Neuro - Rehab Flashcards

1
Q

What does the research show about acute therapy rehabilitation compared to no therapy following a stroke?

A

Patients that end up at therapy rehab care tend to have better outcomes when compared to sub-acute rehab or no therapy

  1. ) More independence in ADLs
  2. ) Less mortality
  3. ) Less re-hospitalizations
  4. ) Gains persist after one year
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2
Q

What is a CVA?

A

Cerebral vascular accident
rapid onset of focal or global cerebral function disturbance lasting >24 hours, with no apparent cause other than vascular origin. “brain attack”

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3
Q

What is TIA?

A

Transient ischemic attack

“mini stroke” <24 hours

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4
Q

What are the epidemiology of stroke?

A
3rd leading cause of death
780,000 strokes annually (75% new)
6 million stroke survivors
most common reason for rehabilitation
Leading cause of disability in US
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5
Q

How did the occurrence of stroke change in the 1950-1975 and from 1975-1985?

A

For the first 25 years there was a 50% decrease in CVA secondary to decrease in CAD, HTN and cigarettes

Then in 1975-1985 there was a 20% increase in stroke secondary to increase in the use of CT which correctly diagnosed stroke

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6
Q

What is the prognosis after a stroke?

A

1st month care CVA 90% deaths secondary to direct effect of brain lesion or complications of immobility

In the 1st 6 months heart disease

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7
Q

What is the goal of stroke rehabilitation?

A

To return maximum recovery of physical,
cognitive and psychological function, thereby, optimizing independence, quality of life, and dignity of the patient

Multidimensional nature of stroke and its
consequences makes an interdisciplinary approach to
patient care the most appropriate strategy

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8
Q

What is the role of the Physiatrist in the interdisciplinary rehabilitation team?

A

Physiatrist- Provides primary medical care, while coordinating interdisciplinary team and medical specialists

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9
Q

What are the natural recovery signs from impairments?

A

Immediately there is Decreased voluntary movement and MSRs

In 48 hrs the are increased MSRs

In one to 38 days there will be Spasticity and Clonus

In 6 - 33 days recovcery of increase voluntary movement and decreased spasticity. MSR’s are still increased

In addition, it was noted that motor control returned proximally (shoulder)
before distally, and LE fnx recovered earlier than UEs.

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10
Q

What are the predictors of motor recovery?

A

Complete paralysis of arm at onset correlates with poor prognosis in
useful hand function (only 9% gain good recovery)

Some movement of hand by 4 weeks correlates with good chance (70%)
of making full recovery

Poor prognosis also associated with severe proximal spasticity

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11
Q

What are types of CVA?

A

Inscemic is thrombotic, embolci and lacunar

Hemorrhagic is intracerebral and subarachnoid

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12
Q

What are the locations of thrombotic CVAs?

A

Larger arteries: ICA, ACA, MCA, PCA VBA

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13
Q

Where do embolisms usually come from?

A

Cardiac source from afib, cardiac catheders, endocarditis (2/2 infection, RA)

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14
Q

Where do lacunar CVAs occur?

A

small vessels usually in the putamen, internal capsule, pons, thalamus, caudate.

Caused from uncontrolled HTN and usually present as pure motor or sensory.

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15
Q

What is a global aphasia?

A

ACA/MCA/PCA infarcts caused my massive hemorrhage

Pt. are not fluent, cannot comprehend, and cannot repeat

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16
Q

Transcortical Mixed aphasia

A

watershed regions

Pt. are not fluent, cannot comprehend, but can repeat

17
Q

Brocca’s apahsa

A

MCA superior divisions

Pt. are not fluent, can comprehend, and cannot repeat

18
Q

Transcotical motor aphasia

A

Superior and anterior to Borca’s area

Pt. are not fluent, can comprehend, and can repeat

19
Q

Wernicke’s aphasia

A

Inferior MCA territory

Pt. are fluent, cannot comprehend, and cannot repeat

20
Q

Transcortical sensory aphasia

A

Area inferior or posterior to Wernicke’s area

Pt. are fluent, cannot comprehend, and can repeat

21
Q

Conduction aphasia

A

Internal arcuate fasciculations

Pt. are fluent, can comprehend, and cannot repeat

22
Q

Anomic aphasia

A

Left temporal & parietal areas. Often the result from recovery of other types of aphasia

Pt. are fluent, can comprehend, and can repeat

23
Q

What is dysarthria?

A

weakness, paralysis, or a lack of coordination of the motor-speech system

24
Q

What is abulia?

A

lack of will or initiative

25
Q

What is apraxia?

A

Deficit in motor planning, not language production. Lesion in located in left anterior corpus collosum