclovir for PHG?
If aged 2-17:
200mg/(oral suspension
200mg per 5ml), 5x daily for 5 days (send 25 tablets)
If 6m-1y:
Half dose
Why is SDF used?
To prevent decay from progressing - more effective than fluoride varnish
Reduces dentine hypersensitvity
Vaseline on ST, dry teeth, apply for 3 mins
What are the indications for SDF?
Non-restorable dentine lesions
MIH - reduces sensitivity
Pre-cooperative
Delay sedation/GA
Asymptomatic cavitated lesions
What are contraindications for SDF?
Allergy to silver or other heavy metals
Painful gums or mouth ulceration
Lesions that involve the pulp
PA pathology
Mucositis/Stomatitis
What are the disadvantages of SDF?
Permanent discolouration
Temporarily stains soft tissues for 1-3 weeks
Discolour composite fillings
Permanent discolouration of clothes
What are the steps if you give FV to an allergic child?
Brush it off with toothbrush
apologise to family - explain what has happened
Have emergency kit on standby
If reaction - send to a&e
If not - watch patient for a few hours in clinic, then send home with the instruction that if the child’s condition deteriorates go to A&E
Get pt in next day for follow up
What alternatives are there to SDF?
Monitoring
FV
Remove decay and restore
Hall technique
XLA
Break bad news of a SCC to a patient.
SPIKES acronym
Setting - sitting down at same lvl as pt, “how have you been since the last time i’ve seen you”
Perceptions - what does the pt understand has happened until now
“ Are you aware of what we’re here to discuss today”
Information - inform pt you have the results of the biopsy
Ask them if they would like you to go through with them - they will say yes
Knowledge - Give them a warning shot - “i wish i had better news”
Give them the knowledge of what you know - “the test we have done has shown some abnormalities of the cells”
“mrs smith im afraid to say that you have mouth cancer”
Let it sink in and let them dictate the pace of the convo
Empathy - words to the effect of
- “I am deeply sorry to break this to you”
- “I understand you must have lots and lots of questions … do you have anything that comes to mind?”
- “Perhaps you would like to bring your husband in with you?”
Summarise and close - Repeat the news
Summarise what you’ve told them and the plan for going forwards
“The good news in all of this is that we’ve and will be able to move forward with tx as soon as possible”
“I’ll be speaking to the surgeons today and they’ll be seeing you in the coming week to discuss tx”
- Offer them a follow-up appt or phone no. for any questions
- Give written material if available
What is the NHS complaints procedure?
- Acknowledge the complaint and provide the patient with the practice complaint procedure
- Inform the dental defence organisation
- Inform the patient of timescales and stages involved
- Acknowledge the complaint in writing, by email or by telephone as soon as you receive it - 3 working days maximum but ideally within 24 hours
- Early resolution 5 working days: For issues that are straightforward and easily resolved, requiring little or no investigation
- Investigation 20 working days: for issues that have not been resolved at the early resolution stage or that are complex, serious or “high risk”
- Independent external review ombudsman: For issues that have not been resolved
Patient was annoyed that they had to wait an hour and receptionist was rude. How would you deal with this situation?
- Take concerns seriously, answer questions as able:
“hello there, what seems to be the problem?”
“can i offer some assistance?” - Acknowledge anger “I can see that you’re upset and I am sorry you’re feeling this way” - this does not accept blame, DO NOT ACCEPT BLAME
- Try to offer practical help - offer investigation with receptionist and provide feedback to the patient
If you can offer another appointment - “do you still have time for us to see you?”
“What would you like us to do, we can work around you”
Making an apology
- Be honest
- Acknowledge the offence
- Explain how it happened - we were running over from previous procedure
- Express remorse - deep guilt, express it (I am so sorry!)
- Ensure amends - “is there anything we can do?”
If formal complaint requested, advice on NHS complaints procedure
- Then, if required: a local resolution (payout)
- If satisfactory: complaint closed
- If unsatisfactory - healthcare commission or health service ombudsmen
Medical emergency - explain hypoglycaemia to a nurse and management.
