Nijs article Flashcards

1
Q

What is the 2011 article by Nijs et al about?

A

Practical guidelines for explaining central sensitization to patients with chronic unexplained msk pain.

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2
Q

When is pain physiology education indicated according to Nijs 2011?

A

1) when central sensitization dominates clinical pictures. 2) maladaptive illness perceptions are present.

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3
Q

What are 2 ways we can help change someone’s chronic pain condition?

A

face to face education about pain physiology and written materials to take home.

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4
Q

What are examples of the chronic conditions that may benefit from pain physiology education?

A

chronic LBP, chronic whiplash, fibromyalgia, chronic fatigue syndrome.

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5
Q

in the 1st session, what 2 types of pain are important to differentiate for the pt?

A

acute nociceptive pain vs. chronic pain.

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6
Q

What can the 2nd session be used for?

A

correct misunderstandings & facilitate change from knowledge of pain to adaptive coping in daily life.

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7
Q

What are the basic neurological changes that characterize central sensitization?

A

decrease in ability of descending pain inhibition. And increase in ascending and descending pain facilitatory pathways.

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8
Q

What areas of the brain are overly active in the altered pain neuromatrix?

A

the normal acute pain centers: insula, anterior cingulate cortex, and prefrontal cortex (not primary or secondary somatosensory cortex) and areas of brain not normally active in acute pain: various brain stem nuclei, dorsolateral frontal cortex, parietal associated cortex.

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9
Q

What is “cognitive emotional sensitization”?

A

Phenomena of forebrain centers powerfully influencing brainstem nuclei involved with descending pathways. Descending pathways can be changed by person’s level of vigilance, attention, and stress.

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10
Q

What is the link between MSK pain and central sensitization?

A

Chronic pain evolves from MSK pain. For example massive peripheral input from whiplash which causes a change in the sensitivity of CNS. Thereafter, any additional trauma can cause bottom-up input on CNS and feed into central sensitization.

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11
Q

A patient with ‘unexplained’ chronic MSK pain who is misinformed about pain demonstrate which tendencies?

A

more catastrophic thoughts and less adaptive coping strategies, and low treatment adherence.

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12
Q

In a 2010 RCT by Ryan et al, was pain education (for chronic LBP patients) alone more beneficial than pain education and group exercise?

A

Yes education alone was more beneficial, in the short term, for pain relief and improving pain self-efficacy.

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13
Q

What can we conclude from studies about pain education and motor performance (Ryan 2010 & Moseley ‘02,’04)?

A

Motor performance may be directly influenced by changes in pain perception.

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14
Q

In studies examining pts with fibromyalgia or chronic fatigue syndrome, was providing the booklets about pain physiology effective?

A

No, but when the booklets were provided along with individually tailored face to face education sessions, they improved.

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15
Q

What are some maladaptive pain cognitions, which can be used to identify a patient with central sensitization?

A

ruminating about pain and hypervigilance to somatic signs.

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16
Q

What tools can be used to identify maladaptive pain cognitions?

A

‘Pain Catastrophizing Scale’, the ‘Pain Vigilance and Awareness Questionnaire’, and the brief ‘Illness Perception Questionnaire.’

17
Q

What is the key concept that chronic pain patients should be convinced of?

A

hypersensitivity of the CNS rather than local tissue damage is the cause of their presenting symptoms.

18
Q

what is the typical progression of a pain education session?

A

info about local nociception mechanisms presented first, then contrasted with central sensitization mechanisms. Patient encouraged to ask questions and info from pt is used to individualize the information.

19
Q

What are common cognitive impairments of patients with chronic pain, and what are the implications?

A

concentration difficulties, impairments in short-term memory, which implies they can forget verbal information. Therefore homework should include a book with info and pictures the same as from the face-to-face session.

20
Q

What can be used to assess the patient’s understanding of pain?

A

the ‘Neurophysiology of Pain Test’. Hand out to pt after first session and tell them to fill it out one day prior to their next session.

21
Q

What are some general recommendation for the 2nd session?

A

Answer questions from Pain Test they got wrong and explain. Identify behaviors they can modify. Like: stop ruminating about aetiology and nature of their pain, reduce stress, increase physical activity levels, decrease hypervigilance, relaxation etc.

22
Q

What can the ‘Pain Reaction Record’ (Sullivan 2003) be used for?

A

To help transition the pt from knowledge of pain, to adaptive behaviors. It is in easily applicable measure facilitating a cognitive approach to pain coping.

23
Q

What is one method to help ensure long term pain belief changes are happening during later treatment sessions?

A

Ask the patient to explain the rational behind a particular treatment. If they can’t explain, then reeducate, and continually look for maladaptive behaviors.

24
Q

If chronic pain patients experience side effects and symptom fluctuations, what should the PT do?

A

Explain these symptoms as part of the central sensitization model and shift the pt’s attention away from the somatic signs and towards adaptive coping strategies and reassurance.

25
Q

What is one patient perception that should be an outcome goal to constantly assess during therapy?

A

The patient’s confidence in the treatment.