Neuro Exam with focus on gait Flashcards

1
Q

MCMC-SRG

A
Mental status
Cranial nerves
Muscle testing
Cerebellar signs
sensation
Reflexes
Gait
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2
Q

Mental Status

A
  • Coma: no sleep/wake
  • PVS: + sleep/wake
  • Minimally conscious: +tracking, episodes of awareness
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3
Q

GOAT

A
  • 100 point scale

- >75 on 2-3 consecutive days signifies out of post-traumatic amnesia

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4
Q

Major Aphasic Syndromes

A
  • Brocas: TC motor except no repetition
  • TC motor: nonfluent with intact repetition
  • Wernicke’s: TC sensory except no repetition
  • TC sensory: fluent with intact repetition
  • Paraphasias: word substitutions
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5
Q

Treatment Arms

A

Medications
Therapy
Injections
Brace alternative medicine

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6
Q

Decorticate vs. Decerebrate

A
  • Decorticate: UE flexed, LE extended (arms to the coronary)

- Decerebrate: UE and LE extended

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7
Q

Alcohol affect on Cerebellar Tests

A

Alcohol use preferentially affects the cerebellar vermis and thus leads to abnormalities in HTS with relatively preserved FTN.

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8
Q

Romberg Test

A
  • differentiates btw proprioception deficit and ataxia
  • If loss of balance occurs only with eyes closed–> proprioception deficit
  • Loss of balance with eyes open and closed–> cerebellar ataxia
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9
Q

Spinal pathways

A

Dorsal Column: light touch, proprioception, vibration (crosses in brain stem)
Spinalthalamic pathway- pain/temperature (crosses at spinal cord in same level

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10
Q

Brown Sequard Syndrome

A

Hemisection–> always trauma related (stab, gun shot)

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11
Q

Reflexes

A
  • graded 0-4
  • Jendrassik maneuver (interlock fingers and pull apart)
  • C5: biceps, brachioradialis
  • C6: pronator
  • C7: triceps
  • L4: patellar
  • L5: medial hamstring
  • S1: achilles
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12
Q

UMN signs

A
  • clonus 4+
  • Babinski, stimulus plantar suface
  • Chaddock, stimulus lateral ankle
  • Stransky, stimulus “flicking” out little toe
  • Oppenheim, stimulus medial surface of tibia
  • Hoffmans, contraction of thumb and index finger
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13
Q

Dix Hallpike Test

A
  • diagnosis BPPV
  • should be fatigable (less nystagmus with repeated testing) and reversible (direction of nystagmus reverses with sitting up again.)
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14
Q

Hoover Sign

A
  • Assess for malingering
  • “synergistic contraction”
  • Patient supine and asked to raise one leg–> normally you should feel pressure in hand if patient is actively trying to life other leg
  • caution in patients with strong hip muscles.
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15
Q

Gower’s Sign

A
  • Duchenne’s muscular dystrophy
  • signifies weakness of proximal muscles
  • patient uses hands to walk up from ground
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16
Q

Beevor’s sign

A
  • Implies spinal cord lesion between T6-T10
  • Can be seens in T6-T10 SCI or motor neuron dz
  • Patient supine, asked to attempt sit up–> belly button moves towards the level of injury (i.e. superiorly) due to weakness of the low ab muscles.
17
Q

Battle’s Sign

A
  • Bruising over mastoid process

- Suggests basilar skull fractures and likely underlying brain injury

18
Q

“Gait is a reflex”

A
  • Gait center/central pattern generator in lumbar spine

- Examples: newborns, cats with transected spinal cords, etc.

19
Q

Gait Definitions

A
  • Gait cycle: heel strike to heel strike in SAME foot
  • Step length: distance between heel strike of one foot to heel strike of other foot.
  • Stride length: distance between heel strikes in the SAME foot (i.e. distance of the gait cycle)
  • Cadence: #steps per time, step/minute
  • Base of support lateral distance between the feet (wide vs. narrow base)
20
Q

Gait phases

A

8 functional phases

  • 5 in stance (60%), 3 in swing (40%)
  • 20% time in “double support” with both legs on the ground

***Remember 60-40-20

21
Q

Where is the center of gravity?