Type 1DM - normal - 5-7mmol, unconcious <3mmol
Assess ABCDE
Signs: Pale, shaky, sweaty, clammy, dizzy, hungry, confusion, blurred vision, loss of consciousness
You must mention loss of consciousness as it defines different tx
If conscious and co-operative - administer oral glucose 10-20g or sugary drink
If unconscious/uncooperative - 1mg IM glucagon injection and oral glucose when regain consciousness
How does glucagon work? - Increases conc. of glucose in the blood by promoting gluconeogenesis to convert glycogen to glucose
After they regain consciousness (15mins - if not 2nd dose) supply oral glucose/sugary drink
IM injection and technique:
- inject diluting solution in vial with glucagon powder
- swirl to mix - dont shake
- syringe solution back into syringe
- use z-track technique into thigh or bicep - spread skin, advance needle in skin 90 degrees, aspirate, inject 30s, pull out, release tension
- say “I would nnormally prepare needle/change needle, remove clothing, alcohol wipe skin, but not going to as life threatening and saves time”
- Reassess ABCDE - assess effect of medication, more oral glucose required?
What is the management of a epileptic fit?
Medication - midazolam - a short acting benzodiazepine - enhances the effect of GABA (neurotransmitter) on GABA receptors resulting in neural inhibition
Signs: loss of consciousness, uncontrollable spasms, drooling, tonic (falls rigid), clonic (sharp jerky movements), hypotension, hypoxia, loss of airway tone
Assess ABCDE
Secure airway
Administration
- Administer 100% O2, 15L/min flow rate
- If the fit is repeated or prolonged (>5min) give midazolam 2ml oromucosal solution, 5mg/ml topically into buccal cavity (10mg) - repeat after 5 mins if not worked
- If subsided, recover position and check airway
- Refer to hospital if - first seizure, seizure is atypical, injury caused or difficult to monitor pt
You overhear a nurse bad-mouthing a pt to a colleague in a public area, they refer to them in a derogatory manner and joke about posting this on social media. Pt and family are easily identifiable from info heard, discuss this issue with your nurse
Introduce yourself and ask the nurse if it is okay to talk = “do you have a minute to talk”
Facts - find out the facts of the situation
Ask the individual for their account of the situation
“So it has come to mt attention that unfortunately there were remarks said publicly about a patient and a talk about posting on social media, I was wondering if you knew anything about this?” - allow nurse to reply
“Is it okay if I hear your side of the story”
Issue - What is the issue here?
Explain the issue to the individual and why it is bad
Quote GDC standards - i.e. breach of confidentiality, brings profession into dispute
“I know it may have been misjudged, but unfortunately, it is not acceptable to say these things publicly about patients or to post on social media”
“As GDC standards state, it is our obligation to have patients best interest and to protect their information. Speaking in the public can breach this confidentiality. The pts are recognisable from the posts and this is not protecting them, for example, if it was someone speaking/posting about your family member, how would you feel?”
“It is also not providing the public with confidence in you, us and the profession as a whole. The practice could be in question and the GDC could be informed of this in the future”
Option:
If involves pt, what options are there to manage this event
What is in the pts best interest?
“There are a few options to rectify this”
- If there is a social media post - delete it and photos immediately
Apologise to the patient if still around - if not, the practice can contact pt to ideally attend for a formal apology
Now:
- What issues do you need to deal with right now?
Inform the nurse that this shouldn’t happen again
Ask/Advise:
- Ask the individual if they would be willing to undertake some training or education on this matter
“It would be acceptable if we had training on this in the future and have meetings about social media to increase awareness”
“Would you be willing to have training on this?”
If problem repeats - Get advice from someone more senior (defence union, VT trainer) how to manage this
Record: document conversation
A 27-year old teacher presents with a bunch of E/O and I/O signs of TMD. Click on both sides, sore
muscles, sore in morning, tongue scalloping and cheek biting (linea alba). Please discuss the diagnosis
with the patient, and conservative management for this condition. You do not need to obtain further
information from the patient.
