neuro/defib Flashcards
Broca’s area
Motor/ expressive speech area that controls the output of words (verbal/written) to ensure they are coordinated/ appropriate
Base of the frontal lobe
Mean arterial pressure (MAP)
70-100 mmHg, <60mmHg can be deadly to organs
Main areas of the spine
Cervical- C1-C7
Thoracic- T1-T12
Lumbar- L1-L5
Sacral- S1-S5
Coccyx- 4
Why does crushing’s reflexes happen
The widening pulse pressure and decreased pulse is from the heart trying to perfuse the brain so the heart must work harder to push past the increased IPC to bring blood to the brain
Irregular respirations are from the pressure being put on the medulla from the ICP which interferes with the respiratory and cardiac centers
major bleeding in neuro pts
direct pressure not too excessive if skull fracture present/ suspected, active bleeding worsens hypoxia, active bleeding decreases CPP (less volume needs more pressure to perfuse)
Hemispheres of the brain
Two hemispheres- left and right, each with 4 major lobes: frontal, parietal, temporal, occipital
Medical TOR criteria in the ALS
Age >16 years old, altered LOA, arrest not witnessed by EMS and no ROSC 20 min of resuscitation and no defibrillation delivered
Parasympathetic nervous system
Dominates digestive system and aids in recovery after sympathetic stimulation
Responsible for slowing of RR, HR, constriction of pupils
Largely innervated by the vagus nerve
Active chemical neurotransmitter is Ach
Antagonistic effects that maintain homeostasis (opposite effects- one is dominating other is blocking ability of the other)
Neurons
Specialized cells that conduct impulses through CNS/PNS that requires glucose/O2 for metabolism
Parts:
Dendrite- receptor site that conducts impulses towards the cell body
Cell body- contains the nucleus
Axon- conducts impulses away from cell body towards effector site or connecting neuron
Myelin sheath- insulates the fiber and speeds up the rate of conduction
Nodes of ranvier- spaces between the myelin sheaths
Acetylcholine
Neurotransmitter present at neuromuscular junctions and in the autonomic nervous system and peripheral nervous system
What are the most common errors in prehospital defibrillation
Improper pad placement and poor adhesive contact
5 main uses of ETCO2 in cardiac arrest
Verify tracheal tube placement, identify tracheal tube displacement, evaluate CPR quality, identify ROSC, determine when ROSC is unlikely
Ninth cranial nerve
Glossopharyngeal- sensory/motor, taste, gag reflux
Serotonin
Neurotransmitter for mood, sexual desire/function, appetite, sleep, memory/learning, temperature regulation, and social behavior
ROSC- post arrest care
Fluid bolus- >2 years old
Ensure chest is clear and BP is hypotensive, 10 ml/kg, max 1000ml, reassess every 100ml >2-<12 years and reassess every 250ml >12 years
Oxygenation- BVM ventilations are required, titrate SPO2 to 94-98%, avoid hyperventilation (ETCO2 40-40mmHg)
What do you do if you obtain a ROSC
Complete assessment of CAB, 12 lead, full set of vitals, continually reassess and treat findings
what changes in vitals would you see with a head injury
irregular respirations, decreased GCS, sluggish/unresponsive pupils, cool clammy skin, increased systolic BP that widens until decreased as compensatory mechanisms fail
How to manage airway
Airway: suctioning/ positioning
Breathing: IPPV, oxygenation (hyperventilation)
Circulation: monitor BP, ECG, IV
Positioning: supine with head elevated 15 degrees
Tension headaches
Muscle contractions of the face, neck, and scalp from stress, persistent noise, poor posture, and eye strain
Most common type of headache
Treatment: analgesics
Ischemic strokes
Occlusions of an artery from plaque buildup or an embolus causing sudden obstruction
S&s: hemiparesis/hemiplegia, contralateral numbness/ facial drooping, aphasia, confusion/coma, convulsions, incontinence, diplopia, dysarthria, headache, dizziness
Cluster headaches
Occurs in short lasting bursts on repeat for days or months (half/two hours) often during sleep due to abnormalities in biological clock (hypothalamus) causing severe pain around one eye, tearing and nasal congestion
Treatment: antihistamines, corticosteroids, calcium channel blockers, analgesics
What are the signs of an obviously dead patient
Decapitation, transection, visible decomposition, putrefaction, absence of vital signs and grossly charred body, an open head/torso wound with gross outpouring of cranial or visceral contents, gross rigor mortis, or dependent lividity
Pia mater
Inner most layer of connective tissue that adheres closely to the convolutions on the brain
Complex partial seizure
Arise from focal seizures in the temporal lobe (psychomotor) that manifest as changes in behavior and present as abnormal repetitive motor behavior and a period of amnesia with a brief duration
Migraines
Severe headaches preceded by visual/ GI disturbances with onset of an intense throbbing pain unilaterally accompanied by nausea and vomiting, constriction and dilation of blood vessels, sensitivity to light, sound or smell
Autonomic nervous system
Branch of the PNS that acts as the involuntary control and has two further branches: SNS (sympathetic nervous system) and PNS (parasympathetic nervous system)
What are the causes of PEA
Hypovolemia, hypoxia, hyper/hypokalemia, hypothermia, H+ (acidosis), trauma, tension, tamponade, toxins, thrombosis
Petit mal
Generalized
Absence seizures around kids 4-12 with a lapse of consciousness, no loss of posture or motor activity, isolated clonic activity for a short period then return to normal.
