Neuro examination Flashcards
What are the different types of mentation?
alert
• normal response to environmental stimuli
disorientated/confused
• abnormal response to environment
depressed/obtunded
• inattentive, less responsive to environment
stuporous
• unconscious but can be roused by painful stimuli
comatose
• unconscious and unresponsive to any environmental stimuli
Stupor and comatose = more brainstem
Depressed either FB or BS
What sort of abnormal behaviours are there?
suggestive of forebrain
- aggression
- compulsive walking/circling
- loss of learnt behaviour
- vocalisation
- hemineglect syndrome – animals with structural forebrain lesions ignore half of their environment (contralateral)
What is decerebrate rigidity?
extension of all limbs and opisthotonus
release of inhibitory UMN descending pathways on LMNs
lesion – rostral brainstem
usually stuporous or comatose
What is decerebellate rigidity?
hyperextension of TLs and opisthotonus
loss of inhibition of stretch reflex mechanism of antigravity muscles
lesion – rostral part of cerebellum
mentation normal; may be episodic or postural
HLs normal
What is Schiff Sherrington?
hyperextension of TLs (maintaining voluntary movement and normal CP) and paralysis of PLs
interference with Border cells – inhibitory neurons in
cranial lumbar spinal cord that inhibit the TL extensor muscles
thoracic or cranial lumbar spine
not prognostic factor but only occurs in serious injuries
Outline sensory/ spinal ataxia
usually more subtle
due to decreased sensory information arriving from the limbs to tell CNS where they are in space at any given time
“legs just don’t know where they
What is vestibular ataxia
loss of orientation of the head with the eyes, neck, trunk and limbs and results in loss of balance
typically with leaning, falling, rolling towards side of lesion
What are the grades of spinal lesions?
Grade 1 no deficits, just pain Grade 2 paresis, ambulatory Grade 3 paresis, non-ambulatory Grade 4 paralysis Grade 5 no pain sensation
How do postural reactions work?
information from proprioceptors (joints, tendons and muscles – GP; inner ear – SP)
→ forebrain ( for conscious perception of information)
problem could be in nerves, spinal cord, all of brain, everywhere!!!
Outline the panniculus/ cutaneous trunci reflex
→ pinching of skin
→ sensory information enters the spinal cord approximately 2 vertebral spaces cranially
→ ascends to C8‐T1
→ bilateral synapse with motor neurons of lateral thoracic nerve
→ brachial plexus
→ cutaneous trunci muscle
→ bilateral contraction of these muscles
useful for T3-L3 lesions and brachial plexus lesions (bad px indicator for brachial plexus lesions)
Outline the palpebral reflex
touch medial and/or lateral canthus of eye → blink
input → Trigeminal (V) ophthalmic and maxillary brainstem
output → Facial (VII)
How does a spinal reflex tell you if the lesion is UMN or LMN
UMN - increased/ the same
LMN - decreased/ absent
Outline the corneal reflex
lightly touch cornea → blink and eye globe retraction
input – Trigeminal (V) ophthalmic, brainstem
output:
Abducens (VI) → globe retraction
Facial (VII) → blink
useful as same input as palpebral
Outline physiological nystagmus
input – vestibulocochlear (VIII) and brainstem
output:
Oculomotor (III)
Trochlear (IV)
Abducens (VI)
when reduced/lost - most commonly due to raised ICP
Why should you turn an animal upside down/ move head when assessing nystagmus?
you’re challenging the vestibular system by changing the position of the head
look for evoked (rather than static):
strabismus
nystagmus