Diagnosis - Mrs Smith, you have a very common condition, in fact around 75% of the population get it at one point in their life, it is called temporomandibular disorder also known as TMD or TMJ
Explanation
“The jaw joint sits in the base of skull and muscles control opening and closing. Now, like any other muscle in the body if they get overworked they can get tired and sore, for example if you climb a mountain you would expect your legs to be sore for a few days”
“However, as your jaw joint gets used all day everyday for speaking and eating it doesnt really get a rest, the muscles become inflamed and sore”
“The fact that you’re sore in the morning also tells us that you clench or grind your teeth at night as well which puts more stress on those muscles and exacerbates the problem even more”
“The clicking by your ear is caused by a small disc that sits between your jaw and skull not moving freely and it becomes trapped in front of the jaw bones and snaps in places”
Management -
Reassurance - “The way we manage this is very simple”
“It involves resting the joints”
- Soft foods/cut in small pieces
- Chewing on both sides
- Avoid chewing gum
- Avoid wide opeining
- Support jaw on yawning
- Avoid grinding during day
- Avoid bad habits e.g. biting nails
- Analgesia
- Heat packs/cold compresses
- Make soft splint to aid bruxism
Summary
- reassurance - common condition with simple conservative management
- further options - will review and if pain still evident may require referral with further options of botox and surgery
- inform that other symptoms like tongue scalloping and linea alba caused by the clenching and will go away with resoltuion of condition
- Any q’s?
A 28-year old female patient who works in television has had an accident in which she injured her face.
There are no other injuries and you have completed the examination as well as taken a radiograph. You
have diagnosed the tooth as having a vertical root fracture and is unrestorable.
Explain your findings to the patient and how you would treat them.
SPIKES approach - bad news
Setting
- Sit down at same lvl as pt
- Gain rapport
- Perception - what does the pt understand has happened until now?
“Are you aware of what might be wrong?” - what is pt expecting from appt? - Information
- Inform patient that you would like to discuss the prognosis of the tooth
- Ask them if they would like to discuss that
Knowledge
- Give them a warning shot
“I wish i had better news”
“I’m afraid the news is not good” - pause for a bit
- Give them the knowledge of what you know
“Your tooth is unrestorable and will be required to be extracted” - big pause
- Let it sink in and let them dictate the pace of the conversation from here - they may want to know a lot of info or they may be in shock
Empathy -
“I am deeply sorry to break this to you and I know this may have come as a shock”
Summary and close:
- Repeat the news - once again the tooth is unrestorable and will need XLA
“We will aim to have this booked in as soon as possible so we can continue with tx”
“In the meantime we would not leave you with an empty space so there are immediate options and then more permanent options around 3-6 months down the line when the bone around the socket has stabilised”
“The immediate options would include - immediate denture, a bridge using your extracted tooth, direct polycarbonate crown bridge”
“More permanent options would be a bridge or a denture, and privately you could consider implant placement”
“Any q’s?” - we will book you a follow-up appt or you can phone the practice if you have any questions
What are common denture faults?
Problems with denture
○ Impression surface:
■ Cause: poor impression (lack of post dam, poor adhesion to tray), damage to cast
■ Solutions: reline/rebase, remake, add post dam using reline
○ Occlusal surface:
■ Cause: premature occlusal contact, centric occlusion/relation not coincident, high lower
occlusal plane restricting the tongue, locked occlusion
■ Solutions: cuspless teeth, selective grinding, re-recording centric occlusion, remake
○ Polished surface:
■ Cause: Overextension, underextended (depth &/or width), not in neutral zone
■ Solutions: remove overextension (esp. lingual lower, use pressure indicating paste, allow
fraenal relief and flange), add greenstick to underextension and reline, remake if extensive
● Problems with denture wearer:
○ Poor neuromuscular control e.g. stroke, Parkinson’s
○ Unstable foundations
■ Anterior flabby ridge
● Solution: perforated trays + light bodied PVS impression (Or special tray with
surgical window and take a wash and cut it out + light bodied PVS)
■ Atrophic lower ridge:
● Solution: admix technique (3 parts imp compound, 7 greenstick)
■ High fraenal attachments
● Solution: provide relief
■ Palatine tori:
● Solution: relief of area on cast before processing
○ Xerostomia
C/C denture provided, please identify 6 faults with this denture and how to rectify.
Anterior flange missing - remove undercuts, build flange w greenstick and reline
Rebase if not possible or remake if necessary
Midline diastema - if want to keep physical aspects of denture, but change aesthetic onnly
- Replica (2 stage putty around denture, vaseline to separate)
- Wax replica used for functional impression + jaw registration
- Ask lab to close diastema for tooth trial stage
Remake if other problems
Underextended posteriorly at tuberosities -
Reline - if functionally good and only one problem
Remake - if everything bad
Locked occlusion - no guidance or group function (issue lateral movements)- remake with replica technique and use cuspless teeth
Base plate too thin - rebase thicker or rebase using high impact resin
Tori - relieve clinically if only problem or ask for tin-foil relief
If too thin or other problems - rebase or remake and ensure lab waxes undercuts
Tooth position wrong - remake
Occlusal table too long - i.e. too many posterior teeth over tuberosities - remove posterior teeth/grind down or remake
What are the advantages of composite?
