Neurogenic Midterm Flashcards

1
Q

Aphasia

A

Acquired language impairment resulting from a focal brain lesion in the absence of other cognitive, motor, and sensory impairments. Breakdown in specific language domains resulting from a focal lesion. Receptive and expressive & multimodal.

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2
Q

Right Hemisphere Syndrome

A

Difficult to detect and diagnose but broadly is considered a communication disorder resulting from damage to the right hemisphere that affects the nonlinguistic aspects of communication with relatively intact communicative abilities.

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3
Q

Effects of Right Hemisphere Syndrome

A
  • prosody
  • discourse production and comprehension
  • pragmatics
  • emotional and nonverbal communication
  • figurative and implied meanings
  • theory of mind
  • comprehension and production of humor
  • visual spatial aspects of reading and writing
  • executive functioning (planning, organization, problem solving, time management)
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4
Q

left and right visual primary

A

see the object first

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5
Q

left and right visual secondary

A

recognize the object; have a visual memory of it

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6
Q

auditory primary (temporal lobe)

A

hear others, hear yourself, connect the visual memory with the auditory memory of it

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7
Q

left and right parietal lobe

A

thought, academic memories

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8
Q

angular gyrus or temporal/occipital/parietal junction

A

tertiary area and translator of thought into components responsible for language, sight into language, touch into language, hearing into language, and emotions into language. Translates one modality to the next.

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9
Q

Wernicke’s area (left secondary auditory cortex)

A

auditory memories for words, word forms, connect to visual memory/auditory memory and name it.

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10
Q

right analogous area to Wernicke’s

A

auditory memories for intonation forms such as rising intonation for questions in English

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11
Q

Broca’s area

A

motor patterns for speech sounds and words, syntax. A pattern generator.

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12
Q

right analogous to Broca’s

A

motor patterns for intonation aspects of speech signal

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13
Q

motor cortex

A

left and right-from it arise Upper Motor Neurons

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14
Q

limbic system

A

emotional memories, emotional words (e.g., cursing), emotional aspects of speech signal and overall communication, emotion comes out in speech, emotional state also affects how you receive information.

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15
Q

Broca’s aphasia

A

lesions in Broca’s area. apraxia, halted and effortful speech without syntax. They recognize their errors, but they can’t find the right words and the right motor patterns to say what they want. Sometimes called an “expressive aphasia” but we shouldn’t call it that. COMPREHENSION RELATIVELY INTACT.

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16
Q

Wernicke’s aphasia

A

lesions in Wernicke’s area. IMPAIRED COMPREHENSION. can’t “make a match” between the sounds and the words. They don’t recognize their own errors. They think they are saying and hearing the right thing, but then the sounds and words become errored on producing the patterns to send to the motor strip when turning the sounds into words. sometimes called “receptive aphasia,” but we shouldn’t.

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17
Q

Conduction aphasia

A

damage of the articulate fasciculous between Broca and Wernicke. Can’t imitate speech, but do better if they think of it themselves

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18
Q

Global aphasia

A

Broca’s, Wernicke’s, and conduction aphasia. caused by a very large stroke because it affects all the other areas.

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19
Q

Anomic aphasia

A

word finding problems. no apraxia, good syntax. can me caused by damage in any of the language-oriented areas.

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20
Q

Transcortical aphasia (sensory)

A

damage between Wernicke’s and angular gyrus. impaired comprehension. can repeat and recognize phonemic patterns, but can’t understand meaning besides very basic stuff.

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21
Q

transcortical aphasia (motor)

A

damage in front of Broca’s; connection between Broca’s and motor strip. don’t talk much, but correct when they do. has to do with initiation of speech. intact language and comprehension, have thoughts they don’t say because part of brain that tells you to talk is permanently inhibited. rare

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22
Q

transcortical mixed

A

mix of transcortical motor and transcortical sensory. have 2 strokes, one right outside Broca’s and one outside Wernicke’s. don’t talk or understand much but they can repeat. mix of transcortical motor and sensory

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23
Q

mixed aphasia

A

most common because brain damage usually doesn’t just stay in one small area

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24
Q

subcortical

A

damage in the thalamus area. often transient

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25
Q

phonemic paraphasia

A

less than half of target sounds are in error (i.e., top, bop)

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26
Q

semantic paraphasia

A

the word you say is an actual word, but not the word you intended to say
related=”boy” for “girl”
unrelated=”car” for “girl”
BUT keep in mind, sometimes may seem unrelated but may be related to them. i.e., if they say “red” for “car” may be because they have a red car.