A

***Remember 2-2

2 inches anterior to S2 vertebrae

When ambulating, COG moves in a sinusoidal pattern in horizontal and vertical planes.

22
Q

What is inman’s gait determinants?

A

Serve to minimize movement of COG to make gait as energy efficient as possible.

23
Q

What are Inman’s 6 determinants of gait?

A

***Know that are 3 at hip, 2 at knee, 1 at foot/ankle

  • Pelvic rotation, pelvic tilt, lateral displacement of pelvis (only one which displaces COG in horizontal plane)
  • Knee flexion in stance/early knee flexion, knee mechanisms (i.e. KE, PF, supination at terminal stance/pre-swing)
  • Ankle/foot mechanisms (i.e. controlled plantarflexion after heel strike)
24
Q

Antalgic gait

A
  • “limping”

- decreased stance time on affected leg

25
Q

What are the 4 compensations to clear an abnormal limb which is “functionally longer” through swing phase?

-May be due to drop foot, PF spasticity, stiff knee, leg length difference, pelvic drop, etc.

A
  1. Circumduction
  2. Steppage gait- increase hip and knee flexion
  3. Hip hike- lifting of hip/trunk to clear leg in swing phase
  4. Vaulting- elevating on toes of sound limb
26
Q

What are the 2 causes of foot slapping after heel strike?

A
  • Proprioception deficits (pt usually has difficulty ambulating in the dark)
  • Mild weakness of dorsiflexors (tibialis anterior)
27
Q

What are the 2 main causes of knee hyperextension/ Genu Recurvatum?

A
  • quadriceps/hamstring weakness–> knee snaps back to avoid falling forward??
  • foot drop/plantar flexion contracture–> plantarflexion a/w knee extension

***may also be seen in ACL deficit knees or ligamentous instability–> hyper-extend at the knee

28
Q

What type of contraction generates most force?

A

Fast eccentric

***Eccentric is lengthening, concentric is shortening

29
Q

What are the 2 main categories of pathologic gait?

A
  • Neurologic

- Musculoskeletal

30
Q

What are the 5 types of neurologic pathologic gaits?

A
  • parkinsons
  • myopathic
  • ataxic
  • spastic diplegia
  • hemiplegic
31
Q

What are the 3 types of msk pathologic gaits?

A
  • hip disorders
  • knee disorders
  • ankle/foot disorders
32
Q

Pakinson gait

A
  • hypokinectic
  • forward flexed
  • decreased arm swing
  • shuffling
  • fenestrating- increased speech w/ increased distance
  • difficulty initiating gait and turning
  • “freezing” episodes
  • often improve with a metronome (clapping, beat)
  • may assess postural stability (retropulsion)
33
Q

Myopathic gait

A
  • Waddling
  • Due to proximal weakness–> hip abduction and hip extension are weak
  • “Trendenlenberg-Weight shift”
  • hyperlordosis for hip stability
34
Q

Ataxic gait

A
  • Due to pathology of cerebellum
  • Wide base of support
  • truncal unsteadiness–> “titubation”
  • Unable to do tandem gait
  • Variable leg placement, appears uncoordinated
35
Q

Spastic Diplegic

A
  • Narrow base
  • “scissoring” due to adductor spasticity
  • “walking through water”–> difficult to advance limb
  • Equinovarus (turns in) feet with decreased/absent heel strike
  • Often a/w increased hip and knee flexion as well
36
Q

Hemiplegic gait

A
  • Circumduction of involved leg during swing phase
  • decreased arm swing on involved side- often held in flexion synergy
  • Slow with decreased step length
  • Decreased/absent heel strike on involved side
  • Often with genu recurvatum (knee hyperextension) on involved side during stance phase
  • Decreased knee flexion during swing phase–> circumduct
37
Q

Trendelenburg gait

A

-Due to gluteus medius weakness/dysfunction

38
Q

Knee Disorders

A

-Painful knee ***

39
Q

Ankle/Foot disorders

A