Better aesthetics
Bonds to tooth
Minimal prep required
On demand Set
Lower thermal conductivity
Suppoers remaining tooth structure
What are the disadvantages of composite?
Under polymerised base - place increments in smaller than 2mm
Polymerisation shrinkage - place composite in small increments (keep c-factor low)
Composite insufficiently cured - cure for longer than 30s
Moisture sensitive - use dam
Post-op sensitivity - use correct technique and bonding
Longer placement time
Less wear resistance
Shorter lifespan
What is the failure rate of composite?
depends on pts OH/diet/parafunction/occlusion/operator skill
5-10 years - 13.7% failure rate at 8 years
What are the constituents of composite?
BIS-GMA
Quartz, silica
Silane coupling agent
Camphorquinone
TEGDMA
What are the requirements of a composite cavity?
Doesn’t require undercuts
Smooth margins
No unsupported enamel
No sharp line angles
Bevelled cavo-surface margin angles - increases bonding area
How can you avoid sensitivity following composite placement?
Reduce polymerisation contraction stress - place composite in increments in less than 2mm to allow for complete curing, also place increments so they are contacting as little surfaces as possible
Place lining material - RMGIC, flowable
Check occlusion after completing restoration with articulating paper
Use FV - 22600ppmF
Use desensitising toothpaste
Use water with high speed when preparing - consider excavator for deep caries
How does composite bond to dentine?
Etch 35% conditions dentine (opens tubules, removes smear layer, decalcifies dentine)
DBA applied - primer and adhesive
Primer (bifunctional) - bonds to dentine with hydrophillic ends and hydrophobic ends are exposed
Adhesive - penetrated primed surface by molecular entaglement binding to primer by hydrophobic interactions
Composite can bond by hydrophobic interactions
What is the purpose of etching enamel as well as dentine?
Increases surface energy
Removes contaminants
Increases surface area by roughening allowing micromechanical interlocking with resin - bond is 20MPA
What are the advantages of amalgam?
Durable
Shorter placement time
Radiopaque
Colour contrast
Self-hardens at mouth temp
Resistant to surface corrosion
Good wear resistance
Good bulk strength
What are the disadvantages of amalgam?
Potential mercury toxicity
Poor aesthetics
Does not usually bond to tooth
High thermal diffusivity
Requires removal of sound tissue during cavity prep
Lichenoid reactions - T4 hypersensitivity
Tooth discolouration
Amalgam tattoos
Creep/marginal breakdown
What are the failure rates of amalgam restorations?
Can last up to 20 years, 5.8% failure rate at 8 years
What are the requirements of an amalgam cavity prep?
Undercuts required - retention and resistance form
CSMA - 90 degrees for butt joint finish - no bevel
Add grooves, isthmus, dove tails for retention
Must be at least 2mm deep
No unsupported enamel
What are components of amalgam?
Silver
Tin
Mercury
Copper
Tend to be zinc free now to prevent hydrogen bubble formation
What does copper in amalgam do?
Increases early strength
Less creep and corrosion
Better marginal integrity
What is the purpose of dental dam?
Eliminate bacterial contamination
Prevent inhalation of instruments
Retract/protect soft tissue
Reduced chairside time
Increased operator and patient confidence
Moisture control
Describe an RCT to a pt
(remember - explanation, procedure, benefits, risks, tx options, follow-up, succesful outcomes)
Aim is to remove the nerve of the tooth, disinfect and shape the root canals using files and a mild bleach and then fill with rubber material to render canal inert/stop the infection spreading
This usually takes 2 appointments as we would access the nerve chamber, remove the nerve, shape and disinfect in one and place a medicament to resolve your symptoms and then place a temporary filling, we would then get you back in and once you are symptom free and everything is nice and clean we can go in again and fill it and then place a permanent filling or crown. These appointments may last slightly longer than a traditional appointment for a filling and if a crown was required that would itself take a few extra visits. The need for a crown depends on how much viable tooth tissue there is at the end of the procedure.