Pregnancy in cardiac arrest
Pregnancy presumed to be >20 weeks’ gestation
Run as regular medical arrest and consider very ealy transport after a minimum 1 analysis and egress plan is organized
TOR contraindicated
Airway obstruction in cardiac arrest
High instances in children, focus on high-quality CPR, consider very early transport (after 1 analysis and egress plan is organized), transport to closest ER since PT is CTAS 1
If airway obstruction is relieved prior to early transport, consider running a longer arrest on scene
TOR is contraindicated
Where should the pt be during cardiac arrest
Hard flat surface with enough space- possibly stretcher and plan extrication
Move pt only if necessary and notify PD and document and do not move pt back
Status epilepticus
prolonged seizure activity that is reoccuring seizures before regained consciousness
Third cranial nerve
Oculomotor- motor, eye movements including eyelids
Spinal cord makeup
Starts at the medulla and ends at L1 (after L1 spinal cord is called cauda equina “horse tail”) and is protected by vertebral column, meninges, and CSF
Consists of nerves that innervate the skeletal muscles with ascending/ descending tracts (afferent/efferent pathways)
What are the signs of a ROSC
Sudden increase in ETCO2, spontaneous respirations, palpable pulses, change in colour, spontaneous movement
Fifth cranial nerve
Trigeminal- sensory/motor, general sensory- eye, nose, face or oral cavity, teeth, speech muscles
Tenth cranial nerve
Vagus- sensory/motor, external ear, parts of taste, heart and lung smooth muscle, glands of GI system, diaphragm
Cerebellum
Coordinates movement, maintain posture and equilibrium from taking impulses from visual pathways, vestibular pathways, and proprioceptors in joints and muscles
Stroke test
B- balance: loss of balance, headache, or dizziness
E- eyes: blurred vision
F- face: one side of face is drooping
A- arms: arm or leg weakness
S- speech: speech difficulty
T- time: all for an ambulance immediately
Trauma TOR conditions
> 16 years, no palpable pulses and no defibrillation delivered and rhythm asystole or no signs of life at any time since fully extricated, or signs of life when fully extricated with the closest ED >30 min transport time away, or rhythm PEA with the closest ED >30 min away
Fourth cranial nerve
Trochlear- motor, eye movements
Brainstem
Pons: contains afferent (incoming) and efferent (outgoing) fibers
Medulla: vital control to regulate respiratory and cardiovascular function
RAS (reticular activating system): determines degree, arousal, awareness of cerebral cortex (decides what sensory impulses brain ignores/notices)
Multiple sclerosis (MS)
Progressive autoimmune disease of the CNS that causes patches of the myelin in brain/spine to be destroyed causing issues in nerve conduction and message impulse
S&s: physical/mental problems, muscle weakness, sensation troubles, vision issues (double/ blindness)
No cure- meds slow progression
Subdural hematoma
Bleeding between the dura and the arachnoid from injury to underlying brain tissues where blood accumulates slowly so diagnosed hours to days later
Tear in the arachnoid can cause loss of CSF in subdural space increased ICP
High risk: alcoholics, people on blood thinners
S&s: headaches, changes in LOC, slurred speech
conditions in medical cardiac arrest
CPR- LOA altered, 2 min intervals
Defib- >24 hours, VF/ pulseless VT
Epi- >24 hours, anaphylaxis as causative event
How does the blood brain barrier work
Cells are tightly joined together to form a barrier that limits the passage of damaging materials into the brain to control balance of electrolytes, glucose, and proteins
Lipid soluble substances can still pass through
How do neurons work
Impulses are transmitted from cell to cell by neurotransmitters involving presynaptic and postsynaptic synapses
Axon and receptor are separated by a synaptic cleft and for impulses to travel the chemical flows a crossed the cleft to the receiving receptor that are specific for each chemical type
what do sluggish pupils indicate
cerebral hypoxia or increased ICP
Hemorrhagic stroke
Hemorrhage in cranial vault from aneurysms, malformations, hypertension, stress/ exertion, cocaine with a sudden onset accompanied by headache, nausea, vomiting, quick deterioration
Basilar skull fracture
A fracture involving the base of the skull
frontal lobe functions
conscious thought and voluntary motor action
TIA (transient ischemic attack)
Focal cerebral dysfunction (temporary reduction in blood flow) that lasts minutes to hours then the patient returns to normal with no permanent damage
Indicate obstruction related to atherosclerosis, can be from spasm of arteries and loss of autoregulation