NEURO Flashcards

(44 cards)

1
Q

Explain the term freezing in Parkinsons

A

A phenomenon that occurs in those with Parkinson when a gait pattern needs go be altered
It happens because they are unable to control movement due to lack of inhibition from thalamus
Patients experience this when they need to stop walking, initiate walking or when they change direction

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2
Q

How do we manage freezing

A

Think of walking as a higher level cognitive task rather than relying on autonomic movements
1. Counting steps or stepping over an imaginary line or brick block when approaching freezing time
2. Action observation training (explain)

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3
Q

What is muscle tone

A

Resistance of muscle to passive stretch

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4
Q

Expected functional recovery: C1-C3

A

Depend on ventilator
Limited talking
C3=limited neck movement

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5
Q

Expected functional recovery: C3-C4

A

Usually have head and neck control
Initially requires ventilator
Ability to shrug shoulders

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6
Q

Expected functional recovery: C5

A

Biceps brachii present
Has neck, shoulder shrug, head and shoulder control
Can bend elbow and face palms up
Independence with eating, drinking, face washing, tooth brushing and hair care
Push a manual wheelchair over short smooth surfaces
Can do self assisted coughs
Can do pressure relief by leaning forwards and to the side
May drive adapted car

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7
Q

Expected functional recovery: C6

A

Tenodesis grip
Able to extend wrist
Can use manual wheelchair for daily chores but may need transfer board for transfers
May independently do bladder and bowel management
Have greater independence with ADLs

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8
Q

Expected functional recovery: C7

A

Have intact triceps brachii
Have major part of latissimus dorsi
May need assistance with cutting food
Able to use manual wheelchair independently and transfers without transfer board

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9
Q

Expected recovery function: C8-T1

A

Have better use of fingers
Have all latissimus dorsi
Independent with ADLs, transfers and bowel and vladder management

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10
Q

Expected functional recovery: T2-T6

A

Good motor function of the head, neck, shoulders and arms with fingers
Better trunk control

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11
Q

Expected functional recovery: T7-T12

A

Better abdominal control
Can have better cough
Ability to sit unsupported for functional activities

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12
Q

4 aims of physiotherapy in acute stage of management.

A
  1. Supportive
    Maintaining airway through adequate airway
    Relief pain
    Maintain MSK
  2. Preventative
  3. Educative (caregivers and pt themselves)
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13
Q

Clinical features of Parkinsons

A

Loss of postural reflexes
Gait problems
Badykinesia
Rigidity
Tremor
Monotonous speech
Blank facial expression
Lack of blinking

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14
Q

Which 4 nerves are likely to be affected by residual paralysis from GBS

A

Median nerves
Peroneal nerves
Ulnar nerve
Tibial nerve

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15
Q

Define Parkinsons

A

A slowly progressive degenerative disorder of the extrapyramidal system

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16
Q

Physiotherapy Principals for end stage parkinsons treatment

A

Pressure sore care if pt wheelchair bound
Passive movements to present contracture and maintain ROM
encourage active movement if available
Education for caregivers
Circulatory exercises to minimize risk of DVT
prevent chest complications by maintaining adequate airway and encourage breathing exercises
Endurance ??

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17
Q

Presentation for MND and psuedobulbar palsy

A

Swallowing problems
Slurred speech
Aspiration
Dysphagia

Treatment:
Facilitating jaw and lip closure with microfiber cloth for sensory input
Elevation of the posterior third of tongue
Elevation of the boarders of the tongue
Cheek closure
Control of breathing in relation to swallowing

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18
Q

Precautions for GBS

A
  1. Do not overstretch (can cause pain and tissue damage)
  2. Beware of postural hypotension
  3. Do not exercise to the point of fatigue (paradoxical weakening)
  4. Autonomic dysreflexia
  5. DVT
  6. Beware of decreased sensation
19
Q

Physiotherapy management to preventing chest complications

A

Increase respiratory muscle strength through incentive spiratory or PEP bottle
Postural drainage with frequent suctioning
Frequently asses breath sounds for added sounds
Monitor patients ABG and chest X-ray
Encourage coughing through ACBT