Study by sorenson and martinoff showed long term 94% of molars with crowns or onlays were succesful compared to 56% with fillings
Benefits of RCT
- removal of infection source
- relief of pain
- keep tooth/bone
- best aestheics
- potential abutment tooth
Risks of RCT
- failure of tx
- ledge creation
- perforation
- pain
- apical zipping
- instrument separation
- hypochlorite incident
- tooth or root fracture
- requires extensive restoration afterwards - added cost
- cost
- missed canals if complex anatomy
Likelihood of success
- up to 90% over 10y w IP
- up to 80% over 10y for dead (necrotic) pulp
Alternative tx options:
- Do nothing and KUO - won’t resolve pain + keeps source of infection
- XLA
We would follow this up via an x-ray in a years time to see whether the lesion has healed and we would assess for up to 4 years
What are succesful outcomes of a RCT?
Successful outcomes
- no pain, swelling, symptoms
- no sinus
- no loss of function
- normal pdl
What are unsuccesful outcomes of RCT?
Associated w signs and symptoms
Lesion has appeared radiographically
Pre-existing lesion has increased in size or remained the same
Signs of continuining RR
What are the tx options following RCT?
- Leave and monitor - no active tx, but may get infection - including abscess which may flare up later
- ReRCT - decreased success, may fail (not guaranteed)
- If post-core crown present - removing may cause VRF
- More complex if fractured instruments, ledges, blockages, severe curvages so consider referral
Periradicular surgery - if re-tx not possible - more difficult to tolerate, invasive, time-consuming, expensive, nerve damage risk, reduced support, scarring
XLA - tooth loss, may need replacement with prosthesis, poor aesthetics
What are the steps involved in re-rct?
Remove GP from coronal 1/3rd - U/S, GG, heat carrier
Remove GP from mid 1/3rd - R25 set at 2/3rd EWL, slow pecking motion - clean flutes every 3 pecks
Continue until GP removed from the middle third of the canal. If necessary use eucalyptus oil to soften GP.
Working length determination with size 10/15 C+/K file
Complete apical preparation to CWL with R25/R25 blue. If no apical tugback with K-25 then enlarge apical preparation with R40/R50 or K-files
What are symptoms of a NAOCL extrusion?
Pain
Swelling
Haemorrhage (bleeding - redness)
Ecchymosis (bruising)
Neurological issues
Airway obstruction
How do you manage a hypochlorite extrusion?
Stop
Inform pt of what has happened
If patient in pain = provide LA block
If bleeding - observe haemostasis - saline irrigation
Place a steroid containing intracanal medicament i.e. odontopaste - don’t use pressure/force
Seal cavity
Encourage analgesia
Consider AB
Recommend hot (hematoma) and cold (swelling) compresses
Review in 24h
Risk factors for NaOCL extrusion?
Excessive pressure (>1ml per 15s)
Needle locking in canal
Loss of WL control
Higher NaOCL conc.
Larger apical diameter (due to resorption, anomalies, perforation)
You were instrumenting a canal and you notice your file is shorter than normal on reconfirming length, what would you do?
Let patient know what you think has happened and apologise
Explain that due to cyclic fatigue and tortional stresses on the instrument that the file has become separated
Ask if you could take an x-ray (PA) of the tooth to reconfirm suspicion
Attempt removal if visible (tweezers or forceps)
If you cannot remove - tell pt this will likely require referral to a specialist
Options:
- Monitor
- Bypass - using small K-files, then using ultrasonics
- Remove file
- Obturate to blockage
- Peri-radicular surgery
- XLA
You are instrumenting a canal when you spot sudden bleeding and the patient is in pain, what do you suspect has happened and what would you do?
Potential perforation
achieve haemostasis - saline
Apologise to patient and explain that during preparation, filing/drilling has accidentally gone outwith the pulp chamber and root canal system and the long term prognosis of the tooth is affected
Repair perforation if accessible with GIC, MTA, or biodentine
Otherwise refer for:
MTA plug with microscope if close to apex
Peri-radicular surgery
Or could perform XLA yourself.
How would you diagnose reversible pulpitis, causes and management?
Pain hx - SOCRATES
Pain to cold+sweet lasting <5 seconds
Not spontaneous
No radiographic changes
Causes
- Exposed dentine
- Caries
- Deep restorations
Tx options:
- Remove the stimulus e.g. the caries
How would you diagnose symptomatic irreversible pulpitis?
