newborn/ premie Flashcards
What does APGAR look at?
heart rate resp effort muscle tone (activity) reflex irritability (grunting) colour (appearance)
APGAR scoring?
HR: 2 if >100. 1 if <100. 0 if absent
Resp effort: 2 regular, strong cry. 1 gasping/ irregular. 0 absent
Muscle tone 2 well flexed, active. 1 some flexion, slightly hypotonic. 0 flaccid
reflex irritability 2 cry, cough. 1 grimace. 0 none
Appearance 2 pink all over. 1 pink w blue extremities 0 blue/ pale
Neonatal hypoglycaemia Mx
Bolus 10% dextrose
if mother received opioid analgesia, infant gets this
naloxone
to prevent resp depression
Newborn severe lactic acidosis mx
sodium bicarb
Nitrogen washout test (Hyperoxia test)
used to differentiate between cardiac and resp causes of cyanosis.
e.g. infant given 100% oxygen for 10min. ABG taken before and after.
If significant improvement, likely problem w oxygenation (resp)
If pO2 still <15kPa indicates cyanotic CHD - TOF, TGA, tricuspid atresia
small white pearls along midline of palate
epstein pearls
resolve spontaneously
port wine stain
present from birth due to vascular malformation of dermal capillaries.
may be assoc w sturge weber syndrome if along distribution of trigeminal n
Guthrie tests for?
SCA CF Congenital hypothyroidism PKU MCADD
Hearing screening
evoked otoacoustic emission to test cochlear function
If abnormal hearing screening?
automated auditory brainstem response (AABR) done
And if abnormal -> refer to paed audiologist
Hypoxic ischaemic encephalopathy causes
significant hypoxic event immediately before/ during labour or delivery
- placental abruption, ruptured uterus, excessive uterine contractions
- cord compression (inc shoulder dystocia), cord prolapse
- preeclampsia w IUGR
- compromised fetus (IUGR, anaemia)
HIE may occur postnatally or be caused by neonatal condition e.g. kernicterus, inborn error of metabolism
Hypoxic ischaemic encephalopathy presentation
severe- comatose, severly hypotonic, needs assisted ventilation, prolonged seizures
mod- lethargic, markedly abnormal tone, requires tube feeding, seizures
mild- irritable, mild hypotonia, poor sucking
Hypoxic ischaemic encephalopathy MX
Resp support
aEEG to detect seizures / early encephalopathy
treat seizures w anticonvulsants
monitoring and treatment of hypoglycaemia and electrolyte imbalance
mild hypothermia by wrapping infant in cooling blanket reduces brain damage due to secondary neuronal death from reperfusion
Conditions premies are at higher risk of
Respiratory distress syndrome (surfactant deficiency)
Patent ductus arteriosus
Necrotizing enterocolitis
Intraventricular haemorrhage
RDS surfactant deficiency IX
CXR- ground glass appearance with air bronchograms
RDS antenatal prevention
Steroids.
IM betamethasone 12mg x2 24 hrs apart
RDS management
surfactant therapy Oxygen therapy (optiflow, CPAP, assisted ventilation)
Intraventricular haemorrhage complications + Mx
may impair drainage and reabsorption of CSF -> hydrocephalus
Mx; VP shunt
initial symptomatic relief by CSF removal using LP or tap
necrotizing enterocolitis what is it?
inflammation of the intestine -> necrosis -> perforation
risk factors of necrotizing enterocolitis
preterm infant
formula fed
necrotizing enterocolitis tx
stop oral feeding, give broad spectrum abx
parenteral nutrition to rest bowel
surgery if bowel perforated
Necrotizing enterocolitis IX
Abdo Xray - distended loops of bowel and thickening of bowel wall w intramural gas
If perforated - can be detected on abdo xray / clinically
commonest cause of resp distress in term infants
transient tachypnoea of newborn
What is transient tachypnoea of newborn?
Risk factors
Caused by delay of resorption of lung fluid
RFs:
elective C-section
maternal diabetes
earlier gestational age
TTN Ix
Xray - fluid in horizontal fissure
Diagnosis of exclusion
- given ambient O2 if required
Meconium aspiration Mx
suction nose, mouth and throat
intubation to suck meconium for lungs
artificial ventilation may be required