Neurologic Impairments Flashcards
Signs
Objective findings determined by physical examination
Symptoms
Subjective reports associated with pathology perceived by patients
Positive Signs/Symptoms
Release of abnormal behavior (i.e. Hyperactive reflex)
Negative Signs/Symptoms
Loss of normal behavior (i.e. paresis)
Primary Effect of Lesion
Impairments causing problems in motor, sensory/perceptual and/or cognitive systems.
Secondary Effect of Lesion
Impairments NOT directly resulting from CNS lesion, developed as result of original problems
Lesion in descending motor
Primary: Paresis, Spasticity
Secondary: Structural & Functional changes in muscles
Motor Weakness (paresis)
Inability to generate sufficient tension in a muscle and a major impairment of motor function in UMN syndrome
Spasticity
Velocity-dependent increase in tonic stretch reflexes
Muscle tone
Muscle’s resistance to a passive stretch
Modified Ashworth
0 = No increase in muscle tone; 1 = slight increase in tone …; 1+ = slight increase in tone; 4 = affected parts rigid in flexion/extension
Coordination Problems
Capacity to generate force does not predict the ability of that muscle to perform a specific task
Abnormal synergies
Loss of ability to recruit a limited number of muscles controlling movement and to control individual joints
Inappropriate muscle activation in sequence, causing unnecessary movements
Flexor Synergy
Scapular retraction/elevation
Shoulder abduction/ER
Elbow Flexion, forearm supination
Wrist/finger flexion
Impairments in Cerebellar Pathology
Hypotonia, Ataxia, Dysmetria, Action/Intention tremor
Impairments in BG
Hypokinetic and Hyperkinetic
Hypokinetic Disorders
PD
Bradykinesia, Rigidity, Resting tremor, Postural Instability
Hyperkinetic Disorders
Huntingtons: Excessive involuntary movement and low muscle tone
Chorea & Dystonia
Somatosensory cortex deficits
Discriminative sensations (Proprioception, 2-point Stereognosis, touch localization)
Visual Deficits based on location of lesion
optic nerve: loss of vision in ipsilateral eye
optic chiasm: loss of vision in temporal visual field from both eyes (bitemporal hemianopsia or tunnel vision)
optic tract (temporal & parietal lobes): loss of vision in contralateral visual fields in both eyes (homonymous hemianopsia)
optic radiation: contralateral homonymous hemianopsia
Assessment of Visual Deficits
Visual Acuity, Depth Perception, Visual Field Confrontation test, Oculomotor Control test
Vestibular Deficits
Gaze Stabilization problem
Impairments in balance and posture
Dizziness or vertigo (BBPV most common)
Motor Learning
Study of acquisition and modification of movement
Conventionally: Learn new skills
Recovery of function: Reacquire lost due to injury
Motor learning and control emerge from interaction between?
Individual, task, environment
Learning
Performance
Practice/experience leading to permanent changes
Temporary changes in motor behavior
Plasticity
Ability to show modification
Nondeclarative learning
Nonassociative, Associative, Procedural
Nonassociative
Habituation: Decreased responsiveness after repeated exposure
Sensitization: Increased responsiveness following threatening stimulus
Associative learning
Classical conditioning: Pair two stimuli
Operant conditioning: associate a response with a consequence
Procedural learning
Learning tasks that can be performed automatically without attention
Develops gradually through repetition of act over many trials
Declarative Learning
Results in knowledge of facts/events, such as awareness, attention, and reflection
*Practice can make transition from declarative to procedural knowledge
Closed versus open loop
Closed: Feedback from movement used for guidance
Open: Movements performed in absence of feedback
Schema Theory
Emphasize open loop
Generalized motor programs: Rules for creating spatial and temporal patterns of muscle activity needed for a task
Five Components to conceptual framework
A model of practice, A model of function and disability, Hypothesis-driven clinical practice, Principles of motor control and learning, Evidence-based practice