Pain Hx - SOCRATES
Sharp pain on thermal stimulus (hot + cold)
Pain lingers for more than 30s after removal of stimulus
Referred or spontaneous pain (accentuated by postural changes)
OTC analgesics - ineffective
No pain to percussion (hasn’t reached apical tissues yet)
Causes:
- Deep caries
- Extensive restorations
- Fracture involving pulp
Tx Options:
- RCT
- XLA
How would you diagnose asymptomatic irreversible pulpitis?
Pulp is vital but incapable of healing and no clinical symptoms
Responds normally to sensibility testing
Would see clinical caries/fracture/compromised restoration and prompt X-ray
Tx options:
- RCT
- XLA
How would you diagnose pulp necrosis?
Death of pulp
Doesn’t respond to sensibility testing
No symptoms
Tx options:
- RCT
- XLA
Other than normal pulp, RP and IP what pulpal diagnoses are there?
Previously initiated - tooth has received partial pulp therapy (extirpation)
Previously treated - has been root canal treated or filled with intracanal medicaments (does not respond to testing)
What periapical diagnoses are there?
Normal
Symptomatic apical periodontitis
Asymptomatic apical peridontitis
Acute apical abscess
Chronic apical abscess
Condensing osteitis
What are the signs of normal periapical tissues?
Not TTP or TTBP
Lamina dura intact - hard bony lining of the alveolus
Uniform PDL
What are the signs of symptomatic apical periodontitis?
TTP, TTBP, Pain on biting (severe indicated degenerating pulp)
May be radiographic changes - width of PDL, PAP
Tx Options:
RCT
XLA
How would you diagnose asymptomatic apical periodontitis?
Inflammation and destruction of the apical periodontium of pulpal origin. Appears as an apical radiolucency and doesnt present clinical symptoms (no pain on percussion or palpation)
Tx options:
RCT
XLA
How would you diagnose a chronic apical abscess?
Gradual onset
Little/no discomfort
Intermitted discharge via sinus
Radiographic signs of osseous destruction
Tx options:
RCT
XLA
How would you diagnose an acute apical abscess?
Rapid onset
Spontaenous pain
Extreme tenderness to pressure
Swelling
Pus formation
Fever, malaise, lymphadenopathy
Mat be no radiographic sign of destruction
Tx options:
RCT
XLA
Consider AB if immunocompromised, spreading infection or systemic symptoms
How would you diagnose condensing osteitis?
Diffuse radiopaque lesions representing localised bony reaction to low grade inflammatory stimulus
Tx options:
RCT
XLA
When making your access cavity what 3 laws should you follow?
Law of symmetry (excludes maxillary molars) -
1. Orifices of canals are equidistant from line drawn in mesial-distal direction across pulp floor
2. Orifices of the canals lie on a line perpendicular to a line drawn in a mesial-distal direction across the centre of pulp floor
Law of colour change - colour of pulp chamber floor is always darker than the vertical surrounding dentine walls
Law of orifice location -
1. Orifices of the root canals are always located at the junction of the dentine walls and the floor of the pulp chamber
2. Orifices of the root canals are located at the angles of the junction of dentine wall to the pulpal floor
3. Orifices of the root canals are located at the terminus of the root developmental fusion lines
What are the functions of provisional restorations?
Maintain aesthetics
Maintain function - speech and mastication
Prevent drifting of teeth
Maintaining gingival margin - contour and health
Prevent sensitivity
Preserve vitality - prevent microleakage
Confirm tooth prep
Ensure optimum home care/optimum OH
Achieve occlusal stability - maintain OVD
Isolation for RCT
For piloting new occlusions and guidance schemes
What are the waste streams?
Black - household waste - packaging, hand towels
Orange - low risk clinical waste - swabs, dressings and other non-sharp clinical waste e.g. dam, micro brush
Yellow - high risk clinical waste (not on clinic) - body parts including teeth
Red - specialist, hazardous waste - dispose of amalgam in white box with red lid, spill/leak proof (mercury vapour suppressant in lid), amalgam waste, amalgam capsules, amalgam filled teeth
Blue - sharps including vials with medication or pharmaceuticals remaining
Dispose of sharps in sharps bin (orange stream) - 3As, 2Ns
- Always dispose of sharps in sharps box immediately after use
- Always keep out of reach of children and non-authorised personnel
- Always close sharps box between use temporary closing mechanism
- Never retrieve anything from sharps box
- Never fill more than 3/4 full
How do you deal with a blood spillage?