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27
Q

anomia

A

difficulty finding a word. ubiquitous among all types of aphasia. word finding problems, loss of fluidity in speech. tip of tongue phenomenon; might provide a description of the word.

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28
Q

stereotypes

A

can only say one word form (i.e., saying “four” for everything) singing may help this.

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29
Q

perseveration

A

stuck on one response (say the same response they just said for the last question) can be verbal, thought, or an action (i.e., crumbling paper); the individual’s brain has captured a behavior and cannot move onto the next.

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30
Q

undifferentiated jargon

A

one sound form that stays for the whole length of the utterance. usually right after a stroke (la la la)

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31
Q

neologistic jargon

A

accessing more of phonemic repertoire, but still unintelligible (la bla who ha) (non words)

32
Q

semantic jargon

A

actually getting some words correct, but they have no collective meaning (make boy jump red)

33
Q

aggramatism

A

absence of grammatical words or syntactical markers, only use content words (boy go store)

34
Q

neologism

A

non words with no intelligible meaning. more than half or target sounds are in error.

35
Q

alexia

A

impaired ability to read

36
Q

agraphia

A

impaired writing

37
Q

depression

A

affects our limbic system, which controls emotion. if they don’t believe they can get better, they won’t. need to start and end therapy with something they’re successful at.

38
Q

CT scan

A

send radiation through the body and record the strength of the signal after it has gone through a foreign body. The less radiation that goes through, the darker the area will be on the picture. Measures density of brain structures.

39
Q

MRI (magnetic resonance imaging)

A

uses a magnetic field to manipulate electrons. when there is no water in an area of the brain due to cell death it will show up white on the pictures.

40
Q

fMRI (functional MRI)

A

measures deoxygenated hemoglobin in the brain. the less hemoglobin blood has the more oxygen that has been used in that particular area of the brain. the more oxygen that is used, the more active the brain is.

41
Q

PET scan (positron emission tomography)

A

radiation is put inside your bloodstream an measures how much oxygen, protein, glucose metabolism, etc is in each region. looks at regions of the brain that are most active (different colors on the image represent activity levels=functioning levels)

42
Q

agnosia

A

sensory disorder resulting in an inability to interpret a sensory stimulus. bizarre situations where patient cannot recognize things that he or she readily recognized before.

43
Q

visual agnosia

A

difficulty recognizing the object when seen

44
Q

prosopagnosia

A

inability to recognize faces

45
Q

auditory agnosia

A

inability to comprehend speech and non speech sounds

46
Q

auditory nonverbal agnosia

A

inability to recognize sounds

47
Q

pure word deafness

A

hearing people speak, but can’t understand them

48
Q

acoustic/auditory agnosia

A

auditory verbal agnosia: words, or music through audition

49
Q

anosognosia

A

unawareness of deficit “conceptual neglect”

50
Q

stimultangosia

A

inability to perceive things as a whole, deficit in spatial visual/spatial bonding

51
Q

tactile agnosia

A

inability to recognize objects by touch

52
Q

spatial agnosia

A

familiar objects through palpation; may not be able to orient themselves or move in familiar surroundings

53
Q

alexia

A

loss of ability to read words

54
Q

agraphia

A

loss of ability to write

55
Q

disability epidemiology

A

study of distribution, determinants, correlates, and outcomes of disability and application of this study to maximizing the health, participation, and quality of life of people with and populations with disability. Look at how people function as a whole and how we can help them with that. Everything that you do to help make the person do a little more on their own assists them and their family. We need to know the epidemiology so that we can maximize the healthy, participation, and quality of life for our patients.

