Normal and abnormal uterine action Flashcards

1
Q

What are the 3 classifications for abnormal uterine action

A
Over-efficient uterus
*Precipitate delivery
*Excessive contraction and retraction
Inefficient uterine action
* Hypotonic inertia
* Hypertonic inertia
* Constriction (contraction) ring
Cervical dystocia
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2
Q

What is precipitate delivery?

A

Precipitate delivery refers to a delivery which results after an unusually rapid labor (combined 1st stage and second stage duration is <2hrs) and culminates in the rapid, spontaneous expulsion of the infant.

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3
Q

What are predisposing factors for precipitate labour

A
Multiparity 
Strong uterine contractions 
Small sized baby
Relaxed pelvic or perineal floor muscles
Roomy pelvis
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4
Q

5 Maternal complications of precipitate labour?

A
Laceration
Hemorrhage from lacerations or hematoma 
Uterine atony from exhaustion
Infection
Uterine inversion
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5
Q

4 Fetal complications of precipitate labour?

A

Intracranial hemorrhage from sudden change in pressure
Asphyxia - lack of resuscitation and placental compressions
Tearing of the umbilical cord
Fetal injury due to falling

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6
Q

How do you manage precipitate labour

A
◦Hospitalize before due date
◦Inhalation anaesthesia: as nitrous oxide and oxygen is given to slow the course of labour.
◦Tocolytic agents.
◦Episiotomy to prevent lacerations
Examine for tears after
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7
Q

What is retraction ring/Bandl’s ring in excessive contraction and retraction

A

It is the retraction ring that rises up during obstructed labour due to marked retraction and thickening of the upper uterine segment while the relatively passive lower segment is markedly stretched and thinned to accommodate the foetus.

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8
Q

How does retraction ring/Bandl’s ring present?

A

Clinical picture: is that of obstructed labour with impending ruptured uterus

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9
Q

How retraction ring/Bandl’s ring diagnosed?

A

The Bandl’s ring is seen and felt abdominally as a transverse groove that may rise to or above the umbilicus

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10
Q

What is the complication of excessive contraction and retraction?

A
  • In the pirmigravid woman, labour comes to a cease due to exhaustion of the muscles
  • In multigravidae retraction continues with progressive thinning of the lower uterine which could cause rupture and hence foetal and maternal compromise
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11
Q

How do you treat excessive contraction and retraction?

A

Adequate pain relief
Cesarean section
Prophylactic antibiotics

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12
Q

What is hypotonic uterine action/inertia?

A

The uterine contractions are infrequent, weak and of short duration

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13
Q

What are general predisposing factors for hypotonic uterine action

A
  • Primigravida particularly elderly
  • Anaemia and asthenia
  • Nervous and emotional as anxiety and fear.
  • Hormonal due to deficient prostaglandins or oxytocin as in induced labour
  • Improperuseofanalgesics
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14
Q

What are local factors of hypotonic uterine action

A
  • Overdistension of the uterus.
  • Developmental anomalies of the uterus e.g. hypoplasia.
  • Myomas
  • Malpresentation
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15
Q

How do you manage hypotonic uterine action

A

Induction of labour - oxytocin, amniotomy
VD with instrumentation
Cesarean section if SVD fail, contraindication to oxytocin, fetal distress

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16
Q

What is hypertonic uterine action/inertia?

A

Uncoordinated uterine action

17
Q

What are the 2 types of hypertonic uterine inertia?

A

Colicky uterus: incoordination of the different parts of the uterus in contractions.
Hyperactive lower uterine segment: so the dominance of the upper segment is lost

18
Q

What is the presentation of hypertonic uterine action?

A

Prolonged labour
More painful and irregular contraction. The pain is felt before and throughout the contractions with marked lower backache.
High resting intrauterine pressure in between uterine contractions detected by tocography
Slow cervical dilatation
Premature rupture of membranes.
Foetal and maternal distress

19
Q

How do you manage hypertonic uterine action

A

Pharmacotherapy:
o Analgesic.
o Epidural analgesia may be of good benefit. 

Caesarean section is indicated in:
o Failure of the previous methods.
o Disproportion.
o Foetal distress before full cervical dilatation

20
Q

What are constriction rings / contraction rings / Schroeder’s rings

A

It is a persistent localised annular spasm of the circular uterine muscles that occurs at any part of the uterus but usually at junction of the upper and lower uterine segments and may appear at any stage during labour

21
Q

What are some associated factors of constriction rings / contraction rings / Schroeder’s rings

A
Mal-presentations and mal-positions.
Improper use of oxytocin e.g.
◦ use of oxytocin in hypertonic inertia.
◦ IM injection of oxytocin
Premature rupture of membrane
Premature attempt in instrumental delivery
22
Q

How do you diagnose constriction rings / contraction rings / Schroeder’s rings

A

The condition is more common in primigravidae and frequently preceded by colicky uterus.
The exact diagnosis is achieved only by feeling the ring with a hand introduced into the uterine cavity.

23
Q

What are 3 possible complications of constriction rings / contraction rings / Schroeder’s rings

A

Prolonged 1st stage: if the ring occurs at the level of the internal os.
Prolonged 2nd stage: if the ring occurs around the foetal neck.
Retained placenta and postpartum haemorrhage: if the ring occurs in the 3rd stage (hour- glass contraction).

24
Q

How do you manage constriction rings / contraction rings / Schroeder’s rings

A
  • In the 1st stage: Pethidine may be of benefit.
  • In the 2nd stage: Deep general anaesthesia and amyl nitrite inhalation are given to relax the constriction ring:
    ◦ If the ring is relaxed, the foetus is delivered immediately by forceps.
    ◦ If the ring does not relax, caesarean section is carried out with lower segment vertical incision to divide the ring.
  • In the 3rd stage: Deep general anaesthesia and amyl nitrite inhalation are given followed by manual removal of the placenta
25
Q

Differences between a constriction ring and a retraction ring

A

Check evernote

26
Q

What is cervical dystocia

A

Failure of the cervix to dilate within a reasonable time in spite of good regular uterine contractions

27
Q

What are the 2 types of cervical dystocia? (just state)

A

Primary (functional) and secondary (organic)

28
Q

What is primary (functional) cervical dystocia?

A
  • In spite of the absence of any organic lesion and the good effacement of the cervix, the external os fails to dilate.
  • This may be due to lack of softening of the cervix during pregnancy or cervical spasm resulted from overactive sympathetic tone
29
Q

What is secondary (organic) cervical dystocia?

A
  • Excessive scarring or rigidity of cervix post previous operation, disease or delivery eg previous amputation, cone biopsy, extensive cauterization or obstetric trauma.
  • Organic lesions as cervical myoma or carcinoma.
30
Q

What are 3 complications of cervical dystocia?

A

1 - Annular detachment of the cervix: surprisingly the bleeding from the cervix is minimal because of fibrosis and avascular pressure necrosis leading to thrombosis of the vessels before detachment.
2 - Rupture uterus secondary to prolonged labour and obstruction
3 - Postpartum haemorrhage: particularly if cervical laceration extends upwards tearing the main uterine vessels.

31
Q

How do you manage primary cervical dystocia (functional)?

A

Pethidine and antispasmodics: may be effective.
Caesarean section: if
* medical treatment fails or
* foetal distress developed

32
Q

How do you manage secondary cervical dystocia (organic)?

A

Caesarean section is the management of choice