NICU Flashcards
What are the goals of developmentally sensitive care?
- Reduce stress and agitation
- Promote growth and healing
- Neuroprotection
- Preventing harm
Name components of developmentally sensitive care
- Eyes: reduce light (dim lights, shield eyes from bright lights, eye covering with bili lights, blanket over isolette, natural light over artificial light, turn lights off when sleeping)
- Hearing: reduce noise (monitor room volume, turn down alarm volumes and silence quickly, avoid tapping on isolettes and teach families this, close portholes slowly and quietly, quiet when around open crib)
- Nose- avoid strong scents such as flowers, perfumes, alcohol swabs, etc.
- Touch: avoid sudden movements and be gentle, warm hands, keep baby well supported and contained, support head and neck, promote positive touch
- Positioning: support MSK development, arms flexed and by face, legs and hips flexed, centrally midline, use swaddling, nesting and positioning supports.
- Pain: limit painful procedures, use adequate pain relief (both pharm and non-pharm measures)
- Sleep: cluster care, watch for stress cues from baby indicating they need a break and sleep
- Skin: duoderm under bipap and cpap masks, pH neutral products, no chlorohex bath wipes, no bathing everyday, watching for skin breakdown, rotating sites where adhesive is used, moisture barrier with diaper changes, diaper change with every handling
- Temp: use warm wipes, limit time baby is uncovered, warm bankets, overbed warmer and isolette warmer when needed, socks/mittens/hat/clothing/blankets
At what age gestation can you start using sucrose?
Above 27 weeks
Characteristics of caput succedaneum?
- Most common, least severe
- Trauma to scalp from delivery- pressure from vaginal delivery
- Superficial hemorrhagic edema, soft and pitting over presenting part of the head
- Crosses suture lines (CS = crosses sutures)
- Max size at birth, does not grow
- Self resolving
Characteristics of cephalohematoma?
- Trauma from birth- usually forceps or vacuum
- Blood collection between periosteum and skull bone (usually parietal)
- Confined by suture lines
- Causes local swelling
- Increases in size after birth, not a significant source of blood loss
- Associated with skull fracture 25% of the time
Characteristics of a subgaleal hemorrhage?
- Least common, most severe
- Caused by rupture of emissary veins, may be accompanied by skull fracture
- Diffuse edema that crosses suture lines
- The subgaleal space can hold a lot of fluid so is a significant loss of blood The space expands through the entire head and down the neck.
- Early signs: diffuse swelling of the scalp, pallor, hypotonia, decreased LOC
- Progressive signs: posterior/lateral spread of swelling, pitting edema, worsening perfusion, further decrease in LOC
- Late signs: hypovolemic shock (hypotension, tachypnea, tachycardia), worsening neuro deterioration, encephalopathy, multiorgan failure, significant anemia
- Nursing interventions: monitor VS, NVS, head circumference closely after birth when vacuum or forcep assisted
- Requires fluid resuscitation and/or blood transfusion, in severe cases surgery may be necessary
Characteristics of an epidural hematoma?
- Opposite of cephalohematoma- on inner side of skull between bone and periosteum
- Does not cross suture lines
- Causes increased ICP, swollen/bulging fontanelles, seizures
- Rapidly expands
- Rare, traumatic birth, linear skull fracture across middle meningeal artery
Characteristics of subarachnoid hemorrhage?
- Most common
- Small and venous
- Caused by birth trauma or hypoxia
- May cause seizures or apnea
- If severe can result in hydrocephalus or neurological damage
Characteristics of a subdural hematoma/hemorrhage?
- Tear in a cerebral vein or sinus, plus tear in the dura
- Secondary to birth trauma, linked to maternal use of aspirin or phenobarb
- Neurological issues at birth, further deterioration after birth
- Slowly expands and crosses suture lines
- Risk of herniation with LP
What are the 2 phases of brain injury in HIE?
Phase 1- initial lack of O2 and blood flow, direct tissue injury from lack of energy and resulting acidosis from metabolite build up
Phase 2- reperfusion injury occurs 8-16 hours after oxygenation/perfusion has been restored. There is a second decrease in high phosphate energy compounds which again results acidic metabolite products building up causing more damage from inflammation and neurotoxic cytokine release.
When must cooling be initiated by in HIE?
Within 6 hrs of life, but the sooner the better to prevent further complications from reperfusion injuries. Physician discretion if it is later than 6 hrs.
Differential diagnosis for HIE
Exclusion of other potential neonatal encephalopathies: meningitis, encephalitis, genetic conditions, thrombophilic disorders
Events consistent with HIE?
-Hypoxic event immediately before or during labour, or sudden and sustained fetal bradycardia/loss of fetal HR with decelerations.
- APGAR scores of 0-3 at 5 min of life
- Multisystem involvement
- Evidence of acute, non-focal cerebral abnormalities shown in early imaging
Signs and symptoms of HIE (mild, moderation and severe)
Mild: hyperalert with normal tone and activity, exaggerated response to stim, reactive pupils, no seizure activity
Moderate: hypotonia, weak suck, constricted but reactive pupils, periodic breathing/apnea. Development of seizures or lethargy indicate deterioration
Severe: stupor or coma, absent reflexes, pupils non reactive, no spontaneous activity, requires mechanical ventilation.
Tests done with HIE
- Blood gas (indicates hypoxemia or hypercarbia)
- ECG
- CSF
- CXR
- EEG
- MRI
- Blood work to assess multiorgan involvement (electrolytes, coagulation, liver function, kidney function)
- Glucose
HIE treatment
- Supportive ventilation
- Maintaining a stable BP
- Temperature regulation
- Maintenance of normal glucose, calcium, and lytes
- Control of seizures
Eligibility for cooling in HIE
35 weeks GA who are less than 6hrs old and meet the following criteria:
- Cord pH <7.0 or base deficit of at least -16 OR
- Cord pH 7.01-7.15 or base deficit -10 to -15.9 or no gas available AND hx of acute perinatal event AND at least 10 min PPV OR Apgar of 5 or less at 10 mins of life
AND
Evidence of moderate to severe encephalopathy as defined by clinical seizures OR the presence of at least 3 of the 6 categories.
Categories:
- LOC: lethargic (mod), stupor/coma (sev)
- Spontaneous activity: decreased (mod), none (sev)
- Posture: distal flexion, full extension (mod), decerebrate (sev)
- Tone: hypotonic (mod), flaccid (severe)
- Primitive reflexes (moro and suck): weak or incomplete (mod), absent (sev)
- Autonomic system: pupils constricted, bradycardic, periodic breathing (mod), dilated and non reactive pupils, variable HR, apnea (sev)
How fast to rewarm a baby who was cooling?
0.5C per hour
How fast do we aim to get the baby’s temp to 33.5?
Within 1-2hrs of admission.
How do we monitor baby’s temp during HIE cooling?
Rectal probe and skin probe
What indications would cue you to stop cooling for HIE early?
- Worsening or severe hypoxemia or hypotension
- Clinically significant coagulopathy despite treatment
- Arrythmia requiring medical treatment
Can babies feed during cooling for HIE?
Trophic feeds can be given to hemodynamically stable babies after 24hrs. OIT can be given as soon as breastmilk is available