Neuro/Sensory Flashcards
CN 10 -Vagus
- Slow HR Vagal stimulation decreases HR
CN 7 - Facial
Puffing out the cheeks
Causes of Paralysis
- Spinal Cord Injury
- Stroke
- Birth Defects
Spinal Shock
-Description
- Characterized by the absence of all voluntary and reflex actifity below level of injury
- Usually lasts less than 48 hrs
Spinal Shock
-S/S
- Flaccid Paralysis
- Loss of reflex activity below level of injury
- Bradycardia & Hypotension
Neurogenic Shock
- Type of shock that occurs after spinal cord injury that results in a massive vasodilation leading to pooling of blood, hypoperfusion, and impaired cellular metabolism
- Treated w/ fluids
Autonomic Dysreflexia
-Patho
- Exagerated Sympathetic response below injury when injury is around T6.
- No message is communicated due to dammage
- Heart Rate Slows down to compensate for vasoconstriction
- Vasoconstriction below injury and vasodilation above level of injury.
Autonomic Dysreflexia
-Treatment /Causes
- PREVENTION
- Caused by distended bladder or impacted rectum
TREAT - SIT UP IMMEDIATELY
- Remove aggravating factor
Autonomic Dysreflexia
-S/S
- Sudden onset, severe throbbing headache
- Severe HTN
- Flushing above injury, Pale below injury
- Nasal Stuffiness
Traumatic Brain Injury
-Assessments
- Neuro Check
- Pupillary response PERLA, A/A x4, CSM, GCS, V/S
GCS
-Measuring
- Eye 1-4
- Verbal 1-5
- Motor 1-6
8-9 Make sure airway is open.. Possible intubation
Components that contribute to ICP
- Cerebral Spinal Fluid
- Brain
- Blood
ICP is a measurement of these three factors
Can be measured w/ Transducer
IICP
-Interventions
- Raise HOB to 30 degrees
- MRI, CT, Intra-ventricular Catheter
- NO LUMBAR PUNCTURE (can cause herniation)
___
-Maintain Oxygenation w/ light hyperoxygenation to maintain paco2 to 35
-Maintain SBP from 100-160 and frequent neuro checks and V/S
-CPP above 70 is ideal
-Adequate sedation to prevent seizure activity and minimize O2 needs
-LIMIT brain stimulation
Response to IICP
- CSF shunts down first CT doesn’t show ventricles
- Vasoconstriction to decrease blood flow to the Brain
- Brain Tissue Herniates
Normal ICP is from 0-15
Monroe-Kelley Hypothesis ICP
- If Brain tissue, CSF, or Blood increases, one of the other factors must decrease to make room for change.
Causes of IICP
- Increased Venous and Arterial Pressures
- Inter-abdominal or Inter-thoracic Pressure. (Coughing)
- Increase Temperature increases metabolic
- Increase CO2 Causes Vasodilation
Cerebral Perfusion Pressure CPP
- Must be maintained at a level above 50 (MAP - ICP)
IICP
-Meds
- Osmotic Diuretics or loop diuretics
-Limit production of CSF - CorticoSteroids
-Used for Trauma, brain injury to decrease CSF and inflammation
3.
Types of Cerebral Bleeds
- Epidural is worst
2. Subdural can be fixed and bleeds slower
IICP
-Late Sign?
- Cushings Triad is a late sign of IICP
- Increased BP
- Decrease HR
- Irregular or increased breathing
Neuro Check
- Check Pupils
- CSM
- GCS
- Reflexes
IICP & Morphine
- Morphine causes vasodilation which would increase ICP
CAGE – Alcohol Withdrawal Warning Signs
- C- Cut Down.. Do you or anyone else feel you should cut down alcohol use
- A- Annoyed.. Do you get annoyed or angry when people talk about your alcohol use?
- G - Do you ever feel guilty about your drinking?
- E- Eye Opener.. Do you drink early in the morning upon awaking
Alcohol Withdrawal
-Treatment
- Ativan, Possible alcohol
- Banana bag
- IV bag that includes electrolytes, vitamines, minerals, thiamine, folic acid
Guillain-Barre Syndrome
- Usually preceded by a mild upper respiratory infection or gastroenteritis
- Slow recovery process that can take years.
- Gradual progressive weakness of upper extremities and facial muscles
- PRIORITY is difficulty breathing::: MONITOR RESPIRATORY STATUS CLOSELY
Parkinson’s Disease
- Degenerative disease caused by depletion of dopamine
- Slow, progressive disease that results in a crippling diability and death
- Assess neuro status and SWALLOW AND CHEW –Aspiration–
Fall risk
Multiple Sclerosis
- Chronic, progressive, non contagious degenerative disease of the CNS characterized by de-myelinization of neurons
- Consists of periods of REMISSIONS & EXACERBATIONS