20
Q

Define the term positive impairments and give examples

A

Any impairment that is considered new or additional to the CNS injury
Spasticity
Babinski response
Extensor spasms
Flexor withdrawal spasms

21
Q

Define negative impairments and give examples

A

Impairments that represent a loss of previous function
Muscle weakness

22
Q

Define Neural plasticity

A

The ability of cells to undergo alterations in their form and function depending on environmental influences

23
Q

Mechanisms of Neural plasticity

A

Latent synapse
Potentiation
Recovery
Collateral sprouting
Terminal regeneration
Denervation supersensitivity

24
Q

Describe 2 supra-spinal level inhibitory influences contributing to spasms

A

Basal Ganglia- Refinement and inhibition movement. Akinesia, Rigidity, dyskinesia. Contraleteral to lesion

Vestibulocerebellum- primarily involved in maintenance of balance. Leads to bilateral ataxia and intention Tremor

25
Define ASIA A
No motor or sensory function is preserved in the sacral segments S4-S5
26
Define ASIA B
Sensory but no motor function preserved believe the neurological level of injury including sacral segments
27
Define ASIA C
Motor function is preserved below neurological level of injury and the majority of key muscles have muscle strength less than 3
28
Define ASIA D
Motor function is preserved below neurological level and at least half of key muscles have muscle strength greater than or equal to 3
29
Define Parkinsons syndrome
Has a known cause (TBI or stroke or drug use) Sudden onset and progression can be altered if the cause if identified Can affect any age
30
Define MS
An immune-mediated demyelinating disease that affects CNS
31
Explain how increase in body temp causes decrease in conduction of nerve impulses in MS
Uhtoff's syndrome Heat increases the hydrolysis of Ach leading to decrease in motor unit recruitment. Decrease in motor recruitment leads to decrease in active motor neurons. Decrease in the no of active motor neurons leads to muscle weakness and fatigue
32
Possible causes of fatigue for MS and how to manage it
1. Working against spastic muscles 2. Heat hydrolysis 3. Respiratory muscle weakness 4. Poor endurance from immobility/deconditioning 5. Loss of proprioception (makes it difficult to conduct coordinated movements)
33
Define GBS
Acute inflammatory demyelinating poly neuropathy Ascending paralysis Demyelination only happens on peripheral nerves and cranial nerves
34
What is Miller Fisher syndrome
Presents like GBS but paralysis starts in the eyes and have ataxic gait
35
Clinical presentation of GBS
Abrupt parenthesia Symmetrical muscle wasting and weakness Decreased tone and reflexes Symptoms present in glove and stalking pattern Autonomic dysfunction I.e unstable BP
36
Medical management for GBS patients
Mechanical ventilation Anticoagulants Injection with immunoglobulins Plasma filtration Flaccid bladder could lead to UTI
37
Causes of MS
Low ultraviolet exposure Autoimmune etiology Recurrent infections Trauma Genetic THE CAUSE FOR MOST CASES IS MOSTLY UNKNOWN
38
Pathology of MS
Mainly affects myelin sheath (Decreased conduction of nerve impulses) Nerve axons are secondarily affected Patchy areas of demyelination are spread throughout the CNS
39
Why would remyelination fail in MS
Deficiency in precursor cell Failure of precursor cell recruitment Failure of precursor cell differentiation Failure of precursor cell maturation
40
Clinical features of MS
Limb weakness (usually asymmetrical) Respiratory muscle weakness Spasticity (can progress to be painful) Fatigue Ataxia Intention Tremor Slurred speech Loss of proprioception Diplopia Urinary retention
41
What are the main reasons for fatigue in MS
Working against spastic muscles Decreased nerve conduction due to decreased internodal distance Heat- hydrolysis of Ach Weakness due to deconditioning or immobility
42
Define motor neuron disease
Progressive paralysis due to loss of motor neurons with no sensory involvement
43
Name MND area of degeneration
Anterior horn cells = LMN Corticospinal tracts = UMN Motor nuclei of brainstem = Bulbar palsy Corticobulbar tracts = Pseudobulbar palsy
44
List the types of MND
Progressive muscle atrophy - LMN Amyotrophic lateral sclerosis - UMN and LMN Pseudobulbar palsy- UMN Progressive Bulbar Palsy - LMN Mixed bulbar palsy