Stop what you’re doing
Apply appropriate PPE
Cover spill with disposable paper towels
Apply sodium hypochlorite/sodium dichloroisocyanurate - liquid/powder/granules (10000ppm)
Leave for 3-5 mins, use scoop to take up the gross contamination and put into orange waste
Clean with water and general purpose neutral detergent disinfectant wipes
What is the system of design for RPD?
Saddles - kennedy classification
Support - resistance to occlusal directed load
Retention - resistance to lifting away from the tissues
Bracing - resistance to horizontal movements achieved by contacting vertical anatomical structures (can be achieved with clasps, plates, major connectors, flanges)
Reciprocation - resistance to displacement by active retention forces
Connectors
Indirect retention - resistance to rotational displacement around the clasp axis
Review and simplify
What are the kennedy classifications?
Class I - bilateral free end saddle
Class II - unilateral free end saddle
Class III - unilateral bounded saddle
Class IV - anterior bounded saddle that crosses the midline
The most posterior saddle defines the class, additional saddles are modifications - the number of modifications don’t include the saddle used to classify the arch
What is RPD support defined as and whats the classification for it?
Resistance to occlusally directed load
Support classification - craddock
- (** preferred) - class 1- tooth-borne
- Class 2 - mucosa-borne
- Class 3 - mixed (commonly in a lower FES)
*Bounded saddles cannot be tooth supported if they are longer than 3 teeth
What provides support for an RPD?
Rests provide support - these are defined by where they’re placed on the teeth
Cingulum rests - use: canines and incisors
- canines provide best support
Incisal rests - use: commonly on lower incisors
- poor aesthetics
- interfere with occlusion
Occlusal rests - use: occlusal surface of molars and premolars
- must extend to the midline to prevent tipping and to ensure the load is transferred down long axis of tooth
- have to cut rest seat if interfering with occlusion
Where should rests be placed?
Bounded saddle - immediately next to saddle (unless abutment teeth are heavily tilted)
Free end saddle - mesial side of abutment tooth
What is retention for an RPD and how do we achieve it?
Resistance to vertical dislodging forces (prevents it falling out)
Mechanical - clasps in undercuts
Muscular - action of patients muscles
Physical - adhesion, cohesion, atmospheric pressure and surface tension
Guide planes - limits POI/R
- Doesn’t deform during insertion and removal
- Provides long term retention
Clasps - terminal end of clasp sits in undercut
types
- occlusal - circumferential or ring clasp (posteriors), clasp comes from rest
- gingival - commonly used in anterior teeth (5 forward), clasp comes from mesh
In Co-Cr RPDs we use 15mm tapering cross sectional diameter clasps which engage a 0.25mm undercut
Mesial undercut - self-reciprocating/ring clasp
Distal undercut - C-clasp (single clasp with reciprocal arm) - may not need reciprocal arm if base plate provides reciprocation
What in indirect retention for an RPD and what can provide it?
Resistance to rotational movement around a clasp axis
can be provided by connectors, rests, saddle, denture base
when planning - look at line between terminal ends of clasps and place indirect retention on the axis 90 degrees to that
What is reciprocation in terms of RPD?
Prevents continual horizontal pressure causing tooth movement
How many reciprocal arms should there be - ideally 3 arms in a triangle shape
What is an RPI?
Stress relieving clasp system
Used on FES (commonly mandible)
Help to eliminate axial torque on abutments
- mesial rest
- proximal guide plate on or above survey line with undercut below
- gingivally approaching I-bar clasp
On loading - clasp and plate disengage to prevent pressure
Reciprocation is provided via minor connector which connects rest and plate
What is the purpose of a connector in an RPD? what are the types and advantages/disadvantages?
Rigid part that joins all components
Major - connects components on one side of arch to the other
Minor - join other components to the major
Types of major
- Plate
- Bar
Plate
- adv: thinner + can be extended onto tooth surface to provide indirect retention
- dis: may have to cover gingival margins, more mucosal coverage
Bar
- adv: less mucosal coverage and relief of gingival margins
- dis: thicker and more edges that can be explored by the tongue
Mandibular connector
- by default- lingual bar but require 8mm space (4mm for bar, 3mm from gingival margin and 1mm from functional FoM)
Avoid horseshoe in upper if FES