56
Q

ICF

A

framework for ASHA scope of practice and preferred practice patterns. includes body function, body structure, activity/participation, environmental factors, personal factors. combination of factors makes one “disabled.” ICF will be tied to reimbursement in the future to prove you’re improving clients. used to determine areas of strength and limitations. helps point out areas in which additional info is required to plan intervention. helps evaluate family/caregivers, co-workers, etc.

57
Q

characteristics of aphasia and right hemisphere syndrome

A

phonemic paraphasias, semantic paraphasias, apraxia, neologism, perseverations, anomia, stereoytpies, jargon, agrammatism, alexia, agraphia, agnosia, aprosodia, anosognosia

58
Q

TIA (transient ischemic attack)

A

symptoms last less than 24 hours. may be no brain damage when symptoms disappear. clot eventually breaks into small pieces and normal blood flow returns. may not show up on neuroimaging. most likely precursors of another, more severe stroke.

59
Q

RIND (reversible ischemic neurological deficit)

A

symptoms last more than 24 hours, but less than 72. subsequent recovery. still at risk for ongoing difficulties.

60
Q

completed stroke

A

the stroke has done its maximum damage

61
Q

evolving stroke

A

series of strokes immediately after each other. the patient shouldn’t be evaluated until they are stabilized.

62
Q

hemorrhagic stroke

A

bleeding occurs. blood doesn’t get to the regions of the brain it needs to. bleeding puts pressure on the brain and can sometimes kill regions of the brain. often lethargic and unable to concentrate. if pressure isn’t relived, pressure can crush brain stem resulting in death.

63
Q

ischemic stroke

A

placement of blockage affects which “branches” of brain die off and what damage will occur. everything beyond the blockage dies. blockages lower in the system will result in severely impaired motor systems and blockages further up in the system will have better outcomes.

64
Q

left hemisphere

A

more linear and better for more logical processes. deals with knowledge, facts, syntax, and literal meaning of words. houses auditory memory for words.

65
Q

right hemisphere

A

holistic processes. metaphors, intonation, social cues, pragmatics, interpreting facial expressions, as well as recognizing faces, pictures, and voices.

66
Q

Boston Diagnostic Aphasia Examination

A

speaking: conversation/expository speech section
listening: auditory comprehension section, commands subtest
reading: reading section: number matching, oral sentence reading, oral sentence comprehension
writing: writing section, written picture naming subtest

67
Q

neuroplasticity

A

brain’s capacity to change either at the micro or macro level, allowing the brain to respond to environmental changes or changes in the organism itself. if therapy is done right, it can change the structure of the brain, which can lead to the brain doing increased behavior, which changes the brain

68
Q

limbic system

A

emotion based. if damaged, don’t lose old memories, but can’t make new memories. hippocampus (memory recall) is in the limbic system and decides what’s important to remember or not. only remember memories important to you. its part of our primitive brain so it can still function after a person loses all other communication

69
Q

aneurysm

A

bulge or ballooning of a blood vessel in the brain. can leak or rupture and cause bleeding into the brain (which could cause a hemorrhagic stroke).

70
Q

transference plasticity

A

plasticity in response to one training experience can enhance the acquisition of similar behaviors (intervention in one region can also make other things better; one intervention experience can generalize)

71
Q

interference in therapy

A

plasticity in response to one experience can interfere with the acquisition of other behaviors (teaching something the wrong way-intervention experience can interfere with acquisition of others).

72
Q

does age matter

A

yes. training-induced plasticity occurs more readily in younger brains; high prognosis with younger population due to neuroplasticity.

73
Q

standardized vs. informal assessment

A

informal is better. formal assessments give you quantitative results, but informal gives you more info and determines what could make therapy more effective.

74
Q

can one “standardize” an informal assessment

A

yes. design a test for each individual client and reassess the client using same eval in given amount of time. allows you to keep track of progress

75
Q

why should you evaluate cognition if patient has aphasia diagnosis

A

cognition may be relatively intact, but cognitive deficits can play a role in behaviors of pts. with aphasia. coexisting cognitive deficits in aphasia can affect pt’s response to intervention. cognitive abilities are critical to positive outcomes including health, medication management and compliance, and discharge planning.